1,500 results on '"Santoro, Massimo"'
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2. Exploiting the metabolic vulnerability of circulating tumour cells
3. Experimental and in silico evaluations of the possible molecular interaction between airborne particulate matter and SARS-CoV-2
4. How to Generate a Vascular-Labelled Transgenic Zebrafish Model to Study Tumor Angiogenesis and Extravasation
5. Oxidative pentose phosphate pathway controls vascular mural cell coverage by regulating extracellular matrix composition
6. Zebrafish from the Alps to Sicily: The 4th Italian Zebrafish Meeting in Palermo.
7. Time to fight: targeting the circadian clock molecular machinery in cancer therapy
8. Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
9. Correction to: Overexpression of the HMGA2 gene in transgenic mice leads to the onset of pituitary adenomas
10. The importance of the RET gene in thyroid cancer and therapeutic implications
11. Pathogenesis of Thyroid Cancer
12. YAP/TAZ–TEAD link angiogenesis to nutrients
13. Compound heterozygosity for an expanded (GAA) and a (GAAGGA) repeat at FXN locus: from a diagnostic pitfall to potential clues to the pathogenesis of Friedreich ataxia
14. Targeted activity of the small molecule kinase inhibitor Pz-1 towards RET and TRK kinases
15. Pathogenesis of Thyroid Carcinoma
16. Role of amino acid metabolism in angiogenesis
17. Sclerotome-derived vascular smooth muscle progenitors contribute to the haematopoietic stem cell specification niche
18. D898_E901 RET Deletion Is Oncogenic, Responds to Selpercatinib, and Treatment Resistance Can Arise Via RET-Independent Mechanisms
19. Serum-Circulating microRNAs in Sporadic Inclusion Body Myositis
20. Efficient clofilium tosylate-mediated rescue of POLG-related disease phenotypes in zebrafish
21. Oncogene-induced senescence and its evasion in a mouse model of thyroid neoplasia
22. Promising rodent models in Parkinson's disease
23. Evaluation of post-natal angiogenesis in a mouse hind limb ischemia model
24. The circadian protein BMAL1 supports endothelial cell cycle during angiogenesis
25. Real-time quantification of subcellular H2O2 and glutathione redox potential in living cardiovascular tissues
26. The molecular basis for RET tyrosine-kinase inhibitors in thyroid cancer
27. Onset of Natural Killer Cell Lymphomas in Transgenic Mice Carrying a Truncated HMGI-C Gene by the Chronic Stimulation of the IL-2 and IL-15 Pathway
28. The binding of 14-3-3 proteins to the Ron receptor is required for its biological activity
29. The RET Receptor Family
30. Supplementary Figure 1 from Activation of the mTOR Pathway in Primary Medullary Thyroid Carcinoma and Lymph Node Metastases
31. Supplementary Tables 1-3 from Activation of the mTOR Pathway in Primary Medullary Thyroid Carcinoma and Lymph Node Metastases
32. Supplementary Figure 1 from A Cell Proliferation and Chromosomal Instability Signature in Anaplastic Thyroid Carcinoma
33. Supplementary Table 4 from A Cell Proliferation and Chromosomal Instability Signature in Anaplastic Thyroid Carcinoma
34. Data from CD44 Proteolysis Increases CREB Phosphorylation and Sustains Proliferation of Thyroid Cancer Cells
35. Supplementary Figure 3 from CD44 Proteolysis Increases CREB Phosphorylation and Sustains Proliferation of Thyroid Cancer Cells
36. Data from Identification of Polo-like Kinase 1 as a Potential Therapeutic Target in Anaplastic Thyroid Carcinoma
37. Supplementary Table 5 from A Cell Proliferation and Chromosomal Instability Signature in Anaplastic Thyroid Carcinoma
38. Supplementary Table 2 from A Cell Proliferation and Chromosomal Instability Signature in Anaplastic Thyroid Carcinoma
39. Supplementary Figure 1 from CD44 Proteolysis Increases CREB Phosphorylation and Sustains Proliferation of Thyroid Cancer Cells
40. Supplementary Table 1 from A Cell Proliferation and Chromosomal Instability Signature in Anaplastic Thyroid Carcinoma
41. Supplementary Figure 2 from Loss of the CBX7 Gene Expression Correlates with a Highly Malignant Phenotype in Thyroid Cancer
42. Supplementary Table 3 from A Cell Proliferation and Chromosomal Instability Signature in Anaplastic Thyroid Carcinoma
43. Supplementary Figure 2 from CD44 Proteolysis Increases CREB Phosphorylation and Sustains Proliferation of Thyroid Cancer Cells
44. Supplementary Figure Legends 1-5 from CD44 Proteolysis Increases CREB Phosphorylation and Sustains Proliferation of Thyroid Cancer Cells
45. Supplementary Methods and Figure Legends 1-2 from Loss of the CBX7 Gene Expression Correlates with a Highly Malignant Phenotype in Thyroid Cancer
46. Supplementary Methods from Identification of Polo-like Kinase 1 as a Potential Therapeutic Target in Anaplastic Thyroid Carcinoma
47. Supplementary Figure 4 from CD44 Proteolysis Increases CREB Phosphorylation and Sustains Proliferation of Thyroid Cancer Cells
48. Supplementary Figure 1 from Identification of Polo-like Kinase 1 as a Potential Therapeutic Target in Anaplastic Thyroid Carcinoma
49. Supplementary Figure 5 from CD44 Proteolysis Increases CREB Phosphorylation and Sustains Proliferation of Thyroid Cancer Cells
50. Supplementary Figure 1 from Loss of the CBX7 Gene Expression Correlates with a Highly Malignant Phenotype in Thyroid Cancer
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