36 results on '"Petrie, Emma J."'
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2. Identification of MLKL membrane translocation as a checkpoint in necroptotic cell death using Monobodies
3. Conformational interconversion of MLKL and disengagement from RIPK3 precede cell death by necroptosis
4. MLKL trafficking and accumulation at the plasma membrane control the kinetics and threshold for necroptosis
5. A missense mutation in the MLKL brace region promotes lethal neonatal inflammation and hematopoietic dysfunction
6. Distinct pseudokinase domain conformations underlie divergent activation mechanisms among vertebrate MLKL orthologues
7. The brace helices of MLKL mediate interdomain communication and oligomerisation to regulate cell death by necroptosis
8. The pseudokinase MLKL mediates programmed hepatocellular necrosis independently of RIPK3 during hepatitis
9. Insane in the membrane: a structural perspective of MLKL function in necroptosis
10. Structural Insights into the Unique Modes of Relaxin-Binding and Tethered-Agonist Mediated Activation of RXFP1 and RXFP2
11. Structural insights into the modes of relaxin-binding and tethered-agonist activation of RXFP1 and RXFP2
12. Membrane permeabilization is mediated by distinct epitopes in mouse and human orthologs of the necroptosis effector, MLKL
13. Resolving the Unconventional Mechanisms Underlying RXFP1 and RXFP2 Receptor Function
14. Viral MLKL Homologs Subvert Necroptotic Cell Death by Sequestering Cellular RIPK3
15. Missense mutations in the MLKL ‘brace’ region lead to lethal neonatal inflammation in mice and are present in high frequency in humans
16. The Structural Basis of Necroptotic Cell Death Signaling
17. Conformational switching of the pseudokinase domain promotes human MLKL tetramerization and cell death by necroptosis
18. Distinct activation modes of the Relaxin Family Peptide Receptor 2 in response to insulin-like peptide 3 and relaxin
19. The secret life of kinases: insights into non‐catalytic functions from pseudokinases
20. Erratum: EspL is a bacterial cysteine protease effector that cleaves RHIM proteins to block necroptosis and inflammation
21. EspL is a bacterial cysteine protease effector that cleaves RHIM proteins to block necroptosis and inflammation
22. Recombinant RXFP1-LDL-A module does not form dimers
23. The complex binding mode of the peptide hormone H2 relaxin to its receptor RXFP1
24. Native Chemical Ligation to Minimize Aspartimide Formation during Chemical Synthesis of Small LDLa Protein
25. In a Class of Their Own – RXFP1 and RXFP2 are Unique Members of the LGR Family
26. Determinants of oligosaccharide specificity of the carbohydrate-binding modules of AMP-activated protein kinase
27. Investigation of Interactions at the Extracellular Loops of the Relaxin Family Peptide Receptor 1 (RXFP1)
28. AMPK β subunits display isoform specific affinities for carbohydrates
29. Mapping Key Regions of the RXFP2 Low-Density Lipoprotein Class-A Module That Are Involved in Signal Activation
30. The Relaxin Receptor (RXFP1) Utilizes Hydrophobic Moieties on a Signaling Surface of Its N-terminal Low Density Lipoprotein Class A Module to Mediate Receptor Activation
31. Chimeric RXFP1 and RXFP2 Receptors Highlight the Similar Mechanism of Activation Utilizing Their N-Terminal Low-Density Lipoprotein Class A Modules
32. Native Chemical Ligation to Minimize Aspartimide Formation during Chemical Synthesis of Small LDLa Protein.
33. Recognition of Mitochondrial Targeting Sequences by the Import Receptors Tom20 and Tom22
34. CD94-NKG2A recognition of human leukocyte antigen (HLA)-E bound to an HLA class I leader sequence
35. Native Chemical Ligation to Minimize Aspartimide Formation during Chemical Synthesis of Small LDLa Protein.
36. Recombinant RXFP1-LDL-A module does not form dimers.
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