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1. A validated heart-specific model for splice-disrupting variants in childhood heart disease

2. Molecular Pathways and Animal Models of Ebstein’s Anomaly

3. Familial multiple discoid fibromas is linked to a locus on chromosome 5 including the FNIP1 gene

4. Identification of novel genetic risk factors of dilated cardiomyopathy: from canine to human

6. Biallelic variants in the calpain regulatory subunit CAPNS1 cause pulmonary arterial hypertension

7. Rare variants in KDR, encoding VEGF Receptor 2, are associated with tetralogy of Fallot

8. Germline variants in HEY2 functional domains lead to congenital heart defects and thoracic aortic aneurysms

9. Biallelic variants in POPDC2 cause a novel autosomal recessive syndrome presenting with cardiac conduction defects and variable hypertrophic cardiomyopathy

11. Common Genetic Variants Contribute to Risk of Transposition of the Great Arteries

13. Loss-of-function variants in myocardin cause congenital megabladder in humans and mice

14. A validated heart-specific model for splice-disrupting variants in childhood heart disease

15. TGFBR1 Variants Can Associate with Non-Syndromic Congenital Heart Disease without Aortopathy

17. Identification of novel genetic risk factors of dilated cardiomyopathy: from canine to human

18. Familial multiple discoid fibromas is linked to a locus on chromosome 5 including the FNIP1 gene

19. Identification of novel genetic risk factors of dilated cardiomyopathy: from canine to human

20. TGFBR1 Variants Can Associate with Non-Syndromic Congenital Heart Disease without Aortopathy

21. Common genetic variants improve risk stratification after the atrial switch operation for transposition of the great arteries

24. Whole Exome Sequencing Reveals the Major Genetic Contributors to Nonsyndromic Tetralogy of Fallot

26. Homozygous frameshift mutations in FAT1 cause a syndrome characterized by colobomatous-microphthalmia, ptosis, nephropathy and syndactyly

27. PRDM10 directs FLCN expression in a novel disorder overlapping with Birt–Hogg–Dubé syndrome and familial lipomatosis

28. No prominent role for complement C1-esterase inhibitor in Marfan syndrome mice

30. GMDS Intragenic Deletions Associate with Congenital Heart Disease including Ebstein Anomaly.

31. Erratum:Germline variants in HEY2 functional domains lead to congenital heart defects and thoracic aortic aneurysms(Genet Med (2021)23(103-110)(s41436020009394)(10.1038/s41436-020-00939-4))

32. Germline variants in HEY2 functional domains lead to congenital heart defects and thoracic aortic aneurysms

33. Germline variants in HEY2 functional domains lead to congenital heart defects and thoracic aortic aneurysms

36. Haploinsufficiency of TAB2 Causes Congenital Heart Defects in Humans

37. Correction to: Rare variants in KDR, encoding VEGF Receptor 2, are associated with tetralogy of Fallot (Genetics in Medicine, (2021), 23, 10, (1952-1960), 10.1038/s41436-021-01212-y)

40. Erratum: Integrative analysis of genomic variants reveals new associations of candidate haploinsufficient genes with congenital heart disease (PLoS Genetics (2021) 17:7 (e1009679) DOI: 10.1371/journal.pgen.1009679)

44. Correction: Integrative analysis of genomic variants reveals new associations of candidate haploinsufficient genes with congenital heart disease

45. Integrative analysis of genomic variants reveals new associations of candidate haploinsufficient genes with congenital heart disease

46. Terminal osseous dysplasia with pigmentary defects and cardiomyopathy caused by a novel FLNA variant

47. Common Genetic Variants Contribute to Risk of Transposition of the Great Arteries

48. Integrative analysis of genomic variants reveals new associations of candidate haploinsufficient genes with congenital heart disease

49. Terminal osseous dysplasia with pigmentary defects and cardiomyopathy caused by a novel FLNA variant

50. Terminal osseous dysplasia with pigmentary defects and cardiomyopathy caused by a novel FLNA variant

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