104 results on '"Perez, Claudio F."'
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2. Estimation of evapotranspiration of a salt marsh in southern South America with coupled Penman-Monteith and surface resistance models
3. First results of energy and mass exchange in a salt marsh on southeastern South America
4. Fluorescence Resonance Energy Transfer-based Structural Analysis of the Dihydropyridine Receptor α1S Subunit Reveals Conformational Differences Induced by Binding of the β1a Subunit
5. Dissimilarity in plant species diversity between salt marsh and neighboring environments decreases as environmental harshness increases
6. Ablation of Triadin Causes Loss of Cardiac Ca²⁺ Release Units, Impaired Excitation-Contraction Coupling, and Cardiac Arrhythmias
7. Bidirectional Signaling between Calcium Channels of Skeletal Muscle Requires Multiple Direct and Indirect Interactions
8. Cyclic ADP-ribose activates caffeine-sensitive calcium channels from sea urchin egg microsomes
9. Functional and structural characterization of a novel malignant hyperthermia-susceptible variant of DHPR-β1a subunit (CACNB1)
10. A focus on extracellular Ca2+ entry into skeletal muscle
11. Functional and structural characterization of a novel malignant hyperthermia-susceptible variant of DHPR-β1a subunit (CACNB1).
12. Contribution of L496-L500-W503 Motif of DHPR-β1A Subunit to Skeletal-Type EC-Coupling
13. FRET-Based Structural Analysis of the Skeletal Muscle DHPR using Biarsenical Labeling
14. Amino Acid Residues 489–503 of Dihydropyridine Receptor (DHPR) β1a Subunit Are Critical for Structural Communication between the Skeletal Muscle DHPR Complex and Type 1 Ryanodine Receptor
15. Whole Body Periodic Acceleration Is an Effective Therapy to Ameliorate Muscular Dystrophy in mdx Mice
16. Myoplasmic resting Ca2+ regulation by ryanodine receptors is under the control of a novel Ca2+-binding region of the receptor
17. Carboxyl-Terminal Domain of DHPR β1A is Essential for DHPR Tetrad Formation
18. Fluorescence Resonance Energy Transfer-based Structural Analysis of the Dihydropyridine Receptor α1S Subunit Reveals Conformational Differences Induced by Binding of the β1a Subunit.
19. Three-Dimensional Localization of the α and β Subunits and of the II-III Loop in the Skeletal Muscle L-type Ca2+ Channel
20. Triadin/Junctin Double Null Mouse Reveals a Differential Role for Triadin and Junctin in Anchoring CASQ to the jSR and Regulating Ca2+ Homeostasis
21. Effect of Oxidative Stress on Intracellular Ca2+ and Na+ Concentration in Duchenne Muscular Dystrophy Myotubes
22. Null Mutations for Triadin and Junctin Reveal Triadin Anchors and the Requirement for Triadin-Junctin-CASQ Complexes in Stablizing RyR Resting Leak
23. Role of C-Term Tail of DHPR β1A in the DHPR/RyR1 Interaction
24. Reduced gain of excitation–contraction coupling in triadin-null myotubes is mediated by the disruption of FKBP12/RyR1 interaction
25. On the footsteps of Triadin and its role in skeletal muscle
26. Ablation of Skeletal Muscle Triadin Impairs FKBP12/RyR1 Channel Interactions Essential for Maintaining Resting Cytoplasmic Ca2+
27. Effect of Triadin on Retrograde and Orthograde Signaling between RyR1 and DHPR in Cultured Myotubes
28. Reactive Cysteines of Ryanodine Receptor Type 1 Influence Function and Response to Oxidative Stress
29. 3D Structure of a Recombinant DHPR Expressed in Mouse
30. Dysregulation of Ca2+ Entry and SR Calcium Leak are Responsible for Elevated Resting Free Ca2+ in Triadin-Null Myotubes
31. Ablation of triadin causes loss of cardiac Ca 2+ release units, impaired excitation–contraction coupling, and cardiac arrhythmias
32. Enantiomeric Specificity of (−)-2,2′,3,3′,6,6′-Hexachlorobiphenyl toward Ryanodine Receptor Types 1 and 2
33. Allosterically coupled calcium and magnesium binding sites are unmasked by ryanodine receptor chimeras
34. Triadins Modulate Intracellular Ca2+ Homeostasis but Are Not Essential for Excitation-Contraction Coupling in Skeletal Muscle
35. Efficient Site-Specific Integration of Large Transgenes by an Enhanced Herpes Simplex Virus/Adeno-Associated Virus Hybrid Amplicon Vector
36. Expression levels of RyR1 and RyR3 control resting free Ca2+in skeletal muscle
37. Amino Acids 1–1,680 of Ryanodine Receptor Type 1 Hold Critical Determinants of Skeletal Type for Excitation-Contraction Coupling
38. Identification of a Key Determinant of Ryanodine Receptor Type 1 Required for Activation by 4-Chloro-m-cresol
39. FKBP12 Binding to RyR1 Modulates Excitation-Contraction Coupling in Mouse Skeletal Myotubes
40. RyR1/RyR3 Chimeras Reveal that Multiple Domains of RyR1 Are Involved in Skeletal-Type E-C Coupling
41. Triadin/Junctin Double Null Mouse Reveals a Differential Role for Triadin and Junctin in Anchoring CASQ to the jSR and Regulating Ca2+ Homeostasis.
42. Efficient Site-Specific Integration of Large Transgenes by an Enhanced Herpes Simplex Virus/Adeno-Associated Virus Hybrid Amplicon Vector.
43. Expression levels of RyR1 and RyR3 control resting free Ca2+ in skeletal muscle.
44. Registro preliminar del depósito atmosférico anual de esporas de helechos de las yungas sobre un gradiente ambiental en la cuenca media del río Perico (Jujuy, Argentina)
45. A focus on extracellular Ca2+entry into skeletal muscle
46. Dysregulation of Ca2+Entry and SR Calcium Leak are Responsible for Elevated Resting Free Ca2+in Triadin-Null Myotubes
47. Null Mutations for Triadin and Junctin Reveal Triadin Anchorsand the Requirement for Triadin-Junctin-CASQ Complexes in Stablizing RyR Resting Leak
48. Effect of Oxidative Stress on Intracellular Ca2+and Na+Concentration in Duchenne Muscular Dystrophy Myotubes
49. Amino Acid Residues 489-503 of Dihydropyridine Receptor (DHPR) β1a Subunit Are Critical for Structural Communication between the Skeletal Muscle DHPR Complex and Type 1 Ryanodine Receptor.
50. Three-Dimensional Localization of the α and β Subunits and of the II-III Loop in the Skeletal Muscle L-type Ca2+ Channel.
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