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466 results on '"Shigeta, K."'

2. Superconducting pairing symmetry on the extended Hubbard model in the presence of the Rashba-type spin-orbit coupling

Author

Shigeta, K., Onari, S., and Tanaka, Y.

Subjects
Condensed Matter - Superconductivity
Abstract

In order to study the pairing symmetry in non-centrosymmetric superconductors, we solve the linearized Eliashberg's equation on the two-dimensional extended Hubbard model in the presence of the Rashba-type spin-orbit coupling (RSOC) within the random phase approximation. In the presence of the RSOC, three types of pairing symmetries appear in the phase diagram with respect to the on-site Coulomb repulsion U and off-site one V. Each of pairing symmetries is admixture of spin-singlet and -triplet ones. On the basis of analytical study, it is found that the admixture of spin-singlet and -triplet components depends on not only the predominant pairing symmetry but also dispersion relation and pairing interaction., Comment: 11 pages, 12 figures

Published
2012
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4. The Impact of Smoking on Pulmonary Metastasis in Colorectal Cancer

Author

Makino A, Tsuruta M, Okabayashi K, Ishida T, Shigeta K, Seishima R, Ikebata A, Koishikawa K, Hasegawa H, Shimoda M, Fukunaga K, Betsuyaku T, and Kitagawa Y

Subjects
icam-1, colorectal cancer, adhesion molecules, lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens, pulmonary metastasis, lcsh:RC254-282, smoking
Abstract

Akitsugu Makino,1 Masashi Tsuruta,1 Koji Okabayashi,1 Takashi Ishida,1 Kohei Shigeta,1 Ryo Seishima,1 Akiyoshi Ikebata,1 Kaoru Koishikawa,1 Hirotoshi Hasegawa,2 Masayuki Shimoda,3 Koichi Fukunaga,4 Tomoko Betsuyaku4 ,* Yuko Kitagawa1 1Department of Surgery, Keio University School of Medicine, Tokyo, Japan; 2Department of Surgery, Tokyo Dental College Ichikawa General Hospital, Chiba, Japan; 3Department of Pathology, Keio University School of Medicine, Tokyo, Japan; 4Division of Pulmonary Medicine, Department of Medicine, Keio University School of Medicine, Tokyo, Japan†Professor Tomoko Betsuyaku passed away on September 1, 2018.Correspondence: Masashi Tsuruta Department of SurgeryKeio University School of Medicine, 35 Shinanomachi Shinjyuku-Ku, Tokyo 160-8582, JapanTel +81-3-3353-1211Fax +81-3-3355-4707Email masashitsuruta@gmail.comIntroduction: Recently, clinical studies have revealed that smoking can contribute to the poor prognosis of colorectal cancer (CRC) and, additionally, can be a risk factor for pulmonary metastasis of CRC. However, there has been no basic research regarding the underlying molecular mechanism. The purpose of this study was to clarify the mechanism by which smoking causes pulmonary metastasis of CRC.Methods: First, pulmonary metastasis model mice inhaled cigarette smoke or air (control) for 1 h once a day for 3 weeks. We attempted to clarify the effect of smoking on the incidence of pulmonary metastasis. On the 15th day, CMT-93 cells were injected into the tail vein. At 6 and 8 weeks following injection, the extent of pulmonary metastasis was evaluated using in vivo micro CT. After the last CT examination, the mice were sacrificed, and the lungs were extracted for pathological examination.Results: The number of mice with pulmonary metastases in the smoking group was significantly higher than in the control group. Three weeks of smoking induced mild inflammation in the lungs, as evidenced by increases in the levels of IL-6 and TNF-α in bronchoalveolar lavage. Moreover, the adhesion-related molecule ICAM-1 was overexpressed in pulmonary tissue, which allowed drained cancer cells to remain in the lung and contribute to the formation of pulmonary metastasis.Conclusion: Collectively, cigarette smoking may contribute to the pathogenesis and development of pulmonary metastasis in CRC through enhancement of adhesion and inflammation.Keywords: smoking, colorectal cancer, pulmonary metastasis, adhesion molecules, ICAM-1

Published
2020

15. Comparing the oncological outcomes between pure laparoscopic radical nephroureterectomy and laparoscopic assisted nephroureterectomy for upper-tract urothelial carcinoma: A multi-center cohort study adjusted by propensity score matching

Author

Shigeta, K., primary, Matsumoto, K., additional, Takeda, T., additional, Hattori, S., additional, Kaneko, G., additional, Matsushima, M., additional, Yasumizu, Y., additional, Tanaka, N., additional, Morita, S., additional, Kosaka, T., additional, Mizuno, R., additional, Asanuma, H., additional, and Oya, M., additional

Published
2020
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16. Evaluating the clinical efficacy of neoadjuvant chemotherapy for node-positive upper tract urothelial carcinoma: A multi-center cohort study

Author

Shigeta, K., primary, Matsumoto, K., additional, Tanaka, N., additional, Murakami, T., additional, Anno, T., additional, Umeda, K., additional, Izawa, M., additional, Sanjo, T., additional, Yasumizu, Y., additional, Takeda, T., additional, Morita, S., additional, Kosaka, T., additional, Mizuno, R., additional, Asanuma, H., additional, and Oya, M., additional

Published
2020
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28. Neuroendocrine responses to psychological stress in patients with myofascial pain.

Author

Yoshihara T, Shigeta K, Hasegawa H, Ishitani N, Masumoto Y, and Yamasaki Y

Abstract

AIMS: To investigate the responses of the hypothalamus-pituitary-adrenocortical and sympathetic-adrenal-medullary systems to experimentally induced psychological stress in patients with myofascial pain. METHODS: To characterize the features of these systems, temporal variations in plasma cortisol, adrenaline, and noradrenaline concentrations in response to psychological stress were measured in 20 patients with myofascial pain and in 20 healthy controls. RESULTS: The concentrations of plasma cortisol, adrenaline, and noradrenaline in response to psychological stress were significantly higher in the pain patients than in the healthy controls. Furthermore, although the plasma cortisol, adrenaline, and noradrenaline concentrations were significantly increased from the basal levels in both groups, the rate of recovery from these levels was significantly slower in patients than in healthy controls. CONCLUSION: These results suggest that both the sympathetic-adrenal-medullary and hypothalamic-pituitary-adrenocortical systems are more highly activated in response to psychological stress in patients with myofascial pain than in healthy individuals. [ABSTRACT FROM AUTHOR]

Published
2005

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