20 results on '"Reckman, Yolan J."'
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2. Circular RNAs open a new chapter in cardiovascular biology
3. Yield and Pitfalls of Ajmaline Testing in the Evaluation of Unexplained Cardiac Arrest and Sudden Unexplained Death: Single-Center Experience With 482 Families
4. circRNAprofiler: an R-based computational framework for the downstream analysis of circular RNAs
5. The role of non-coding RNA/microRNAs in cardiac disease
6. Titin Circular RNAs Create a Back-Splice Motif Essential for SRSF10 Splicing
7. Quantification of Myocardial Creatine and Triglyceride Content in the Human Heart: Precision and Accuracy of in vivo Proton Magnetic Resonance Spectroscopy
8. Variants in the 3′ untranslated region of the KCNQ1-encoded Kv7.1 potassium channel modify disease severity in patients with type 1 long QT syndrome in an allele-specific manner
9. SCN5A mutation type and topology are associated with the risk of ventricular arrhythmia by sodium channel blockers
10. Formins Emerge as a Cause of Hypertrophic Cardiomyopathy: New Genes for Thick Hearts ∗
11. Therapeutic Delivery of miR-148a Suppresses Ventricular Dilation in Heart Failure
12. Yield and pitfalls of ajmaline testing in the evaluation of unexplained cardiac arrest and sudden unexplained death: Single centre experience of 482 families
13. Cardiac circRNAs arise mainly from constitutive exons rather than alternatively spliced exons
14. Circulating Circles Predict Postoperative Atrial Fibrillation
15. RBM20 Regulates Circular RNA Production From the Titin Gene
16. Abstract 28: In Vivo Knockdown of Mirna-15b Results in Increased Cardiac Hypertrophy and Fibrosis in Response to Pressure Overload of the Mouse Heart
17. Variants in the 3′ untranslated region of the KCNQ1-encoded Kv7.1 potassium channel modify disease severity in patients with type 1 long QT syndrome in an allele-specific manner
18. Abstract 28.
19. Therapeutic Delivery of miR-148a Suppresses Ventricular Dilation in Heart Failure
20. Formins Emerge as a Cause of Hypertrophic Cardiomyopathy: New Genes for Thick Hearts.
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