Search

Your search keyword '"Salzmann, Karin"' showing total 21 results
Start Over "Salzmann, Karin"
21 results on '"Salzmann, Karin"'

6. Empagliflozin protects mice against diet-induced obesity, insulin resistance and hepatic steatosis

Author

Radlinger, Bernhard, primary, Ress, Claudia, additional, Folie, Sabrina, additional, Salzmann, Karin, additional, Lechuga, Ana, additional, Weiss, Bernhard, additional, Salvenmoser, Willi, additional, Graber, Michael, additional, Hirsch, Jakob, additional, Holfeld, Johannes, additional, Kremser, Christian, additional, Moser, Patrizia, additional, Staudacher, Gabriele, additional, Jelenik, Tomas, additional, Roden, Michael, additional, Tilg, Herbert, additional, and Kaser, Susanne, additional

Published
2022
Full Text
View/download PDF

13. Metabolic effects of reduced growth hormone action in fatty liver disease

Author

Rufinatscha, Kerstin, primary, Ress, Claudia, additional, Folie, Sabrina, additional, Haas, Simone, additional, Salzmann, Karin, additional, Moser, Patrizia, additional, Dobner, Jochen, additional, Weiss, Guenter, additional, Iruzubieta, Paula, additional, Arias-Loste, María Teresa, additional, Crespo, Javier, additional, Tilg, Herbert, additional, and Kaser, Susanne, additional

Published
2018
Full Text
View/download PDF

14. Fat-enriched rather than high-fructose diets promote whitening of adipose tissue in a sex-dependent manner

Author

Dobner, Jochen, primary, Ress, Claudia, additional, Rufinatscha, Kerstin, additional, Salzmann, Karin, additional, Salvenmoser, Willi, additional, Folie, Sabrina, additional, Wieser, Verena, additional, Moser, Patrizia, additional, Weiss, Guenter, additional, Goebel, Georg, additional, Tilg, Herbert, additional, and Kaser, Susanne, additional

Published
2017
Full Text
View/download PDF

18. Serum prolactin in advanced chronic liver disease

Author

Ress, Claudia, primary, Maeser, Pirmin, additional, Tschoner, Alexander, additional, Loacker, Lorin, additional, Salzmann, Karin, additional, Staudacher, Gabriele, additional, Melmer, Andreas, additional, Zoller, Heinz, additional, Vogel, Wolfgang, additional, Griesmacher, Andrea, additional, Tilg, Herbert, additional, Graziadei, Ivo, additional, and Kaser, Susanne, additional

Published
2014
Full Text
View/download PDF

19. CDTect-RIA and CDTect-EIA for determination of serum carbohydrate-deficient transferrin compared.

Author

Arndt, Torsten, Kropf, Juergen, Brandt, Ragnhild, Gressner, Axel M., Hackler, Rolf, Herold, Manfred, Van Pelt, Johannes, Mårtensson, Ola, Salzmann, Karin, and Velmans, Mathieu H.

Subjects
SERUM, TRANSFERRIN, RADIOIMMUNOASSAY, ENZYME-linked immunosorbent assay, LABORATORIES
Abstract

CDTect-RIA and CDTect-EIA for determination of serum carbohydrate-deficient transferrin (CDT) by radioimmunossay and enzyme immunoassay respectively were tested for equality and precision in four European laboratories. For correlational studies, serum samples with CDT concentrations up to 130 U/I were analysed in accordance with a uniform trial schedule. The regression of CDT values obtained by the two procedures was computed for each laboratory using the method of Passing and Bablok. Slopes and intercepts of the regression functions did not differ significantly from the values 1 or 0, as proved by the corresponding 95% confidence intervals. Precision studies were computed using analysis of variance. For CDT concentrations at the upper reference limit for men, the within-day coefficients of variation (CVs) ranged between 0.7 and 6.4% (median 5.2%) for CDTect-RIA and from 4.3 to 9.2% (median 6.2%) for CDTect-EIA. The corresponding pure between-day CVs were 5.0-18.5% (median 9.8%) and 3.5-14.5% (median 10.9%). The study demonstrates the equality of CDT values obtained by CDTect-RIA and CDTect-EIA. According to this study, the two methods can be used interchangeably without getting fluctuating CDT values, e.g. in longitudinal studies. [ABSTRACT FROM PUBLISHER]

Published
1998
Full Text
View/download PDF

20. Effect of Obesity and Insulin Sensitivity on Adiponectin Isoform Distribution.

Author

Kaser, Susanne, Tatarczyk, Tobias, Salzmann, Karin, Stadlmayr, Andreas, Iglseder, Bernhard, Mackevics, Vitolds, Ciardi, Christian, Engl, Julia, Ress, Claudia, Laimer, Markus, Sandhofer, Anton, Weber, Bernhard Paul, Ebenbichler, Christoph F., and Patsch, Josef R.

Subjects
OBESITY, INSULIN resistance, ADIPOSE tissues, BLOOD plasma, REGRESSION analysis, OVERWEIGHT persons
Abstract

Adiponectin is an insulin-sensitizing, antiatherogenic and antiinflammatory adipocytokine that circulates in plasma in three isoforms: a trimer (low-molecular weight (LMW)), a hexamer (trimer-dimer) of medium-molecular weight (MMW) and a multimeric high-molecular weight (HMW) isoform. Evidence suggests that HMW adiponectin is the active isoform the adipocytokine. We investigated the impact of adipose tissue and insulin sensitivity on adiponectin isoform distribution. Altogether 187 normolipidemic, non-diabetic, female and male, lean and obese subjects with and without insulin resistance participating in the Salzburg Atherosclerosis Prevention program in subjects at High Individual Risk (SAPHIR) were included in the study. Insulin sensitivity was determined by the short insulin tolerance test and the homeostasis model assessment (HOMA) index. Serum adiponectin isoform distribution was determined by ELISA. Total adiponectin as well as HMW/total adiponectin ratio was significantly increased in female subjects. Circulating adiponectin declined with raising body mass index due to reductions in HMW isoform. As determined by stepwise regression analysis, besides age and HDL cholesterol, amount of visceral adipose tissue and waist-to-hip ratio predicted concentration of HMW adiponectin, while insulin sensitivity had no influence on either total adiponectin or its isoforms. Our data suggest that the HMW isoform of adiponectin is a more sensitive marker of cardiovascular risk than total adiponectin concentration. Visceral adipose tissue mass - but not insulin sensitivity - is a strong determinant of adiponectin secretion so that we hypothesize that insulin resistance is rather a consequence than the cause of hypoadiponectinemia in obese subjects. [ABSTRACT FROM AUTHOR]

Published
2007

Catalog

Books, media, physical & digital resources
See catalog results