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2. Supplementary Data from Lynch Syndrome–Associated Breast Cancers: Clinicopathologic Characteristics of a Case Series from the Colon Cancer Family Registry

Author

Walsh, Michael D., primary, Buchanan, Daniel D., primary, Cummings, Margaret C., primary, Pearson, Sally-Ann, primary, Arnold, Sven T., primary, Clendenning, Mark, primary, Walters, Rhiannon, primary, McKeone, Diane M., primary, Spurdle, Amanda B., primary, Hopper, John L., primary, Jenkins, Mark A., primary, Phillips, Kerry D., primary, Suthers, Graeme K., primary, George, Jill, primary, Goldblatt, Jack, primary, Muir, Amanda, primary, Tucker, Kathy, primary, Pelzer, Elise, primary, Gattas, Michael R., primary, Woodall, Sonja, primary, Parry, Susan, primary, Macrae, Finlay A., primary, Haile, Robert W., primary, Baron, John A., primary, Potter, John D., primary, Le Marchand, Loic, primary, Bapat, Bharati, primary, Thibodeau, Stephen N., primary, Lindor, Noralane M., primary, McGuckin, Michael A., primary, and Young, Joanne P., primary

Published
2023
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3. Data from Lynch Syndrome–Associated Breast Cancers: Clinicopathologic Characteristics of a Case Series from the Colon Cancer Family Registry

Author

Walsh, Michael D., primary, Buchanan, Daniel D., primary, Cummings, Margaret C., primary, Pearson, Sally-Ann, primary, Arnold, Sven T., primary, Clendenning, Mark, primary, Walters, Rhiannon, primary, McKeone, Diane M., primary, Spurdle, Amanda B., primary, Hopper, John L., primary, Jenkins, Mark A., primary, Phillips, Kerry D., primary, Suthers, Graeme K., primary, George, Jill, primary, Goldblatt, Jack, primary, Muir, Amanda, primary, Tucker, Kathy, primary, Pelzer, Elise, primary, Gattas, Michael R., primary, Woodall, Sonja, primary, Parry, Susan, primary, Macrae, Finlay A., primary, Haile, Robert W., primary, Baron, John A., primary, Potter, John D., primary, Le Marchand, Loic, primary, Bapat, Bharati, primary, Thibodeau, Stephen N., primary, Lindor, Noralane M., primary, McGuckin, Michael A., primary, and Young, Joanne P., primary

Published
2023
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5. Expression of MUC2, MUC5AC, MUC5B, and MUC6 mucins in colorectal cancers and their association with the CpG island methylator phenotype

Author

Walsh, Michael D, Clendenning, Mark, Williamson, Elizabeth, Pearson, Sally-Ann, Walters, Rhiannon J, Nagler, Belinda, Packenas, David, Win, Aung K, Hopper, John L, Jenkins, Mark A, Haydon, Andrew M, Rosty, Christophe, English, Dallas R, Giles, Graham G, McGuckin, Michael A, Young, Joanne P, and Buchanan, Daniel D

Published
2013
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8. Immunohistochemical testing of conventional adenomas for loss of expression of mismatch repair proteins in Lynch syndrome mutation carriers: a case series from the Australasian site of the colon cancer family registry

Author

Walsh, Michael D, Buchanan, Daniel D, Pearson, Sally-Ann, Clendenning, Mark, Jenkins, Mark A, Win, Aung Ko, Walters, Rhiannon J, Spring, Kevin J, Nagler, Belinda, Pavluk, Erika, Arnold, Sven T, Goldblatt, Jack, George, Jill, Suthers, Graeme K, Phillips, Kerry, Hopper, John L, Jass, Jeremy R, Baron, John A, Ahnen, Dennis J, Thibodeau, Stephen N, Lindor, Noralane, Parry, Susan, Walker, Neal I, Rosty, Christophe, and Young, Joanne P

Published
2012
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10. Tumor CD155 Expression Is Associated with Resistance to Anti-PD1 Immunotherapy in Metastatic Melanoma

Author

Lepletier, Ailin, primary, Madore, Jason, additional, O'Donnell, Jake S., additional, Johnston, Rebecca L., additional, Li, Xian-Yang, additional, McDonald, Elizabeth, additional, Ahern, Elizabeth, additional, Kuchel, Anna, additional, Eastgate, Melissa, additional, Pearson, Sally-Ann, additional, Mallardo, Domenico, additional, Ascierto, Paolo A., additional, Massi, Daniela, additional, Merelli, Barbara, additional, Mandala, Mario, additional, Wilmott, James S., additional, Menzies, Alexander M., additional, Leduc, Charles, additional, Stagg, John, additional, Routy, Bertrand, additional, Long, Georgina V., additional, Scolyer, Richard A., additional, Bald, Tobias, additional, Waddell, Nicola, additional, Dougall, William C., additional, Teng, Michele W.L., additional, and Smyth, Mark J., additional

Published
2020
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13. Oncogenic BRAF mutation induces DNA methylation changes in a murine model for human serrated colorectal neoplasia

Author

Bond, Catherine E., primary, Liu, Cheng, additional, Kawamata, Futoshi, additional, McKeone, Diane M., additional, Fernando, Winnie, additional, Jamieson, Saara, additional, Pearson, Sally-Ann, additional, Kane, Alexandra, additional, Woods, Susan L., additional, Lannagan, Tamsin R. M., additional, Somashekar, Roshini, additional, Lee, Young, additional, Dumenil, Troy, additional, Hartel, Gunter, additional, Spring, Kevin J., additional, Borowsky, Jennifer, additional, Fennell, Lochlan, additional, Bettington, Mark, additional, Lee, Jason, additional, Worthley, Daniel L., additional, Leggett, Barbara A., additional, and Whitehall, Vicki L. J., additional

Published
2018
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14. Copy number profiles of paired primary and metastatic colorectal cancers

Author

Kawamata, Futoshi, primary, Patch, Ann-Marie, additional, Nones, Katia, additional, Bond, Catherine, additional, McKeone, Diane, additional, Pearson, Sally-Ann, additional, Homma, Shigenori, additional, Liu, Cheng, additional, Fennell, Lochlan, additional, Dumenil, Troy, additional, Hartel, Gunter, additional, Kobayasi, Nozomi, additional, Yokoo, Hideki, additional, Fukai, Moto, additional, Nishihara, Hiroshi, additional, Kamiyama, Toshiya, additional, Burge, Matthew E., additional, Karapetis, Christos S., additional, Taketomi, Akinobu, additional, Leggett, Barbara, additional, Waddell, Nicola, additional, and Whitehall, Vicki, additional

Published
2017
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15. Abstract PR04: Wnt and MAPK pathway activation in conventional and serrated colorectal neoplasia

Author

Whitehall, Vicki, primary, Borowsky, Jennifer, additional, Bond, Catherine, additional, Bettington, Mark, additional, Pearson, Sally-Ann, additional, Kalimutho, Murugan, additional, Liu, John, additional, Dumenil, Troy, additional, McKeone, Diane, additional, Jamieson, Saara, additional, Fernando, Winnie, additional, Fennell, Lochlan, additional, Clouston, Andrew, additional, Rosty, Christophe, additional, Brown, Ian, additional, Walker, Neal, additional, and Leggett, Barbara, additional

Published
2017
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17. Oncogenic <italic>BRAF</italic> mutation induces DNA methylation changes in a murine model for human serrated colorectal neoplasia.

Author

Bond, Catherine E., Liu, Cheng, Kawamata, Futoshi, McKeone, Diane M., Fernando, Winnie, Jamieson, Saara, Pearson, Sally-Ann, Kane, Alexandra, Woods, Susan L., Lannagan, Tamsin R. M., Somashekar, Roshini, Lee, Young, Dumenil, Troy, Hartel, Gunter, Spring, Kevin J., Borowsky, Jennifer, Fennell, Lochlan, Bettington, Mark, Lee, Jason, and Worthley, Daniel L.

Abstract

Colorectal cancer is a major cause of cancer death and approximately 20% arises within serrated polyps, which are under-recognized and poorly understood. Human serrated colorectal polyps frequently exhibit both oncogenic BRAF mutation and widespread DNA methylation changes, which are important in silencing genes restraining neoplastic progression. Here, we investigated whether in vivo induction of mutant Braf is sufficient to result in coordinated promoter methylation changes for multiple cancer-related genes. The BrafV637E mutation was induced in murine intestine on an FVB;C57BL/6J background and assessed for morphological and DNA methylation changes at multiple time points from 10 days to 14 months. Extensive intestinal hyperplasia developed by 10 days post-induction of the mutation. By 8 months, most mice had murine serrated adenomas with dysplasia and invasive cancer developed in 40% of mice by 14 months. From 5 months onwards, Braf mutant mice showed extensive, gene-specific increases in DNA methylation even in hyperplastic mucosa without lesions. This demonstrates that persistent oncogenic Braf signaling is sufficient to induce widespread DNA methylation changes. This occurs over an extended period of time, mimicking the long latency followed by rapid progression of human serrated neoplasia. This study establishes for the first time that DNA methylation arises slowly in direct response to prolonged oncogenic Braf signaling in serrated polyps; this finding has implications both for chemoprevention and for understanding the origin of DNA hypermethylation in cancer generally. [ABSTRACT FROM AUTHOR]

Published
2018
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19. Serrated tubulovillous adenoma of the large intestine

Author

Bettington, Mark, primary, Walker, Neal, additional, Rosty, Christophe, additional, Brown, Ian, additional, Clouston, Andrew, additional, McKeone, Diane, additional, Pearson, Sally‐Ann, additional, Klein, Kerenaftali, additional, Leggett, Barbara, additional, and Whitehall, Vicki, additional

Published
2015
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21. Reply to J. Moline et al

Author

Buchanan, Daniel D., primary, Tan, Yen Y., additional, Walsh, Michael D., additional, Clendenning, Mark, additional, Metcalf, Alexander M., additional, Ferguson, Kaltin, additional, Arnold, Sven T., additional, Thompson, Bryony A., additional, Lose, Felicity A., additional, Parsons, Michael T., additional, Walters, Rhiannon J., additional, Pearson, Sally-Ann, additional, Cummings, Margaret, additional, Oehler, Martin K., additional, Blomfield, Penelope B., additional, Quinn, Michael A., additional, Kirk, Judy A., additional, Stewart, Colin J., additional, Obermair, Andreas, additional, Young, Joanne P., additional, Webb, Penelope M., additional, and Spurdle, Amanda B., additional

Published
2014
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22. Tumor Mismatch Repair Immunohistochemistry and DNA MLH1 Methylation Testing of Patients With Endometrial Cancer Diagnosed at Age Younger Than 60 Years Optimizes Triage for Population-Level Germline Mismatch Repair Gene Mutation Testing

Author

Buchanan, Daniel D., primary, Tan, Yen Y., additional, Walsh, Michael D., additional, Clendenning, Mark, additional, Metcalf, Alexander M., additional, Ferguson, Kaltin, additional, Arnold, Sven T., additional, Thompson, Bryony A., additional, Lose, Felicity A., additional, Parsons, Michael T., additional, Walters, Rhiannon J., additional, Pearson, Sally-Ann, additional, Cummings, Margaret, additional, Oehler, Martin K., additional, Blomfield, Penelope B., additional, Quinn, Michael A., additional, Kirk, Judy A., additional, Stewart, Colin J., additional, Obermair, Andreas, additional, Young, Joanne P., additional, Webb, Penelope M., additional, and Spurdle, Amanda B., additional

Published
2014
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23. The role of APCin WNT pathway activation in serrated neoplasia

Author

Borowsky, Jennifer, Dumenil, Troy, Bettington, Mark, Pearson, Sally-Ann, Bond, Catherine, Fennell, Lochlan, Liu, Cheng, McKeone, Diane, Rosty, Christophe, Brown, Ian, Walker, Neal, Leggett, Barbara, and Whitehall, Vicki

Abstract

Conventional adenomas are initiated by APCgene mutation that activates the WNT signal. Serrated neoplasia is commonly initiated by BRAFor KRASmutation. WNT pathway activation may also occur, however, to what extent this is owing to APCmutation is unknown. We examined aberrant nuclear β-catenin immunolocalization as a surrogate for WNT pathway activation and analyzed the entire APCgene coding sequence in serrated and conventional pathway polyps and cancers. WNT pathway activation was a common event in conventional pathway lesions with aberrant nuclear immunolocalization of β-catenin and truncating APCmutations in 90% and 89% of conventional adenomas and 82% and 70% of BRAFwild-type cancers, respectively. WNT pathway activation was seen to a lesser extent in serrated pathway lesions. It occurred at the transition to dysplasia in serrated polyps with a significant increase in nuclear β-catenin labeling from sessile serrated adenomas (10%) to sessile serrated adenomas with dysplasia (55%) and traditional serrated adenomas (9%) to traditional serrated adenomas with dysplasia (39%) (P=0.0001). However, unlike the conventional pathway, truncating APCmutations were rare in the serrated pathway lesions especially sessile serrated adenomas even when dysplastic (15%) and in the BRAFmutant cancers with microsatellite instability that arise from them (8%). In contrast, APCmissense mutations that were rare in conventional pathway adenomas and cancers (3% in BRAFwild-type cancers) were more frequent in BRAFmutant cancers with microsatellite instability (32%). We conclude that increased WNT signaling is important in the transition to malignancy in the serrated pathway but that APCmutation is less common and the spectrum of mutations is different than in conventional colorectal carcinogenesis. Moderate impact APCmutations and non-APC-related causes of increased WNT signaling may have a more important role in serrated neoplasia than the truncating APCmutations common in conventional adenomas.

Published
2018
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24. Oncogenic BRAFmutation induces DNA methylation changes in a murine model for human serrated colorectal neoplasia

Author

Bond, Catherine E., Liu, Cheng, Kawamata, Futoshi, McKeone, Diane M., Fernando, Winnie, Jamieson, Saara, Pearson, Sally-Ann, Kane, Alexandra, Woods, Susan L., Lannagan, Tamsin R. M., Somashekar, Roshini, Lee, Young, Dumenil, Troy, Hartel, Gunter, Spring, Kevin J., Borowsky, Jennifer, Fennell, Lochlan, Bettington, Mark, Lee, Jason, Worthley, Daniel L., Leggett, Barbara A., and Whitehall, Vicki L. J.

Abstract

ABSTRACTColorectal cancer is a major cause of cancer death and approximately 20% arises within serrated polyps, which are under-recognized and poorly understood. Human serrated colorectal polyps frequently exhibit both oncogenic BRAFmutation and widespread DNA methylation changes, which are important in silencing genes restraining neoplastic progression. Here, we investigated whether in vivoinduction of mutant Brafis sufficient to result in coordinated promoter methylation changes for multiple cancer-related genes. The BrafV637Emutation was induced in murine intestine on an FVB;C57BL/6J background and assessed for morphological and DNA methylation changes at multiple time points from 10 days to 14 months. Extensive intestinal hyperplasia developed by 10 days post-induction of the mutation. By 8 months, most mice had murine serrated adenomas with dysplasia and invasive cancer developed in 40% of mice by 14 months. From 5 months onwards, Brafmutant mice showed extensive, gene-specific increases in DNA methylation even in hyperplastic mucosa without lesions. This demonstrates that persistent oncogenic Brafsignaling is sufficient to induce widespread DNA methylation changes. This occurs over an extended period of time, mimicking the long latency followed by rapid progression of human serrated neoplasia. This study establishes for the first time that DNA methylation arises slowly in direct response to prolonged oncogenic Brafsignaling in serrated polyps; this finding has implications both for chemoprevention and for understanding the origin of DNA hypermethylation in cancer generally.

Published
2018
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25. Cancer risks for relatives of patients with serrated polyposis

Author

Win, Aung Ko, Walters, Rhiannon, Buchanan, Daniel, Jenkins, Mark, Sweet, Kevin, Frankel, Wendy, De La Chapelle, Albert, Mckeone, Diane, Walsh, Michael, Clendenning, Mak, Pearson, Sally-Ann, Pavluk, Erica, Nagler, Belinda, Hopper, John, Gattas, Michael, Goldblatt, Jack, George, Jill, Suthers, Graeme, Phillips, Kerry D, Woodall, Sonia, Arnold, Julie, Tucker, Kathy, Field, Michael, Greening, Sian, Gallinger, Steve, Aronson, Melyssa, Perrier, Renee, Woods, Michael, Green, Jane, Walker, Neal, Rosty, Christophe, Parry, Susan, Young, Joanne P, Win, Aung Ko, Walters, Rhiannon, Buchanan, Daniel, Jenkins, Mark, Sweet, Kevin, Frankel, Wendy, De La Chapelle, Albert, Mckeone, Diane, Walsh, Michael, Clendenning, Mak, Pearson, Sally-Ann, Pavluk, Erica, Nagler, Belinda, Hopper, John, Gattas, Michael, Goldblatt, Jack, George, Jill, Suthers, Graeme, Phillips, Kerry D, Woodall, Sonia, Arnold, Julie, Tucker, Kathy, Field, Michael, Greening, Sian, Gallinger, Steve, Aronson, Melyssa, Perrier, Renee, Woods, Michael, Green, Jane, Walker, Neal, Rosty, Christophe, Parry, Susan, and Young, Joanne P

Abstract

OBJECTIVES; Serrated polyposis (hyperplastic polyposis) is characterized by multiple polyps with serrated architecture in the colorectum. Although patients with serrated polyposis are known to be at increased risk of colorectal cancer (CRC) and possibly extracolonic cancers, cancer risk for their relatives has not been widely explored. The aim of this study was to estimate the risks of CRC and extracolonic cancers for relatives of patients with serrated polyposis. METHODS; A cohort of the 1,639 first- and second-degree relatives of 100 index patients with serrated polyposis recruited regardless of a family history of polyps or cancer from genetic clinics in Australia, New Zealand, Canada, and the USA, were retrospectively analyzed to estimate the country-, age-, and sex-specific standardized incidence ratios (SIRs) for relatives compared with the general population. RESULTS; A total of 102 CRCs were observed in first- and second-relatives (SIR 2.25, 95% confidence interval (CI) 1.75-2.93; P

Published
2012

26. Lynch syndrome-associated breast cancers: Clinicopathologic characteristics of a case series from the colon cancer family registry

Author

Walsh, Michael, Buchanan, Daniel, Cummings, Margaret, Pearson, Sally-Ann, Arnold, Sven, Clendenning, Mark, Walters, Rhiannon, McKeone, Diane, Spurdle, Amanda, Hopper, John, Jenkins, Mark, Phillips, Kerry, Suthers, Graeme, George, Jill, Goldblatt, Jack, Muir, Amanda, Tucker, Katherine, Pelzer, Elise, other, and, Walsh, Michael, Buchanan, Daniel, Cummings, Margaret, Pearson, Sally-Ann, Arnold, Sven, Clendenning, Mark, Walters, Rhiannon, McKeone, Diane, Spurdle, Amanda, Hopper, John, Jenkins, Mark, Phillips, Kerry, Suthers, Graeme, George, Jill, Goldblatt, Jack, Muir, Amanda, Tucker, Katherine, Pelzer, Elise, and other, and

Abstract

Purpose: The recognition of breast cancer as a spectrum tumor in Lynch syndrome remains controversial. The aim of this study was to explore features of breast cancers arising in Lynch syndrome families. Experimental Design: This observational study involved 107 cases of breast cancer identified from the Colorectal Cancer Family Registry (Colon CFR) from 90 families in which (a) both breast and colon cancer co-occurred, (b) families met either modified Amsterdam criteria, or had at least one early-onset (<50 years) colorectal cancer, and (c) breast tissue was available within the biospecimen repository for mismatch repair (MMR) testing. Eligibility criteria for enrollment in the Colon CFR are available online. Breast cancers were reviewed by one pathologist. Tumor sections were stained for MLH1, PMS2, MSH2, and MSH6, and underwent microsatellite instability testing. Results: Breast cancer arose in 35 mutation carriers, and of these, 18 (51%) showed immunohistochemical absence of MMR protein corresponding to the MMR gene mutation segregating the family. MMR-deficient breast cancers were more likely to be poorly differentiated (P = 0.005) with a high mitotic index (P = 0.002), steroid hormone receptor–negative (estrogen receptor, P = 0.031; progesterone receptor, P = 0.022), and to have peritumoral lymphocytes (P = 0.015), confluent necrosis (P = 0.002), and growth in solid sheets (P < 0.001) similar to their colorectal counterparts. No difference in age of onset was noted between the MMR-deficient and MMR-intact groups. Conclusions: MMR deficiency was identified in 51% of breast cancers arising in known mutation carriers. Breast cancer therefore may represent a valid tissue option for the detection of MMR deficiency in which spectrum tumors are lacking

Published
2010

27. Family History of Colorectal Cancer in BRAF p.V600E-Mutated Colorectal Cancer Cases

Author

Buchanan, Daniel D., primary, Win, Aung K., additional, Walsh, Michael D., additional, Walters, Rhiannon J., additional, Clendenning, Mark, additional, Nagler, Belinda, additional, Pearson, Sally-Ann, additional, Macrae, Finlay A., additional, Parry, Susan, additional, Arnold, Julie, additional, Winship, Ingrid, additional, Giles, Graham G., additional, Lindor, Noralane M., additional, Potter, John D., additional, Hopper, John L., additional, Rosty, Christophe, additional, Young, Joanne P., additional, and Jenkins, Mark A., additional

Published
2013
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28. Multiplicity and Molecular Heterogeneity of Colorectal Carcinomas in Individuals With Serrated Polyposis

Author

Rosty, Christophe, primary, Walsh, Michael D., additional, Walters, Rhiannon J., additional, Clendenning, Mark, additional, Pearson, Sally-Ann, additional, Jenkins, Mark A., additional, Win, Aung Ko, additional, Hopper, John L., additional, Sweet, Kevin, additional, Frankel, Wendy L., additional, Aronson, Melyssa, additional, Gallinger, Steve, additional, Goldblatt, Jack, additional, Tucker, Kathy, additional, Greening, Sian, additional, Gattas, Michael R., additional, Woodall, Sonja, additional, Arnold, Julie, additional, Walker, Neal I., additional, Parry, Susan, additional, Young, Joanne P., additional, and Buchanan, Daniel D., additional

Published
2013
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29. Phenotype and Polyp Landscape in Serrated Polyposis Syndrome

Author

Rosty, Christophe, primary, Buchanan, Daniel D., additional, Walsh, Michael D., additional, Pearson, Sally-Ann, additional, Pavluk, Erika, additional, Walters, Rhiannon J., additional, Clendenning, Mark, additional, Spring, Kevin J., additional, Jenkins, Mark A., additional, Win, Aung K., additional, Hopper, John L., additional, Sweet, Kevin, additional, Frankel, Wendy L., additional, Aronson, Melyssa, additional, Gallinger, Steve, additional, Goldblatt, Jack, additional, Woodall, Sonja, additional, Arnold, Julie, additional, Walker, Neal I., additional, Jass, Jeremy R., additional, Parry, Susan, additional, and Young, Joanne P., additional

Published
2012
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30. Cancer Risks for Relatives of Patients With Serrated Polyposis

Author

Win, Aung Ko, primary, Walters, Rhiannon J, additional, Buchanan, Daniel D, additional, Jenkins, Mark A, additional, Sweet, Kevin, additional, Frankel, Wendy L, additional, de la Chapelle, Albert, additional, McKeone, Diane M, additional, Walsh, Michael D, additional, Clendenning, Mark, additional, Pearson, Sally-Ann, additional, Pavluk, Erika, additional, Nagler, Belinda, additional, Hopper, John L, additional, Gattas, Michael R, additional, Goldblatt, Jack, additional, George, Jill, additional, Suthers, Graeme K, additional, Phillips, Kerry D, additional, Woodall, Sonja, additional, Arnold, Julie, additional, Tucker, Kathy, additional, Field, Michael, additional, Greening, Sian, additional, Gallinger, Steve, additional, Aronson, Melyssa, additional, Perrier, Renee, additional, Woods, Michael O, additional, Green, Jane S, additional, Walker, Neal, additional, Rosty, Christophe, additional, Parry, Susan, additional, and Young, Joanne P, additional

Published
2012
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31. Su1851 Family History and Pathology Features in Early-Onset Colorectal Cancer Cases With a BRAF P.V600e Mutation

Author

Buchanan, Daniel D., primary, Win, Aung Ko, additional, Walters, Rhiannon J., additional, Walsh, Michael D., additional, Clendenning, Mark, additional, Nagler, Belinda N., additional, Pavluk, Erika, additional, Pearson, Sally-Ann, additional, Rosty, Christophe, additional, Parry, Susan, additional, Hopper, John, additional, Jenkins, Mark A., additional, and Young, Joanne P., additional

Published
2012
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32. Risk Factors for Colorectal Cancer in Patients with Multiple Serrated Polyps: A Cross-Sectional Case Series from Genetics Clinics

Author

Buchanan, Daniel D., primary, Sweet, Kevin, additional, Drini, Musa, additional, Jenkins, Mark A., additional, Win, Aung Ko, additional, English, Dallas R., additional, Walsh, Michael D., additional, Clendenning, Mark, additional, McKeone, Diane M., additional, Walters, Rhiannon J., additional, Roberts, Aedan, additional, Pearson, Sally-Ann, additional, Pavluk, Erika, additional, Hopper, John L., additional, Gattas, Michael R., additional, Goldblatt, Jack, additional, George, Jill, additional, Suthers, Graeme K., additional, Phillips, Kerry D., additional, Woodall, Sonja, additional, Arnold, Julie, additional, Tucker, Kathy, additional, Muir, Amanda, additional, Field, Michael, additional, Greening, Sian, additional, Gallinger, Steven, additional, Perrier, Renee, additional, Baron, John A., additional, Potter, John D., additional, Haile, Robert, additional, Frankel, Wendy, additional, de la Chapelle, Albert, additional, Macrae, Finlay, additional, Rosty, Christophe, additional, Walker, Neal I., additional, Parry, Susan, additional, and Young, Joanne P., additional

Published
2010
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33. S1677 Over-Expression of the Chromosome 11 Mucins is Associated With CIMP and BRAF Mutation in Colorectal Cancer

Author

Walsh, Michael D., primary, Buchanan, Daniel D., additional, Pearson, Sally-Ann, additional, Walters, Rhiannon J., additional, McKeone, Diane M., additional, Clendenning, Mark, additional, Hopper, John, additional, Jenkins, Mark A., additional, Giles, Graham G., additional, English, Dallas, additional, Rosty, Christophe, additional, McGuckin, Michael A., additional, and Young, Joanne P., additional

Published
2010
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34. Lynch Syndrome–Associated Breast Cancers: Clinicopathologic Characteristics of a Case Series from the Colon Cancer Family Registry

Author

Walsh, Michael D., primary, Buchanan, Daniel D., additional, Cummings, Margaret C., additional, Pearson, Sally-Ann, additional, Arnold, Sven T., additional, Clendenning, Mark, additional, Walters, Rhiannon, additional, McKeone, Diane M., additional, Spurdle, Amanda B., additional, Hopper, John L., additional, Jenkins, Mark A., additional, Phillips, Kerry D., additional, Suthers, Graeme K., additional, George, Jill, additional, Goldblatt, Jack, additional, Muir, Amanda, additional, Tucker, Kathy, additional, Pelzer, Elise, additional, Gattas, Michael R., additional, Woodall, Sonja, additional, Parry, Susan, additional, Macrae, Finlay A., additional, Haile, Robert W., additional, Baron, John A., additional, Potter, John D., additional, Le Marchand, Loic, additional, Bapat, Bharati, additional, Thibodeau, Stephen N., additional, Lindor, Noralane M., additional, McGuckin, Michael A., additional, and Young, Joanne P., additional

Published
2010
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36. Equality in Lynch Syndrome Screening: Why Should We Hold Patients With Endometrial Cancer to a Different Standard?

Author

Buchanan, Daniel D., Tan, Yen Y., Walsh, Michael D., Clendenning, Mark, Metcalf, Alexander M., Ferguson, Kaltin, Arnold, Sven T., Thompson, Bryony A., Lose, Felicity A., Parsons, Michael T., Walters, Rhiannon J., Pearson, Sally-Ann, Cummings, Margaret, Oehler, Martin K., Blomfield, Penelope B., Quinn, Michael A., Kirk, Judy A., Stewart, Colin J., Obermair, Andreas, and Young, Joanne P.

Published
2014
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