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2. Islet Transplantation: Current Limitations and Challenges for Successful Outcomes.

Author

Langlois, Allan, Pinget, Michel, Kessler, Laurence, and Bouzakri, Karim

Subjects
*ISLANDS, *MITOCHONDRIA, *PANCREAS, *IMMUNOSUPPRESSION, *ISCHEMIA, *ISLANDS of Langerhans
Abstract

Islet transplantation is a promising approach for treating patients with unstable T1DM. However, it is confronted with numerous obstacles throughout the various stages of the transplantation procedure. Significant progress has been made over the last 25 years in understanding the mechanisms behind the loss of functional islet mass and in developing protective strategies. Nevertheless, at present, two to three pancreases are still needed to treat a single patient, which limits the maximal number of patients who can benefit from islet transplantation. Thus, this publication provides an overview of recent scientific findings on the various issues affecting islet transplantation. Specifically, we will focus on the understanding of the mechanisms involved and the strategies developed to alleviate these problems from the isolation stage to the post-transplantation phase. Finally, we hope that this review will highlight new avenues of action, enabling us to propose pancreatic islet transplantation to a maximum number of patients with T1DM. [ABSTRACT FROM AUTHOR]

Published
2024
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7. Le diabète : Mieux le comprendre pour mieux vivre

Author

Pinget, Michel, Gerson, Michel, Pinget, Michel, Pinget, Michel, Gerson, Michel, and Pinget, Michel

Abstract

Le diabète est une maladie chronique, devenue un véritable enjeu de santé publique, pouvant intervenir à tout âge de la vie. Sous l'impulsion du Centre Européen d'Etude du Diabète (CEED), les auteurs ont réussi la gageure de rendre accessible au patient et à sa famille : des informations scientifiques et médicales pour mieux comprendre les mécanismes de la maladie; des conseils sur l'alimentation et l'activité physique pour mieux vivre au quotidien; l'explication des traitements actuels et de demain. Un guide, très documenté et pratique.

Published
2022

9. Orthostatic hypotension and orthostatic hypertension are both associated with lower cognitive function: The S.AGES cohort.

Author

Strumia, M., Vidal, J. S., Cestac, P., Sallerin, B., Hanon, O., Rouch, L., Benattar‐Zibi, Linda, Bertin, Philippe, Berrut, Gilles, Corruble, Emmanuelle, Derumeaux, Geneviève, Falissard, Bruno, Forette, Françoise, Pasquier, Florence, Pinget, Michel, Ourabah, Rissane, and Danchin, Nicolas

Subjects
COGNITION disorder risk factors, HYPERTENSION, CARDIOVASCULAR diseases risk factors, CONFIDENCE intervals, ORTHOSTATIC hypotension, FISHER exact test, RISK assessment, DESCRIPTIVE statistics, CHI-squared test, RESEARCH funding, DATA analysis software, COGNITION in old age, DISEASE complications, OLD age
Abstract

Background: Blood pressure (BP) postural changes, both orthostatic hypotension (OHYPO) and orthostatic hypertension (OHYPER) are common in older adults. Few studies have investigated their association with cognition, particularly for OHYPER, an emerging cardiovascular risk factor. We aimed to assess the association between OHYPO, OHYPER and cognition in non‐institutionalized older subjects. Methods: The S.AGES (Sujets ÂGES, Aged Subjects) cohort followed every 6 months for 3 years non‐institutionalized subjects aged ≥65 years without dementia at inclusion, in France. OHYPO and OHYPER were respectively defined as a fall or an increase of ≥20 mmHg in systolic BP and/or ≥10 mmHg in diastolic BP after standing from a sitting position. Cognition was assessed using the Mini‐Mental State Examination (MMSE). Linear mixed models were used for the analyses. Results: Among the 3170 subjects included (mean age 78 years, 56% women), 209 (6.5%) had OHYPO and 226 (7.1%) had OHYPER at baseline. After adjustment for demographics, cardiovascular risk factors and disease, seated SBP/DBP and BP lowering treatment, mean MMSE was 0.52 point lower in participants with OHYPER compared to those with normal BP postural changes (β adjusted [95% CI] = −0.52 [−0.96; −0.09], p = 0.02) and 0.50 point lower in participants with OHYPO compared to those with normal BP postural changes (β adjusted [95% CI] = −0.50 [−0.95; −0.06], p = 0.03). Sensitivity analyses showed a dose–response relationship between OHYPO and cognition. Conclusion: Although the absolute differences in MMSE were small, both OHYPO and OHYPER were associated with lower cognition. Orthostatic BP measurements could help identify patients with risk of cognitive impairment. Further studies are needed to assess whether controlling orthostatic BP could be a promising interventional target in preserving cognition among older adults. [ABSTRACT FROM AUTHOR]

Published
2023
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11. Primary care management of non-institutionalized elderly diabetic patients: The S.AGES cohort – Baseline data

Author

Bucher, Sophie, Bauduceau, Bernard, Benattar-Zibi, Linda, Bertin, Philippe, Berrut, Gilles, Corruble, Emmanuelle, Danchin, Nicolas, Delespierre, Tiba, Derumeaux, Geneviève, Doucet, Jean, Falissard, Bruno, Forette, Francoise, Hanon, Olivier, Ourabah, Rissane, Pasquier, Florence, Piedvache, Celine, Pinget, Michel, Ringa, Virginie, and Becquemont, Laurent

Published
2015
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13. sj-docx-1-cll-10.1177_09636897231179642 – Supplemental material for Beneficial Effects of Two Marine Oxygen Carriers, M101 and M201, on Human Islet Quality in Hypoxic Culture Conditions

Author

Lemaire, Florent, Sigrist, Séverine, Brassard, Jonathan, Demini, Leila, Zal, Franck, Jeandidier, Nathalie, Pinget, Michel, and Maillard, Elisa

Subjects
Medicine, Cell Biology
Abstract

Supplemental material, sj-docx-1-cll-10.1177_09636897231179642 for Beneficial Effects of Two Marine Oxygen Carriers, M101 and M201, on Human Islet Quality in Hypoxic Culture Conditions by Florent Lemaire, Séverine Sigrist, Jonathan Brassard, Leila Demini, Franck Zal, Nathalie Jeandidier, Michel Pinget and Elisa Maillard in Cell Transplantation

Published
2023
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19. Crosstalk Communications Between Islets Cells and Insulin Target Tissue: The Hidden Face of Iceberg

Author

Langlois, Allan, Dumond, Aurore, Vion, Julie, Pinget, Michel, and Bouzakri, Karim

Subjects
islets, Beta cell (B-cell), Endocrinology, Diabetes and Metabolism, RC648-665, Diseases of the endocrine glands. Clinical endocrinology, adipose tissue, Islets of Langerhans, Liver, Insulin-Secreting Cells, Insulin Secretion, Diabetes Mellitus, Humans, Insulin, skeletal muscle
Abstract

The regulation of insulin secretion is under control of a complex inter-organ/cells crosstalk involving various metabolites and/or physical connections. In this review, we try to illustrate with current knowledge how β-cells communicate with other cell types and organs in physiological and pathological contexts. Moreover, this review will provide a better understanding of the microenvironment and of the context in which β-cells exist and how this can influence their survival and function. Recent studies showed that β-cell insulin secretion is regulated also by a direct and indirect inter-organ/inter-cellular communication involving various factors, illustrating the idea of “the hidden face of the iceberg”. Moreover, any disruption on the physiological communication between β-cells and other cells or organs can participate on diabetes onset. Therefore, for new anti-diabetic treatments’ development, it is necessary to consider the entire network of cells and organs involved in the regulation of β-cellular function and no longer just β-cell or pancreatic islet alone. In this context, we discuss here the intra-islet communication, the β-cell/skeletal muscle, β-cell/adipose tissue and β-cell/liver cross talk.

Published
2022
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22. Cardiovascular Drugs and Metformin Drug Dosage According to Renal Function in Non-Institutionalized Elderly Patients

Author

Becquemont, Laurent, Bauduceau, Bernard, Benattar-Zibi, Linda, Al-Salameh, Abdallah, Berrut, Gilles, Bertin, Philippe, Bucher, Sophie, Corruble, Emmanuelle, Danchin, Nicolas, Derumeaux, Geneviève, Doucet, Jean, Falissard, Bruno, Forette, Francoise, Hanon, Olivier, Pasquier, Florence, Pinget, Michel, Ourabah, Rissane, and Piedvache, Celine

Published
2016
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24. Association between Cardiovascular Drugs and Chronic Kidney Disease in Non-Institutionalized Elderly Patients

Author

Becquemont, Laurent, Bauduceau, Bernard, Benattar-Zibi, Linda, Berrut, Gilles, Bertin, Philippe, Bucher, Sophie, Corruble, Emmanuelle, Danchin, Nicolas, al-Salameh, Abdallah, Derumeaux, Geneviève, Doucet, Jean, Falissard, Bruno, Forette, Francoise, Hanon, Olivier, Pasquier, Florence, Pinget, Michel, Ourabah, Rissane, and Piedvache, Celine

Published
2015
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25. Beneficial Effects of Two Marine Oxygen Carriers, M101 and M201, on Human Islet Quality in Hypoxic Culture Conditions.

Author

Lemaire, Florent, Sigrist, Séverine, Brassard, Jonathan, Demini, Leila, Zal, Franck, Jeandidier, Nathalie, Pinget, Michel, and Maillard, Elisa

Subjects
OXYGEN carriers, OXYGEN content of seawater, ISLANDS, ISLANDS of Langerhans, MARINE worms
Abstract

High pancreatic islet sensitivity to hypoxia is an important issue in the field of pancreatic islet transplantation. A promising strategy to improve islet oxygenation in hypoxic conditions is to leverage the properties of hemoglobin as a natural carrier of oxygen. Studies using human or bovine hemoglobin have failed to demonstrate efficacy, probably due to the molecule being unstable in the absence of protective erythrocytes. Recently, marine worm hemoglobins have been shown to be more stable and to possess higher oxygen carrier potential, with 156 oxygen binding sites per molecule compared to four in humans. Previous studies have shown the beneficial effects of two marine worm hemoglobins, M101 and M201, on nonhuman pancreatic islets. However, their effects on human islets have not been tested or compared. In this study, we assessed the impact of both molecules during human islet culture in vitro under hypoxic conditions. Human islets were exposed to both molecules for 24 h in high islet density-induced hypoxia [600 islet equivalents (IEQ)/cm²]. M101 and M201 reduced the release of hypoxic (VEGF) and apoptotic (cyt c) markers in the medium after 24-h culture. Human islet function or viability was improved in vitro in the presence of these oxygen carriers. Thus, the utilization of M101 or M201 could be a safe and easy way to improve human islet oxygenation and survival in hypoxic conditions as observed during islet culture prior to transplantation or islet encapsulation. [ABSTRACT FROM AUTHOR]

Published
2023
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27. STAT3 Regulates Mitochondrial Gene Expression in Pancreatic β-Cells and its Deficiency Induces Glucose Intolerance in Obesity

Author

Schaschkow, Anaïs, primary, Pang, Lokman, primary, Vandenbempt, Valerie, primary, Elvira, Bernat, primary, Litwak, Sara A., primary, Vekeriotaite, Beata, primary, Maillard, Elisa, primary, Vermeersch, Marjorie, primary, Paula, Flavia MM, primary, Pinget, Michel, primary, Perez-Morga, David, primary, Gough, Daniel J., primary, and Gurzov, Esteban N., primary

Published
2021
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28. Meal replacement by formula diet reduces weight more than a lifestyle intervention alone in patients with overweight or obesity and accompanied cardiovascular risk factors—the ACOORH trial

Author

Halle, Martin, Röhling, Martin, Banzer, Winfried, Braumann, Klaus Michael, Kempf, Kerstin, McCarthy, David, Schaller, Nina, Predel, Hans Georg, Scholze, Jürgen, Führer-Sakel, Dagmar, Toplak, Hermann, Berg, Aloys, Martin, Stephan, Tan, Susanne, Koohkan, Sadaf, Pinget, Michel, and ACOORH Study Group

Subjects
0301 basic medicine, medicine.medical_specialty, Meal, 030109 nutrition & dietetics, Nutrition and Dietetics, Meal replacement, Weight change, Medizin, dewey610, Medicine (miscellaneous), 030209 endocrinology & metabolism, Overweight, medicine.disease, Obesity, law.invention, 03 medical and health sciences, 0302 clinical medicine, Randomized controlled trial, law, Weight loss, Internal medicine, medicine, medicine.symptom, Metabolic syndrome, dewey570
Abstract

Background:\ud As formula diets have demonstrated to be effective in reducing weight, we hypothesised that in patients with overweight or obesity and accompanied cardiovascular risk factors, combining a liquid formula diet with a lifestyle intervention is superior in reducing weight and improving cardiovascular risk factors than lifestyle intervention alone. \ud \ud Methods:\ud In this multicenter RCT 463 participants with overweight or obesity (BMI: 27–35 kg/m²; at least one additional comorbidity of the metabolic syndrome) were randomised (1:2) into either a control group with lifestyle intervention only (CON, n = 155) or a lifestyle intervention group including a liquid meal replacement (INT, n = 308). Both groups used telemonitoring devices (scales and pedometers), received information on healthy diet and were instructed to increase physical activity. Telemonitoring devices automatically transferred data into a personalised online portal and acquired data were discussed. INT obtained a liquid meal replacement substituting three meals/day (~1200 kcal) within the first week. During weeks 2–4, participants replaced two meals/day and during weeks 5–26 only one meal/day was substituted\ud (1300–1500 kcal/day). Follow-up was conducted after 52 weeks. Intention-to-treat analyses were performed. Primary outcome was weight change. Secondary outcomes comprised changes in cardiometabolic risk factors including body composition and laboratory parameters.\ud \ud Results:\ud From the starting cohort 360 (78%, INT: n = 244; CON: n = 116) and 317 (68%, INT: n = 216; CON: n = 101) participants completed the 26-weeks intervention phase and the 52-weeks follow-up. The estimated treatment difference (ETD) between both groups was −3.2 kg [−4.0; −2.5] (P < 0.001) after 12 weeks and −1.8 kg [−2.8; −0.8] (P < 0.001) after 52 weeks.\ud \ud Conclusions:\ud A low-intensity lifestyle intervention combined with a liquid meal replacement is superior regarding weight reduction and improvement of cardiovascular risk factors than lifestyle intervention alone.

Published
2020

29. STAT3 Regulates Mitochondrial Gene Expression in Pancreatic Beta-Cells and Its Deficiency Induces Glucose Intolerance in Obesity

Author

Schaschkow, Anaïs, Pang, Lokman, Vandenbempt, Valerie, Elvira Jimenez, Bernat, Litwak, Sara A, Vekeriotaite, Beata, Maillard, Elisa, Vermeersch, Marjorie, Moura De Paula, Flavia, Pinget, Michel, Perez-Morga, David, Gough, Daniel J, Gurzov, Esteban Nicolas, Schaschkow, Anaïs, Pang, Lokman, Vandenbempt, Valerie, Elvira Jimenez, Bernat, Litwak, Sara A, Vekeriotaite, Beata, Maillard, Elisa, Vermeersch, Marjorie, Moura De Paula, Flavia, Pinget, Michel, Perez-Morga, David, Gough, Daniel J, and Gurzov, Esteban Nicolas

Abstract

info:eu-repo/semantics/published

Published
2021

30. Diabetes and associated disorders in Cambodia: two epidemiological surveys

Author

King, Hilary, Keuky, Lim, Seng, Serey, Khun, Touch, Roglic, Gojka, and Pinget, Michel

Subjects
World Health Organization, Diabetes -- Research, Diabetes -- Growth, Diabetes -- Analysis, Diabetes -- Statistics, Cardiovascular diseases -- Growth, Cardiovascular diseases -- Analysis, Obesity -- Growth, Obesity -- Analysis, Obesity -- Statistics, Cambodia -- Economic aspects, Cambodia -- Health policy, Company growth
Published
2005

34. A circular RNA generated from an intron of the insulin gene controls insulin secretion

Author

Stoll, Lisa, primary, Rodríguez-Trejo, Adriana, additional, Guay, Claudiane, additional, Brozzi, Flora, additional, Bayazit, Mustafa Bilal, additional, Gattesco, Sonia, additional, Menoud, Véronique, additional, Sobel, Jonathan, additional, Marques, Ana Claudia, additional, Venø, Morten Trillingsgaard, additional, Esguerra, Jonathan Lou S., additional, Barghouth, Mohammad, additional, Suleiman, Mara, additional, Marselli, Lorella, additional, Kjems, Jørgen, additional, Eliasson, Lena, additional, Renström, Erik, additional, Bouzakri, Karim, additional, Pinget, Michel, additional, Marchetti, Piero, additional, and Regazzi, Romano, additional

Published
2020
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35. Beta-Cell-Specific Expression of Nicotinamide Adenine Dinucleotide Phosphate Oxidase 5 Aggravates High-Fat Diet-Induced Impairment of Islet Insulin Secretion in Mice

Author

Bouzakri, Karim, primary, Veyrat-Durebex, Christelle, additional, Holterman, Chet, additional, Arous, Caroline, additional, Barbieux, Charlotte, additional, Bosco, Domenico, additional, Altirriba, Jordi, additional, Alibashe, Mohamed, additional, Tournier, Benjamin B., additional, Gunton, Jenny E., additional, Mouche, Sarah, additional, Bietiger, William, additional, Forterre, Alexis, additional, Berney, Thierry, additional, Pinget, Michel, additional, Christofori, Gerhard, additional, Kennedy, Christopher, additional, and Szanto, Ildiko, additional

Published
2020
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36. Beta-cell-specific expression of NOX5 aggravates high fat diet-induced impairment of islet insulin secretion in mice

Author

Bouzakri, Karim, Veyrat-Durebex, Christelle, Holterman, Chet E, Arous, Caroline, Barbieux, Charlotte, Bosco, Domenico, Altirriba Gutierrez, Jorge, Alibashe Ahmed, Mohamed, Tournier, Benjamin, Gunton, Jenny E, Mouche, Sarah, Bietiger, William, Forterre, Alexis, Berney, Thierry, Pinget, Michel, Christofori, Gerhard, Kennedy, Christopher, and Szanto, Ildiko

Subjects
Beta cell, Islet, endocrine system, ddc:616.89, NOX5, ddc:617, NADPH oxidase, Diabetes, Insulin, ddc:576.5, ddc:612, Somatostatin
Abstract

NADPH oxidases (NOX-es) produce reactive oxygen species and modulate β-cell insulin secretion. Islets of Type2 diabetic subjects present elevated expression of NOX5. Here we sought to characterize regulation of NOX5 expression in human islets in vitro and to uncover the relevance of NOX5 in islet function in vivo using a novel mouse model expressing NOX5 in doxycycline-inducible, β-cell-specific manner (RIP/rtTA/NOX5 mice). In situ hybridization and immunohistochemistry employed on pancreatic sections demonstrated NOX5 mRNA and protein expressions in human islets. In cultures of dispersed islets NOX5 protein was observed in somatostatin-positive (δ) cells in basal (2.8mM glucose) conditions. siRNA-mediated knock-down of NOX5 in human islets cultured in basal glucose concentrations resulted in diminished glucose-induced insulin secretion in vitro. However, when islets were preincubated in high (16.7mM) glucose media for 12 hours, NOX5 appeared also in insulin-positive (β) cells. In vivo, mice with β-cell NOX5 expression developed aggravated impairment of glucose-induced insulin secretion compared to control mice when challenged with 14 weeks of high-fat diet. Similarly, in vitro palmitate pre-incubation resulted in more severe reduction of insulin release in islets of RIP/rtTA/NOX5 mice compared to their control littermates. Decreased insulin secretion was most distinct in response to theophylline stimulation suggesting impaired cAMP-mediated signaling due to increased phophodiesterase activation. Our data provide the first insight into the complex regulation and function of NOX5 in islets implying an important role for NOX5 in δ-cell mediated intra-islet crosstalk in physiological circumstances but also identifying it as an aggravating factor in β-cell failure in diabetic conditions.

Published
2020

44. Les stratégies thérapeutiques médicamenteuses et non médicamenteuses de l’aide à l’arrêt du tabac

Author

Lagrue, Gilbert, Le Foll, Bernard, Melihan-Cheinin, Pascal, Rostoker, Guy, Ades, Jean, de Beaurepaire, Renaud, Berlin, Yvan, Borgne, Anne, Dautzenberg, Bertrand, Dally, Sylvain, Divine, Catherine, Denis, Catherine, Dumarcet, Nathalie, Dupont, Patrick, Jeanjean, Anne, Lagier, Georges, Lebargy, François, Leder, Jean-Marc, Legeron, Patrick, Le Pen, Claude, Mallaret, Michel, Menard, Joël, Messina, Catherine, Molimard, Robert, Mussetta, Bertrand, Peiffer, Gérard, Pons, Florence, Robine, Isabelle, Saint-Salvi, Béatrice, Stoebner, Anne, Bouvenot, Gilles, Bergmann, Jean-François, Caulin, Charles, Dupuis, Bernard, Aubier, Michel, Bannwarth, Bernard, Camelli, Bruno, Castot, Anne, Funck-Brentano, Christian, Le Jeunne, Claire, Meyer, François, Petit, Michel, Reveillaud, Olivier, Riche, Christian, Thery, Claude, Tremolieres, François, Trouvin, Jean-Hugues, Wong, Olivier, Acquaviva, Jean-Louis, Atlan, Pierre, Aubert, Jean-Pierre, Aubin, Henri-Jean, Baumelou, Alain, Borys, Jean-Michel, Bozzi, Jean-François, Clement, Claude Augustin, Cogneau, Joël, Coletti, Michel, Demeaux, Jean-Louis, Detilleux, Michel, Dumel, François, Gallouedec, Nathalie, Godin, Guy, Klotz, Pierre, Lalande-Errard, Gabrielle, Lemasson, Jean-Louis, Le Roux, Gérard, L’Huillier, Jean-Pierre, Liard, François, Lievre, Michel, Martinet, Yves, Menu, Gilles, Meyrand, Bruno, Mortal, Philippe, Olaya, Emile, Partouche, Henri, Pinget, Michel, Pitras, Jérôme, Said, Gérard, Thibon, René, Thomas, Daniel, and Vrigneaud, Jean

Published
2003
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45. STAT3 Regulates Mitochondrial Gene Expression in Pancreatic β-Cells and Its Deficiency Induces Glucose Intolerance in Obesity.

Author

Schaschkow, Anaïs, Pang, Lokman, Vandenbempt, Valerie, Elvira, Bernat, Litwak, Sara A., Vekeriotaite, Beata, Maillard, Elisa, Vermeersch, Marjorie, Paula, Flavia M.M., Pinget, Michel, Perez-Morga, David, Gough, Daniel J., Gurzov, Esteban N., and Paula, Flavia Mm

Subjects
STAT proteins, GLUCOSE intolerance, GENE expression, HIGH-fat diet, MITOCHONDRIA, OBESITY, RESEARCH, ANIMAL experimentation, RESEARCH methodology, ANIMAL nutrition, BLOOD sugar, MEDICAL cooperation, EVALUATION research, ISLANDS of Langerhans, INSULIN, COMPARATIVE studies, GENES, CARRIER proteins, MICE
Abstract

Most obese and insulin-resistant individuals do not develop diabetes. This is the result of the capacity of β-cells to adapt and produce enough insulin to cover the needs of the organism. The underlying mechanism of β-cell adaptation in obesity, however, remains unclear. Previous studies have suggested a role for STAT3 in mediating β-cell development and human glucose homeostasis, but little is known about STAT3 in β-cells in obesity. We observed enhanced cytoplasmic expression of STAT3 in severely obese subjects with diabetes. To address the functional role of STAT3 in adult β-cells, we generated mice with tamoxifen-inducible partial or full deletion of STAT3 in β-cells and fed them a high-fat diet before analysis. Interestingly, β-cell heterozygous and homozygous STAT3-deficient mice showed glucose intolerance when fed a high-fat diet. Gene expression analysis with RNA sequencing showed that reduced expression of mitochondrial genes in STAT3 knocked down human EndoC-β1H cells, confirmed in FACS-purified β-cells from obese STAT3-deficient mice. Moreover, silencing of STAT3 impaired mitochondria activity in EndoC-β1H cells and human islets, suggesting a mechanism for STAT3-modulated β-cell function. Our study postulates STAT3 as a novel regulator of β-cell function in obesity. [ABSTRACT FROM AUTHOR]

Published
2021
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46. Supplementary_Table_4._GIP – Supplemental material for Gastrointestinal Hormones Manipulation to Counteract Metabolic Syndrome Using Duodenal Targeted Embolization

Author

Pop, Raoul, Kong, Seong-Ho, Langlois, Allan, Marchegiani, Francesco, Shlomovitz, Eran, Legnèr, András, Bietiger, William, Pinget, Michel, Beaujeux, Rémy, Mutter, Didier, Marescaux, Jacques, and Diana, Michele

Subjects
FOS: Materials engineering, FOS: Other engineering and technologies, FOS: Clinical medicine, 110323 Surgery, 110604 Sports Medicine, 91299 Materials Engineering not elsewhere classified, FOS: Health sciences, 110314 Orthopaedics, 99999 Engineering not elsewhere classified
Abstract

Supplemental material, Supplementary_Table_4._GIP for Gastrointestinal Hormones Manipulation to Counteract Metabolic Syndrome Using Duodenal Targeted Embolization by Raoul Pop, Seong-Ho Kong, Allan Langlois, Francesco Marchegiani, Eran Shlomovitz, András Legnèr, William Bietiger, Michel Pinget, Rémy Beaujeux, Didier Mutter, Jacques Marescaux and Michele Diana in Surgical Innovation

Published
2019
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47. Supplementary_Table_8._RNA_GLUT-2 – Supplemental material for Gastrointestinal Hormones Manipulation to Counteract Metabolic Syndrome Using Duodenal Targeted Embolization

Author

Pop, Raoul, Kong, Seong-Ho, Langlois, Allan, Marchegiani, Francesco, Shlomovitz, Eran, Legnèr, András, Bietiger, William, Pinget, Michel, Beaujeux, Rémy, Mutter, Didier, Marescaux, Jacques, and Diana, Michele

Subjects
FOS: Materials engineering, FOS: Other engineering and technologies, FOS: Clinical medicine, 110323 Surgery, 110604 Sports Medicine, 91299 Materials Engineering not elsewhere classified, FOS: Health sciences, 110314 Orthopaedics, 99999 Engineering not elsewhere classified
Abstract

Supplemental material, Supplementary_Table_8._RNA_GLUT-2 for Gastrointestinal Hormones Manipulation to Counteract Metabolic Syndrome Using Duodenal Targeted Embolization by Raoul Pop, Seong-Ho Kong, Allan Langlois, Francesco Marchegiani, Eran Shlomovitz, András Legnèr, William Bietiger, Michel Pinget, Rémy Beaujeux, Didier Mutter, Jacques Marescaux and Michele Diana in Surgical Innovation

Published
2019
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48. Supplementary_Table_7._RNA_SGLT-1 – Supplemental material for Gastrointestinal Hormones Manipulation to Counteract Metabolic Syndrome Using Duodenal Targeted Embolization

Author

Pop, Raoul, Kong, Seong-Ho, Langlois, Allan, Marchegiani, Francesco, Shlomovitz, Eran, Legnèr, András, Bietiger, William, Pinget, Michel, Beaujeux, Rémy, Mutter, Didier, Marescaux, Jacques, and Diana, Michele

Subjects
FOS: Materials engineering, FOS: Other engineering and technologies, FOS: Clinical medicine, 110323 Surgery, 110604 Sports Medicine, 91299 Materials Engineering not elsewhere classified, FOS: Health sciences, 110314 Orthopaedics, 99999 Engineering not elsewhere classified
Abstract

Supplemental material, Supplementary_Table_7._RNA_SGLT-1 for Gastrointestinal Hormones Manipulation to Counteract Metabolic Syndrome Using Duodenal Targeted Embolization by Raoul Pop, Seong-Ho Kong, Allan Langlois, Francesco Marchegiani, Eran Shlomovitz, András Legnèr, William Bietiger, Michel Pinget, Rémy Beaujeux, Didier Mutter, Jacques Marescaux and Michele Diana in Surgical Innovation

Published
2019
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