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1. Case Reports Presentations

Author

Mustafa Dinçer, Cemil Çelik, Elif Ecem Helvalı, Şahin Bodur, Emine Tuğçe Akçaer, Serdar Süleyman Can, Görkem Karakaş Uğurlu, Sümeyye İslamoğlu, Semra Ulusoy Kaymak, Yasemin Taştorun, Oğuz Peker, Ali Çayköylü, Zehra Ece Soğucak, Aslı Sürer Adanır, Esin Özatalay, Arif Önder, Abdurrahman Erdem Başaran, Ayşen Bingöl, Berhan Akdağ, Murat İlhan Atagün, Yakup Doğan, Öznur Bilaç, Canem Kavurma, Gülseren Taşkıran, Aybike Erdem, Zehra Başar Kocagöz, Adnan Özçetin, Ahmet Ataoğlu, Merve Çavdar Toraman, Özgen Özçelik, Hüseyin Kara, Talya Tomar, Buket Cinemre, Yusuf Tokgöz, Taner Öznur, Abdullah Bolu, Özcan Uzun, İkbal İnanlı, Deniz Altunova, Ali Metehan Çalışkan, İbrahim Eren, Tüba Şerife Elmas, Yasemin Gökçenoğlu, Saliha Çalışır, Ali Baran Tanrıkulu, Şenay Yıldız Bozdoğan, Fatma Şahin, Ceren Çamur, İbrahim Gündoğmuş, Abdulkadir Karagöz, Ayhan Algül, Ebru Doneray, Ipek Percinel Yazici, Kemal Utku Yazici, Aslı Adanır, Yetiş Işıldar, Ebru Sağlam, Ayhan Bilgiç, Betül Akbaş, Çağla Çelikkol, Seher Serez Öztürrk, Hilal Seven, Dudu Demiröz, Seda Özbek, İsmet Esra Çiçek, Fatma Coşkun, Ömer Faruk Akça, Doğa Sevinçok, Çağdaş Öykü Memiş, Burcu Çakaloz, Bilge Çetin İlhan, Tuba Şerife Elmas, Azra Sehure Yaşar, Nafiye Yağlı, Osman Ak, Recep Başaran, Mehmet Murat Balcı, Mehmet Murat Kuloğlu, İbrahim Taş, Sehure Azra Yaşar, Mustafa Çağrı Yıldız, Ebru Çiftçi, Hasan Ali Guler, Ali Kandeger, Dilara Guler, Serhat Turkoglu, Önder Küçük, Ferhat Yaylacı, Handan Özek Erkuran, Betül Kurtses Gürsoy, Seher Serez Öztürk, Berrin Ünal, Gizem Aral, Evrim Özkorumak Karagüzel, Demet Sağlam Aykut, Filiz Civil Arslan, Ezgi Karagöz, Neslihan Emir İnaloğlu, Necati Uzun, Mutlu Muhammed Özbek, Mustafa Tolga Tunagür, Bilge Doğan, Levent Sevinçok, Abdullah Akgün, Kamil Nahit Özmenler, Tayfun Kara, İsmail Akaltun, Ayşe Erdoğan Kaya, Esra Yazıcı, Muhammed Nurullah Sezer, Çağlar Turan, Engin Sert, Yusuf Ezel Yıldırım, Pınar Çetinay Aydın, Sevilay Kunt, Tonguç Demir Berkol, Erman Esnafoglu, Öznur Adıgüzel, Serhat Tunc, Hamit Serdar Basbug, Selçuk Dalyan, Şermin Bilgen Ulgar, Hamza Ayaydın, Sema Bozbey, Merve Yazıcı, Çiğdem Yektaş, Enes Sarıgedik, Mehmet Asoğlu, Betül Uyar Ekmen, Hasan Akçalı, Cuma Taş, Cansu Mercan Işık, Belde Demirci, Seda Aybüke Sarı, Ayla Uzun Çiçek, Ezgi Eynalli, Ozge Metin, Perihan Cam Ray, Aysegul Yolga Tahiroglu, Gonca Gul Celik, Tuğçe Akçaer, Elif Merve Kurt, İbrahim Özkan Göncüoğlu, İsmail Ak, Merve Tsakir Chasan, Lut Tamam, Soner Çakmak, Mehmet Emin Demirkol, Erdem Örnek, Ayşe Sakallı Kani, Volkan Topçuoğlu, Rukiye Çolak Sivri, Nihal Yurteri Çetin, Merve Sertdemir, Emre Ürer, Gökçen İlçioğlu Ekici, Birim Günay Kılıç, Keziban Turgut, Canan Kuygun Karci, Ayse Avci, Azra Yaşar, Sümeyra Elif Kaplan, Hazal Muhsinoglu, Alper Zıblak, Ayşe Nur İnci Kenar, Cantekin Can, Canan Kuygun Karcı, Gamze Kutlu, Çağlar Soykan, Cansu Pınar Şen, Mehmet Fatih Ceylan, Selma Tural Hesapçıoğlu, Özden Şükran Öneri, Abdullah Karataş, Hatice Altun, Umut Karaaslan, Nurdan Kasar, Nilfer Şahin, Damla Balkan, Aslıhan Okan İbiloğlu, Abdullah Atli, Rabia Erdogan, Esra Yazici, Tugba Mutu, Ozlem Akcay Ciner, Ali Savas Cilli, Atila Erol, Esra Porgalı Zayman, Cengiz Darılmaz, İsmail Reyhani, Rıfat Karlıdağ, Kübra Yıldırım, Yunus Emre Dönmez, Serdar Karatoprak, Özlem Özcan, Ali Hakan Öztürk, Özden Şükran Üneri, Perihan Turhan Gürbüz, Mustafa Uğurlu, Özlem Doğan, Tahir Kurtuluş Yoldaş, Nuran Bilgen, Vesile Altınyazar, Muhammed Mutlu Özbek, Ebru Ulu, Esra Demirci, Sevgi Özmen, Ümit Haluk Yeşilkaya, Ozge Sahmelikoglu Onur, Omer Akay, Yasin Hasan Balcioglu, Fatih Oncu, Çiğdem Toklu Yalvaç, Ümit Işık, Erol Erkan, Mehmet Hamdi Örüm, Tezan Bildik, Mahmut Zabit Kara, Helin Yılmaz, Hasan Akın Tahıllıoğlu, Aysun Kalenderoğlu, Oğuzhan Bekir Eğilmez, Murat Eren Özen, Yaşar Kapıcı, Ümit Kılıçoğlu, Murad Atmaca, Gulgaz Karimova, Asiye Arıcı, Feyza Hatice Sevgen, Zehra Alğan, Fadime Dalboy, Mehmet Ak, Faruk Uğuz, Kübra Kılınç, Fatih Hilmi Çetin, Serhat Türkoğlu, Semra Yılmaz, İbrahim İbiloğlu, Mustafa Özkan, Osman Bertizlioğlu, Ece Merve Yazar, Ahmet Özercan, Mehmet Kemal Kuşçu, Muhammet Akbolat, Gazanfer Ekinci, İpek Midi, Fatih Mücahit Harmankaya, Adem Aydın, Neslihan Yazar, Busra Bahar Ataoğlu, Neşe Yorguner Küpeli, Necati Serkut Bulut, Kaan Kora, Cihad Yükselir, Serkan Zincir, Dilşad Yıldız Miniksar, Pelin Çon Bayhan, Faruk Pirinççioğlu, İsmail Karka, Meltem Göbelek, Öznur Akıl, Sümeyra Güngören, Deniz Deniz Özturan, Zeynep Bebek Yılmaz, Derya Deniz Kürekçi, Aykut Özturan, Mihriban Ünay, Hasan Mervan Aytaç, Nazan Aydın, Can Tuncer, Volkan Seneger, Burcu Bakar Kahraman, Selma Hilal Avcı, Hasan Turan Karatepe, Mehmet Arslan, Sıla Çalışkan, Yusuf Çokünlü, Zeynep Yücehan, Seher Serez Öztrük, Hatice Yardım Özayhan, Özlem Karakaya, Gökçen Turan, Burak Elbeyli Ahmet, Safiye Bahar Ölmez, Merve Çavdar, Evrim Aktepe, Pınar Aydoğan Avşar, Yakup Erdoğan, Özlem Beğinoğlu, Rümeysa Yeni Elbay, Hayriye Hızarcıoğlu Gülşen, Arzu Yılmaz, Yasemin İmrek, Mesut Sari, Büşra Pala, Yusuf Öztürk, Güler Göl, Mehmet Akif Cansız, Uğur Savcı, Berna Gündüz Çıtır, Hatice Aksu, Sema Çam Salihoğlu, Nurhak Çağatay Birer, Güler Özkula, Ercan Altınörs, Fatih Baz, Mesut Yıldız, Leyla Bozatlı, Hasan Cem Aykutlu, Işık Görker, Oğuzhan Sapdüzen, Çiçek Hocaoğlu, Alphan Anak, Mesut Yılmaz, Yeliz Doymaz, Ece Ayyıldız, Mehmet Baltacıoğlu, Selvi Ceran Kayıpmaz, Ali Ercan Altınöz, Arzu Oğuz, Sema Çam Salihoglu, Hacer Gizem Gerçek, Mehmet Ayhan Cöngöloğlu, Emre Subas, Selin Alkan, Suat Yalcin, Suleyman Donmezler, Sevilay Umut Kilinc, Burcu Hamurisci Yalcin, Guliz Ozgen, Ahmet Turkcan, Ayse Ceren Kaypak, Nese Yorguner Kupeli, Ali İnaltekin, İbrahim Yağcı, Yüksel Kıvrak, Emine Füsun Akyüz Çim, Leyla Delikanli, Mustafa Tuncturk, Oguz Bilal Karakus, Ali Guven Kılıcoglu, Gul Karacetin, Merve Okuyan, Halil İbrahim İvelik, Burak Okumuş, Rukiye Tekdemir, Memduha Aydın, Nihal Taştekin, Betül Kırşavoğlu, Murat Yalçın, Sibel Ayvaz, Ayşe Gülşah Kırımlı, Ayşegül Taşdelen Kul, Huda Pasli, Mine Ozkan, and Ferda Volkan

Subjects
Neurosciences. Biological psychiatry. Neuropsychiatry, RC321-571
Published
2018
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2. Does coupling to ADP ribosylation factor 6 explain differences between muscarinic and other receptors in interaction with β-adrenoceptor-mediated smooth muscle relaxation?

Author

Martin Michel and BETUL RABIA ERDOGAN

Subjects
Pharmacology, ADP-Ribosylation Factor 6, Muscle Relaxation, General Medicine, Adrenergic beta-Agonists, Muscarinic Agonists, Muscle Contraction, Receptors, Adrenergic
Abstract

Numerous studies in airways, ileum, and urinary bladder have demonstrated that relaxation by β-adrenoceptor agonists has lower potency and/or efficacy when contraction was elicited by muscarinic receptor agonists as compared to other G-protein-coupled receptors, KCl, or basal tone, but the molecular mechanisms behind this relative resistance remain unclear. A paper by Huang et al. in this issue demonstrates that NAV2729, an inhibitor of ADP ribosylation factor 6, inhibits contraction of isolated blood vessels elicited by muscarinic receptor agonists, but not by α1-adrenoceptor agonists or KCl. Against this background, we discuss the role of ADP ribosylation factor 6 in cellular responses to G-protein-coupled receptor stimulation. While ADP ribosylation factor 6 apparently is the only promising molecular explanation for the relative resistance of smooth muscle contraction elicited by muscarinic agonists, the existing data are insufficient for a robust conclusion.

Published
2022
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3. The Effects of Low-Dose Empagliflozin on Cardiac Function and Β-Adrenoceptor Responses in a Rat Model of Streptozotocin-Induced Diabetes

Author

Zeynep Elif Yesilyurt, Irem Karaomerlioglu, Betul Rabia Erdogan, Gaye Ozturk, Martin C. Michel, and Ebru Arioglu-Inan

Subjects
cardiology
Abstract

Diabetes mellitus leads to cardiovascular complications including impaired cardiac β-adrenoceptor (β-AR) function. Sodium-glucose cotransporter-2 (SGLT2) inhibitors, such as empagliflozin (EMPA) improve outcomes in heart failure patients and animal models thereof. Therefore, we have investigated the effects of EMPA on in vivo cardiac function (pressure-volume loop analysis) and β-AR-mediated contractile responses (papillary strips) in streptozotocin (STZ; 40 mg/kg, i.p.)-induced diabes in male Sprague Dawley rats (control, EMPA-treated control, diabetic, EMPA-treated diabetic) in a design reflecting late-onset treatment. 13-16 weeks after STZ injection treatment with a low dose of EMPA (10 mg/kg/day, daily oral gavage) or vehicle was administered for another 8 weeks. EMPA did not change cardiac function in control rats. Diabetic rats had a reduced heart rate, cardiac output, stroke work, rate of contration and rate of relaxation and increased isovolumic relaxation, whereas in vitro responses were not markedly attenuated. Treatment with EMPA showed a trend for improvement of some but not all parameters. Our results indicate that low dose EMPA treatment had limited effects on cardiac impairment despite reducing blood glucose when initiated after diabetes is manifest. Future studies with a higher dose and greater sample sizes could help to clarify the possible benefits of EMPA on the diabetic heart.

Published
2022
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4. Iron in infectious diseases friend or foe?: The role of gut microbiota

Author

Zinnet Şevval AKSOYALP, AYBALA TEMEL, and BETUL RABIA ERDOGAN

Subjects
Inorganic Chemistry, Iron Overload, Iron, Humans, Molecular Medicine, Communicable Diseases, Biochemistry
Abstract

Iron is a trace element involved in metabolic functions for all organisms, from microorganisms to mammalians. Iron deficiency is a prevalent health problem that affects billions of people worldwide, and iron overload could have some hazardous effect. The complex microbial community in the human body, also called microbiota, influences the host immune defence against infections. An imbalance in gut microbiota, dysbiosis, changes the host's susceptibility to infections by regulating the immune system. In recent years, the number of studies on the relationship between infectious diseases and microbiota has increased. Gut microbiota is affected by different parameters, including mode of delivery, hygiene habits, diet, drugs, and plasma iron levels during the lifetime. Gut microbiota may influence iron levels in the body, and iron overload and deficiency can also affect gut microbiota composition. Novel researches on microbiota shed light on the fact that the bidirectional interactions between gut microbiota and iron play a role in the pathogenesis of many diseases, especially infections. A better understanding of these interactions may help us to comprehend the pathogenesis of many infectious and metabolic diseases affecting people worldwide and following the development of more effective preventive and/or therapeutic strategies. In this review, we aimed to present the iron-mediated host-gut microbiota interactions, susceptibility to bacterial infections, and iron-targeted therapy approaches for infections.

Published
2023
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5. Urinary Bladder Weight and Function in a Rat Model of Mild Hyperglycemia and Its Treatment With Dapagliflozin

Author

Betiil Rabia Erdogan, Ayhanim Elif Muderrisoglu, Irem Karaomerlioglu, Zeynep Elif Yesilyurt, Ebru Arioglu-Inan, and Martin C. Michel

Subjects
0301 basic medicine, medicine.medical_specialty, Type 2 diabetes, Muscle hypertrophy, Contractility, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, relaxation, Diabetes mellitus, Internal medicine, Medicine, Pharmacology (medical), Dapagliflozin, Fenoterol, Original Research, Pharmacology, Urinary bladder, diabetes, business.industry, lcsh:RM1-950, contraction, medicine.disease, Streptozotocin, 030104 developmental biology, medicine.anatomical_structure, Endocrinology, lcsh:Therapeutics. Pharmacology, chemistry, 030220 oncology & carcinogenesis, hyperglycemia, business, hypertrophy, urinary bladder, medicine.drug
Abstract

Hypertrophy and dysfunction of the urinary bladder are consistently observed in animal models of type 1 and less consistently in those of type 2 diabetes. We have tested the effects of mild hyperglycemia (n = 10 per group) in a randomized, blinded study and, in a blinded pilot study, of type 2 diabetes (n = 6 per group) and its treatment with dapagliflozin (1 mg/kg per day) on weight, contraction, and relaxation of the rat bladder. Based on a combination of high-fat diet and a low dose of streptozotocin, animals in the main study reached a mean peak blood glucose level of about 300 mg/dl, which declined to 205 mg/dl at study end. This was associated with a small, if any, increase in bladder weight. In a pooled analysis of all animals of the main and the pilot study, we detected a correlation of moderate strength between blood glucose and bladder weight (r(2) = 0.2013; P = 0.0003 for Pearson correlation coefficient). Neither the main nor the pilot study found evidence for an altered contractility (responses to carbachol or KCl) or relaxation (responses to isoprenaline, fenoterol, CL 316,243, or forskolin). Treatment with dapagliflozin in the absence of hyperglycemia increased diuresis in the main study by 43% relative to control and increased bladder weight by 15% in the pooled groups of both studies (post hoc analysis). We conclude that mild hyperglycemia has no major effects on bladder hypertrophy or function.

Published
2019
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