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43 results on '"Sakatani, Tomohiko"'

2. Impact of physician interpretation of at-home ECG on clinically-detected AF

Author

Senoo, Keitaro, primary, Yukawa, Arito, additional, Iwakoshi, Hibiki, additional, Nishimura, Tetsuro, additional, Shimoo, Satoshi, additional, Inoue, Keiji, additional, Sakatani, Tomohiko, additional, Kakita, Ken, additional, Hattori, Tetsuhisa, additional, Kitajima, Hiroki, additional, Nakai, Kentaro, additional, Nishiuchi, Suguru, additional, Nakata, Mitsuko, additional, Teramukai, Satoshi, additional, Shiraishi, Hirokazu, additional, and Matoba, Satoaki, additional

Published
2024
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7. The impact of home electrocardiograph measurement rate on the detection of atrial fibrillation recurrence after ablation: A prospective multicenter observational study

Author

Senoo, Keitaro, primary, Yukawa, Arito, additional, Ohkura, Takashi, additional, Iwakoshi, Hibiki, additional, Nishimura, Tetsuro, additional, Shimoo, Satoshi, additional, Inoue, Keiji, additional, Sakatani, Tomohiko, additional, Kakita, Ken, additional, Hattori, Tetsuhisa, additional, Kitajima, Hiroki, additional, Nakai, Kentaro, additional, Nishiuchi, Suguru, additional, Nakata, Mitsuko, additional, Teramukai, Satoshi, additional, Shiraishi, Hirokazu, additional, and Matoba, Satoaki, additional

Published
2023
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11. IVUS-Guided Wiring Improves the Clinical Outcomes of Angioplasty for Long Femoropopliteal CTO Compared with the Conventional Intraluminal Approach

Author

Tsubakimoto, Yoshinori, primary, Isodono, Koji, additional, Fujimoto, Tomotaka, additional, Kirii, Yosuke, additional, Shiraga, Akiko, additional, Kasahara, Takeru, additional, Ariyoshi, Makoto, additional, Irie, Daisuke, additional, Sakatani, Tomohiko, additional, Matsuo, Akiko, additional, Inoue, Keiji, additional, and Fujita, Hiroshi, additional

Published
2021
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13. Enhanced activity of ventricular [Na.sup.+]-HC[O.sup.-.sub.3] cotransport in pressure overload hypertrophy

Author

Yamamoto, Taku, Shirayama, Takeshi, Sakatani, Tomohiko, Takahashi, Tomosaburo, Tanaka, Hideo, Takamatsu, Tetsuro, Spitzer, Kenneth W., and Matsubara, Hiroaki

Subjects
Heart enlargement -- Physiological aspects, Heart cells -- Physiological aspects, Messenger RNA -- Properties, Polymerase chain reaction -- Observations, Hydrogen-ion concentration -- Measurement, Biological sciences
Abstract

The [Na.sup.+]-HC[O.sup.-.sub.3] cotransporter (NBC) plays a key role in intracellular pH (p[H.sub.i] regulation in normal ventricular muscle. However, the state of NBC in nonischemic hypertrophied hearts is unresolved. In this study, we examined functional and molecular properties of NBC in adult rat ventricular myocytes. The cells were enzymatically isolated from both normal and hypertrophied hearts. Ventricular hypertrophy was induced by pressure overload created by suprarenal abdominal aortic constriction of 50% for 7 wk. p[H.sub.i] was measured in single cells using the fluorescent pH indicator 2',7'-bis(2-carboxyethyl)5-(6)carboxy-fluorescein. Real-time PCR analysis was used to quantitatively assess expression of NBC-encoding mRNA, including SLC4A4 (encoding electrogenic NBC, NBCel) and SLC4A7 (electroneutral NBC, NBCnl). Our results demonstrate that: 1) mRNA levels of both the electrogenic NBCel (SLC4A4) and electroneutral NBCnl (SLC4A7) forms of NBC were increased by aortic constriction, 2) the onset of NBC upregulation occurred within 3 days after constriction, 3) normal and hypertrophied ventricles displayed regional differences in NBC expression, 4) acid extrusion via NBC ([J.sub.NBC]) was increased significantly in hypertrophied myocytes, 5) although acid extrusion via [Na.sup.+]/[H.sup.+] exchange was also increased in hypertrophied myocytes, the relative enhancement of [J.sub.NBC] was larger, 6) membrane depolarization markedly increased [J.sub.NBC] in hypertrophied myocytes, and 7) losartan, an ANG II A[T.sub.1] receptor antagonist, significantly attenuated the upregulation of both NBCs induced by 3 wk of aortic constriction. Enhanced NBC activity during hypertrophic development provides a mechanism for intracellular [Na.sup.+] overload, which may render the ventricles more vulnerable to [Ca.sup.2+] overload during ischemia-reperfusion. cardiac myocytes; intracellular pH regulation; messenger ribonucleic acid; polymerase chain reaction

Published
2007

15. The frequency and clinical characteristics of in-stent restenosis due to calcified nodule development after coronary stent implantation

Author

Isodono, Koji, primary, Fujii, Kenichi, additional, Fujimoto, Tomotaka, additional, Kasahara, Takeru, additional, Ariyoshi, Makoto, additional, Irie, Daisuke, additional, Tsubakimoto, Yoshinori, additional, Sakatani, Tomohiko, additional, Matsuo, Akiko, additional, Inoue, Keiji, additional, and Fujita, Hiroshi, additional

Published
2020
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16. Diagnostic Value of Atrial Fibrillation by Built-in Electrocardiogram Technology in a Blood Pressure Monitor

Author

Senoo, Keitaro, primary, Miki, Tomonori, additional, Okura, Takashi, additional, Shiraishi, Hirokazu, additional, Shirayama, Takeshi, additional, Inoue, Keiji, additional, Sakatani, Tomohiko, additional, Kakita, Ken, additional, Hattori, Tetsuhisa, additional, Nakai, Kentaro, additional, Ikeda, Takanori, additional, and Matoba, Satoaki, additional

Published
2020
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20. Cholesterol crystal depth in coronary atherosclerotic plaques: A novel index of plaque vulnerability using optical frequency domain imaging

Author

Koide, Masahiro, primary, Matsuo, Akiko, additional, Shimoo, Satoshi, additional, Takamatsu, Kazuaki, additional, Kyodo, Atsushi, additional, Tsuji, Yumika, additional, Mera, Kayoko, additional, Tsubakimoto, Yoshinori, additional, Isodono, Koji, additional, Sakatani, Tomohiko, additional, Inoue, Keiji, additional, and Fujita, Hiroshi, additional

Published
2017
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21. Successful Complete Revascularization With PCI Using Super-Low Volume of Contrast Medium in a Patient With Three-Vessel Disease Including 2 Chronic Total Occlusions With Severe Renal Dysfunction

Author

Koide, Masahiro, primary, Kato, Masayuki, additional, Usui, Kimihito, additional, Mera, Kayoko, additional, Tsubakimoto, Yoshinori, additional, Isodono, Koji, additional, Sakatani, Tomohiko, additional, Matsuo, Akiko, additional, Inoue, Keiji, additional, and Fujita, Hiroshi, additional

Published
2017
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22. Visualization of the improvement of myocardial perfusion after coronary intervention using motorized fractional flow reserve pullback curve

Author

Matsuo, Akiko, primary, Shimoo, Satoshi, additional, Takamatsu, Kazuaki, additional, Tsuji, Yumika, additional, Kyodo, Atsushi, additional, Mera, Kayoko, additional, Koide, Masahiro, additional, Isodono, Koji, additional, Tsubakimoto, Yoshinori, additional, Sakatani, Tomohiko, additional, Inoue, Keiji, additional, and Fujita, Hiroshi, additional

Published
2016
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30. A case report: 201Tl/99mTc-Pyrophosphate dual-isotope myocardial SPECT for detecting annular subendocardial infarction induced by a transient shock

Author

Tanaka, Tetsuya, primary, Fujita, Hiroshi, additional, Seto, Shinji, additional, Yamamoto, Kazuto, additional, Kondou, Morihiko, additional, Ootomo, Toshiyuki, additional, Shimoda, Yoshiaki, additional, Makita, Naoki, additional, Matsuo, Kiyonari, additional, Shiono, Yasutsugu, additional, Tsubakimoto, Yoshinori, additional, Sakatani, Tomohiko, additional, Matsuo, Akiko, additional, Inoue, Keiji, additional, and Kitamura, Makoto, additional

Published
2010
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41. Enhanced activity of ventricular Na+-HCO-3 cotransport in pressure overload hypertrophy.

Author

Yamamoto, Taku, Shirayama, Takeshi, Sakatani, Tomohiko, Takahashi, Tomosaburo, Tanaka, Hideo, Takamatsu, Tetsuro, Spitzer, Kenneth W., and Matsubara, Hiroaki

Subjects
SODIUM cotransport systems, HYPERTROPHY, MESSENGER RNA, AORTA abnormalities, FLUORESCENCE
Abstract

The Na+-HCO+ cotransporter (NBC) plays a key role in intracellular pH (pHi) regulation in normal ventricular muscle. However, the state of NBC in nonischemic hypertrophied hearts is unresolved. In this study, we examined functional and molecular properties of NBC in adult rat ventricular myocytes. The cells were enzymatically isolated from both normal and hypertrophied hearts. Ventricular hypertrophy was induced by pressure overload created by suprarenal abdominal aortic constriction of 50% for 7 wk. pHi was measured in single cells using the fluorescent pH indicator 2′,7′-bis(2-carboxyethyl)5-(6)carboxyfluorescein. Real-time PCR analysis was used to quantitatively assess expression of NBC-encoding mRNA, including SLC4A4 (encoding electrogenic NBC, NBCeI) and SLC4A7 (electroneutral NBC, NBCn1). Our results demonstrate that: 1) mRNA levels of both the electrogenic NBCeI (SLC4A4) and electroneutral NBCn1 (SLC4A7) forms of NBC were increased by aortic constriction, 2) the onset of NBC upregulation occurred within 3 days after constriction, 3) normal and hypertrophied ventricles displayed regional differences in NBC expression, 4) acid extrusion via NBC (JNBC) was increased significantly in hypertrophied myocytes, 5) although acid extrusion via Na+/H+ exchange was also increased in hypertrophied myocytes, the relative enhancement of JNBC was larger, 6) membrane depolarization markedly increased JNBC in hypertrophied myocytes, and 7) losartan, an ANG II AT1 receptor antagonist, significantly attenuated the upregulation of both NBCs induced by 3 wk of aortic constriction. Enhanced NBC activity during hypertrophic development provides a mechanism for intracellular Na+ overload, which may render the ventricles more vulnerable to Ca2+ overload during ischemia-reperfusion. [ABSTRACT FROM AUTHOR]

Published
2007
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42. A case report: 201Tl/99mTc-Pyrophosphate dual-isotope myocardial SPECT for detecting annular subendocardial infarction induced by a transient shock.

Author

Tanaka T, Fujita H, Seto S, Yamamoto K, Kondou M, Ootomo T, Shimoda Y, Makita N, Matsuo K, Shiono Y, Tsubakimoto Y, Sakatani T, Matsuo A, Inoue K, and Kitamura M

Abstract

The patient was a 74-year-old woman with angina pectoris, who had undergone percutaneous coronary intervention with stent placement in the right coronary artery on October 2, 2007. On November 12 of the same year, she suffered from paroxysmal atrial fibrillation. She was treated with pilsicainide hydrochloride administered by intravenous injection, which was followed by a sudden sinus standstill, with marked bradycardia and a shock state. The patient was then treated with a catecholamine, however, the shock state persisted for about an hour. An electrocardiogram revealed persistent ST depression in leads V 4-6 along with elevation of the serum creatinine kinase. A coronary angiography performed on the admission day revealed no abnormality. On the third hospital day, a dual-isotope myocardial SPECT using 201 Tl and 99m Tc-pyrophosphate demonstrated an annular accumulation of 99m Tc-pyrophosphate concordant with the endocardium from apex to the mid-portion of the left ventricle, suggestive of subendocardial infarction. The case is reported here, as there are few reports of subendocardial infarction developing due to ischemia arising from a shock state.

Published
2016
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43. [Chronic pulmonary thromboembolism originating from left forearm deep vein thrombosis: a case report].

Author

Mani H, Sakatani T, Hadase M, Kawasaki T, Kamitani T, Kawasaki S, and Sugihara H

Subjects
Aged, Chronic Disease, Female, Humans, Forearm blood supply, Pulmonary Embolism etiology, Venous Thrombosis complications
Abstract

Upper extremity deep venous thromboembolism has become increasingly common due to the use of subclavian venous access. However, forearm deep venous thrombosis is rare. We report a case of chronic pulmonary thromboembolism originating from left forearm deep venous thrombosis. A 66-year-old woman was admitted to our hospital because of dyspnea, which had worsened over the previous 2 months. Echocardiography showed severe right ventricular enlargement and pulmonary hypertension. Perfusion lung scintigraphy revealed multisegmental perfusion defects. No likely factors responsible for hypercoagulability were found. The patient had a history of left radial fracture and had been treated with external fixation. Venography of the upper and lower extremities revealed total occlusion of the deep veins of the left forearm with collateral vessels, but no evidence of other upper or lower extremity venous thrombosis.

Published
2002

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