468 results on '"Rock, Kenneth L."'
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2. GENETIC ABLATION OF THE C-TYPE LECTIN RECEPTOR CLEC2D INCREASES PERITONITIS MORTALITY, INFLAMMATION, AND PHYSIOLOGY WITHOUT DIMINISHING ORGAN INJURY.
3. Cellular Protein Is the Source of Cross-Priming Antigen in vivo
4. Poliovirus Vaccine Vectors Elicit Antigen-Specific Cytotoxic T Cells and Protect Mice against Lethal Challenge with Malignant Melanoma Cells Expressing a Model Antigen
5. Two Distinct Proteolytic Processes in the Generation of a Major Histocompatibility Complex Class I-Presented Peptide
6. Pathways of MHC I cross-presentation of exogenous antigens
7. Major Histocompatibility Class I Presentation of Soluble Antigen Facilitated by Mycobacterium tuberculosis Infection
8. Present Yourself! By MHC Class I and MHC Class II Molecules
9. Tetraspanin-5–mediated MHC class I clustering is required for optimal CD8 T cell activation
10. Re-examining class-I presentation and the DRiP hypothesis
11. Using intein catalysis to probe the origin of major histocompatibility complex class I-presented peptides
12. IL-1 Generated Subsequent to Radiation-Induced Tissue Injury Contributes to the Pathogenesis of Radiodermatitis
13. Cytotoxic T Lymphocytes in Resistance to Tuberculosis
14. Endoplasmic Reticulum Aminopeptidase 1 (ERAP1) Trims MHC Class I-Presented Peptides in vivo and Plays an Important Role in Immunodominance
15. A genome wide screen identifies a new peptidase involved in class I antigen processing and presentation
16. A genome-wide SiRNA screen reveals a novel class I-like protein involved in cross-presentation
17. Novel genes involved in MHC I antigen presentation in dendritic cells
18. The xanthine oxidase inhibitor Febuxostat reduces tissue uric acid content and inhibits injury-induced inflammation in the liver and lung
19. Presentation of Exogenous Antigen with Class I Major Histocompatibility Complex Molecules
20. Overexpressed Ly-6A.2 Mediates Cell-Cell Adhesion by Binding a Ligand Expressed on Lymphoid Cells
21. Efficient Major Histocompatibility Complex Class I Presentation of Exogenous Antigen Upon Phagocytosis by Macrophages
22. Gene Encoding T-Cell-Activating Protein TAP Maps to the Ly-6 Locus
23. Phospholipase Treatment of Accessory Cells that Have Been Exposed to Antigen Selectively Inhibits Antigen-Specific Ia-Restricted, but not Allospecific, Stimulation of T Lymphocytes
24. Expression of T-Cell-Activating Protein in Peripheral Lymphocyte Subsets
25. Characterization of Antigen Association with Accessory Cells: Specific Removal of Processed Antigens from the Cell Surface by Phospholipases
26. Peptidase Activities of Proteasomes are Differentially Regulated by the Major Histocompatibility Complex-Encoded Genes for LMP2 and LMP7
27. Analysis of Antigen Presentation by Metabolically Inactive Accessory Cells and Their Isolated Membranes
28. Reassociation with β 2 -Microglobulin is Necessary for K b Class I Major Histocompatibility Complex Binding of Exogenous Peptides
29. Reassociation with β 2 -Microglobulin is Necessary for D b Class I Major Histocompatibility Complex Binding of an Exogenous Influenza Peptide
30. Two Genetically Identical Antigen-Presenting Cell Clones Display Heterogeneity in Antigen Processing
31. Low Temperature and Peptides Favor the Formation of Class I Heterodimers on RMA-S Cells at the Cell Surface
32. Biosynthesis, Glycosylation, and Partial N-terminal Amino Acid Sequence of the T-Cell-Activating Protein TAP
33. Cloning and Expression of a cDNA for the T-Cell-Activating Protein TAP
34. Serum Proteases Alter the Antigenicity of Peptides Presented by Class I Major Histocompatibility Complex Molecules
35. Analysis of the Association of Peptides of Optimal Length to Class I Molecules on the Surface of Cells
36. Thymic T Cells are Driven to Expand upon Interaction with Self-Class II Major Histocompatibility Complex Gene Products on Accessory Cells
37. Mice completely lacking immunoproteasomes show major changes in antigen presentation
38. NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals
39. Uric acid promotes an acute inflammatory response to sterile cell death in mice
40. Cancer Immune Evasion Through Loss of MHC Class I Antigen Presentation
41. Microbiota signalling through MyD88 is necessary for a systemic neutrophilic inflammatory response
42. Expression of a novel oncofetal mRNA-binding protein IMP3 in endometrial carcinomas: diagnostic significance and clinicopathologic correlations
43. Identification of a key pathway required for the sterile inflammatory response triggered by dying cells
44. IMP3 is a novel biomarker for adenocarcinoma in situ of the uterine cervix: an immunohistochemical study in comparison with p16INK4a expression
45. Natural endogenous adjuvants
46. MyD88-dependent IL-1 receptor signaling is essential for gouty inflammation stimulated by monosodium urate crystals
47. Mammaglobin and CRxA-01 in pleural effusion cytology; potential utility of distinguishing metastatic breast carcinomas from other cytokeratin 7-positive/cytokeratin 20-negative carcinomas
48. Uric acid as a danger signal in gout and its comorbidities
49. The ER aminopeptidase ERAP1 enhances or limits antigen presentation by trimming epitopes to 8–9 residues
50. Priming of T cells by exogenous antigen cross-presented on MHC class I molecules
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