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2. Airway mucin MUC5AC and MUC5B concentrations and the initiation and progression of chronic obstructive pulmonary disease: an analysis of the SPIROMICS cohort

3. Airway Mucin Concentration as a Marker of Chronic Bronchitis

4. Proximal and Distal Bronchioles Contribute to the Pathogenesis of Non-Cystic Fibrosis Bronchiectasis (NCFB)

5. Airway mucins promote immunopathology in virus-exacerbated chronic obstructive pulmonary disease

6. Proximal and Distal Bronchioles Contribute to the Pathogenesis of Non–Cystic Fibrosis Bronchiectasis.

7. Genetic regulators of sputum mucin concentration and their associations with COPD phenotypes

9. Genetic regulators of sputum mucin concentration and their associations with COPD phenotypes

12. Genetic Regulators of Sputum Mucin Concentration and Their Associations with COPD Phenotypes

13. Cystic Fibrosis Airway Mucus Hyperconcentration Produces a Vicious Cycle of Mucin, Pathogen, and Inflammatory Interactions that Promotes Disease Persistence

15. Hereditary Mucin Deficiency Caused by Biallelic Loss of Function of MUC5B

16. Genetic regulators of sputum mucin concentration and their associations with COPD phenotypes.

17. Hereditary Mucin Deficiency Caused by Biallelic Loss of Function of MUC5B

18. Mucus concentration–dependent biophysical abnormalities unify submucosal gland and superficial airway dysfunction in cystic fibrosis

19. Hyperconcentrated Mucus Unifies Submucosal Gland and Superficial Airway Dysfunction in Cystic Fibrosis

20. Airway Mucus Hyperconcentration in Non–Cystic Fibrosis Bronchiectasis

21. Endotracheal tube mucus as a source of airway mucus for rheological study

22. Localization of Secretory Mucins MUC5AC and MUC5B in Normal/Healthy Human Airways

25. Antagonistic Effect of a Salivary Proline-Rich Peptide on the Cytosolic Ca2+ Mobilization Induced by Progesterone in Oral Squamous Cancer Cells.

26. Structural studies and SH3 domain binding properties of a human antiviral salivary proline-rich peptide

27. Allergic asthma is distinguished by sensitivity of allergen-specific CD4+T cells and airway structural cells to type 2 inflammation

28. Structural studies and SH3 domain binding properties of a human antiviral salivary proline-rich peptide

29. Antagonistic Effect of a Salivary Proline-Rich Peptide on the Cytosolic Ca2+ Mobilization Induced by Progesterone in Oral Squamous Cancer Cells

32. Allergic asthma is distinguished by sensitivity of allergen-specific CD4+ T cells and airway structural cells to type 2 inflammation.

33. Antagonistic Effect of a Salivary Proline-Rich Peptide on the Cytosolic Ca2+ Mobilization Induced by Progesterone in Oral Squamous Cancer Cells.

34. Airway Mucin Concentration as a Marker of Chronic Bronchitis.

35. Dysregulated Airway Host Defense in Hyper IgE Syndrome due to STAT3 Mutations.

36. Endotracheal tube mucus as a source of airway mucus for rheological study.

37. The innate immune properties of airway mucosal surfaces are regulated by dynamic interactions between mucins and interacting proteins: the mucin interactome.

38. Allergic asthma is distinguished by sensitivity of allergen-specific CD4+ T cells and airway structural cells to type 2 inflammation.

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