Search

Your search keyword '"Riccardi, Daniela"' showing total 389 results
Start Over Author "Riccardi, Daniela"
389 results on '"Riccardi, Daniela"'

2. Cachexia causes time‐dependent activation of the inflammasome in the liver

Author

das Neves, Rodrigo Xavier, primary, Yamashita, Alex S., additional, Riccardi, Daniela M.R., additional, Köhn‐Gaone, Julia, additional, Camargo, Rodolfo G., additional, Neto, Nelson I., additional, Caetano, Daniela, additional, Gomes, Silvio P., additional, Santos, Felipe H., additional, Lima, Joanna D.C.C., additional, Batista, Miguel L., additional, Rosa‐Neto, José Cesar, additional, Martins De Alcântara, Paulo Sérgio, additional, Maximiano, Linda F., additional, Otoch, José P., additional, Trinchieri, Giorgio, additional, Tirnitz‐Parker, Janina E.E., additional, and Seelaender, Marília, additional

Published
2023
Full Text
View/download PDF

5. Calcium-sensing receptor in GtoPdb v.2023.1

Author

Bikle, Daniel, primary, Bräuner-Osborne, Hans, primary, Brown, Edward M., primary, Chang, Wenhan, primary, Conigrave, Arthur, primary, Hannan, Fadil, primary, Leach, Katie, primary, Riccardi, Daniela, primary, Shoback, Dolores, primary, Ward, Donald T., primary, and Yarova, Polina, primary

Published
2023
Full Text
View/download PDF

7. Cachexia causes time‐dependent activation of the inflammasome in the liver

Author

Neves, Rodrigo Xavier, Yamashita, Alex S., Riccardi, Daniela M.R., Köhn‐Gaone, Julia, Camargo, Rodolfo G., Neto, Nelson I., Caetano, Daniela, Gomes, Silvio P., Santos, Felipe H., Lima, Joanna D.C.C., Batista, Miguel L., Rosa‐Neto, José Cesar, Martins De Alcântara, Paulo Sérgio, Maximiano, Linda F., Otoch, José P., Trinchieri, Giorgio, Tirnitz‐Parker, Janina E.E., and Seelaender, Marília

Abstract

Cachexia is a wasting syndrome associated with systemic inflammation and metabolic disruption. Detection of the early signs of the disease may contribute to the effective attenuation of associated symptoms. Despite playing a central role in the control of metabolism and inflammation, the liver has received little attention in cachexia. We previously described relevant disruption of metabolic pathways in the organ in an animal model of cachexia, and herein, we adopt the same model to investigate temporal onset of inflammation in the liver. The aim was thus to study inflammation in rodent liver in the well‐characterized cachexia model of Walker 256 carcinosarcoma and, in addition, to describe inflammatory alterations in the liver of one cachectic colon cancer patient, as compared to one control and one weight‐stable cancer patient. Colon cancer patients (one weight stable [WSC] and one cachectic [CC]) and one patient undergoing surgery for cholelithiasis (control, n= 1) were enrolled in the study, after obtainment of fully informed consent. Eight‐week‐old male rats were subcutaneously inoculated with a Walker 256 carcinosarcoma cell suspension (2 × 107cells in 1.0 mL; tumour‐bearing [T]; or phosphate‐buffered saline—controls [C]). The liver was excised on Days 0 (n= 5), 7 (n= 5) and 14 (n= 5) after tumour cell injection. In rodent cachexia, we found progressively higher numbers of CD68+myeloid cells in the liver along cancer‐cachexia development. Similar findings are described for CC, whose liver showed infiltration of the same cell type, compared with both WSC and control patient organs. In advanced rodent cachexia, hepatic phosphorylated c‐Jun N‐terminal kinase protein content and the inflammasome pathway protein expression were increased in relation to baseline (P< 0.05). These changes were accompanied by augmented expression of the active interleukin‐1β (IL‐1β) form (P< 0.05 for both circulating and hepatic content). The results show that cancer cachexia is associated with an increase in the number of myeloid cells in rodent and human liver and with modulation of hepatic inflammasome pathway. The latter contributes to the aggravation of systemic inflammation, through increased release of IL‐1β.

Published
2023
Full Text
View/download PDF

8. Impaired Mineral Ion Metabolism in a Mouse Model of Targeted Calcium-Sensing Receptor (CaSR) Deletion from Vascular Smooth Muscle Cells

Author

Schepelmann, Martin, primary, Ranieri, Marianna, additional, Lopez-Fernandez, Irene, additional, Webberley, Thomas S., additional, Brennan, Sarah C., additional, Yarova, Polina L., additional, Graca, Joao, additional, Hanif, Umar-Khetaab, additional, Müller, Christian, additional, Manhardt, Teresa, additional, Salzmann, Martina, additional, Quasnichka, Helen, additional, Price, Sally A., additional, Ward, Donald T., additional, Gilbert, Thierry, additional, Matchkov, Vladimir V., additional, Fenton, Robert A., additional, Herberger, Amanda, additional, Hwong, Jenna, additional, Santa Maria, Christian, additional, Tu, Chia-Ling, additional, Kallay, Enikö, additional, Valenti, Giovanna, additional, Chang, Wenhan, additional, and Riccardi, Daniela, additional

Published
2022
Full Text
View/download PDF

9. In Search of the Acute Oxygen Sensor : Functional proteomics and acute regulation of large-conductance, calcium-activated potassium channels by hemeoxygenase-2

Author

KEMP, PAUL J., PEERS, CHRIS, RICCARDI, DANIELA, ILES, DAVID E., MASON, HELEN S., WOOTTON, PHILLIPPA, WILLIAMS, SANDILE E., BACK, NATHAN, editor, COHEN, IRUN R., editor, KRITCHEVSKY, DAVID, editor, LAJTHA, ABEL, editor, PAOLETTI, RODOLFO, editor, Hayashida, Yoshiaki, editor, Gonzalez, Constancio, editor, and Kondo, Hisatake, editor

Published
2006
Full Text
View/download PDF

14. Stereo-Specific Modulation of the Extracellular Calcium-Sensing Receptor in Colon Cancer Cells

Author

Schepelmann, Martin, Kupper, Nadja, Sladczyk, Marta, Mansfield, Bethan, Manhardt, Teresa, Piatek, Karina, Iamartino, Luca, Riccardi, Daniela, Kariuki, Benson M., Bassetto, Marcella, and Kallay, Enikö

Subjects
Models, Molecular, QH301-705.5, calcium-sensing receptor, Green Fluorescent Proteins, Molecular Conformation, Article, stereospecificity, enantiomer, Humans, calcilytic, Biology (General), QD1-999, IL-8, Interleukin-8, technology, industry, and agriculture, Stereoisomerism, calcimimetic, Chemistry, HEK293 Cells, Gene Expression Regulation, colon cancer, HT-29, inflammation, Colonic Neoplasms, Extracellular Space, HT29 Cells, Receptors, Calcium-Sensing
Abstract

Pharmacological allosteric agonists (calcimimetics) of the extracellular calcium-sensing receptor (CaSR) have substantial gastro-intestinal side effects and induce the expression of inflammatory markers in colon cancer cells. Here, we compared the effects of both CaSR-specific (R enantiomers) and -unspecific (S enantiomers) enantiomers of a calcimimetic (NPS 568) and a calcilytic (allosteric CaSR antagonists, NPS 2143) to prove that these effects are indeed mediated via the CaSR, rather than via off-target effects, e.g., on β-adrenoceptors or calcium channels, of these drugs. The unspecific S enantiomer of NPS 2143 and NPS S-2143 was prepared using synthetic chemistry and characterized using crystallography. NPS S-2143 was then tested in HEK-293 cells stably transfected with the human CaSR (HEK-CaSR), where it did not inhibit CaSR-mediated intracellular Ca2+ signals, as expected. HT29 colon cancer cells transfected with the CaSR were treated with both enantiomers of NPS 568 and NPS 2143 alone or in combination, and the expression of CaSR and the pro-inflammatory cytokine interleukin 8 (IL-8) was measured by RT-qPCR and ELISA. Only the CaSR-selective enantiomers of the calcimimetic NPS 568 and NPS 2143 were able to modulate CaSR and IL-8 expression. We proved that pro-inflammatory effects in colon cancer cells are indeed mediated through CaSR activation. The non-CaSR selective enantiomer NPS S-2143 will be a valuable tool for investigations in CaSR-mediated processes.

Published
2021
Full Text
View/download PDF

15. Transcriptome profiling reveals a potential role for antagonism of the calcium-sensing receptor in the prophylaxis of idiopathic pulmonary fibrosis

Author

Wolffs, Kasope L., primary, Pass, Daniel A., additional, Mansfield, Beth, additional, Bruce, Richard T., additional, Popov, Petar A., additional, Corrigan, Christopher J., additional, Ward, Jeremy P.T., additional, Kemp, Paul J., additional, Hope-Gill, Benjamin, additional, and Riccardi, Daniela, additional

Published
2021
Full Text
View/download PDF

16. Calcium-sensing receptor in GtoPdb v.2021.3

Author

Bikle, Daniel, primary, Bräuner-Osborne, Hans, primary, Brown, Edward M., primary, Chang, Wenhan, primary, Conigrave, Arthur, primary, Hannan, Fadil, primary, Leach, Katie, primary, Riccardi, Daniela, primary, Shoback, Dolores, primary, Ward, Donald T., primary, and Yarova, Polina, primary

Published
2021
Full Text
View/download PDF

22. Physiology and pathophysiology of the calcium-sensing receptor in the kidney

Author

Riccardi, Daniela and Brown, Edward M.

Subjects
Cell receptors -- Research, Kidneys -- Physiological aspects, Autoimmunity -- Research, Biological sciences
Abstract

The extracellular calcium-sensing receptor (CaSR) plays a major role in the maintenance of a physiological serum ionized calcium ([Ca.sup.2+]) concentration by regulating the circulating levels of parathyroid hormone. It was molecularly identified in 1993 by Brown et al. in the laboratory of Dr. Steven Hebert with an expression cloning strategy. Subsequent studies have demonstrated that the CaSR is highly expressed in the kidney, where it is capable of integrating signals deriving from the tubular fluid and/or the interstitial plasma. Additional studies elucidating inherited and acquired mutations in the CaSR gene, the existence of activating and inactivating autoantibodies, and genetic polymorphisms of the CaSR have greatly enhanced our understanding of the role of the CaSR in mineral ion metabolism. Allosteric modulators of the CaSR are the first drugs in their class to become available for clinical use and have been shown to treat successfully hyperparathyroidism secondary to advanced renal failure. In addition, preclinical and clinical studies suggest the possibility of using such compounds in various forms of hypercalcemic hyperparathyroidism, such as primary and lithium-induced hyperparathyroidism and that occurring after renal transplantation. This review addresses the role of the CaSR in kidney physiology and pathophysiology as well as current and in-the-pipeline treatments utilizing CaSR-based therapeutics. proximal tubule; thick ascending limb; distal convoluted tubule; collecting duct; 1,25-dihydroxyvitamin [D.sub.3]; parathyroid hormone; hypercalcemia; hypocalcemia; hypercalciuria; calcimimetic; hyperparathyroidism; inactivating mutation; activating mutation; polymorphism; familial hypocalciuric hypercalcemia; neonatal severe primary hyperparathyroidism; autosomal dominant hypoparathyroidism doi:10.1152/ajprenal.00608.2009

Published
2010

30. Characterization of Negative Allosteric Modulators of the Calcium-Sensing Receptor for Repurposing as a Treatment of Asthma

Author

Yarova, Polina L., primary, Huang, Ping, additional, Schepelmann, Martin W., additional, Bruce, Richard, additional, Ecker, Rupert, additional, Nica, Robert, additional, Telezhkin, Vsevolod, additional, Traini, Daniela, additional, Gomes dos Reis, Larissa, additional, Kidd, Emma J., additional, Ford, William R., additional, Broadley, Kenneth J., additional, Kariuki, Benson M., additional, Corrigan, Christopher J., additional, Ward, Jeremy P.T., additional, Kemp, Paul J., additional, and Riccardi, Daniela, additional

Published
2020
Full Text
View/download PDF

31. Calcium-sensing receptor antagonists, calcilytics, prevent activation of human dendritic and epithelial cells by urban particulate matter

Author

Mansfield, Bethan, primary, Ho, Tzer-Ren, additional, Mudway, Ian, additional, Lugg, Sebastian. T, additional, Corrigan, Chris. J, additional, Ward, Jeremy P.T., additional, Thickett, David, additional, Prytherch, Zoe, additional, Scofield, Simon, additional, Kemp, Paul. J, additional, Lewis, Keir. E, additional, Mur, Luis. A, additional, Hawrylowicz, Catherine, additional, and Riccardi, Daniela, additional

Published
2020
Full Text
View/download PDF

32. List of Contributors

Author

Abbate, Mauro, primary, Adrogué, Horacio J., additional, Alper, Seth L., additional, Alpern, Robert J., additional, Baliga, Radhakrishna, additional, Batlle, Daniel, additional, Bernardo, José F., additional, Berndt, Theresa J., additional, Bevensee, Mark O., additional, Biber, Jürg, additional, Bindels, René J.M., additional, Birn, Henrik, additional, Boron, Walter F., additional, Craig Brater, D., additional, Brown, Edward M., additional, Burckhardt, Gerhard, additional, Camargo, Simone M.R., additional, Caplan, Michael J., additional, Cárdenas, Andrés, additional, Chen, Sheldon, additional, Ilsø Christensen, Erik, additional, Chung-Park, Moonja, additional, Coe, Fredric L., additional, Conrad, Kirk P., additional, Cooper, Christopher J., additional, Curthoys, Norman P., additional, Devarajan, Prasad, additional, Diamond, Matthew, additional, Dumitru, Cristina, additional, Dworkin, Lance D., additional, Eckardt, Kai-Uwe, additional, Huba Endre, Zoltán, additional, Evan, Andrew, additional, Falk, Ronald J., additional, Forster, Ian C., additional, Friedman, Peter A., additional, Kazue Fujihara, Clarice, additional, Geibel, John P., additional, Giacomini, Kathleen M., additional, Ginès, Pere, additional, Goral, Simin, additional, Halperin, Mitchell, additional, Hamm, L. Lee, additional, Haque, Syed K., additional, Hebert, Steven C., additional, Harold Helderman, J., additional, Henrich, William L., additional, Hernando, Nati, additional, Hoenderop, Joost G.J., additional, Homeister, Jonathon W., additional, Hummel, Charles S., additional, Jennette, J. Charles, additional, Kamel, Kamel S., additional, Ananth Karumanchi, S., additional, Kashtan, Clifford E., additional, Khoury, Charbel, additional, Kleta, Robert, additional, Koepsell, Hermann, additional, Konrad, Martin, additional, Krapf, Reto, additional, Kumar, Rajiv, additional, Kurtz, Armin, additional, Kurtz, Ira, additional, Langone, Anthony J., additional, Lin, Shih-Hua, additional, Lindheimer, Marshall D., additional, Lu, Christopher Y., additional, Macconi, Daniela, additional, Madaio, Michael, additional, Madias, Nicolaos E., additional, Makrides, Victoria, additional, Manoharon, Anup, additional, Matsson, Pär, additional, Mitch, William E., additional, Moe, Orson W., additional, Molitoris, Bruce A., additional, Murer, Heini, additional, Nattie, Eugene, additional, Nielsen, Rikke, additional, Palmer, Biff F., additional, Preisig, Patricia A., additional, Quamme, Gary A., additional, Quarles, L. Darryl, additional, Rajapurkar, Mohan, additional, Remuzzi, Giuseppe, additional, Riccardi, Daniela, additional, Schaefer, Heidi, additional, Schelling, Jeffrey R., additional, Schlingmann, Karl P., additional, Schrier, Robert W., additional, Sedor, John R., additional, Segal, Yoav, additional, Seldin, Donald W., additional, Shah, Sudhir V., additional, Sharfuddin, Asif, additional, Somlo, Stefan, additional, Stewart, Andrew K., additional, Tebben, Peter J., additional, Torres, Vicente E., additional, Verrey, François, additional, James Walker, Robert, additional, Wenger, Roland H., additional, Worcester, Elaine, additional, Workeneh, Biruh T., additional, Wright, Ernest M., additional, Wysolmerski, John, additional, Yang, Sung-Sen, additional, Zatz, Roberto, additional, Ziyadeh, Fuad N., additional, and Zoja, Carla, additional

Published
2012
Full Text
View/download PDF

33. Hemoxygenase-2 is an oxygen sensor for a calcium-sensitive potassium channel

Author

Williams, Sandile E.J., Wootton, Phillippa, Mason, Helen S., Bould, Jonathan, Iles, David E., Riccardi, Daniela, Peers, Chris, and Kemp, Paul J.

Subjects
Ion channels -- Research, Human biology -- Research, Science and technology, Research
Abstract

Modulation of calcium-sensitive potassium (BK) channels by oxygen is important in several mammalian tissues, and in the carotid body it is crucial to respiratory control. However, the identity of the oxygen sensor remains unknown. We demonstrate that hemoxygenase-2 (HO-2) is part of the BK channel complex and enhances channel activity in normoxia. Knockdown of HO-2 expression reduced channel activity, and carbon monoxide, a product of HO-2 activity, rescued this loss of function. Inhibition of BK channels by hypoxia was dependent on HO-2 expression and was augmented by HO-2 stimulation. Furthermore, carotid body cells demonstrated HO-2-dependent hypoxic BK channel inhibition, which indicates that HO-2 is an oxygen sensor that controls channel activity during oxygen deprivation., Large-conductance, [Ca.sup.2+]-sensitive potassium (BK) channels are strongly implicated in the acute [O.sub.2] signaling cascade of a number of cellular systems. In carotid body chemoreceptors (1, 2), low arterial [pO.sub.2] is [...]

Published
2004

35. Altered dietary iron intake is a strong modulator of renal DMT1 expression

Author

Wareing, Mark, Ferguson, Carole J., Delannoy, Mathieu, Cox, Alan G., McMahon, Raymond F.T., Green, Roger, Riccardi, Daniela, and Smith, Craig P.

Subjects
Iron in the body -- Research, Kidneys -- Research, Food -- Iron content, Food -- Research, Biological sciences
Abstract

Divalent metal transporter1 (DMT1; also known as DCT1 or NRAMP2) is an important component of the cellular machinery responsible for dietary iron absorption in the duodenum. DMT1 is also highly expressed in the kidney where it has been suggested to playa role in urinary iron handling. In this study, we determined the effect on renal DMT1 expression of feeding an iron-restricted diet (50 mg/kg) or an iron-enriched diet (5 g/kg) for 4 wk and measured urinary and fecal iron excretion rates. Feeding the low-iron diet caused a reduction in serum iron concentration and fecal iron output rate with an increase in renal DMT1 expression. Feeding an iron-enriched diet had the converse effect. Therefore, DMT1 expression in the kidney is sensitive to dietary iron intake, and the level of expression is inversely related to the dietary iron content. Changes in DMT1 expression occurred intracellularly in the proximal tubule and in the apical membrane and subapical region of the distal convoluted tubule. Increased DMT1 expression was accompanied by a decrease in urinary iron excretion rate and vice versa when DMT1 expression was reduced. Together, these findings suggest that modulation of renal DMT1 expression may influence renal iron excretion rate. serum iron level; kidney; iron regulatory protein; SLC11A2; NRAMP2

Published
2003

36. Lung Organogenesis

Author

Warburton, David, primary, El-Hashash, Ahmed, additional, Carraro, Gianni, additional, Tiozzo, Caterina, additional, Sala, Frederic, additional, Rogers, Orquidea, additional, Langhe, Stijn De, additional, Kemp, Paul J., additional, Riccardi, Daniela, additional, Torday, John, additional, Bellusci, Saverio, additional, Shi, Wei, additional, Lubkin, Sharon R, additional, and Jesudason, Edwin, additional

Published
2010
Full Text
View/download PDF

41. Contributors

Author

ABBATE, MAURO, primary, ABRAHAMSON, DALE R., additional, ADAMCZAK, MARCIN, additional, ADROGUÉ, HORACIO J., additional, ALPER, SETH L., additional, ALPERN, ROBERT J., additional, ANDREOLI, THOMAS E., additional, APERIA, ANITA C., additional, BAILEY, MATTHEW A., additional, BATLLE, DANIEL, additional, BAUM, MICHEL, additional, BERNDT, THERESA J., additional, BEVENSEE, MARK O., additional, BIBER, JÜRG, additional, BICHET, DANIEL G., additional, BINDELS, RENÉ J.M., additional, BLANTZ, ROLAND C., additional, BORON, WALTER F., additional, BRATER, D. CRAIG, additional, BRIGGS, JOSEPHINE P., additional, BROWN, ALEX, additional, BRUNSKILL, NIGEL J., additional, BURCKHARDT, GERHARD, additional, BURNSTOCK, GEOFFREY, additional, CANTLEY, LLOYD, additional, CAO, CHUNHUA, additional, CAPASSO, GIOVAMBATTISTA, additional, CAPLAN, MICHAEL J., additional, CARROLL, HUGH J., additional, CHAN, LAURENCE, additional, CHUNG-PARK, MOONJA, additional, COE, FREDRIC L., additional, COFFMAN, THOMAS M., additional, COMPER, WAYNE D., additional, CONRAD, KIRK P., additional, CROWLEY, STEVEN D., additional, CURTHOYS, NORMAN P., additional, CUTILLAS, PEDRO R., additional, DANOFF, THEODORE M., additional, DEBNAM, EDWARD S., additional, DIMKE, HENRIK, additional, DOUCET, ALAIN, additional, DUBEY, RAGHVENDRA K., additional, DUSSO, ADRIANA, additional, ECKARDT, KAI-UWE, additional, ELLISON, DAVID H., additional, ENDOU, HITOSHI, additional, ENDRE, ZOLTÁN HUBA, additional, EPSTEIN, FRANKLIN H., additional, EVAN, ANDREW, additional, FALK, RONALD J., additional, FARBAKHSH, KAMBIZ, additional, FERRERI, NICHOLAS R., additional, FONG, PEYING, additional, FONTELES, MANASSES CLAUDINO, additional, FORSTER, IAN, additional, FORTE, LEONARD RALPH, additional, FRANCH, HAROLD A., additional, FRASSETTO, LYNDA A., additional, FRIEDMAN, PETER A., additional, FRØKLÆR, JØRGEN, additional, GEIBEL, JOHN P., additional, GEKLE, MICHAEL, additional, GIEBISCH, GERHARD, additional, GINÈS, PERE, additional, GOLDSTEIN, STEVE A.N., additional, GORAL, SIMIN, additional, GRIFFIN, SIAN V., additional, GUGGINO, WILLIAM B., additional, GUISE, THERESA A., additional, GURLEY, SUSAN B., additional, HALL, STEPHEN D., additional, HALPERIN, MITCHELL L., additional, HAMM, L. LEE, additional, HEBERT, STEVEN C., additional, HEDIGER, MATTHIAS A., additional, HELDERMAN, J. HAROLD, additional, HENRICH, WILLIAM L., additional, HERAS-HERZIG, AILLEEN, additional, HERNANDO, NATI, additional, HOENDEROP, JOOST G.J., additional, HOLTBÄCK, ULLA, additional, HORISBERGER, JEAN-DANIEL, additional, HUMMLER, EDITH, additional, HUNLEY, TRACY E., additional, JACKSON, EDWIN K., additional, JENNETTE, J. CHARLES, additional, JOHNS, EDWARD J., additional, JOHNSON, JOHN P., additional, KAISSLING, BRIGITTE, additional, KAMEL, KAMEL S., additional, KARUMANCHI, S. ANANTH, additional, KASHTAN, CLIFFORD E., additional, KASISKE, BERTRAM L., additional, KATZ, ADRIAN I., additional, KING, BRIAN F., additional, KLAHR, SAULO, additional, KLEYMAN, THOMAS R., additional, KOEPSELL, HERMANN, additional, KON, VALENTINA, additional, KONRAD, MARTIN, additional, KOPP, ULLA C., additional, KRAPF, RETO, additional, KRIZ, WILHELM, additional, KUMAR, RAJIV, additional, KURSCHAT, CHRISTINE E., additional, KURTZ, ARMIN, additional, KWON, TAE-HWAN, additional, LANDOWSKI, CHRISTOPHER P., additional, LANGONE, ANTHONY J., additional, LANG, FLORIAN, additional, LAYTON, HAROLD E., additional, LE, THU H., additional, LEVY, DANIEL I., additional, LIN, SHIH-HUA, additional, LINDHEIMER, MARSHALL D., additional, LU, CHRISTOPHER Y., additional, MADAIO, MICHAEL P., additional, MADIAS, NICOLAOS E., additional, MALNIC, GERHARD, additional, MATLIN, KARL S., additional, McCLELLAN, WILLIAM C., additional, MCGIFF, JOHN C., additional, MICHEL, C. CHARLES, additional, MINER, JEFFREY H., additional, MITCH, WILLIAM E., additional, MIYAZAKI, HIROKI, additional, MOE, ORSON W., additional, MOLITORIS, BRUCE A., additional, MORRIS, R. CURTIS, additional, MUJAIS, SALIM K., additional, MURER, HEINI, additional, MUTO, SHIGEAKI, additional, NATTIE, EUGENE, additional, NEILSON, ERIC G., additional, NIELSEN, SØREN, additional, NIGAM, SANJAY K., additional, NOGUEIRA, JOSEPH M., additional, OH, MAN S., additional, OKUSA, MARK D., additional, OSICKA, TANYA M., additional, PALLONE, THOMAS L., additional, PALMER, BIFF F., additional, PALMER, LAWRENCE G., additional, PARKER, MARK D., additional, PARKS, JOAN H., additional, PREISIG, PATRICIA A., additional, QUAMME, GARY A., additional, QUARLES, L. DARRYL, additional, QUIGLEY, RAYMOND, additional, REEVES, W. BRIAN, additional, REMUZZI, GIUSEPPE, additional, REUSS, LUIS, additional, RICCARDI, DANIELA, additional, RINGHOFER, BRIAN, additional, RITZ, EBERHARD, additional, RIVARD, CHRISTOPHER J., additional, ROBERTSON, GARY L., additional, ROSA, ROBERT M., additional, ROSSIER, BERNARD C., additional, RUSSO, LEILEATA M., additional, SACKIN, HENRY, additional, SAKURAI, HIROYUKI, additional, SANDS, JEFF M., additional, SATLIN, LISA M., additional, SCHAEFER, HEIDI, additional, SCHELLING, JEFFREY R., additional, SCHILD, LAURENT, additional, SCHLINGMANN, KARL P., additional, SCHNERMANN, JÜRGEN B., additional, SCHRIER, ROBERT W., additional, SEBASTIAN, ANTHONY, additional, SEDOR, JOHN R., additional, SEGAL, YOAV, additional, SEKINE, TAKASHI, additional, SELDIN, DONALD W., additional, SENITKO, MARTIN, additional, SHAH, MALATHI, additional, SHAH, SUDHIR V., additional, SHANKLAND, STUART J., additional, SHARFUDDIN, ASIF A., additional, SHARMA, KUMAR, additional, SHENG, SHAOHU, additional, SHIRLEY, DAVID G., additional, SILBERNAGL, STEFAN, additional, SILVER, STEPHEN M., additional, SILVERMAN, MEL, additional, SLATOPOLSKY, EDUARDO, additional, SOMLO, STEFAN, additional, STERNS, RICHARD H., additional, STEWART, ANDREW K., additional, SUZUKI, YOSHIRO, additional, TEBBEN, PETER J., additional, THOMSON, SCOTT C., additional, TORRES, VICENTE E., additional, UNWIN, ROBERT J., additional, VERREY, FRANÇOIS, additional, VESELY, DAVID L., additional, WAGNER, CARSTEN A., additional, WALDMAN, MERYL, additional, WALKER, ROBERT JAMES, additional, WANG, WEI, additional, WANG, WEN-HUI, additional, WANG, YINGHONG, additional, WEINSTEIN, ALAN M., additional, WELLING, PAUL A., additional, WOLF, GUNTER, additional, WORCESTER, ELAINE, additional, ZIYADEH, FUAD N., additional, and ZOJA, CARLA, additional

Published
2008
Full Text
View/download PDF

43. International Union of Basic and Clinical Pharmacology. CVIII. Calcium-Sensing Receptor Nomenclature, Pharmacology, and Function

Author

Leach, Katie, primary, Hannan, Fadil M., additional, Josephs, Tracy M., additional, Keller, Andrew N., additional, Møller, Thor C., additional, Ward, Donald T., additional, Kallay, Enikö, additional, Mason, Rebecca S., additional, Thakker, Rajesh V., additional, Riccardi, Daniela, additional, Conigrave, Arthur D., additional, and Bräuner-Osborne, Hans, additional

Published
2020
Full Text
View/download PDF

44. Calcium-sensing receptor antagonism as a novel therapeutic for pulmonary fibrosis

Author

Wolffs, Kasope L., primary, Mansfield, Bethan, additional, Bruce, Richard T., additional, Huang, Ping, additional, Schepelmann, Martin, additional, Brennan, Sarah C, additional, Verckist, Line, additional, Adriaensen, Dirk, additional, De Araújo, Rachel Paes, additional, Mur, Luis A.J., additional, Attanoos, Richard, additional, Lewis, Keir E., additional, Ward, Jeremy P.T., additional, Corrigan, Christopher J., additional, Kemp, Paul J., additional, Hope-Gill, Benjamin D., additional, and Riccardi, Daniela, additional

Published
2020
Full Text
View/download PDF

45. Plasma Lipid Profile and Systemic Inflammation in Patients With Cancer Cachexia

Author

Riccardi, Daniela Mendes dos Reis, primary, das Neves, Rodrigo Xavier, additional, de Matos-Neto, Emidio Marques, additional, Camargo, Rodolfo Gonzalez, additional, Lima, Joanna Darck Carola Correia, additional, Radloff, Katrin, additional, Alves, Michele Joana, additional, Costa, Raquel Galvão Figuerêdo, additional, Tokeshi, Flávio, additional, Otoch, José Pinhata, additional, Maximiano, Linda Ferreira, additional, de Alcantara, Paulo Sérgio Martins, additional, Colquhoun, Alison, additional, Laviano, Alessandro, additional, and Seelaender, Marilia, additional

Published
2020
Full Text
View/download PDF

49. Calcium-sensing receptor (CaSR) drives lung ECM remodelling and fibrosis in murine asthma models and in ageing mice

Author

Bruce, Richard, primary, Huang, Ping, additional, Yarova, Polina, additional, Schepelmann, Martin, additional, Wolffs, Kasope, additional, Mansfield, Bethan, additional, Ford, William, additional, Kidd, Emma, additional, Chang, Wenhan, additional, Ecker, Rupert, additional, Nica, Robert, additional, Ying, Sun, additional, Kemp, Paul, additional, and Riccardi, Daniela, additional

Published
2019
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results