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2. Herausgeber

4. Oxytocin receptor gene methylation as a molecular marker for severity of depressive symptoms in affective disorder patients

7. Associations Between Attention-Deficit/Hyperactivity Disorder and Various Eating Disorders: A Swedish Nationwide Population Study Using Multiple Genetically Informative Approaches

8. Genome-wide association study identifies eight risk loci and implicates metabo-psychiatric origins for anorexia nervosa

9. Was heißt signifikant – geheilt oder nur etwas besser? Ein Vergleich verschiedener Berechnungsmethoden zur Bewertung von Veränderungen der depressiven Symptomatik bei ambulanten Rehabilitanden

12. Corrigendum to ‘BDNF gene polymorphisms predicting treatment response to CBT-based rehabilitation of depression’ European Neuropsychopharmacology Volume 58, May 2022, Pages 103-108

13. BDNF gene polymorphisms predicting treatment response to CBT-based rehabilitation of depression

16. Additional file 2 of Oxytocin receptor gene methylation as a molecular marker for severity of depressive symptoms in affective disorder patients

17. Additional file 1 of Oxytocin receptor gene methylation as a molecular marker for severity of depressive symptoms in affective disorder patients

18. Additional file 3 of Oxytocin receptor gene methylation as a molecular marker for severity of depressive symptoms in affective disorder patients

19. Oxytocin Receptor Gene Methylation as a Molecular Marker for Severity of Depressive Symptoms in Affective Disorder Patients

20. The “DGPPN-Cohort”: a national collaboration initiative by the German Association for Psychiatry and Psychotherapy (DGPPN) for establishing a large-scale cohort of psychiatric patients

22. Monoamino Oxidase A Gene Single-Nucleotide Polymorphisms and Methylation Status and the Risk of Violent Suicide Attempts in Affective Disorder Patients

23. The impact of Cytochrome P450 CYP1A2, CYP2C9, CYP2C19 and CYP2D6 genes on suicide attempt and suicide risk—a European multicentre study on treatment-resistant major depressive disorder

24. Shared genetic risk between eating disorder- and substance-use-related phenotypes: Evidence from genome-wide association studies

26. Genetic identification of cell types underlying brain complex traits yields insights into the etiology of Parkinson’s disease

27. Shared genetic risk between eating disorder- and substance-use-related phenotypes:Evidence from genome-wide association studies

30. Shared genetic risk between eating disorder‐ and substance‐use‐related phenotypes: Evidence from genome‐wide association studies

33. Analysis of shared heritability in common disorders of the brain

34. Early maladaptive schemas as predictors for depression severity and therapy outcome in ambulant psychiatric rehabilitation

35. Exploration of Shared Genetic Architecture Between Subcortical Brain Volumes and Anorexia Nervosa

37. Results of the European Group for the Study of Resistant Depression (GSRD) — basis for further research and clinical practice

38. Significant locus and metabolic genetic correlations revealed in genome-wide association study of anorexia nervosa

40. Associations Between Attention-Deficit/Hyperactivity Disorder and Various Eating Disorders: A Swedish Nationwide Population Study Using Multiple Genetically Informative Approaches

41. Shared Genetic Risk between Eating Disorder- and Substance-Use-Related Phenotypes: Evidence from Genome-Wide Association Studies

42. Results of the European Group for the Study of Resistant Depression (GSRD) — basis for further research and clinical practice

43. Utility of the pooling approach as applied to whole genome association scans with high-density Affymetrix microarrays

44. Analysis of shared heritability in common disorders of the brain

45. Analysis of shared heritability in common disorders of the brain

46. Erratum: A genome-wide association study of anorexia nervosa suggests a risk locus implicated in dysregulated leptin signaling

47. A genome-wide association study of anorexia nervosa suggests a risk locus implicated in dysregulated leptin signaling

48. Evidence for three genetic loci involved in both anorexia nervosa risk and variation of body mass index

49. Identifying tissues implicated in Anorexia Nervosa using Transcriptomic Imputation

50. The Impact of COMT and Childhood Maltreatment on Suicidal Behaviour in Affective Disorders

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