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5. A multicentre, patient- and assessor-blinded, non-inferiority, randomised and controlled phase II trial to compare standard and torque teno virus-guided immunosuppression in kidney transplant recipients in the first year after transplantation: TTVguideIT

Author

Haupenthal, Frederik, Rahn, Jette, Maggi, Fabrizio, Gelas, Fanny, Bourgeois, Philippe, Hugo, Christian, Jilma, Bernd, Böhmig, Georg A., Herkner, Harald, Wolzt, Michael, Doberer, Konstantin, Vossen, Matthias, Focosi, Daniele, Neuwirt, Hannes, Banas, Miriam, Banas, Bernhard, Budde, Klemens, Viklicky, Ondrej, Malvezzi, Paolo, Rostaing, Lionel, Rotmans, Joris I., Bakker, Stephan J. L., Eller, Kathrin, Cejka, Daniel, Pérez, Alberto Molina, Rodriguez-Arias, David, König, Franz, and Bond, Gregor

Published
2023
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9. Predicting Kidney Failure, Cardiovascular Disease and Death in Advanced CKD Patients

Author

Cupisti, Adamasco, Sagliocca, Adelia, Ferraro, Alberto, Musiała, Aleksandra, Mele, Alessandra, Naticchia, Alessandro, Còsaro, Alex, Woodman, Alistair, Ranghino, Andrea, Stucchi, Andrea, Jonsson, Andreas, Schneider, Andreas, Pignataro, Angelo, Schrander, Anita, Torp, Anke, McKeever, Anna, Szymczak, Anna, Blom, Anna-Lena, De Blasio, Antonella, Pani, Antonello, Tsalouichos, Aris, Ullah, Asad, McLaren, Barbara, van Dam, Bastiaan, Iwig, Beate, Antonio, Bellasi, Di Iorio, Biagio Raffaele, Rogland, Björn, Perras, Boris, Alessandra, Butti, Harron, Camille, Wallquist, Carin, Siegert, Carl, Barrett, Carla, Gaillard, Carlo, Garofalo, Carlo, Abaterusso, Cataldo, Beerenhout, Charles, O'Toole, Charlotte, Somma, Chiara, Marx, Christian, Summersgill, Christina, Blaser, Christof, D'alessandro, Claudia, Emde, Claudia, Zullo, Claudia, Pozzi, Claudio, Geddes, Colin, Verburgh, Cornelis, Bergamo, Daniela, Ciurlino, Daniele, Motta, Daria, Glowski, Deborah, McGlynn, Deborah, Vargas, Denes, Krieter, Detlef, Russo, Domenico, Fuchs, Dunja, Sands, Dympna, Hoogeveen, Ellen, Irmler, Ellen, Dimény, Emöke, Favaro, Enrico, Platen, Eva, Olczyk, Ewelina, Hoorn, Ewout, Vigotti, Federica, Ansali, Ferruccio, Conte, Ferruccio, Cianciotta, Francesca, Giacchino, Francesca, Cappellaio, Francesco, Pizzarelli, Francesco, Sundelin, Fredrik, Uhlin, Fredrik, Greco, Gaetano, Roy, Geena, Porto, Gaetana, Bigatti, Giada, Marinangeli, Giancarlo, Cabiddu, Gianfranca, Hirst, Gillian, Fumagalli, Giordano, Caloro, Giorgia, Piccoli, Giorgina, Capasso, Giovanbattista, Gambaro, Giovanni, Tognarelli, Giuliana, Bonforte, Giuseppe, Conte, Giuseppe, Toscano, Giuseppe, Del Rosso, Goffredo, Welander, Gunilla, Augustyniak-Bartosik, Hanna, Boots, Hans, Schmidt-Gürtler, Hans, King, Hayley, McNally, Helen, Schlee, Hendrik, Boom, Henk, Naujoks, Holger, Masri-Senghor, Houda, Murtagh, Hugh, Rayner, Hugh, Miśkowiec-Wiśniewska, Ilona, Schlee, Ines, Capizzi, Irene, Hernandez, Isabel Bascaran, Baragetti, Ivano, Manitius, Jacek, Turner, Jane, Eijgenraam, Jan-Willem, Kooman, Jeroen, Beige, Joachim, Pondel, Joanna, Wilcox, Joanne, Berdeprado, Jocelyn, Röthele, Jochen, Wong, Jonathan, Rotmans, Joris, Banda, Joyce, Mazur, Justyna, Hahn, Kai, Jędrzejak, Kamila, Nowańska, Katarzyna, Blouin, Katja, Neumeier, Katrin, Jones, Kirsteen, Anding-Rost, Kirsten, Gröntoft, Knut-Christian, Oldrizzi, Lamberto, Haydock, Lesley, Vogt, Liffert, Wilkinson, Lily, Gesualdo, Loreto, Schramm, Lothar, Biancone, Luigi, Nowak, Łukasz, Raasveld, Maarten, Durlik, Magdalena, Magnano, Manuela, Vervloet, Marc, Ricardi, Marco, Carmody, Margaret, Di Bari, Maria, Laudato, Maria, Sirico, Maria Luisa, Stendahl, Maria, Svensson, Maria, Weetman, Maria, van Buren, Marjolijn, Joinson, Martin, Ferraresi, Martina, Dutton, Mary, van Diepen, Merel, Matthews, Michael, Provenzano, Michele, Hopf, Monika, Malaguti, Moreno, Wuttke, Nadja, Morgan, Neal, Palmieri, Nicola, Frischmuth, Nikolaus, Bleakley, Nina, Murrone, Paola, Cockwell, Paul, Leurs, Paul, Roderick, Paul, Voskamp, Pauline, Kashioulis, Pavlos, Ichtiaris, Pawlos, Blankestijn, Peter, Kirste, Petra, Schulz, Petra, Mason, Phil, Kalra, Philip, Cirillo, Pietro, Dattolo, Pietro, Acampora, Pina, Sajith, Rincy, Nigro, Rita, Boero, Roberto, Scarpioni, Roberto, Sicoli, Rosa, Malandra, Rosella, Aign, Sabine, Cäsar, Sabine, van Esch, Sadie, Chapman, Sally, Biribauer, Sandra, Navjee, Santee, Crosbie, Sarah, Brown, Sharon, Tickle, Sheila, Manan, Sherin, Röser, Silke, Savoldi, Silvana, Bertoli, Silvio, Borrelli, Silvio, Boorsma, Siska, Heidenreich, Stefan, Melander, Stefan, Maxia, Stefania, Maffei, Stefano, Mangano, Stefano, Palm, Stephanie, Konings, Stijn, Mathavakkannan, Suresh, Schwedler, Susanne, Delrieux, Sylke, Renker, Sylvia, Schättel, Sylvia, Dorota, Szyszkowska, Cicchetti, Teresa, Nieszporek, Teresa, Stephan, Theresa, Schmiedeke, Thomas, Weinreich, Thomas, Leimbach, Til, Rappa, Tiziana, Almquist, Tora, Stövesand, Torsten, Bahner, Udo, Jensen, Ulrika, Palazzo, Valentina, De Simone, Walter, Seeger, Wolfgang, Kuan, Ying, Heleniak, Zbigniew, Aydin, Zeynep, Ramspek, Chava L., Boekee, Rosemarijn, Evans, Marie, Heimburger, Olof, Snead, Charlotte M., Caskey, Fergus J., Torino, Claudia, Szymczak, Maciej, Krajewska, Magdalena, Drechsler, Christiane, Wanner, Christoph, Chesnaye, Nicholas C., Jager, Kitty J., Dekker, Friedo W., Snoeijs, Maarten G.J., and Rotmans, Joris I.

Published
2022
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10. Regional Variation in Hemoglobin Distribution Among Individuals With CKD: the ISN International Network of CKD Cohorts

Author

Ahn, Curie, Berger, Stefan P., Caskey, Fergus J., Cho, Min Hyun, Cho, Heeyeon, Dekker, Friedo W., Diwan, Vishal, Drechsler, Christiane, Eckardt, Kai-Uwe, Evans, Marie, Ferreiro, Alejandro, Floege, Jürgen, Gadola, Liliana, Haller, Hermann, Han, Kyung Hee, Healy, Helen G., Heerspink, Hiddo Lambers, Hemmelder, Marc, Hiemstra, Thomas, Hilbrands, Luuk, Kim, Seong Heon, Klyprayong, Pinkaew, Köttgen, Anna, Kronenberg, Florian, Lamadrid, Veronica, Lee, Joo Hoo, Mark, Patrick, Matheson, Matt, Mi, Eun, Noppakun, Kajohnsak, Oefner, Peter, Panaput, Thanachai, Park, Young Seo, Prokosch, Hans-Ulrich, Reis, André, Rios, Pablo, Rosivall, Laszlo, Rotmans, Joris I., Sackeyfio, Alfred, Sangthawan, Pornpen, Schmid, Matthias, Shin, Jae Il, Silavarino, Ricardo, Sitter, Thomas, Sommerer, Claudia, Szymczak, Maciej, Torino, Claudia, Toth, Janos, van Ittersum, Frans J., Venuthurupalli, Sree Krishna, Verhaar, Marianne C., Wang, Zaimin, Wanner, Christoph, Wiecek, Andrzej, Wolf, Gunter, de Zeeuw, Dick, Zhang, Luxia, Zheng, Yuyan, Zhao, Ming-Hui, Zietse, Robert, Canney, Mark, Induruwage, Dilshani, Tang, Mila, Alencar de Pinho, Natalia, Er, Lee, Zhao, Yinshan, Djurdjev, Ognjenka, Ahn, Yo Han, Behnisch, Rouven, Calice-Silva, Viviane, Chesnaye, Nicholas C., de Borst, Martin H., Dember, Laura M., Dionne, Janis, Ebert, Natalie, Eder, Susanne, Fenton, Anthony, Fukagawa, Masafumi, Furth, Susan L., Hoy, Wendy E., Imaizumi, Takahiro, Jager, Kitty J., Jha, Vivekanand, Kang, Hee Gyung, Kitiyakara, Chagriya, Mayer, Gert, Oh, Kook-Hwan, Onu, Ugochi, Pecoits-Filho, Roberto, Reichel, Helmut, Richards, Anna, Schaefer, Franz, Schaeffner, Elke, Scheppach, Johannes B., Sola, Laura, Ulasi, Ifeoma, Wang, Jinwei, Yadav, Ashok K., Zhang, Jianzhen, Feldman, Harold I., Taal, Maarten W., Stengel, Bénédicte, and Levin, Adeera

Published
2023
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11. Potassium and the kidney: a reciprocal relationship with clinical relevance

Author

Wieërs, Michiel L. A. J., Mulder, Jaap, Rotmans, Joris I., and Hoorn, Ewout J.

Subjects
Chronic kidney failure -- Risk factors -- Development and progression, Potassium in the body -- Physiological aspects -- Health aspects, Health
Abstract

By controlling urinary potassium excretion, the kidneys play a key role in maintaining whole-body potassium homeostasis. Conversely, low urinary potassium excretion (as a proxy for insufficient dietary intake) is increasingly recognized as a risk factor for the progression of kidney disease. Thus, there is a reciprocal relationship between potassium and the kidney: the kidney regulates potassium balance but potassium also affects kidney function. This review explores this relationship by discussing new insights into kidney potassium handling derived from recently characterized tubulopathies and studies on sexual dimorphism. These insights reveal a central but non-exclusive role for the distal convoluted tubule in sensing potassium and subsequently modifying the activity of the sodium-chloride cotransporter. This is another example of reciprocity: activation of the sodium-chloride cotransporter not only reduces distal sodium delivery and therefore potassium secretion but also increases salt sensitivity. This mechanism helps explain the well-known relationship between dietary potassium and blood pressure. Remarkably, in children, blood pressure is related to dietary potassium but not sodium intake. To explore how potassium deficiency can cause kidney injury, we review the mechanisms of hypokalemic nephropathy and discuss if these mechanisms may explain the association between low dietary potassium intake and adverse kidney outcomes. We discuss if potassium should be repleted in patients with kidney disease and what role dietary potassium plays in the risk of hyperkalemia. Supported by data and physiology, we reach the conclusion that we should view potassium not only as a potentially dangerous cation but also as a companion in the battle against kidney disease., Author(s): Michiel L. A. J. Wieërs [sup.1] , Jaap Mulder [sup.2] [sup.3] , Joris I. Rotmans [sup.4] , Ewout J. Hoorn [sup.1] Author Affiliations: (1) grid.5645.2, 000000040459992X, Department of Internal [...]

Published
2022
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20. Autologous Mandril-Based Vascular Grafts

Author

Geelhoed, Wouter J., Moroni, Lorenzo, Rotmans, Joris I., Walpoth, Beat H., editor, Bergmeister, Helga, editor, Bowlin, Gary L., editor, Kong, Deling, editor, Rotmans, Joris I., editor, and Zilla, Peter, editor

Published
2020
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23. Nutritional Management of Chronic Kidney Disease

Author

Hoorn, Ewout J, Vogt, Liffert, and Rotmans, Joris I

Subjects
Humans, Kidney Failure, Chronic, Renal Insufficiency, Chronic, Medical and Health Sciences, General & Internal Medicine, Biomedical and clinical sciences, Health sciences
Published
2018

33. An Implantable Magnetic Drive Mechanism for Non-Invasive Arteriovenous Conduit Blood Flow Control

Author

White, Nicholas A., van der Kroft, Sander L., van der Bogt, Koen E.A., Vrielink, Timo J.C. Oude, Camenzuli, Christian, Calleja-Agius, Jean, Sanchez-Margallo, Juan A., Sanchez-Margallo, Francisco M., van de Stadt, Huybert J.F., Dankelman, Jenny, Rotmans, Joris I., and Horeman, Tim

Abstract

Objective: Hemodialysis patients usually receive an arteriovenous fistula (AVF) in the arm as vascular access conduit to allow dialysis 2–3 times a week. This AVF introduces the high flow necessary for dialysis, but over time the ever-present supraphysiological flow is the leading cause of complications. This study aims to develop an implantable device able to non-invasively remove the high flow outside dialysis sessions. Methods: The developed prototype features a magnetic ring allowing external coupling and torque transmission to non-invasively control an AVF valve. Mock-up devices were implanted into arm and sheep cadavers to test sizes and locations. The transmission torque, output force, and valve closure are measured for different representative skin thicknesses. Results: The prototype was placed successfully into arm and sheep cadavers. In the prototype, a maximum output force of 78.9 ± 4.2 N, 46.7 ± 1.9 N, 25.6 ± 0.7 N, 13.5 ± 0.6 N and 6.3 ± 0.4 N could be achieved non-invasively through skin thicknesses of 1–5 mm respectively. The fistula was fully collapsible in every measurement through skin thickness up to the required 4 mm. Conclusion: The prototype satisfies the design requirements. It is fully implantable and allows closure and control of an AVF through non-invasive torque transmission. In vivo studies are pivotal in assessing functionality and understanding systemic effects. Significance: A method is introduced to transfer large amounts of energy to a medical implant for actuation of a mechanical valve trough a closed surface. This system allows non-invasive control of an AVF to reduce complications related to the permanent high flow in conventional AVFs.

Published
2024
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35. SGLT2 inhibition promotes glomerular repopulation by cells of renin lineage in experimental kidney disease

Author

van der Pluijm, Loïs A.K., Koudijs, Angela, Stam, Wendy, Roelofs, Joris J.T.H., Danser, A. H.Jan, Rotmans, Joris I., Gross, Kenneth W., Pieper, Michael P., van Zonneveld, Anton Jan, Bijkerk, Roel, van der Pluijm, Loïs A.K., Koudijs, Angela, Stam, Wendy, Roelofs, Joris J.T.H., Danser, A. H.Jan, Rotmans, Joris I., Gross, Kenneth W., Pieper, Michael P., van Zonneveld, Anton Jan, and Bijkerk, Roel

Abstract

Aim: Sodium glucose co-transporter-2 (SGLT2) inhibitors stimulate renal excretion of sodium and glucose and exert renal protective effects in patients with (non-)diabetic chronic kidney disease (CKD) and may as well protect against acute kidney injury (AKI). The mechanism behind this kidney protective effect remains unclear. Juxtaglomerular cells of renin lineage (CoRL) have been demonstrated to function as progenitors for multiple adult glomerular cell types in kidney disease. This study assesses the impact of SGLT2 inhibition on the repopulation of glomerular cells by CoRL and examines their phenotypic commitment. Methods: Experiments were performed in Ren1cre-tdTomato lineage-trace mice. Either 5/6 nephrectomy (5/6NX) modeling CKD or bilateral ischaemia reperfusion injury (bIRI) mimicking AKI was applied, while the SGLT2 inhibitor empagliflozin (10 mg/kg) was administered daily via oral gavage for 14 days. Results: Both 5/6NX and bIRI-induced kidney injury increased the number of glomerular CoRL-derived cells. SGLT2 inhibition improved kidney function after 5/6NX, indicated by decreased blood creatinine and urea levels, but not after bIRI. In line with this, empagliflozin in 5/6NX animals resulted in less glomerulosclerosis, while it did not affect histopathological features in bIRI. Treatment with empagliflozin resulted in an increase in the number of CoRL-derived glomerular cells in both 5/6NX and bIRI conditions. Interestingly, SGLT2 inhibition led to more CoRL-derived podocytes in 5/6NX animals, whereas empagliflozin-treated bIRI mice presented with increased levels of parietal epithelial and mesangial cells derived from CoRL. Conclusion: We conclude that SGLT2 inhibition by empagliflozin promotes CoRL-mediated glomerular repopulation with selective CoRL-derived cell types depending on the type of experimental kidney injury. These findings suggest a previously unidentified mechani

Published
2024

39. Chronic kidney disease increases the susceptibility to negative effects of low and high potassium intake.

Author

Gritter, Martin, Wei, Kuang-Yu, Wouda, Rosa D, Musterd-Bhaggoe, Usha M, Dijkstra, Kyra L, Kers, Jesper, Ramakers, Christian, Vogt, Liffert, Borst, Martin H de, Danser, Alexander H J, Hoorn, Ewout J, and Rotmans, Joris I

Subjects
CHRONIC kidney failure, DISEASE susceptibility, RENAL fibrosis, KIDNEY physiology, GLOMERULAR filtration rate
Abstract

Background Dietary potassium (K+) has emerged as a modifiable factor for cardiovascular and kidney health in the general population, but its role in people with chronic kidney disease (CKD) is unclear. Here, we hypothesize that CKD increases the susceptibility to the negative effects of low and high K+ diets. Methods We compared the effects of low, normal and high KChloride (KCl) diets and a high KCitrate diet for 4 weeks in male rats with normal kidney function and in male rats with CKD using the 5/6th nephrectomy model (5/6Nx). Results Compared with rats with normal kidney function, 5/6Nx rats on the low KCl diet developed more severe extracellular and intracellular K+ depletion and more severe kidney injury, characterized by nephromegaly, infiltration of T cells and macrophages, decreased estimated glomerular filtration rate and increased albuminuria. The high KCl diet caused hyperkalemia, hyperaldosteronism, hyperchloremic metabolic acidosis and severe hypertension in 5/6Nx but not in sham rats. The high KCitrate diet caused hypochloremic metabolic alkalosis but attenuated hypertension despite higher abundance of the phosphorylated sodium chloride cotransporter (pNCC) and similar levels of plasma aldosterone and epithelial sodium channel abundance. All 5/6Nx groups had more collagen deposition than the sham groups and this effect was most pronounced in the high KCitrate group. Plasma aldosterone correlated strongly with kidney collagen deposition. Conclusions CKD increases the susceptibility to negative effects of low and high K+ diets in male rats, although the injury patterns are different. The low K+ diet caused inflammation, nephromegaly and kidney function decline, whereas the high K+ diet caused hypertension, hyperaldosteronism and kidney fibrosis. High KCitrate attenuated the hypertensive but not the pro-fibrotic effect of high KCl, which may be attributable to K+-induced aldosterone secretion. Our data suggest that especially in people with CKD it is important to identify the optimal threshold of dietary K+ intake. [ABSTRACT FROM AUTHOR]

Published
2024
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40. Correction to: Outcomes of parathyroidectomy versus calcimimetics for secondary hyperparathyroidism and kidney transplantation: a propensity-matched analysis

Author

Koh, Ezra Y., van der Plas, Willemijn Y., Dulfer, Roderick R., Pol, Robert A., Kruijff, Schelto, Rotmans, Joris I., Appelman-Dijkstra, Natasha, Schepers, Abbey, de Borst, Martin H., Hoorn, Ewout J., van Ginhoven, Tessa M., Nieveen van Dijkum, Els J. M., Vogt, Liffert, and Engelsman, Anton F.

Published
2022
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41. FLOW: Flow dysfunction of hemodialysis vascular access: A randomized controlled trial on the effectiveness of surveillance of arteriovenous fistulas and grafts

Author

Zomer, Bianca, primary, Ruiter, Matthijs S, additional, Dekker, Friedo W, additional, Goertz, Ellen GD, additional, de Haan, Michiel W, additional, Hemmelder, Marc HH, additional, Hiligsmann, Mickaël JC, additional, Konijn, Wanda S, additional, van Loon, Magda M, additional, Maessen, José MC, additional, Mees, Barend ME, additional, Rotmans, Joris I, additional, Schurink, Geert WH, additional, Vleugels, Marie-José JPJ, additional, and Snoeijs, Maarten GJ, additional

Published
2024
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43. Nephrology Partnership for Advancing Technology in Healthcare (N-PATH) program: the teachers’ perspective

Author

Lomonte, Carlo, primary, Rossini, Michele, additional, Ibeas, Josè, additional, Forcella, Mauro, additional, Ponikvar, Jadranka Buturovic, additional, Gallieni, Maurizio, additional, Russo, Roberto, additional, Goumenos, Dimitris, additional, Tesar, Vladimir, additional, Hruskova, Zdenka, additional, Roelofs, Joris, additional, Florquin, Sandrine, additional, Snoeijs, Maarten, additional, Giusto, Antonio, additional, Shemesh, David, additional, Rotmans, Joris, additional, Torra, Roser, additional, Wanner, Christoph, additional, and Gesualdo, Loreto, additional

Published
2023
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47. Chronic kidney disease increases the susceptibility to negative effects of low and high potassium intake

Author

Gritter, Martin, primary, Wei, Kuang-Yu, additional, Wouda, Rosa D, additional, Musterd-Bhaggoe, Usha M, additional, Dijkstra, Kyra L, additional, Kers, Jesper, additional, Ramakers, Christian, additional, Vogt, Liffert, additional, de Borst, Martin H, additional, Danser, Alexander H J, additional, Hoorn, Ewout J, additional, and Rotmans, Joris I, additional

Published
2023
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49. Regional Variation in Hemoglobin Distribution Among Individuals With CKD: the ISN International Network of CKD Cohorts

Author

Canney, Mark, primary, Induruwage, Dilshani, additional, Tang, Mila, additional, Alencar de Pinho, Natalia, additional, Er, Lee, additional, Zhao, Yinshan, additional, Djurdjev, Ognjenka, additional, Ahn, Yo Han, additional, Behnisch, Rouven, additional, Calice-Silva, Viviane, additional, Chesnaye, Nicholas C., additional, de Borst, Martin H., additional, Dember, Laura M., additional, Dionne, Janis, additional, Ebert, Natalie, additional, Eder, Susanne, additional, Fenton, Anthony, additional, Fukagawa, Masafumi, additional, Furth, Susan L., additional, Hoy, Wendy E., additional, Imaizumi, Takahiro, additional, Jager, Kitty J., additional, Jha, Vivekanand, additional, Kang, Hee Gyung, additional, Kitiyakara, Chagriya, additional, Mayer, Gert, additional, Oh, Kook-Hwan, additional, Onu, Ugochi, additional, Pecoits-Filho, Roberto, additional, Reichel, Helmut, additional, Richards, Anna, additional, Schaefer, Franz, additional, Schaeffner, Elke, additional, Scheppach, Johannes B., additional, Sola, Laura, additional, Ulasi, Ifeoma, additional, Wang, Jinwei, additional, Yadav, Ashok K., additional, Zhang, Jianzhen, additional, Feldman, Harold I., additional, Taal, Maarten W., additional, Stengel, Bénédicte, additional, Levin, Adeera, additional, Ahn, Curie, additional, Berger, Stefan P., additional, Caskey, Fergus J., additional, Cho, Min Hyun, additional, Cho, Heeyeon, additional, Dekker, Friedo W., additional, Diwan, Vishal, additional, Drechsler, Christiane, additional, Eckardt, Kai-Uwe, additional, Evans, Marie, additional, Ferreiro, Alejandro, additional, Floege, Jürgen, additional, Gadola, Liliana, additional, Haller, Hermann, additional, Han, Kyung Hee, additional, Healy, Helen G., additional, Heerspink, Hiddo Lambers, additional, Hemmelder, Marc, additional, Hiemstra, Thomas, additional, Hilbrands, Luuk, additional, Kim, Seong Heon, additional, Klyprayong, Pinkaew, additional, Köttgen, Anna, additional, Kronenberg, Florian, additional, Lamadrid, Veronica, additional, Lee, Joo Hoo, additional, Mark, Patrick, additional, Matheson, Matt, additional, Mi, Eun, additional, Noppakun, Kajohnsak, additional, Oefner, Peter, additional, Panaput, Thanachai, additional, Park, Young Seo, additional, Prokosch, Hans-Ulrich, additional, Reis, André, additional, Rios, Pablo, additional, Rosivall, Laszlo, additional, Rotmans, Joris I., additional, Sackeyfio, Alfred, additional, Sangthawan, Pornpen, additional, Schmid, Matthias, additional, Shin, Jae Il, additional, Silavarino, Ricardo, additional, Sitter, Thomas, additional, Sommerer, Claudia, additional, Szymczak, Maciej, additional, Torino, Claudia, additional, Toth, Janos, additional, van Ittersum, Frans J., additional, Venuthurupalli, Sree Krishna, additional, Verhaar, Marianne C., additional, Wang, Zaimin, additional, Wanner, Christoph, additional, Wiecek, Andrzej, additional, Wolf, Gunter, additional, de Zeeuw, Dick, additional, Zhang, Luxia, additional, Zheng, Yuyan, additional, Zhao, Ming-Hui, additional, and Zietse, Robert, additional

Published
2023
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