19 results on '"Schulte, Dörte"'
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2. A Novel Splice-Site Mutation in VEGFC Is Associated with Congenital Primary Lymphoedema of Gordon
3. An Evolutionarily Conserved Role for Polydom/Svep1 During Lymphatic Vessel Formation
4. Endothelial Basement Membrane Laminin 511 Contributes to Endothelial Junctional Tightness and Thereby Inhibits Leukocyte Transmigration
5. Functional Dissection of the CCBE1 Protein
6. Fusing VE-Cadherin to α-Catenin Impairs Fetal Liver Hematopoiesis and Lymph but Not Blood Vessel Formation
7. Ccbe1 regulates Vegfc-mediated induction of Vegfr3 signaling during embryonic lymphangiogenesis
8. Mutation in Vascular Endothelial Growth Factor-C, a Ligand for Vascular Endothelial Growth Factor Receptor-3, Is Associated With Autosomal Dominant Milroy-Like Primary Lymphedema
9. Locking endothelial junctions blocks leukocyte extravasation, but not in all tissues
10. The Sphingosine-1-Phosphate Receptor S1PR1 Restricts Sprouting Angiogenesis by Regulating the Interplay between VE-Cadherin and VEGFR2
11. Dissociation of VE-PTP from VE-cadherin is required for leukocyte extravasation and for VEGF-induced vascular permeability in vivo
12. Stabilizing the VE-cadherin-catenin complex blocks leukocyte extravasation and vascular permeability
13. Cortactin deficiency is associated with reduced neutrophil recruitment but increased vascular permeability in vivo
14. Cortactin deficiency is associated with reduced leukocyte recruitment but increased vascular permeability in vivo
15. Control of endothelial barrier function by regulating vascular endothelial-cadherin
16. Jakmip1 Is Expressed upon T Cell Differentiation and Has an Inhibitory Function in Cytotoxic T Lymphocytes
17. Functional Dissection of the CCBE1 Protein.
18. Dissociation of VE-PTP from VE-cadherin is required for leukocyte extravasation and for VEGF-induced vascular permeability in vivo
19. Cortactin deficiency is associated with reduced neutrophil recruitment but increased vascular permeability in vivo.
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