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1. LMO2-Associated Clonal T Cell Proliferation in Two Patients after Gene Therapy for SCID-X1

2. An intragenic ERG deletion is a marker of an oncogenic subtype of B-cell precursor acute lymphoblastic leukemia with a favorable outcome despite frequent IKZF1 deletions

4. Clinical, cytogenetic and molecular characteristics of 14 T-ALL patients carrying the TCRβ-HOXA rearrangement: a study of the Groupe Francophone de Cytogénétique Hématologique

7. LMO2-associated clonal T cell proliferation in two patients after gene therapy for SCID-X1

22. Carcinome épidermoïde cutané associé à des cellules tumorales p53 positives mutées dérivant de cellules épithéliales du rein transplanté

23. An intragenic ERG deletion is a marker of an oncogenic subtype of B-cell precursor acute lymphoblastic leukemia with a favorable outcome despite frequent IKZF1 deletions

25. Lack of evidence of HTLV-I/II infection in T CD8 malignant or reactive lymphoproliferative disorders in France: a serological and/or molecular study of 169 cases

26. 260 Myelodysplasia and leukemia of Fanconi anemia are associated with a specific pattern of genomic abnormalities that includes RUNX1/AML1 lesions

28. MTCP-1: a novel gene on the human chromosome Xq28 translocated to the T cell receptor alpha/delta locus in mature T cell proliferations

30. Impact of genotype on survival of children with T-cell acute lymphoblastic leukemia treated according to the French protocol FRALLE-93: the effect of TLX3/HOX11L2 gene expression on outcome

31. Clinical, cytogenetic and molecular characteristics of 14 T-ALL patients carrying the TCRβ-HOXA rearrangement: a study of the Groupe Francophone de Cytogénétique Hématologique

32. Comparison of alpha beta and gamma delta expressing CD3+ acute lymphoblastic leukemias

41. Expression of HLA-E in transgenic mice

45. The majority of myeloid‐antigen‐positive (My + ) childhood B‐cell precursor acute lymphoblastic leukaemias express TEL‐AML1 fusion transcripts

49. Activation of the human homologue of the Drosophila sina gene in apoptosis and tumor suppression.

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