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6. Chronic kidney disease in propionic acidemia

Author

Shchelochkov, Oleg A., Manoli, Irini, Sloan, Jennifer L., Ferry, Susan, Pass, Alexandra, Van Ryzin, Carol, Myles, Jennifer, Schoenfeld, Megan, McGuire, Peter, Rosing, Douglas R., Levin, Mark D., Kopp, Jeffrey B., and Venditti, Charles P.

Published
2019
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9. Supplementary Figure S1 from Impact of ABCB1 Allelic Variants on QTc Interval Prolongation

Author

Sissung, Tristan M., primary, Gardner, Erin R., primary, Piekarz, Richard L., primary, Howden, Reuben, primary, Chen, Xiaohong, primary, Woo, Sukyung, primary, Franke, Ryan, primary, Clark, James A., primary, Miller-DeGraff, Laura, primary, Steinberg, Seth M., primary, Venzon, David, primary, Liewehr, David, primary, Kleeberger, Steven R., primary, Bates, Susan E., primary, Price, Douglas K., primary, Rosing, Douglas R., primary, Cabell, Christopher, primary, Sparreboom, Alex, primary, and Figg, William D., primary

Published
2023
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10. Data from Impact of ABCB1 Allelic Variants on QTc Interval Prolongation

Author

Sissung, Tristan M., primary, Gardner, Erin R., primary, Piekarz, Richard L., primary, Howden, Reuben, primary, Chen, Xiaohong, primary, Woo, Sukyung, primary, Franke, Ryan, primary, Clark, James A., primary, Miller-DeGraff, Laura, primary, Steinberg, Seth M., primary, Venzon, David, primary, Liewehr, David, primary, Kleeberger, Steven R., primary, Bates, Susan E., primary, Price, Douglas K., primary, Rosing, Douglas R., primary, Cabell, Christopher, primary, Sparreboom, Alex, primary, and Figg, William D., primary

Published
2023
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11. Supplementary Methods from Impact of ABCB1 Allelic Variants on QTc Interval Prolongation

Author

Sissung, Tristan M., primary, Gardner, Erin R., primary, Piekarz, Richard L., primary, Howden, Reuben, primary, Chen, Xiaohong, primary, Woo, Sukyung, primary, Franke, Ryan, primary, Clark, James A., primary, Miller-DeGraff, Laura, primary, Steinberg, Seth M., primary, Venzon, David, primary, Liewehr, David, primary, Kleeberger, Steven R., primary, Bates, Susan E., primary, Price, Douglas K., primary, Rosing, Douglas R., primary, Cabell, Christopher, primary, Sparreboom, Alex, primary, and Figg, William D., primary

Published
2023
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12. Supplementary Results from Impact of ABCB1 Allelic Variants on QTc Interval Prolongation

Author

Sissung, Tristan M., primary, Gardner, Erin R., primary, Piekarz, Richard L., primary, Howden, Reuben, primary, Chen, Xiaohong, primary, Woo, Sukyung, primary, Franke, Ryan, primary, Clark, James A., primary, Miller-DeGraff, Laura, primary, Steinberg, Seth M., primary, Venzon, David, primary, Liewehr, David, primary, Kleeberger, Steven R., primary, Bates, Susan E., primary, Price, Douglas K., primary, Rosing, Douglas R., primary, Cabell, Christopher, primary, Sparreboom, Alex, primary, and Figg, William D., primary

Published
2023
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15. Succinate Dehydrogenase Gene Mutations in Cardiac Paragangliomas

Author

Martucci, Victoria L., Emaminia, Abbas, del Rivero, Jaydira, Lechan, Ronald M., Magoon, Bindiya T., Galia, Analyza, Fojo, Tito, Leung, Steve, Lorusso, Roberto, Jimenez, Camilo, Shulkin, Barry L., Audibert, Jennifer L., Adams, Karen T., Rosing, Douglas R., Vaidya, Anand, Dluhy, Robert G., Horvath, Keith A., and Pacak, Karel

Published
2015
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17. Effects of interrupting daily sedentary behavior on children's glucose metabolism: A 6‐day randomized controlled trial

Author

Broadney, Miranda M., primary, Belcher, Britni R., additional, Ghane, Nejla, additional, Sheni, Risha, additional, Jayson, Michael J., additional, Trenschel, Robert W., additional, Collins, Shavonne M., additional, Brychta, Robert J., additional, Davis, Elisabeth K., additional, Brady, Sheila M., additional, Yang, Shanna B., additional, Courville, Amber B., additional, Smith, Kevin P., additional, Rosing, Douglas R., additional, Chen, Kong Y., additional, and Yanovski, Jack A., additional

Published
2022
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20. The Effects of Interrupting Sitting Time on Affect and State Anxiety in Children of Healthy Weight and Overweight: A Randomized Crossover Trial.

Author

Zink, Jennifer, Berrigan, David A., Broadney, Miranda M., Shareef, Faizah, Papachristopoulou, Alexia, Brady, Sheila M., Bernstein, Shanna B., Brychta, Robert J., Hattenbach, Jacob D., Tigner, Ira L., Courville, Amber B., Drinkard, Bart E., Smith, Kevin P., Rosing, Douglas R., Wolters, Pamela L., Chen, Kong Y., Yanovski, Jack A., and Belcher, Britni R.

Subjects
ANXIETY, AFFECT (Psychology), BODY weight, CONFIDENCE intervals, CROSSOVER trials, EXPERIMENTAL design, CHILDHOOD obesity, STATISTICAL sampling, SITTING position, TIME, RANDOMIZED controlled trials, TREATMENT effectiveness, PRE-tests & post-tests, SEDENTARY lifestyles, DESCRIPTIVE statistics, CHILDREN
Abstract

Purpose: Sedentary time relates to higher anxiety and more negative affect in children. This study assessed whether interrupting sitting over 3 hours is sufficient to influence state anxiety, positive affect, or negative affect, and tested weight status as a moderator. Methods: Analyses were the second (preplanned) purpose of a larger study. Children (N = 61; age: mean [SD] = 9.5 [1.3]; 43% healthy weight) completed 2 experimental conditions: continuous sitting for 3 hours and sitting for 3 hours interrupted with walking for 3 minutes in every 30 minutes. State anxiety, positive affect, and negative affect were reported at pretest and posttest. Multilevel models for repeated measures assessed whether experimental condition predicted posttest scores. Results: Experimental condition was unrelated to posttest state anxiety or positive affect. Weight status moderated how experimental condition influenced posttest negative affect (P =.003). Negative affect was lower in the children of healthy weight after interrupted sitting (vs continuous sitting; β = −0.8; 95% confidence interval, −1.5 to 0.0, P =.05), but it was higher in the children with overweight/obesity after interrupted sitting (vs continuous sitting; β = 0.6; 95% confidence interval, 0.0 to 1.2, P =.06). Conclusions: Interrupting sitting acutely reduced negative affect in children of healthy weight, but not in children with overweight. Further research is needed to better understand the potential emotional benefits of sitting interruptions in youth. [ABSTRACT FROM AUTHOR]

Published
2020
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27. Prospective phenotyping of long-term survivors of generalized arterial calcification of infancy (GACI)

Author

Ferreira, Carlos R., primary, Hackbarth, Mary E., additional, Ziegler, Shira G., additional, Pan, Kristen S., additional, Roberts, Mary S., additional, Rosing, Douglas R., additional, Whelpley, Margaret S., additional, Bryant, Joy C., additional, Macnamara, Ellen F., additional, Wang, Sisi, additional, Müller, Kerstin, additional, Hartley, Iris R., additional, Chew, Emily Y., additional, Corden, Timothy E., additional, Jacobsen, Christina M., additional, Holm, Ingrid A., additional, Rutsch, Frank, additional, Dikoglu, Esra, additional, Chen, Marcus Y., additional, Mughal, M. Zulf, additional, Levine, Michael A., additional, Gafni, Rachel I., additional, and Gahl, William A., additional

Published
2021
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29. Cardiac toxicity in a pilot study of duvelisib and ibrutinib combination therapy for chronic lymphocytic leukaemia.

Author

Paul, Shira R., Rosing, Douglas R., Haigney, Mark C., Peer, Cody J., Figg, William D., Wiestner, Adrian, and Sun, Clare

Subjects
*LYMPHOCYTIC leukemia, *CARDIOTOXICITY, *CHRONIC leukemia, *BRUTON tyrosine kinase, *ARRHYTHMIA
Abstract

After the first patient's sudden death, EKG, ambulatory EKG, exercise stress test, and echocardiogram were obtained. Keywords: B lymphocytes; Bruton tyrosine kinase inhibitor; cardiology; CLL; non-Hodgkin lymphoma EN B lymphocytes Bruton tyrosine kinase inhibitor cardiology CLL non-Hodgkin lymphoma 261 263 3 01/16/23 20230115 NES 230115 Bruton tyrosine kinase inhibitor (BTKi), ibrutinib, and B-cell lymphoma 2 inhibitor, venetoclax, have revolutionized the treatment of chronic lymphocytic leukaemia (CLL). After review by the Data and Safety Monitoring Board and IRB, patient #3, who had a history of atrial fibrillation (AF) and tachycardia/bradycardia syndrome with pacemaker placement, discontinued duvelisib and ibrutinib after cycle 1. [Extracted from the article]

Published
2023
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35. Association between fetal lymphedema and congenital cardiovascular defects in Turner syndrome

Author

Loscalzo, Melissa L., Van, Phillip L., Ho, Vincent B., Bakalov, Vladimir K., Rosing, Douglas R., Malone, Carol A., Dietz, Harry C., and Bondy, Carolyn A.

Subjects
Turner Syndrome Society of the United States, Turner syndrome -- Complications and side effects, Turner syndrome -- Research, Mitral valve -- Statistics, Aortic coarctation -- Statistics, Fetus -- Growth, X chromosome, Lymphedema, Congenital heart disease
Abstract

Objectives. Turner syndrome (TS) is associated with congenital cardiovascular defects (CCVDs), most commonly bicuspid aortic valve (BAV) and aortic coarctation (COARC), congenital renal anomalies, and fetal lymphedema. It has been theorized that compressive or obstructive effects of fetal lymphedema may actually cause cardiovascular and renal dysmorphogenesis in TS. The objective of this study was to determine whether there is a specific association between a history of fetal lymphedema and CCVDs in monosomy X, or TS, independent of karyotype or general severity of the phenotype. Methods. This was a prospective study of 134 girls and women who have TS (mean age: 30 years) and were clinically evaluated for evidence of fetal lymphedema, classified as central (signified by the presence of neck webbing) or peripheral (current or perinatal, or dysplastic fingernails). The presence of BAV and/or COARC was detected by magnetic resonance imaging combined with echocardiography, and renal anomalies were determined by ultrasound. Results. There is a strong association between developmental central lymphedema, signified by neck webbing, and the presence of BAV ([chi square] = 10) and COARC ([chi square] = 8). The association between webbed neck and CCVDs was independent of karyotype. There was, in contrast, no significant association between renal anomalies and webbed neck or CCVDs. Conclusions. The strong, statistically significant association between neck webbing and the presence of BAV and COARC in TS suggests a pathogenetic connection between fetal lymphatic obstruction and defective aortic development. The presence of neck webbing in TS should alert the clinician to the possibility of congenital cardiovascular defects. Pediatrics 2005;115:732-735; X-chromosome, aortic coarctation, bicuspid aortic valve, lymphedema., ABBREVIATIONS. CCVD, congenital cardiovascular defect; BAV, bicuspid aortic valve; COARC, coarctation; TS, Turner syndrome. Congenital cardiovascular defects (CCVDs), including most commonly bicuspid aortic valve (BAV) and coarctation of the aorta [...]

Published
2005

44. Cardiomyopathy in congenital and acquired generalized lipodystrophy: a clinical assessment

Author

Lupsa, Beatrice C., Sachdev, Vandana, Lungu, Andreea O., Rosing, Douglas R., and Gorden, Phillip

Subjects
Lipodystrophy -- Research, Cardiomyopathy -- Development and progression, Cardiomyopathy -- Diagnosis, Cardiomyopathy -- Research, Heart diseases -- Development and progression, Heart diseases -- Diagnosis, Heart diseases -- Research
Published
2010

50. Prolongation of the cardiac QTc interval in turner syndrome

Author

Bondy, Carolyn A., Van, Phillip L., Bakalov, Vladimir K., Sachdev, Vandana, Malone, Carol A., Ho, Vincent B., and Rosing, Douglas R.

Subjects
Turner syndrome -- Physiological aspects, Turner syndrome -- Complications and side effects, Congenital heart disease -- Causes of, Congenital heart disease -- Physiological aspects, Congenital heart disease -- Risk factors, Long QT syndrome -- Research
Published
2006

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