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4. Hypertensionologist' hypertension. Multiple-salvo manual BP recordings-medical harakiri procedures must be banned by legistlations via ESH, ISH and WHO. This phenomenon may be named Sagliker Effect (SE)

Author

Sagliker, Y., Paylar, N., Ocal, F., Yildiz, I., Sagliker, H. S., Ozkaynak, P. Sagliker, Sagliker, C., and Çukurova Üniversitesi

Abstract

18th Scientific Meeting of the European-Society-of-Hypertension/22nd Scientific Meeting of the International-Society-of-Hypertension -- JUN 14-19, 2008 -- Berlin, GERMANY WOS: 000257197003419 … Euorpean Soc Hyperten, Deutsch Hochdruckliga

Published
2008

6. Sagliker effect(SE) and hypertensionologist hypertension (HH) in medical doctors and nurses. Multiple-salvo manual BP recordings which are medical Harakiri procedures must be banned by legislation and via ESH, ISH and WHO

Author

Sagliker, Y, Balal, M, Sagliker, C, Ozkaynak, PS, Paylar, N, Sagliker, HS, Paydas, S, and Çukurova Üniversitesi

Abstract

15th European Meeting on Hypertension -- JUN 17-21, 2005 -- Milan, ITALY WOS: 000230639802470 … European Soc Hypertens, AstraZeneca, Bristol-Myers Squibb Co, Boehringer Ingelheim, MSD, NOVARTIS, RECORDATI, SANKYO, Sanofi Aventis, Bayer Healthcare AG, Pfizer Inc, Solvay Pharmaceut GmbH

Published
2005

8. 867 A NEW PHENOMENON

Author

Ocal, F., primary, Sagliker, Y., additional, Paylar, N., additional, Sagliker, C., additional, Inal, T., additional, Kolasin, P., additional, Ozkaynak, P.S., additional, and Sagliker, H.S., additional

Published
2012
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10. Mineral and bone disease - CKD 1-5

Author

Loh, Z. Y., primary, Yap, C. W., additional, Anantharaman, V., additional, How, P., additional, Hirata, M., additional, Aizawa, K., additional, Yogo, K., additional, Tashiro, Y., additional, Takeda, S., additional, Endo, K., additional, Fukagawa, M., additional, Serizawa, K.-I., additional, Fujii, H., additional, Kono, K., additional, Nakai, K., additional, Goto, S., additional, Shinohara, M., additional, Kitazawa, R., additional, Kitazawa, S., additional, Nishi, S., additional, Oruc, A., additional, Korkmaz, S., additional, Bal, O., additional, Yilmaztepe Oral, A., additional, Ersoy, A., additional, Gullulu, M., additional, Ketteler, M., additional, Martin, K., additional, Amdahl, M., additional, Cozzolino, M., additional, Goldsmith, D., additional, Sharma, A., additional, Khan, S., additional, Chitalia, N., additional, Afzali, B., additional, Edozie, F., additional, Manghat, P., additional, Wierzbicki, A., additional, Hampson, G., additional, Corradini, M., additional, Iannuzzella, F., additional, Manenti, L., additional, Ciarrocchi, A., additional, Albertazzi, L., additional, Somenzi, D., additional, Pasquali, S., additional, Calabria Baxmann, A., additional, Barcellos Menon, V., additional, Froeder, L., additional, Medina-Pestana, J. O., additional, Barbosa Carvalho, A., additional, Pfeferman Heilberg, I., additional, Sola, L., additional, De Souza, N., additional, Flores, J., additional, Perico, N., additional, Yuste, C., additional, Garcia DE Vinuesa, M. S., additional, Luno, J., additional, Goicoechea, M. A., additional, Barraca, D., additional, Panizo, N., additional, Quiroga, B., additional, Kim, S. M., additional, Kwon, S. K., additional, Kim, H.-Y., additional, Cournoyer, S., additional, Bell, R., additional, Berbiche, D., additional, Menard, L., additional, Viaene, L., additional, Evenepoel, P., additional, Meijers, B., additional, Overbergh, L., additional, Mathieu, C., additional, Pasquali, M., additional, Rotondi, S., additional, Conte, C., additional, Pirro, G., additional, Mazzaferro, S., additional, Frasheri, A., additional, Marangella, M., additional, Tartaglione, L., additional, Park, J.-S., additional, Koo, T. Y., additional, Kim, G.-H., additional, Kang, C. M., additional, Lee, C.-H., additional, Hiemstra, T. F., additional, Casian, A., additional, Boraks, P., additional, Jayne, D., additional, Schoenmakers, I., additional, Schmiedeke, B., additional, Niemann, M., additional, Schmiedeke, D., additional, Davydenko, I., additional, Emmert, A., additional, Pilz, S., additional, Obermayer-Pietsch, B., additional, Weidemann, F., additional, Breunig, F., additional, Wanner, C., additional, Drechsler, C., additional, Shiizaki, K., additional, Ito, C., additional, Onishi, A., additional, Nakazawa, E., additional, Ogura, M., additional, Kusano, E., additional, Ermolenko, V., additional, Mikhaylova, N., additional, Vartanjan, K., additional, Levchuk, D., additional, Dobrina, E., additional, Capusa, C., additional, Stancu, S., additional, Maria, D., additional, Vladu, I., additional, Barsan, L., additional, Garneata, L., additional, Mota, E., additional, Mircescu, G., additional, Ilyes, A., additional, Dorobantu, N., additional, Petrescu, L., additional, Martinez-Gallardo, R., additional, Ferreira, F., additional, Garcia-Pino, G., additional, Luna, E., additional, Caravaca, F., additional, De Jager, D. J., additional, Grootendorst, D. C., additional, Postmus, I., additional, De Goeij, M. C. M., additional, Boeschoten, E. W., additional, Sijpkens, Y. W. J., additional, Dekker, F. W., additional, Halbesma, N., additional, Wuthrich, R. P., additional, Covic, A., additional, Gaillard, S., additional, Rakov, V., additional, Louvet, L., additional, Buchel, J., additional, Steppan, S., additional, Passlick-Deetjen, J., additional, Massy, Z. A., additional, Akalin, N., additional, Altiparmak, M. R., additional, Trabulus, S., additional, Yalin, A. S., additional, Seyahi, N., additional, Ataman, R., additional, Serdengecti, K., additional, Donate-Correa, J., additional, Martinez-Sanz, R., additional, Muros-de-Fuentes, M., additional, Garcia, J., additional, Garcia, P., additional, Cazana, V., additional, Mora-Fernandez, C., additional, Navarro-Gonzalez, J. F., additional, Berutti, S., additional, Marranca, D., additional, Soragna, G., additional, Erroi, L., additional, Migliardi, M., additional, Belloni, L., additional, Parmeggiani, M., additional, Camerini, C., additional, Pezzotta, M., additional, Zani, R., additional, Movilli, E., additional, Cancarini, G., additional, Anwar, S., additional, Pruthi, R., additional, Kenchayikoppad, S., additional, Reyes, J., additional, Dasilva, I., additional, Furlano, M., additional, Calero, F., additional, Montanes, R., additional, Ayasreh, N., additional, Del Pozo, M., additional, Estorch, M., additional, Rousaud, F., additional, Ballarin, J. A., additional, Bover, J., additional, Resende, A., additional, Dias, C. B., additional, Dos Reis, L., additional, Jorgetti, V., additional, Woronik, V., additional, Panuccio, V., additional, Enia, G., additional, Tripepi, R., additional, Cutrupi, S., additional, Pizzini, P., additional, Aliotta, R., additional, Zoccali, C., additional, Yildiz, I., additional, Sagliker, Y., additional, Demirhan, O., additional, Tunc, E., additional, Inandiklioglu, N., additional, Tasdemir, D., additional, Acharya, V., additional, Zhang, L., additional, Golea, O., additional, Sabry, A., additional, Ookalkar, D., additional, Radulescu, D., additional, Ben Maiz, H., additional, Chen, C. H., additional, Rome, J. P., additional, Benzegoutta, M., additional, Paylar, N., additional, Eyupoglu, K., additional, Karatepe, E., additional, Esenturk, M., additional, Yavascan, O., additional, Grzegorzevska, A., additional, Shilo, V., additional, M-Mazdeh, M., additional, Francesco, R. C., additional, Gouda, Z., additional, Adam, S. M., additional, Emir, I., additional, Ocal, F., additional, Usta, E., additional, Kiralp, N., additional, Sagliker, C., additional, S Ozkaynak, P., additional, Sagliker, H. S., additional, Bassuoni, M., additional, El-Wakil, H. S., additional, Akar, H., additional, Yenicerioglu, Y., additional, Kose, E., additional, and Sekin, O., additional

Published
2012
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12. International evaluation of unrecognizably uglifying human faces in late and severe secondary hyperparathyroidism in chronic kidney disease. Sagliker syndrome. A unique catastrophic entity, cytogenetic studies for chromosomal abnormalities, calcium-sensing receptor gene and GNAS1 mutations. Striking and promising missense mutations on the GNAS1 gene exons 1, 4, 10, 4.

Author

Yildiz I, Sagliker Y, Demirhan O, Tunc E, Inandiklioglu N, Tasdemir D, Acharya V, Zhang L, Golea O, Sabry A, Ookalkar DS, Capusa C, Radulescu D, Garneata L, Mircescu G, Ben Maiz H, Chen CH, Prado Rome J, Benzegoutta M, Paylar N, Eyuboglu K, Karatepe E, Esenturk M, Yavascan O, Grzegorzevska A, Shilo V, Mazdeh MM, Francesco RC, Gouda Z, Adam SM, Emir I, Ocal F, Usta E, Kiralp N, Sagliker C, Ozkaynak PS, Sagliker HS, Bassuoni M, and Sekin O

Subjects
Chromogranins, Exons genetics, Humans, Hyperparathyroidism, Secondary pathology, Hyperparathyroidism, Secondary physiopathology, Syndrome, Facial Bones pathology, GTP-Binding Protein alpha Subunits, Gs genetics, Hyperparathyroidism, Secondary genetics, Kidney Failure, Chronic complications, Mutation, Missense genetics, Receptors, Calcium-Sensing genetics
Abstract

Hypotheses explaining pathogenesis of secondary hyperparathyroidism (SH) in late and severe CKD as a unique entity called Sagliker syndrome (SS) are still unclear. This international study contains 60 patients from Turkey, India, Malaysia, China, Romania, Egypt, Tunisia, Taiwan, Mexico, Algeria, Poland, Russia, and Iran. We examined patients and first degree relatives for cytogenetic chromosomal abnormalities, calcium sensing receptor (Ca SR) genes in exons 2 and 3 abnormalities and GNAS1 genes mutations in exons 1, 4, 5, 7, 10, 13. Our syndrome could be a new syndrome in between SH, CKD, and hereditary bone dystrophies. We could not find chromosomal abnormalities in cytogenetics and on Ca SR gene exons 2 and 3. Interestingly, we did find promising missense mutations on the GNAS1 gene exons 1, 4, 10, 4. We finally thought that those catastrophic bone diseases were severe SH and its late treatments due to monetary deficiencies and iatrogenic mistreatments not started as early as possible. This was a sine qua non humanity task. Those brand new striking GNAS1 genes missense mutations have to be considered from now on for the genesis of SS., (Copyright © 2012 National Kidney Foundation, Inc. Published by Elsevier Inc. All rights reserved.)

Published
2012
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13. International study on Sagliker syndrome and uglifying human face appearance in severe and late secondary hyperparathyroidism in chronic kidney disease patients.

Author

Sagliker Y, Acharya V, Ling Z, Golea O, Sabry A, Eyupoglu K, Ookalkar DS, Tapiawala S, Durugkar S, Khetan P, Capusa C, Univar R, Yildiz I, Cengiz K, Bali M, Ozkaynak PS, Sagliker HS, Paylar N, Adam SM, Balal M, Paydas S, Demirhan O, Tasdemir D, Ben Maiz H, Redulescu D, Garneata L, Mircescu G, Hong-Liang R, Lun L, Yildizer K, Emir I, Yuksekgonul M, Yenicerioglu Y, Akar H, Sagliker C, Esenturk M, and Kiralp N

Subjects
Adult, Body Height, Facial Bones abnormalities, Female, Humans, Hyperparathyroidism, Secondary epidemiology, Kidney Failure, Chronic psychology, Male, Siblings, Skull anatomy & histology, Spine abnormalities, Face abnormalities, Hyperparathyroidism, Secondary psychology, Kidney Failure, Chronic complications, Mental Disorders epidemiology
Abstract

Objective: It is known that skeletal changes due to secondary hyperparathyroidism (SH) can be severe in chronic kidney disease (CKD). Recently described Sagliker syndrome (SS) is a very striking and prominent feature of SH in CKD, including an uglifying appearance to the face, short stature, extremely severe maxillary and mandibulary changes, soft tissue in the mouth, teeth/dental abnormalities, fingertip changes, knee and scapula deformities, hearing abnormalities, and neurological and, more important, severe psychological problems., Design, Setting, Patients: In the past 8 years, we have encountered 40 cases of SS in SH and CKD by performing an international study in Turkey, India, Romania, Egypt, Maleysia, Tunis, and China., Results: The medical history of these patients showed that they did not receive proper therapy. Changes, particularly in children and teenagers, become irreversible, which was disastrous for the patients both aesthetically and psychologically., Conclusion: Treatment must begin early and be the appropriate treatment given in centers with sophisticated skills. Otherwise, the inability to correct all the changes in the skull and face, to remodel a new face, to extending the height, and, most important, to convince the patients to face the dramatic psychological problems can be catastrophic for those patients.

Published
2008
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14. Neurologic manifestations in Sagliker syndrome: uglifying human face appearance in severe and late secondary hyperparathyroidism in chronic renal failure patients.

Author

Giray S, Sagliker Y, Yildiz I, Halvaci I, Paylar N, Ocal F, Balal M, Ozkaynak PS, Paydas S, Sagliker C, Sagliker HS, Kiralp N, Adam SM, Esenturk M, Gocmez E, Taskapan H, Yuksekgonul M, Emir I, Guler T, Yakar H, Sekin O, Kayali E, Caliskan S, Eskiocak AF, Ogruk B, Guzelyurt T, and Kurt C

Subjects
Adolescent, Adult, Cranial Nerve Diseases epidemiology, Fatigue, Female, Headache epidemiology, Humans, Kidney Failure, Chronic therapy, Male, Mental Disorders epidemiology, Nervous System Diseases complications, Nervous System Diseases diagnosis, Polyneuropathies epidemiology, Renal Dialysis, Syndrome, Bone Diseases complications, Facial Bones, Hyperparathyroidism, Secondary complications, Kidney Failure, Chronic complications, Nervous System Diseases epidemiology
Abstract

Patients with chronic renal failure (CRF) often have signs and symptoms related to fluid and electrolyte disturbances, anemia, malnutrition, bone disease, and gastrointestinal problems. Vascular and neurologic impairment in particular remain an important source of morbidity and mortality in this vulnerable patient population. Sagliker syndrome is a novel syndrome that was recently described in 2004 in patients with CRF and severe and late secondary hyperparathyroidism who suffered from severe skull and facial bone changes, particularly from uglifying human face appearances and neuropsychiatric disorders. The goal of this study was to assess neuropsychiatric manifestations occurring in CRF patients with Sagliker syndrome. Four female and 8 male patients with CRF on regular dialysis at the hemodialysis units of the Internal Medicine Departments around southern Turkey participated in the study. All patients underwent a clinical neurologic examination performed by the same neurologist. Neuropsychiatric signs and symptoms were found in all cases. The results showed that the most frequent neurologic manifestations in CRF patients with Sagliker syndrome were headache, polyneuropathy, cranial neuropathy, fatigue, and psychiatric disorders.

Published
2006
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15. Cephalometric evaluation of patients with Sagliker syndrome: uglifying human face appearance in severe and late secondary hyperparathyroidism in chronic renal failure patients.

Author

Uzel A, Uzel I, Sagliker Y, Yildiz I, Halvaci I, Paylar N, Ocal F, Balal M, Ozkaynak PS, Paydas S, Sagliker C, Sagliker HS, Kiralp N, Adam SM, Esenturk M, Gocmez E, Taskapan H, Yuksekgonul M, Emir I, Guler T, Yakar H, Sekin O, Kayali E, Caliskan S, Eskiocak AF, Ogruk B, Guzelyurt T, and Kurt C

Subjects
Adolescent, Adult, Female, Humans, Male, Malocclusion, Angle Class II diagnosis, Malocclusion, Angle Class II epidemiology, Syndrome, Bone Diseases etiology, Cephalometry, Facial Bones, Hyperparathyroidism, Secondary complications, Kidney Failure, Chronic complications
Abstract

It is well known that secondary hyperparathyroidism may be an extremely severe condition in chronic renal failure, and almost all patients with chronic kidney disease, even in the well-developed countries, encounter every kind of bone abnormalities if they are not treated properly. Although some sporadic cases have been reported of unique facial bone changes, the largest collection of this phenomenon has been reported by Sagliker et al. We also have found 6 of 9 patients who have these changes (Sagliker syndrome) to manifest class II malocclusion of the upper and lower jaws according to dental universally accepted criteria by performing cephalometric studies, x-ray plain films, tomographic procedures, and drawing technology.

Published
2006
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16. Sagliker syndrome: uglifying human face appearance in late and severe secondary hyperparathyroidism in chronic renal failure.

Author

Sagliker Y, Balal M, Sagliker Ozkaynak P, Paydas S, Sagliker C, Sabit Sagliker H, Kiralp N, Mumin Adam S, Tuncer I, Gonlusen G, Esenturk M, Gocmez E, Taskapan H, Yeksan M, Kobaner E, Ozkaya O, Yuksekgonul M, Emir I, Cengiz N, Onder Isik I, Bilginer O, Guler T, Yakar H, Sarsmaz N, Dilaver S, Akoglu B, Basgumus M, and Chirik E

Subjects
Age Factors, Child, Female, Humans, Male, Severity of Illness Index, Syndrome, Abnormalities, Multiple, Face abnormalities, Facial Bones abnormalities, Hyperparathyroidism, Secondary complications, Kidney Failure, Chronic complications
Abstract

Almost every patient with chronic renal failure (CRF) eventually develops secondary hyperparathyroidism (SH) unless they are treated with proper and novel medications in advanced medical centers by skilled medical personnel. Every kind of bone abnormality including skull deformities has been described in detail by almost every concerned researcher and textbook, but descriptions of this phenomenon are limited in the medical literature to the years from 1973 to 1977. To our knowledge, extensive data regarding uglifying human face appearances have not been defined so far in the literature. We are therefore making this addition to the clinical nephrology field by accumulating such data. After we found 2 consecutive peculiar and unique patients with uglifying human face appearances in 2000, we attempted to inform and draw attention to this new entity to all hemodialysis (HD) centers in Turkey, as well as in other developing countries around the world to collect data on this phenomenon. Accordingly, we visited dialysis centers and patients' houses to collect detailed information, including medical clinical histories, physical examinations, laboratory data, biographies, current medications, and so forth. We found 25 patients who had CRF, SH, short stature, extremely severe skull changes, maxillary and mandibular bone changes, teeth/dental abnormalities, and soft and innocuous tumoral tissues in the mouth (hence, uglifying the appearance of the face), fingertip changes, severe psychologic problems, and depression. It appears that patients with CRF may have a new syndrome of bone deformities that have long been neglected, ignored, and forgotten since the mid-1970s when they were first described. This is vital and critical information for the clinical status of patients who suffered from the syndrome that we have named Sagliker syndrome (SS), and we believe there are many more patients in the world who are suffering from it.

Published
2004
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