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4. Publisher Correction: MethCORR modelling of methylomes from formalin-fixed paraffin-embedded tissue enables characterization and prognostication of colorectal cancer

Author

Mattesen, Trine B., Rasmussen, Mads H., Sandoval, Juan, Ongen, Halit, Árnadóttir, Sigrid S., Gladov, Josephine, Martinez-Cardus, Anna, de Moura, Manuel Castro, Madsen, Anders H., Laurberg, Søren, Dermitzakis, Emmanouil T., Esteller, Manel, Andersen, Claus L., and Bramsen, Jesper B.

Published
2020
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6. Supplementary Figure 4 from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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7. Supplementary Figure 2 from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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8. Supplementary Figure 3 from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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9. Supplementary Table 1 from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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10. Supplementary Table 3 from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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11. Data from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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12. Supplementary Table 2 from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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13. Supplementary information from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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14. Supplementary Figure 1 from Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., primary, Laurberg, Jens R., primary, Boysen, Anders T., primary, Bramsen, Jesper B., primary, Primdal-Bengtson, Bjarke, primary, Hendrix, An, primary, Lamy, Philippe, primary, Dagnaes-Hansen, Frederik, primary, Rasmussen, Mads H., primary, Bui, Khan H., primary, Fristrup, Niels, primary, Christensen, Erik I., primary, Nordentoft, Iver, primary, Morth, Jens P., primary, Jensen, Jørgen B., primary, Pedersen, Jakob S., primary, Beck, Martin, primary, Theodorescu, Dan, primary, Borre, Michael, primary, Howard, Kenneth A., primary, Dyrskjøt, Lars, primary, and Ørntoft, Torben Falck, primary

Published
2023
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15. Additional file 1 of DREAMS: deep read-level error model for sequencing data applied to low-frequency variant calling and circulating tumor DNA detection

Author

Christensen, Mikkel H., Drue, Simon O., Rasmussen, Mads H., Frydendahl, Amanda, Lyskjær, Iben, Demuth, Christina, Nors, Jesper, Gotschalck, Kåre A., Iversen, Lene H., Andersen, Claus L., and Pedersen, Jakob Skou

Abstract

Additional file 1. Supplementary material, Supplementary methods, tables, and figures [12, 16, 24, 33, 35–43].

Published
2023
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17. DREAMS: Deep Read-level Error Model for Sequencing data applied to low-frequency variant calling and circulating tumor DNA detection

Author

Christensen, Mikkel H., primary, Drue, Simon, additional, Rasmussen, Mads H., additional, Frydendahl, Amanda, additional, Lyskjær, Iben, additional, Demuth, Christina, additional, Nors, Jesper, additional, Gotschalck, Kåre A., additional, Iversen, Lene H., additional, Andersen, Claus L., additional, and Pedersen, Jakob Skou, additional

Published
2022
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18. A genetically inducible porcine model of intestinal cancer

Author

Callesen, Morten M., Árnadóttir, Sigrid S., Lyskjær, Iben, Ørntoft, Mai‐Britt W., Høyer, Søren, Dagnæs‐Hansen, Frederik, Liu, Ying, Li, Rong, Callesen, Henrik, Rasmussen, Mads H., Berthelsen, Martin F., Thomsen, Martin K., Schweiger, Pawel J., Jensen, Kim B., Laurberg, Søren, Ørntoft, Torben F., Elverløv‐Jakobsen, Jannik E., and Andersen, Claus L.

Published
2017
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20. Putative cis-regulatory drivers in colorectal cancer

Author

Ongen, Halit, Andersen, Claus L., Bramsen, Jesper B., Oster, Bodil, Rasmussen, Mads H., Ferreira, Pedro G., Sandoval, Juan, Vidal, Enrique, Whiffin, Nicola, Planchon, Alexandra, Padioleau, Ismael, Bielser, Deborah, Romano, Luciana, Tomlinson, Ian, Houlston, Richard S., Esteller, Manel, Orntoft, Torben F., and Dermitzakis, Emmanouil T.

Subjects
Oncology, Experimental, Gene expression -- Research, Genetic research, Quantitative trait loci -- Identification and classification, Colorectal cancer -- Development and progression -- Genetic aspects, Cancer -- Research, Environmental issues, Science and technology, Zoology and wildlife conservation
Abstract

The cis-regulatory effects responsible for cancer development have not been as extensively studied as the perturbations of the protein coding genome in tumorigenesis (1,2). To better characterize colorectal cancer (CRC) development we conducted an RNA-sequencing experiment of 103 matched tumour and normal colon mucosa samples from Danish CRC patients, 90 of which were germline-genotyped. By investigating allele-specific expression (ASE) we show that the germline genotypes remain important determinants of allelic gene expression in tumours. Using the changes in ASE in matched pairs of samples we discover 71 genes with excess of somatic cis-regulatory effects in CRC, suggesting a cancer driver role. We correlate genotypes and gene expression to identify expression quantitative trait loci (eQTLs) and find 1,693 and 948 eQTLs in normal samples and tumours, respectively. We estimate that 36% of the tumour eQTLs are exclusive to CRC and show that this specificity is partially driven by increased expression of specific transcription factors and changes in methylation patterns. We show that tumour-specific eQTLs are more enriched for low CRC genome-wide association study (GWAS) P values than shared eQTLs, which suggests that some of the GWAS variants are tumour specific regulatory variants. Importantly, tumour-specific eQTL genes also accumulate more somatic mutations when compared to the shared eQTL genes, raising the possibility that they constitute germline-derived cancer regulatory drivers. Collectively the integration of genome and the transcriptome reveals a substantial number of putative somatic and germline cis-regulatory cancer changes that may have a role in tumorigenesis., The non-coding genome has so far been overlooked in the search for drivers in cancer, except for some isolated examples (1,2). The genome contains regulatory non-coding germline variation affecting gene [...]

Published
2014

21. Establishment and characterization of models of chemotherapy resistance in colorectal cancer: Towards a predictive signature of chemoresistance

Author

Jensen, Niels F., Stenvang, Jan, Beck, Mette K., Hanáková, Barbora, Belling, Kirstine C., Do, Khoa N., Viuff, Birgitte, Nygård, Sune B., Gupta, Ramneek, Rasmussen, Mads H., Tarpgaard, Line S., Hansen, Tine P., Budinská, Eva, Pfeiffer, Per, Bosman, Fred, Tejpar, Sabine, Roth, Arnaud, Delorenzi, Mauro, Andersen, Claus L., Rmer, Maria U., Brünner, Nils, and Moreira, José M.A.

Published
2015
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22. Performance characteristics of thromboelastometry assays using incompletely filled and prolonged stored samples

Author

Paarup, Helene M., Rasmussen, Mads H., Yazer, Mark H., and Sprogøe, Ulrik

Abstract

BACKGROUND: Thromboelastometry (TEM) is often used to guide transfusion therapy in patients with massive bleeding. The effect of testing incompletely filled samples and those stored for a prolonged time at 4°C was investigated. METHODS: Whole blood samples were collected from 15 healthy blood donors and were pooled according to ABO group. From these pools, aliquots were taken and diluted to produce final whole blood:citrate buffer ratios ranging from 90:10 (fully filled sample) to 40:60 (extremely under filled samples). These samples were then tested by EXTEM, INTEM, and FIBTEM on calibrated ROTEM delta machines. Separately, the four samples at 90:10 dilution were kept at 4°C for 16-20 hours and then retested on the ROTEM machines. RESULTS: All of the samples at the 90:10 and 80:20 (half-filled sample) whole blood:citrate buffer dilutions demonstrated ROTEM parameters within their respective reference ranges, although the samples from the 80:20 dilution tended to demonstrate slightly longer or slower times, depending on each ROTEM parameter, compared to the completely filled samples. All of the samples with more dilute whole blood to citrate buffer ratios (i.e., 70:30 to 40:60) yielded abnormal TEM results. The TEM results for the 90:10 dilution samples exposed to 16-20 hours of storage at 4°C were within the reference intervals. CONCLUSION: Completely and half-filled samples, and completely filled samples after prolonged cold storage, produced normal ROTEM results. Tubes that are less than half-filled should not be used for ROTEM testing.

Published
2020

23. MiRNA-362-3p induces cell cycle arrest through targeting of E2F1, USF2 and PTPN1 and is associated with recurrence of colorectal cancer

Author

Christensen, Lise Lotte, Tobiasen, Heidi, Holm, Anja, Schepeler, Troels, Ostenfeld, Marie S, Thorsen, Kasper, Rasmussen, Mads H., Birkenkamp-Demtroeder, Karin, Sieber, Oliver M., Gibbs, Peter, Lubinski, Jan, Lamy, Philippe, Laurberg, Sren, ster, Bodil, Hansen, Kristian Q., Hagemann-Madsen, Rikke, Byskov, Kristina, rntoft, Torben F., and Andersen, Claus L.

Published
2013
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26. Abstract 466: A novel DNA methylation-based approach for molecular subtyping and improved prognostication of colorectal cancer using formalin-fixed and paraffin-embedded tissue

Author

Mattesen, Trine B., primary, Rasmussen, Mads H., additional, Sandoval, Juan, additional, Ongen, Halit, additional, Árnadóttir, Sigrid S., additional, Madsen, Anders H., additional, Laurberg, Søren, additional, Dermitzakis, Emmanouil T., additional, Esteller, Manel, additional, Andersen, Claus L., additional, and Bramsen, Jesper B., additional

Published
2019
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27. Tumor-specific usage of alternative transcription start sites in colorectal cancer identified by genome-wide exon array analysis

Author

Laurila Kirsti, Mansilla Francisco, Eller Asger, Pedersen Jakob, Lamy Philippe, Hansen Kristian Q, Wang Kai, Vang Søren, Rasmussen Mads H, Øster Bodil, Schepeler Troels, Thorsen Kasper, Wiuf Carsten, Laurberg Søren, Dyrskjøt Lars, Ørntoft Torben F, and Andersen Claus L

Subjects
Biotechnology, TP248.13-248.65, Genetics, QH426-470
Abstract

Abstract Background Approximately half of all human genes use alternative transcription start sites (TSSs) to control mRNA levels and broaden the transcriptional output in healthy tissues. Aberrant expression patterns promoting carcinogenesis, however, may arise from alternative promoter usage. Results By profiling 108 colorectal samples using exon arrays, we identified nine genes (TCF12, OSBPL1A, TRAK1, ANK3, CHEK1, UGP2, LMO7, ACSL5, and SCIN) showing tumor-specific alternative TSS usage in both adenoma and cancer samples relative to normal mucosa. Analysis of independent exon array data sets corroborated these findings. Additionally, we confirmed the observed patterns for selected mRNAs using quantitative real-time reverse-transcription PCR. Interestingly, for some of the genes, the tumor-specific TSS usage was not restricted to colorectal cancer. A comprehensive survey of the nine genes in lung, bladder, liver, prostate, gastric, and brain cancer revealed significantly altered mRNA isoform ratios for CHEK1, OSBPL1A, and TCF12 in a subset of these cancer types. To identify the mechanism responsible for the shift in alternative TSS usage, we antagonized the Wnt-signaling pathway in DLD1 and Ls174T colorectal cancer cell lines, which remarkably led to a shift in the preferred TSS for both OSBPL1A and TRAK1. This indicated a regulatory role of the Wnt pathway in selecting TSS, possibly also involving TP53 and SOX9, as their transcription binding sites were enriched in the promoters of the tumor preferred isoforms together with their mRNA levels being increased in tumor samples. Finally, to evaluate the prognostic impact of the altered TSS usage, immunohistochemistry was used to show deregulation of the total protein levels of both TCF12 and OSBPL1A, corresponding to the mRNA levels observed. Furthermore, the level of nuclear TCF12 had a significant correlation to progression free survival in a cohort of 248 stage II colorectal cancer samples. Conclusions Alternative TSS usage in colorectal adenoma and cancer samples has been shown for nine genes, and OSBPL1A and TRAK1 were found to be regulated in vitro by Wnt signaling. TCF12 protein expression was upregulated in cancer samples and correlated with progression free survival.

Published
2011
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28. Evaluation of two commercial global miRNA expression profiling platforms for detection of less abundant miRNAs

Author

Ørntoft Torben F, Dyrskjøt Lars, Ostenfeld Marie S, Rasmussen Mads H, Lamy Philippe, Jensen Steffen G, and Andersen Claus L

Subjects
Biotechnology, TP248.13-248.65, Genetics, QH426-470
Abstract

Abstract Background microRNAs (miRNA) are short, endogenous transcripts that negatively regulate the expression of specific mRNA targets. miRNAs are found both in tissues and body fluids such as plasma. A major perspective for the use of miRNAs in the clinical setting is as diagnostic plasma markers for neoplasia. While miRNAs are abundant in tissues, they are often scarce in plasma. For quantification of miRNA in plasma it is therefore of importance to use a platform with high sensitivity and linear performance in the low concentration range. This motivated us to evaluate the performance of three commonly used commercial miRNA quantification platforms: GeneChip miRNA 2.0 Array, miRCURY Ready-to-Use PCR, Human panel I+II V1.M, and TaqMan Human MicroRNA Array v3.0. Results Using synthetic miRNA samples and plasma RNA samples spiked with different ratios of 174 synthetic miRNAs we assessed the performance characteristics reproducibility, recovery, specificity, sensitivity and linearity. It was found that while the qRT-PCR based platforms were sufficiently sensitive to reproducibly detect miRNAs at the abundance levels found in human plasma, the array based platform was not. At high miRNA levels both qRT-PCR based platforms performed well in terms of specificity, reproducibility and recovery. At low miRNA levels, as in plasma, the miRCURY platform showed better sensitivity and linearity than the TaqMan platform. Conclusion For profiling clinical samples with low miRNA abundance, such as plasma samples, the miRCURY platform with its better sensitivity and linearity would probably be superior.

Published
2011
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29. Abstract 1590: Personalized circulating tumor DNA analysis to monitor colorectal cancer

Author

Reinert, Thomas, primary, Henriksen, Tenna V., additional, Rasmussen, Mads H., additional, Sethi, Himanshu, additional, Sharma, Shruti, additional, Wu, Hsin-Ta, additional, Hafez, Dina, additional, Natarajan, Prashanthi, additional, Dashner, Scott, additional, Balcioglu, Mustafa, additional, Nguyen, Ann, additional, Renner, Derrick, additional, Zimmermann, Bernhard, additional, Iversen, Lene H-, additional, Madsen, Mogens R., additional, Lin, Cheng-Ho Jimmy, additional, and Andersen, Claus L., additional

Published
2018
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33. Abstract 2927: miR-625-3p regulates oxaliplatin resistance by targeting MAP2K6-p38 signalling in human colorectal adenocarcinoma cell

Author

Rasmussen, Mads H., primary, Lyskjær, Iben, additional, Jersie-Christensen, Rosa R., additional, Tarpgaard, Line S., additional, Primdal-Bengtson, Bjarke, additional, Nielsen, Morten M., additional, Pedersen, Jakob S., additional, Hansen, Tine P., additional, Hansen, Flemming, additional, Olsen, Jesper V., additional, Pfeiffer, Per, additional, Ørntoft, Torben F., additional, and Andersen, Claus L., additional

Published
2016
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34. Cellular Disposal of miR23b by RAB27-Dependent Exosome Release Is Linked to Acquisition of Metastatic Properties

Author

Ostenfeld, Marie Stampe, primary, Jeppesen, Dennis K., additional, Laurberg, Jens R., additional, Boysen, Anders T., additional, Bramsen, Jesper B., additional, Primdal-Bengtson, Bjarke, additional, Hendrix, An, additional, Lamy, Philippe, additional, Dagnaes-Hansen, Frederik, additional, Rasmussen, Mads H., additional, Bui, Khan H., additional, Fristrup, Niels, additional, Christensen, Erik I., additional, Nordentoft, Iver, additional, Morth, Jens P., additional, Jensen, Jørgen B., additional, Pedersen, Jakob S., additional, Beck, Martin, additional, Theodorescu, Dan, additional, Borre, Michael, additional, Howard, Kenneth A., additional, Dyrskjøt, Lars, additional, and Ørntoft, Torben Falck, additional

Published
2014
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36. Functional Screening Identifies miRNAs Influencing Apoptosis and Proliferation in Colorectal Cancer

Author

Christensen, Lise Lotte, primary, Holm, Anja, additional, Rantala, Juha, additional, Kallioniemi, Olli, additional, Rasmussen, Mads H., additional, Ostenfeld, Marie S., additional, Dagnaes-Hansen, Frederik, additional, Øster, Bodil, additional, Schepeler, Troels, additional, Tobiasen, Heidi, additional, Thorsen, Kasper, additional, Sieber, Oliver M., additional, Gibbs, Peter, additional, Lamy, Philippe, additional, Hansen, Torben F., additional, Jakobsen, Anders, additional, Riising, Eva M., additional, Helin, Kristian, additional, Lubinski, Jan, additional, Hagemann-Madsen, Rikke, additional, Laurberg, Søren, additional, Ørntoft, Torben F., additional, and Andersen, Claus L., additional

Published
2014
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37. Abstract 1157: MicroRNA-625-3p is associated with response to first-line oxaliplatin-based treatment of metastatic colorectal cancer.

Author

Rasmussen, Mads H., primary, Jensen, Niels F., additional, Tarpgaard, Line S., additional, Qvortrup, Camilla, additional, Rømer, Maria U., additional, Stenvang, Jan, additional, Hansen, Tine P., additional, Christensen, Lise-Lotte, additional, Lindebjerg, Jan, additional, Hansen, Flemming, additional, Jensen, Benny V., additional, Hansen, Torben F., additional, Jakobsen, Anders M., additional, Pfeiffer, Per, additional, Brünner, Nils, additional, Ørntoft, Torben F., additional, and Andersen, Claus L., additional

Published
2013
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38. Abstract 4138: miRNA-362-3p is associated with recurrence of colorectal cancer and targets E2F1, USF2 and PTPN1

Author

Tobiasen, Heidi, primary, Christensen, Lise Lotte, additional, Holm, Anja, additional, Schepeler, Troels, additional, Thorsen, Kasper, additional, Rasmussen, Mads H., additional, Birkenkamp-Demtroeder, Karin, additional, Sieber, Oliver M., additional, Gibbs, Peter, additional, Lamy, Philippe, additional, Laurberg, Søren, additional, Øster, Bodil, additional, Hansen, Kristian Q., additional, Hagemann-Madsen, Rikke, additional, Byskov, Kristina, additional, Ørntoft, Torben F., additional, and Andersen, Claus L., additional

Published
2012
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39. Tumor-specific usage of alternative transcription start sites in colorectal cancer identified by genome-wide exon array analysis

Author

Thorsen, Kasper, primary, Schepeler, Troels, additional, Øster, Bodil, additional, Rasmussen, Mads H, additional, Vang, Søren, additional, Wang, Kai, additional, Hansen, Kristian Q, additional, Lamy, Philippe, additional, Pedersen, Jakob Skou, additional, Eller, Asger, additional, Mansilla, Francisco, additional, Laurila, Kirsti, additional, Wiuf, Carsten, additional, Laurberg, Søren, additional, Dyrskjøt, Lars, additional, Ørntoft, Torben F, additional, and Andersen, Claus L, additional

Published
2011
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41. Alternative Splicing of SLC39A14 in Colorectal Cancer is Regulated by the Wnt Pathway

Author

Thorsen, Kasper, primary, Mansilla, Francisco, additional, Schepeler, Troels, additional, Øster, Bodil, additional, Rasmussen, Mads H., additional, Dyrskjøt, Lars, additional, Karni, Rotem, additional, Akerman, Martin, additional, Krainer, Adrian R., additional, Laurberg, Søren, additional, Andersen, Claus L., additional, and Ørntoft, Torben F., additional

Published
2011
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