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2. Disease Mechanisms

3. Autoimmune Myasthenia Gravis

8. Sex-specific analysis in Behçet's disease reveals higher genetic risk in male patients

9. Disease Mechanisms

16. Genetic Association of a Gain‐of‐Function IFNGR1 Polymorphism and the Intergenic Region LNCAROD/DKK1 With Behçet’s Disease

19. Immune alterations in subacute sclerosing panencephalitis reflect an incompetent response to eliminate the measles virus

20. Identification of susceptibility loci for Takayasu arteritis through a large multi-ancestral genome-wide association study

27. CD4+ T Cells of Myasthenia Gravis Patients Are Characterized by Increased IL-21, IL-4, and IL-17A Productions and Higher Presence of PD-1 and ICOS

35. Corrigendum to: A genome-wide association study identifies nucleotide variants at SIGLEC5 and DEFA1A3 as risk loci for periodontitis

37. Correction: Corrigendum: Analysis of the common genetic component of large-vessel vasculitides through a meta-Immunochip strategy

39. CD4+ T Cells of Myasthenia Gravis Patients Are Characterized by Increased IL-21, IL-4, and IL-17A Productions and Higher Presence of PD-1 and ICOS.

40. A genome-wide association study identifies nucleotide variants at SIGLEC5 and DEFA1A3 as risk loci for periodontitis

41. A genome-wide association study identifies nucleotide variants at SIGLEC5 and DEFA1A3 as risk loci for periodontitis

43. A putative functional variant within the ubiquitin-associated domain-containing protein 2 gene (UBAC2) is associated with increased risk of Behçet’s disease

44. cFLIP overexpression in T cells in thymoma‐associated myasthenia gravis

45. Differential Cytokine Changes in Patients with Myasthenia Gravis with Antibodies against AChR and MuSK

48. A Decrease of Regulatory T Cells and Altered Expression of NK Receptors Are Observed in Subacute Sclerosing Panencephalitis

50. Late-onset myasthenia gravis – CTLA4low genotype association and low-for-age thymic output of naïve T cells

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