364 results on '"Pollard, Steven M."'
Search Results
2. Imaging extrachromosomal DNA (ecDNA) in cancer
3. RNA lipid nanoparticles as efficient in vivo CRISPR-Cas9 gene editing tool for therapeutic target validation in glioblastoma cancer stem cells
4. TAK1 inhibition leads to RIPK1-dependent apoptosis in immune-activated cancers
5. Autophagy supports PDGFRA-dependent brain tumor development by enhancing oncogenic signaling
6. Elevated FOXG1 in glioblastoma stem cells cooperates with Wnt/β-catenin to induce exit from quiescence
7. Engineering Genetic Predisposition in Human Neuroepithelial Stem Cells Recapitulates Medulloblastoma Tumorigenesis
8. Myeloid cell interferon secretion restricts Zika flavivirus infection of developing and malignant human neural progenitor cells
9. Author Correction: The white matter is a pro-differentiative niche for glioblastoma
10. Benchmarking brain organoid recapitulation of fetal corticogenesis
11. Regional identity of human neural stem cells determines oncogenic responses to histone H3.3 mutants
12. Simultaneous disruption of PRC2 and enhancer function underlies histone H3.3-K27M oncogenic activity in human hindbrain neural stem cells
13. Glioblastomas acquire myeloid-affiliated transcriptional programs via epigenetic immunoediting to elicit immune evasion
14. Post-translational modification of SOX family proteins: Key biochemical targets in cancer?
15. The white matter is a pro-differentiative niche for glioblastoma
16. LRIG1 is a gatekeeper to exit from quiescence in adult neural stem cells
17. Inositol treatment inhibits medulloblastoma through suppression of epigenetic-driven metabolic adaptation
18. Reprogramming of Fibroblasts to Oligodendrocyte Progenitor-like Cells Using CRISPR/Cas9-Based Synthetic Transcription Factors
19. EGFR amplification and EGFRvIII predict and participate in TAT-Cx43266–283 antitumor response in preclinical glioblastoma models.
20. Author Reply to Peer Reviews of CDK12/CDK13 inhibition disrupts a transcriptional program critical for glioblastoma survival
21. Genome Editing in Human Neural Stem and Progenitor Cells
22. Neural G0: a quiescent‐like state found in neuroepithelial‐derived cells and glioma
23. Challenges to curing primary brain tumours
24. Accelerating glioblastoma drug discovery: Convergence of patient-derived models, genome editing and phenotypic screening
25. Transcriptional and epigenetic regulatory mechanisms in glioblastoma stem cells
26. Contributors
27. Synthetic Biology, Stem Cells, and Regenerative Medicine
28. A novel ILK/STAT3 pathway controls plasticity in a neural stem cell model of glioblastoma.
29. The tumour ecology of quiescence: Niches across scales of complexity
30. Corrigendum: Hierarchical reactivation of transcription during mitosis-to-G1 transition by Brn2 and Ascl1 in neural stem cells
31. A novel ILK/STAT3 pathway controls plasticity in a neural stem cell model of glioblastoma
32. Supplementary Figure 5 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
33. Supplementary Figure 4 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
34. Supplementary Figure 1 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
35. Data from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
36. Supplementary Figure 7 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
37. Supplementary Table 1 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
38. Supplementary Figure 8 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
39. Supplementary Figure 2 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
40. Supplementary Figure 6 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
41. Supplementary Figure 9 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
42. Supplementary Figure 3 from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
43. Supplementary Methods from Cancer-Specific Requirement for BUB1B/BUBR1 in Human Brain Tumor Isolates and Genetically Transformed Cells
44. Genome-wide CRISPR-Cas9 Screens Reveal Loss of Redundancy between PKMYT1 and WEE1 in Glioblastoma Stem-like Cells
45. Glioblastoma Stem Cells Respond to Differentiation Cues but Fail to Undergo Commitment and Terminal Cell-Cycle Arrest
46. Oncogene expression from extrachromosomal DNA is driven by copy number amplification and does not require spatial clustering in glioblastoma stem cells
47. STEM-22. EFFECT OF A SRC INHIBITORY PEPTIDE BASED ON CONNEXIN43 ON NEURAL STEM CELLS WITH GLIOMA-DRIVER MUTATIONS
48. STAR: A Simple TAL Effector Assembly Reaction Using Isothermal Assembly
49. Reply to ‘Assembling the brain trust: the multidisciplinary imperative in neuro-oncology’
50. Lrig1 regulates the balance between proliferation and quiescence in glioblastoma stem cells
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