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5. MSH2-MSH3 promotes DNA end resection during homologous recombination and blocks polymerase theta-mediated end-joining through interaction with SMARCAD1 and EXO1

6. Supplementary Data from NSD3-Induced Methylation of H3K36 Activates NOTCH Signaling to Drive Breast Tumor Initiation and Metastatic Progression

7. Supplementary figure legends from A Novel Chemotherapeutic Agent to Treat Tumors with DNA Mismatch Repair Deficiencies

8. Data from A Novel Chemotherapeutic Agent to Treat Tumors with DNA Mismatch Repair Deficiencies

9. Data from NSD3-Induced Methylation of H3K36 Activates NOTCH Signaling to Drive Breast Tumor Initiation and Metastatic Progression

10. Supplementary methods from A Novel Chemotherapeutic Agent to Treat Tumors with DNA Mismatch Repair Deficiencies

11. Supplementary Figures from A Novel Chemotherapeutic Agent to Treat Tumors with DNA Mismatch Repair Deficiencies

13. Precision targeting tumor cells using cancer-specific InDel mutations with CRISPR-Cas9

14. MSH2-MSH3 promotes DNA end resection during HR and blocks TMEJ through interaction with SMARCAD1 and EXO1

15. NSD3-Induced Methylation of H3K36 Activates NOTCH Signaling to Drive Breast Tumor Initiation and Metastatic Progression

17. CDK12 drives breast tumor initiation and trastuzumab resistance viaWNTandIRS1‐ErbB‐PI3K signaling

20. A Novel Chemotherapeutic Agent to Treat Tumors with DNA Mismatch Repair Deficiencies

38. CDK12 drives breast tumor initiation and trastuzumab resistance via WNT and IRS1‐ErbB‐PI3K signaling.

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