180 results on '"Shelkovnikova, Tatyana"'
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2. Stress-induced TDP-43 nuclear condensation causes splicing loss of function and STMN2 depletion
3. The master energy homeostasis regulator PGC-1α exhibits an mRNA nuclear export function
4. The Batten disease protein CLN3 is important for stress granules dynamics and translational activity
5. Deep learning modeling of rare noncoding genetic variants in human motor neurons definesCCDC146as a therapeutic target for ALS
6. TDP-43 in nuclear condensates: where, how, and why.
7. C9orf72 poly-PR condensation induces nuclear TDP-43 pathology and is inhibited by RNA in an optogenetic cell model
8. TDP-43 is a Master Regulator of Paraspeckle Condensation
9. Toxic gain of function from mutant FUS protein is crucial to trigger cell autonomous motor neuron loss
10. Long non-coding RNA Neat1 regulates adaptive behavioural response to stress in mice
11. Frameshift peptides alter the properties of truncated FUS proteins in ALS-FUS
12. ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles
13. Model of Artificial Neural Network of Biological Type and Simulation of the Network Operation
14. Assessment of radiological techniques application possibility for non-invasive diagnostics of latent inflammatory processes in myocardium in patients with atrial fibrillation
15. Oligonucleotide-Recognizing Topoisomerase Inhibitors (OTIs): Precision Gene Editors for Neurodegenerative Diseases?
16. A toolkit for the identification of NEAT1_2/paraspeckle modulators
17. AtypicalTDP‐43 protein expression in anALSpedigree carrying a p.Y374Xtruncation mutation inTARDBP
18. Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
19. MRI POTENTIAL FOR PREDICTION OF THE ANTIHYPERTENSIVE EFFECT OF RFA IN PATIENTS WITH TREATMENT-RESISTANT HYPERTENSION
20. MRI IN THE ASSESSMENT OF CEREBRAL INJURY AND CEREBROPROTECTIVE EFFECTS OF RENAL DENERVATION IN RESISTANT ARTERIAL HYPERTENSION
21. NEPHROPROTECTIVE EFFECT OF RD ACCORDING TO 5-YEAR FOLLOW-UP
22. Atypical TDP‐43 protein expression in an ALS pedigree carrying a p.Y374X truncation mutation in TARDBP.
23. ALS-linked cytoplasmic FUS assemblies are compositionally different from physiological stress granules and sequester hnRNPA3, a novel modifier of FUS toxicity
24. Processing and Recognition of Two-Coordinate Reference Images in a Scanning Probe Microscopy
25. Connecting the “dots”: RNP granule network in health and disease
26. Multistep process of FUS aggregation in the cell cytoplasm involves RNA-dependent and RNA-independent mechanisms
27. Dimebon Slows Progression of Proteinopathy in γ-Synuclein Transgenic Mice
28. Compromised paraspeckle formation as a pathogenic factor in FUSopathies
29. Compositional analysis of ALS-linked stress granule-like structures reveals factors and cellular pathways dysregulated by mutant FUS under stress
30. Long non-coding RNA NEAT1_1 ameliorates TDP-43 toxicity in in vivo models of TDP-43 proteinopathy
31. Additional file 1 of Frameshift peptides alter the properties of truncated FUS proteins in ALS-FUS
32. Stress granules regulate paraspeckles: RNP granule continuum at work
33. Antiviral Immune Response as a Trigger of FUS Proteinopathy in Amyotrophic Lateral Sclerosis
34. Stress granules regulate stress-induced paraspeckle assembly
35. Additional file 1: of ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles
36. Additional file 3: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
37. Additional file 6: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
38. Additional file 4: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
39. Additional file 1: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
40. Additional file 5: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
41. Additional file 2: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
42. Long non-coding RNA Neat1 regulates adaptive behavioural response to stress in mice
43. Calcium-responsive transactivator (CREST) toxicity is rescued by loss of PBP1/ATXN2 function in a novel yeast proteinopathy model and in transgenic flies
44. Antiviral Immune Response as a Trigger of FUS Proteinopathy in Amyotrophic Lateral Sclerosis
45. NEAT1 and paraspeckles in neurodegenerative diseases: A missing lnc found?
46. Chronically stressed or stress-preconditioned neurons fail to maintain stress granule assembly
47. Magnetic resonance imaging syndromes in patients with suspected myocarditis and theyr' relations to histological data
48. Calcium-responsive transactivator (CREST) toxicity is rescued by loss of PBP1/ATXN2 function in a novel yeast proteinopathy model and in transgenic flies
49. Modulation of p-eIF2α cellular levels and stress granule assembly/disassembly by trehalose
50. Cdkn1c Boosts the Development of Brown Adipose Tissue in a Murine Model of Silver Russell Syndrome
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