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2. Relationship between earlier introduction of tocilizumab and glucocorticoid-sparing effects on the acute phase of adult-onset Still's disease.

Author

Yoshihara, R, Tsuchiya, H, Shoda, H, and Fujio, K

Subjects
STILL'S disease, TOCILIZUMAB, MACROPHAGE activation syndrome
Abstract

This article discusses the relationship between the timing of tocilizumab (TCZ) introduction and its glucocorticoid-sparing effects on adult-onset Still's disease (AOSD). A retrospective observational study was conducted on AOSD patients who received TCZ and high-dose glucocorticoids for remission-induction therapy. The study found that early introduction of TCZ within 3 weeks of initiating glucocorticoid treatment effectively reduced the glucocorticoid dose requirement without increasing adverse events or the risk of macrophage activation syndrome. However, further research is needed to determine the efficacy of early TCZ introduction in AOSD treatment. [Extracted from the article]

Published
2024
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3. A Prospective Cohort Study to Define the Clinical Features and Outcome of Lung Cancers Harboring HER2 Aberration in Japan (HER2-CS STUDY)

Author

Gemba, K., Ikeda, G., Yasugi, M., Kurimoto, E., Nakano, K., Moritaka, T., Inoue, K., Miyoshi, S., Hamaguchi, N., Ito, R., Sano, Y., Takata, I., Mitani, A., Nishisaka, T., Shoda, H., Nishida, A., Tamamoto, S., Fujitaka, K., Masuda, T., Miyamoto, S., Hattori, N., Sugimoto, K., Fujii, S., Ueda, Y., Sakugawa, M., Fukamatsu, N., Ogata, Y., Bandoh, S., Kanaji, N., Takigawa, N., Yamane, H., Ochi, N., Honda, Y., Oka, M., Kittaka, M., Kubota, T., Yokoyama, A., Yokoyama, T., Sato, E., Shiota, Y., Horita, N., Kanematsu, T., Awaya, Y., Nakamasu, A., Murakami, I., Kuyama, S., Kudo, K., Tamura, T., Umeno, T., Morichika, D., Fujiwara, K., Sato, K., Harada, D., Nogami, N., Nishii, K., Fuchimoto, Y., Kishimoto, T., Kawai, H., Watanabe, K., Tokumo, K., Isobe, T., Tsubata, Y., Inoue, M., Ichikawa, H., Nishioka, Y., Hanibuchi, M., Goto, H., Sumikawa, T., Kodani, M., Suyama, H., Makino, H., Kinosita, N., Shimizu, E., Obata, H., Ikegami, H., Chikamori, K., Maeda, T., Kishino, T., Kamei, H., Ueoka, H., Kunihiro, Y., Kobayashi, T., Ueda, K., Hayashi, M., Kamiya, M., Murakami, J., Sato, A., Ichihara, E., Kubo, T., Ninomiya, T., Hirata, T., Minami, D., Kato, Y., Higo, H., Makimoto, G., Toyota, Y., Oda, N., Nakanishi, M., Kayatani, H., Senoo, S., Kano, H., Watanabe, H., Ando, T., Nakasuka, T., Hara, N., Itano, J., Nakashima, H., Tabata, M., Ninomiya, Kiichiro, Hata, Tae, Yoshioka, Hiroshige, Ohashi, Kadoaki, Bessho, Akihiro, Hosokawa, Shinobu, Ishikawa, Nobuhisa, Yamasaki, Masahiro, Shibayama, Takuo, Aoe, Keisuke, Kozuki, Toshiyuki, Harita, Shingo, Ueda, Yutaka, Murakami, Toshi, Fujimoto, Nobukazu, Yanai, Hiroyuki, Toyooka, Shinichi, Takata, Minoru, Hotta, Katsuyuki, and Kiura, Katsuyuki

Published
2019
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6. OP0202 IMMUNOMICS ANALYSIS OF RHEUMATOID ARTHRITIS IDENTIFIED PRECURSOR DENDRITIC CELLS AS A KEY CELL SUBSET OF TREATMENT RESISTANCE

Author

Yamada, S., primary, Nagafuchi, Y., additional, Wang, M., additional, Ota, M., additional, Hatano, H., additional, Takeshima, Y., additional, Okubo, M., additional, Kobayashi, S., additional, Sugimori, Y., additional, Nakano, M., additional, Yoshida, R., additional, Hanata, N., additional, Suwa, Y., additional, Tsuchida, Y., additional, Iwasaki, Y., additional, Sumitomo, S., additional, Kubo, K., additional, Shimane, K., additional, Setoguchi, K., additional, Azuma, T., additional, Kanda, H., additional, Shoda, H., additional, Zhang, X., additional, Yamamoto, K., additional, Ishigaki, K., additional, Okamura, T., additional, and Fujio, K., additional

Published
2023
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7. 029 Significance of anti-transcobalamin receptor antibodies in cutaneous arteritis unveiled by proteome-wide autoantibody screening

Author

Matsuda, K.M., primary, Kotani, H., additional, Yamaguchi, K., additional, Okumura, T., additional, Fukuda, E., additional, Kono, M., additional, Hisamoto, T., additional, Fukayama, M., additional, Okamura, T., additional, Shoda, H., additional, Fujio, K., additional, Goshima, N., additional, Shinichi, S., additional, and Yoshizaki, A., additional

Published
2023
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8. Resolution of exacerbated rheumatoid arthritis-associated interstitial lung disease under baricitinib treatment.

Author

Komai, T, Sawada, T, Tsuchiya, H, Harada, H, Shoda, H, and Fujio, K

Subjects
INTERSTITIAL lung diseases, BARICITINIB, SARS-CoV-2
Abstract

This article discusses a case study of a patient with rheumatoid arthritis-associated interstitial lung disease (RA-ILD) who experienced an acute exacerbation of their condition. The patient was treated with high-dose corticosteroids and immunosuppressive therapy, but their symptoms worsened. However, when the patient was given baricitinib, a Janus kinase (JAK) inhibitor, in combination with intensive immunosuppressive therapy, their respiratory symptoms improved and their lung infiltrates decreased. The article suggests that JAK inhibitors may be effective in treating acute exacerbations of RA-ILD and reducing the risk of fatal outcomes. [Extracted from the article]

Published
2024
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12. OP0110 CELL-TYPE-SPECIFIC TRANSCRIPTOME ARCHITECTURE UNDERLYING THE ESTABLISHMENT AND EXACERBATION OF SYSTEMIC LUPUS ERYTHEMATOSUS

Author

Nakano, M., primary, Ota, M., additional, Takeshima, Y., additional, Iwasaki, Y., additional, Hatano, H., additional, Nagafuchi, Y., additional, Kim, K., additional, Bang, S. Y., additional, Lee, H. S., additional, Shoda, H., additional, Zhang, X., additional, Bae, S. C., additional, Terao, C., additional, Yamamoto, K., additional, Okamura, T., additional, Ishigaki, K., additional, and Fujio, K., additional

Published
2022
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15. Efficacy and safety of plasma exchange in interstitial lung diseases with anti-melanoma differentiation-associated 5 gene antibody positive clinically amyopathic dermatomyositis.

Author

Komai, T, Iwasaki, Y, Tsuchida, Y, Hanata, N, Tsuchiya, H, Harada, Hiroaki, Hamasaki, Y, Nangaku, M, Shoda, H, and Fujio, K

Subjects
INTERSTITIAL lung diseases, MYOSITIS, DERMATOMYOSITIS, PULMONARY function tests, SERUM albumin, CYTOMEGALOVIRUS diseases, ANTIBODY titer
Abstract

Clinically amyopathic dermatomyositis (CADM) patients frequently develop refractory interstitial lung disease (ILD), with a poor prognosis. We aimed to verify the efficacy and safety of plasma exchange (PE) treatment for ILD in CADM. A retrospective case–control study was conducted to compare clinical outcomes with and without PE treatment in CADM-ILD patients refractory to combination therapy of high-dose glucocorticoids, calcineurin inhibitors, and cyclophosphamide. Among 19 enrolled patients, 11 were further treated with PE. We compared survival rates and other clinical characteristics. PE consisted of either fresh-frozen plasma or albumin as a replacement solution. Basal clinical characteristics at diagnosis, including age, gender, serum ferritin, Krebs von den Lungen-6 (KL-6), C-reactive protein, and respiratory function tests, did not differ between the two groups. The survival rate for treatment with PE was higher than for treatment without PE (91% and 50%, respectively, p < 0.05). Among PE-treated patients, anti-melanoma differentiation-associated gene-5 (anti-MDA-5) antibody titre, ferritin, and KL-6 as serological activity markers were sustainably reduced only after initiating PE. Therapeutic intervention with PE reduced the frequency of exacerbation of ILD requiring methylprednisolone pulse therapy. The occurrence of bacterial, fungal, and cytomegalovirus infection did not differ between the groups with and without PE, and adverse events associated with PE resolved with appropriate intervention. Combination therapy with PE was associated with an improved survival rate, and may be effective for the management of refractory ILD in CADM patients. A personalized therapeutic strategy including PE could be introduced for fatal rapidly progressive ILD. [ABSTRACT FROM AUTHOR]

Published
2023
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28. Varicella zoster virus myelitis in a patient with rheumatoid arthritis treated by tofacitinib.

Author

Itamiya, T, Komai, T, Tsuchida, Y, Shoda, H, and Fujio, K

Subjects
VARICELLA-zoster virus, HERPES zoster, RHEUMATOID arthritis, MYELITIS, MAGNETIC resonance imaging, MEDICAL personnel, SPINAL cord diseases
Abstract

Focal muscle weakness (zoster paresis) and sequential VZV myelitis is a rare neurological complication of VZV infection, with a frequency of approximately 0.5-5% in patients with VZV reactivation ([7]). Janus kinase (JAK) inhibitors, a type of targeted synthetic disease-modifying anti-rheumatic drug (DMARD), are emerging as useful therapeutic agents for a variety of autoimmune diseases, including rheumatoid arthritis (RA) ([1]). [Extracted from the article]

Published
2021
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31. A Prospective Cohort Study to Define the Clinical Features and Outcome of Lung Cancers Harboring HER2 Aberration in Japan (HER2-CS STUDY)

Author

Ninomiya, Kiichiro, Hata, Tae, Yoshioka, Hiroshige, Ohashi, Kadoaki, Bessho, Akihiro, Hosokawa, Shinobu, Ishikawa, Nobuhisa, Yamasaki, Masahiro, Shibayama, Takuo, Aoe, Keisuke, Kozuki, Toshiyuki, Harita, Shingo, Ueda, Yutaka, Murakami, Toshi, Fujimoto, Nobukazu, Yanai, Hiroyuki, Toyooka, Shinichi, Takata, Minoru, Hotta, Katsuyuki, Kiura, Katsuyuki, Gemba, K., Ikeda, G., Yasugi, M., Kurimoto, E., Nakano, K., Moritaka, T., Inoue, K., Miyoshi, S., Hamaguchi, N., Ito, R., Sano, Y., Takata, I., Mitani, A., Nishisaka, T., Shoda, H., Nishida, A., Tamamoto, S., Fujitaka, K., Masuda, T., Miyamoto, S., Hattori, N., Sugimoto, K., Fujii, S., Ueda, Y., Sakugawa, M., Fukamatsu, N., Ogata, Y., Bandoh, S., Kanaji, N., Takigawa, N., Yamane, H., Ochi, N., Honda, Y., Oka, M., Kittaka, M., Kubota, T., Yokoyama, A., Yokoyama, T., Sato, E., Shiota, Y., Horita, N., Kanematsu, T., Awaya, Y., Nakamasu, A., Sano, Y., Murakami, I., Kuyama, S., Kudo, K., Tamura, T., Umeno, T., Morichika, D., Fujiwara, K., Sato, K., Harada, D., Nogami, N., Nishii, K., Fuchimoto, Y., Kishimoto, T., Kawai, H., Watanabe, K., Tokumo, K., Isobe, T., Tsubata, Y., Inoue, M., Ichikawa, H., Nishioka, Y., Hanibuchi, M., Goto, H., Sumikawa, T., Kodani, M., Suyama, H., Makino, H., Ueda, Y., Kinosita, N., Shimizu, E., Obata, H., Ikegami, H., Chikamori, K., Maeda, T., Kishino, T., Kamei, H., Ueoka, H., Kunihiro, Y., Kobayashi, T., Ueda, K., Hayashi, M., Kamiya, M., Murakami, J., Sato, A., Ichihara, E., Kubo, T., Ninomiya, T., Hirata, T., Minami, D., Kato, Y., Higo, H., Makimoto, G., Toyota, Y., Oda, N., Nakanishi, M., Kayatani, H., Nishii, K., Senoo, S., Kano, H., Watanabe, H., Ando, T., Nakasuka, T., Hara, N., Itano, J., Nakashima, H., and Tabata, M.

Abstract

Human epidermal growth factor 2 (HER2) is a potential driver oncogene. Although HER2-targeted precision therapy has been tested in non-small cell lung cancer (NSCLC), the demographic characteristics of HER2-positive NSCLC have not been systematically defined.

Published
2019
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34. AB0536 Immunosuppressive Therapy is Promising for Pulmonary Arterial Hypertension Associated with Connective Tissue Disease (CTD-PAH)

Author

Takeshima, Y., primary, Iwasaki, Y., additional, Kato, R., additional, Sumitomo, S., additional, Shirai, H., additional, Natsumoto, B., additional, Ota, M., additional, Teruya, S., additional, Tsuchiya, H., additional, Komai, T., additional, Hanata, N., additional, Shoda, H., additional, Shibuya, M., additional, Fujio, K., additional, Maki, H., additional, Hatano, M., additional, and Yamamoto, K., additional

Published
2015
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36. Orbital apex syndrome on initial presentation of giant cell arteritis: a case report and review of the literature.

Author

Tsuzuki, S, Tsuchiya, H, Shoda, H, and Fujio, K

Subjects
GIANT cell arteritis, SYNDROMES, CONTRAST-enhanced magnetic resonance imaging, MULTINUCLEATED giant cells, SENSORY disorders, LITERATURE reviews
Abstract

Giant cell arteritis (GCA) is an inflammatory vascular syndrome that is characterized by cranial and/or large vessel vasculitis, causing stenoses and aneurysms of affected vessels ([1]). We report our experience with a GCA patient with visual deterioration due to orbital apex syndrome, a rare complication of GCA. Presumably, GCA with orbital apex syndrome is included in the latter group; the pathological findings would differ from GCA with PMR. [Extracted from the article]

Published
2022
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39. A 40Gb/s multi-data-rate CMOS transceiver chipset with SFI-5 interface for optical transmission systems

Author

Amamiya, Y., primary, Kaeriyama, S., additional, Noguchi, H., additional, Yamazaki, Z., additional, Yamase, T., additional, Hosoya, K., additional, Okamoto, M., additional, Tomari, S., additional, Yamaguchi, H., additional, Shoda, H., additional, Ikeda, H., additional, Tanaka, S., additional, Takahashi, T., additional, Ohhira, R., additional, Noda, A., additional, Hijioka, K., additional, Tanabe, A., additional, Fujita, S., additional, and Kawahara, N., additional

Published
2009
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44. LETTER TO THE EDITOR.

Author

Okubo, M., Shoda, H., Bannai, E., Kubo, K., Kanda, H., Fujio, K., and Yamamoto, K.

Subjects
*PLASMA exchange (Therapeutics), *MYCOPHENOLIC acid, *COLON diverticulum, *COLITIS
Abstract

The article presents case studies of systemic lupus erythematosus (SLE)/ thrombotic microangiopathy (TMA) patients with plasma exchange (PE), steroid and mycophenolate mofetil (MMF). It mentions case study of 52-year-old male who was admitted because of sigmoid colon diverticulitis with microperforation, 43-year-old female who was admitted because of nephritic syndrome and 20-year-old female was admitted because of colitis.

Published
2017
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45. Characteristics of antisynthetase syndrome patients with hyperferritinaemia.

Author

Kobayashi, S, Shoda, H, Seri, Y, Nagafuchi, Y, Sasaki, O, Kubo, K, Fujio, K, and Yamamoto, K

Subjects
*ANTISYNTHETASE syndrome, *AUTOIMMUNE diseases, *IRON in the body, *TRANSFER RNA, *MYOSITIS, *TUMOR classification, *CYTOKINES, *ENZYMES, *FERRITIN, *IMMUNOGLOBULINS, *IRON metabolism disorders, *LONGITUDINAL method, *PROTEINS, *TUMORS, *DISEASE management, *DISEASE complications, *DIAGNOSIS, *THERAPEUTICS
Abstract

The article focuses on a study which compares antisynthetase syndrome (ASS) cases with hyperferritinaemia to those without hyperferritinaemia to reveal their characteristics. Topics discussed include the presence anti-aminoacyl-tRNA synthetase (ARS) antibodies in some patients with inflammatory myopathy or myositis, the definition of hyperferritinaemia, and the association of hyperferritinaemia in ASS with evident myositis, systemic inflammation, anti-EJ antiboides, and malignancy.

Published
2017
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50. Subacute cerebellar ataxia and atrophy developed in a young woman with systemic lupus erythematosus whose cerebrospinal fluid was positive for antineuronal cell antibody.

Author

Iwasaki, Y, Okamoto, A, Shoda, H, Takahashi, Y, Fujio, K, Kawahata, K, and Yamamoto, K

Subjects
CEREBELLAR ataxia, ATROPHY, CENTRAL nervous system, LUPUS erythematosus
Abstract

Subacute cerebellar ataxia in combination with cerebellar atrophy has rarely been reported as one of the manifestations of lupus in the central nervous system (CNS). We describe a 27-year-old woman with systemic lupus erythematosus who developed subacute cerebellar ataxia. Computed tomography and magnetic resonance imaging of her brain showed cerebellar atrophy in both hemispheres, particularly on the right side. Moreover, increased antineuronal cell antibody levels were detected in her cerebrospinal fluid. The cerebellar ataxia improved markedly following high-dose corticosteroid administration. This suggests that a relationship exists between autoantibodies and subacute atrophic processes in CNS lupus. [ABSTRACT FROM AUTHOR]

Published
2012
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