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2. Native-state proteomics of Parvalbumin interneurons identifies unique molecular signatures and vulnerabilities to early Alzheimer’s pathology

4. Integrative proteomics identifies a conserved Aβ amyloid responsome, novel plaque proteins, and pathology modifiers in Alzheimer’s disease

6. Cerebrovascular events and outcomes in hospitalized patients with COVID-19: The SVIN COVID-19 Multinational Registry.

8. Unique molecular characteristics and microglial origin of Kv1.3 channel–positive brain myeloid cells in Alzheimer’s disease

9. Telemedicine impact on post-stroke outpatient follow-up in an academic healthcare network during the COVID-19 pandemic

10. Kv1.3 modulates neuroinflammation and neurodegeneration in Parkinson's disease.

15. Kv1.3 inhibition as a potential microglia-targeted therapy for Alzheimer’s disease: preclinical proof of concept

18. Development and validation of a SCORing systEm for pre-thrombectomy diagnosis of IntraCranial Atherosclerotic Disease (Score-ICAD)

19. Proteomic changes in the human cerebrovasculature in Alzheimer's disease and related tauopathies linked to peripheral biomarkers in plasma and cerebrospinal fluid

21. Efficacy and safety of nerinetide for the treatment of acute ischaemic stroke (ESCAPE-NA1): a multicentre, double-blind, randomised controlled trial

22. Abstract 87: Predictive Scoring System for Pre-Thrombectomy Diagnosis of Intracranial Atherosclerotic Disease LVOS

23. Abstract WP313: Towards Molecular Profiling of Transient Ischemic Attack Mimics for Triage

25. Large-scale proteomic analysis of Alzheimer’s disease brain and cerebrospinal fluid reveals early changes in energy metabolism associated with microglia and astrocyte activation

27. Potassium Channel Kv1.3 Is Highly Expressed by Microglia in Human Alzheimer's Disease

28. Proximity labeling proteomics reveals Kv1.3 potassium channel immune interactors in microglia.

30. In vivo TurboID proximity labeling to capture astrocyte proteomic and transcriptomic signatures under homeostatic and neuroinflammatory conditions

34. Entorhinal cortex vulnerability to human APP expression promotes hyperexcitability and tau pathology

35. Entorhinal cortex vulnerability to human APP expression promotes hyperexcitability and tau pathology

36. Abstract 223: Pre‐Thrombectomy Diagnosis of Large Vessel Occlusion with Underlying Intracranial Atherosclerotic Disease

44. Native-state proteomics of Parvalbumin interneurons identifies novel molecular signatures and metabolic vulnerabilities to early Alzheimer's disease pathology

45. APOE expression and secretion are modulated by mitochondrial dysfunction

47. Corrigendum to Cincinnati Prehospital Stroke Scale Implementation of an Urban County Severity-Based Stroke Triage Protocol: Impact and Outcomes on a Comprehensive Stroke Center Journal of Stroke and Cerebrovascular Diseases Volume 31, Issue 8, August 2022, 106575

48. Profiling the neuroimmune cascade in 3xTg mice exposed to successive mild traumatic brain injuries

49. Central Nervous System and Peripheral Nerves

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