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3. Development of an eReaxFF Force Field for BZY20 Solid Oxide Electrocatalysis

4. On the origin of non-classical ripples in draped graphene sheets

7. varVAMP: automated pan-specific primer design for tiled full genome sequencing and qPCR of highly diverse viral pathogens.

8. SARS-CoV-2 infection triggers profibrotic macrophage responses and lung fibrosis

10. eReaxFF force field development for BaZr0.8Y0.2O3-δ solid oxide electrolysis cells applications.

16. SARS-CoV-2 variant Alpha has a spike-dependent replication advantage over the ancestral B.1 strain in human cells with low ACE2 expression

19. Human lungs show limited permissiveness for SARS-CoV-2 due to scarce ACE2 levels but virus-induced expansion of inflammatory macrophages

20. Functional Analysis of Influenza A virus interactions with host surface proteins in influenza pneumonia

21. Functional Analysis of Influenza A virus interactions with host surface proteins in influenza pneumonia

23. Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV

24. Analysis of SARS-CoV-2 replication in explant cultures of the human upper respiratory tract reveals broad tissue tropism of wild-type and B.1.1.7 variant viruses

25. Analysis of BNT162b2‐ and CVnCoV‐elicited sera and of convalescent sera toward SARS‐CoV‐2 viruses

26. A Remote Secondary Binding Pocket Promotes Heteromultivalent Targeting of DC-SIGN

28. Specific Protein Antigen Delivery to Human Langerhans Cells in Intact Skin

29. Post-entry, spike-dependent replication advantage of B.1.1.7 and B.1.617.2 over B.1 SARS-CoV-2 in an ACE2-deficient human lung cell line

30. Analysis of Severe Acute Respiratory Syndrome 2 Replication in Explant Cultures of the Human Upper Respiratory Tract Reveals Broad Tissue Tropism of Wild-Type and B.1.1.7 Variant Viruses

31. PROJECT MANAGEMENT: ENGINEERING VS. NON-ENGINEERING.

32. Analysis of BNT162b2‐ and CVnCoV‐elicited sera and of convalescent sera toward SARS‐CoV‐2 viruses.

33. Human Lungs Show Limited Permissiveness for SARS-CoV-2 Due to Scarce ACE2 Levels But Strong Virus-Induced Immune Activation in Alveolar Macrophages

34. Influenza A Virus Infection Induces Apical Redistribution of Na+, K+-ATPase in Lung Epithelial Cells In Vitro and In Vivo

35. A Specific, Glycomimetic Langerin Ligand for Human Langerhans Cell Targeting

37. A Specific, Glycomimetic Langerin Ligand for Human Langerhans Cell Targeting

38. A Specific, Glycomimetic Langerin Ligand for Human Langerhans Cell Targeting

40. A specific, glycomimetic Langerin ligand for human Langerhans cell targeting

41. Influenza A Virus Infection Induces Apical Redistribution of Na+, K+-ATPase in Lung Epithelial Cells In Vitro and In Vivo.

43. Influenza A Virus Infection Induces Apical Redistribution of Na + , K + -ATPase in Lung Epithelial Cells In Vitro and In Vivo .

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