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1. Fine-mapping analysis including over 254,000 East Asian and European descendants identifies 136 putative colorectal cancer susceptibility genes

2. Multiomic Network Analysis Identifies Dysregulated Neurobiological Pathways in Opioid Addiction

3. Probing the diabetes and colorectal cancer relationship using gene – environment interaction analyses

5. Convergence of case-specific epigenetic alterations identify a confluence of genetic vulnerabilities tied to opioid overdose

6. Robust Hi-C Maps of Enhancer-Promoter Interactions Reveal the Function of Non-coding Genome in Neural Development and Diseases

7. Cell Type-Specific Intralocus Interactions Reveal Oligodendrocyte Mechanisms in MS

8. Two genome-wide interaction loci modify the association of nonsteroidal anti-inflammatory drugs with colorectal cancer

9. Two genome-wide interaction loci modify the association of nonsteroidal anti-inflammatory drugs with colorectal cancer

10. Fine-mapping analysis including over 254 000 East Asian and European descendants identifies 136 putative colorectal cancer susceptibility genes

11. Functional Enhancers Shape Extrachromosomal Oncogene Amplifications

12. Ex vivo screen identifies CDK12 as a metastatic vulnerability in osteosarcoma

13. Genetic variant predictors of gene expression provide new insight into risk of colorectal cancer

16. Correction to: Genetic variant predictors of gene expression provide new insight into risk of colorectal cancer

18. Discovery of common and rare genetic risk variants for colorectal cancer

19. Data from A Genetic Locus within the FMN1/GREM1 Gene Region Interacts with Body Mass Index in Colorectal Cancer Risk

20. Supplementary Data from A Genetic Locus within the FMN1/GREM1 Gene Region Interacts with Body Mass Index in Colorectal Cancer Risk

21. Table 2 from A Genetic Locus within the FMN1/GREM1 Gene Region Interacts with Body Mass Index in Colorectal Cancer Risk

22. Table 1 from A Genetic Locus within the FMN1/GREM1 Gene Region Interacts with Body Mass Index in Colorectal Cancer Risk

24. A Genetic Locus within the FMN1/GREM1 Gene Region Interacts with Body Mass Index in Colorectal Cancer Risk

25. Genome-wide interaction study with smoking for colorectal cancer risk identifies novel genetic loci related to tumor suppression, inflammation, and immune response

26. Positively selected enhancer elements endow osteosarcoma cells with metastatic competence

27. Therapeutic targeting of ependymoma as informed by oncogenic enhancer profiling

28. A Genetic Locus within the FMN1/GREM1 Gene Region Interacts with Body Mass Index in Colorectal Cancer Risk

29. Oncogenic reactivation of young L1s is a hallmark of colon cancer

30. Supplementary Figure 2 from mTOR Inhibition Mitigates Enhanced mRNA Translation Associated with the Metastatic Phenotype of Osteosarcoma Cells In Vivo

31. Supplementary Figure 1 from mTOR Inhibition Mitigates Enhanced mRNA Translation Associated with the Metastatic Phenotype of Osteosarcoma Cells In Vivo

32. Supplementary_Methods from mTOR Inhibition Mitigates Enhanced mRNA Translation Associated with the Metastatic Phenotype of Osteosarcoma Cells In Vivo

33. Data from Chemotherapy-Induced Distal Enhancers Drive Transcriptional Programs to Maintain the Chemoresistant State in Ovarian Cancer

34. Supplementary Table 1 from mTOR Inhibition Mitigates Enhanced mRNA Translation Associated with the Metastatic Phenotype of Osteosarcoma Cells In Vivo

35. Supplementary Data from Chemotherapy-Induced Distal Enhancers Drive Transcriptional Programs to Maintain the Chemoresistant State in Ovarian Cancer

36. Transcription elongation factors represent in vivo cancer dependencies in glioblastoma

37. Genome-wide Interaction Study with Smoking for Colorectal Cancer Risk Identifies Novel Genetic Loci Related to Tumor Suppression, Inflammation, and Immune Response

39. Cellular Maturation of Oligodendrocytes is Governed by Transient Gene Melting

41. Probing the diabetes and colorectal cancer relationship using gene – environment interaction analyses

45. Epigenomic Enhancer Profiling Defines a Signature of Colon Cancer

46. Convergence of case-specific epigenetic alterations identify a confluence of genetic vulnerabilities tied to opioid overdose

49. Beyond GWAS of Colorectal Cancer: Evidence of Interaction with Alcohol Consumption and Putative Causal Variant for the 10q24.2 Region

50. Correction: Corrigendum: Positively selected enhancer elements endow osteosarcoma cells with metastatic competence

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