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1. Genetic influences on plasma CFH and CFHR1 concentrations and their role in susceptibility to age-related macular degeneration.

Author

Moore, Anthony, Ansari, M, McKeigue, PM, Skerka, C, Hayward, C, Rudan, I, Vitart, V, Polasek, O, Armbrecht, A-M, Yates, JRW, and Vatavuk, Z

Abstract

It is a longstanding puzzle why non-coding variants in the complement factor H (CFH) gene are more strongly associated with age-related macular degeneration (AMD) than functional coding variants that directly influence the alternative complement pathway. T

Published
2013

3. Langzeitdaten von zwei Schwestern mit C3-Glomerulonephritis (C3-GN) aufgrund einer homozygoten CFH-Mutation und Auto-Antikörpern

Author

Hackl, A., primary, Erger, F., additional, Skerka, C., additional, Wenzel, A., additional, Tschernoster, N., additional, Ehren, R., additional, Burgmaier, K., additional, Riehmer, V., additional, Licht, C., additional, Kirschfink, M., additional, Weber, L.T., additional, Altmueller, J., additional, Zipfel, P.F., additional, and Habbig, S., additional

Published
2021
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14. Genotypisierung bei exsudativer AMD - auf dem Weg zur personalisierten Medizin?

Author

Busch, Catharina, Jacob, S., Lin, Y., Pilger, D., Zipfel, P. F., Strauss, O., Skerka, C., and Joussen, A. M.

Subjects
ddc: 610, 610 Medical sciences, Medicine
Abstract

Hintergrund: Die intravitreale anti-VEGF Therapie stellt seit vielen Jahren die Therapie der Wahl der exsudativen AMD dar. Handlungsspielraum im Rahmen der intravitrealen Therapie beschränken sich zurzeit auf die Auswahl zwischen verschiedenen applizierbaren anti-VEGF Substanzen (Bevacizumab, Ranibizumab,[zum vollständigen Text gelangen Sie über die oben angegebene URL], Wintertagung der Berlin-Brandenburgischen Augenärztlichen Gesellschaft 2016

Published
2016
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17. Komplement Wirkung auf das retinale Pigmentepithel: Intrazelluläre Signalverarbeitung der Anaphylatoxin Rezeptoren

Author

Strauß, Olaf, Busch, C., Huber, C., Wildner, G., Skerka, C., Zipfel, P., and Rohrer, B.

Subjects
ddc: 610, 610 Medical sciences, Medicine
Abstract

Hintergrund: Akkumulation von Komplementproteinen in Drusen und Polymorphismen in Genen für das Komplementsystem, die zur verminderten inhibitorischen Kontrolle des alternativen Aktivierungsweges führen, deuten auf eine Beteiligung des Komplementsystems an der AMD. Trotz dieser Erkenntnisse[zum vollständigen Text gelangen Sie über die oben angegebene URL], 29. Jahrestagung der Retinologischen Gesellschaft

Published
2016

18. Phänotyp-Genotyp-Korrelation in Patienten mit altersabhängiger Makuladegeneration

Author

Busch, Catharina, Jacob, S., Lin, Y., Pilger, D., Zipfel, P. F., Skerka, C., Strauss, O., and Joussen, A. M.

Subjects
ddc: 610, 610 Medical sciences, Medicine
Abstract

Hintergrund: Zahlreiche genetische Meta-Analysen konnten eine starke Assoziation von Polymorphismen im Komplement Faktor H (CFH)- und Age-related maculopathy susceptibility 2 (ARMS2)- Gen mit der altersabhängigen Makuladegeneration (AMD) aufzeigen. Über eine mögliche Korrelation zwischen[zum vollständigen Text gelangen Sie über die oben angegebene URL], 29. Jahrestagung der Retinologischen Gesellschaft

Published
2016

19. Age-related Maculopathy Susceptibility Protein 2 (ARMS2) interacts with human properdin

Author

Lin, Yuchen, Karlstetter, M., Jacob, S., Micklisch, S., Fauser, S., Pauly, D., Langmann, T., Zipfel, P.F., and Skerka, C.

Subjects
genetic structures, ddc: 610, sense organs, 610 Medical sciences, Medicine, eye diseases
Abstract

Background: The formation of drusen at the macula and the degeneration of RPE cells is characteristic for AMD and represents the most common cause of blindness in developed countries. The ARMS2 variant at 10q26 (A69S, rs10490924) is strongly associated with AMD. This polymorphism is linked to a mutation[for full text, please go to the a.m. URL], VI. International Symposium on AMD – Age-Related Macular Degeneration – Emerging Concepts – Exploring known and Identifying new Pathways

Published
2015

20. Complement signaling at the RPE

Author

Strauß, Olaf, Busch, C., Genewsky, A., Huber, C., Skerka, C., Zipfel, P., and Rohrer, B.

Subjects
ddc: 610, sense organs, 610 Medical sciences, Medicine, eye diseases
Abstract

The reduced efficiency to control the alternative pathway of the complement system by complement proteins with polymorphisms associated with AMD risk led to the hypothesis that the constant attack of the RPE by the terminal complement complex (TCC) is a major disease mechanism. To test this hypothesis[for full text, please go to the a.m. URL], VI. International Symposium on AMD – Age-Related Macular Degeneration – Emerging Concepts – Exploring known and Identifying new Pathways

Published
2015

21. Effect of AMD patients' sera on ARPE-19 cells in comparison to normal human serum

Author

Busch, Catharina, Jacob, S., Huber, C., Skerka, C., Zipfel, P.F., Joussen, A.M., and Strauß, O.

Subjects
ddc: 610, sense organs, 610 Medical sciences, Medicine, eye diseases
Abstract

Background: AMD patients’ sera present an altered systemic complement profile, characterized by elevated levels of complement split products and an increased activity of the alternative complement pathway (Reynolds R et al. 2009, Hecker LA et al. 2010). Among AMD patients CFH and ARMS2 risk genotypes[for full text, please go to the a.m. URL], VI. International Symposium on AMD – Age-Related Macular Degeneration – Emerging Concepts – Exploring known and Identifying new Pathways

Published
2015
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25. Liver-kidney transplantation to cure atypical hemolytic uremic syndrome

Author

Saland, JM, Ruggenenti, P, Remuzzi, G, Bekassy, Z, Bensman, A, Bresin, E, Colledan, M, Camilla, R, Coppo, R, Cruzado-Garrit, JM, Daina, E, Fremeaux-Bacchi, V, Goodship, TJ, Gridelli, B, Hugo, C, Karpman, D, Jalanko, H, Loirat, C, Hijosa, MM, Mc Kiernan, PJ, Noris, M, De Cordoba, SR, Rota, G, Sanchez-Corral, P, Skerka, C, Tartufari, A, Zipfel, PF, Saland, J, Ruggenenti, P, Remuzzi, G, Bekassy, Z, Bensman, A, Bresin, E, Colledan, M, Camilla, R, Coppo, R, Cruzado-Garrit, J, Daina, E, Fremeaux-Bacchi, V, Goodship, T, Gridelli, B, Hugo, C, Karpman, D, Jalanko, H, Loirat, C, Hijosa, M, Mc Kiernan, P, Noris, M, De Cordoba, S, Rota, G, Sanchez-Corral, P, Skerka, C, Tartufari, A, and Zipfel, P

Subjects
Nephrology, Hemolytic anemia, medicine.medical_specialty, Anemia, Hemolytic, Kidney Disease, Complement factor I, Bioinformatics, Internal medicine, Atypical hemolytic uremic syndrome, medicine, Humans, Kidney transplantation, Kidney, business.industry, General Medicine, medicine.disease, Combined Modality Therapy, Kidney Transplantation, Thrombocytopenia, Surgery, Liver Transplantation, Transplantation, medicine.anatomical_structure, Complement Factor H, Hemolytic-Uremic Syndrome, Mutation, Practice Guidelines as Topic, Kidney Diseases, business, Kidney disease, Human
Abstract

10 páginas, 8 tablas -- PAGS nros. 940-949, Atypical hemolytic uremic syndrome is often associated with mutations in genes encoding complement regulatory proteins and secondary disorders of complement regulation. Progression to kidney failure and recurrence with graft loss after kidney transplantation are frequent. The most common mutation is in the gene encoding complement factor H. Combined liver-kidney transplantation may correct this complement abnormality and prevent recurrence when the defect involves genes encoding circulating proteins that are synthesized in the liver, such as factor H or I. Good outcomes have been reported when surgery is associated with intensified plasma therapy. A consensus conference to establish treatment guidelines for atypical hemolytic uremic syndrome was held in Bergamo in December 2007. The recommendations in this article are the result of combined clinical experience, shared research expertise, and a review of the literature and registry information. This statement defines groups in which isolated kidney transplantation is extremely unlikely to be successful and a combined liver-kidney transplant is recommended and also defines those for whom kidney transplant remains a viable option. Although combined liver-kidney or isolated liver transplantation is the preferred therapeutic option in many cases, the gravity of risk associated with the procedure has not been eliminated completely, and assessment of risk and benefit requires careful and individual attention. Hemolytic uremic syndrome (HUS) is a rare disease of microangiopathic hemolytic anemia, thrombocytopenia, and renal impairment. Typical HUS follows a diarrheal illness caused by a verocytotoxin-producing bacterium, commonly Escherichia coli O157:H7, and resolves in most cases without sequelae.1 Atypical HUS (aHUS) is much less common and has a considerably worse prognosis in terms of both mortality and development of ESRD.2, Mutations in genes encoding complement regulatory proteins and secondary disorders of complement regulation seem to play a central role in the pathogenesis of most cases of aHUS. Triggering of an unregulated complement cascade is thought to damage endothelium, producing microangiopathic hemolytic anemia and thrombosis. Gene mutations are identified in at least 50% of cases. The most frequently reported mutations are in the gene encoding complement factor H (CFH) and account for 50 to 60% of cases associated with documented genetic abnormalities. Less frequent are mutations in the genes encoding membrane co-factor protein (MCP) and complement factor I (CFI) and are observed in approximately 20% and 10 to 15% of the overall disease-associated mutations, respectively.3–6 More recently, gain-of-function mutations in the genes encoding the complement-activating protein complement factor B (CFB) as well as complement C3 (C3) have been reported in a few cases with aHUS.7,8 Antibodies against factor H have also been observed in patients with aHUS, particularly children, and in most cases in association with homozygous deletion of the genes encoding complement factor H–related proteins 1 and 3 (CFHR1 and CFHR3).9 Indeed, there is a growing list of the mutations, polymorphisms, and other complement abnormalities that alone or in combination may lead to aHUS.8–13, This work has been partially supported by a grant from Fondazione ART Per La Ricerca Sui Trapianti (Milan, Italy)

Published
2009

26. Mechanism of complement evasion by malaria parasites

Author

Rosa, TFA, Ngwa, CJ, Simon, N, Kuehn, A, Lasonder, E, Agarwal, V, Blom, A, Zipfel, PF, Skerka, C, and Pradel, G

Subjects
ddc: 610, chemical and pharmacologic phenomena, 610 Medical sciences, Medicine
Abstract

The complement system represents a crucial component of the innate immune response against invading pathogens. However, a variety of pathogens evade the complement attack by binding to host complement regulators such factor H, C4b-binding protein or vitronectin. In a recently study we showed that in[for full text, please go to the a.m. URL], 12th Malaria Meeting

Published
2014
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27. Complement signaling at the RPE

Author

Strauß, O, Busch, C, Genewsky, A, Huber, C, Skerka, C, Zipfel, P, Rohrer, B, Strauß, O, Busch, C, Genewsky, A, Huber, C, Skerka, C, Zipfel, P, and Rohrer, B

Published
2015

30. The pH-regulated antigen 1 of Candida albicans binds the human complement inhibitor C4b-binding protein and mediates fungal complement evasion

Author

Luo, S., Blom, A.M., Rupp, S., Hipler, U.-C., Hube, B., Skerka, C., Zipfel, P.F., and Publica

Abstract

Candida albicans binds and utilizes human complement inhibitors, such as C4b-binding protein (C4BP), Factor H, and FHL-1 for immune evasion. Here, we identify Candida pH-regulated antigen 1 (Pra1) as the first fungal C4BP-binding protein. Recombinant Pra1 binds C4BP, as shown by ELISA and isothermal titration calorimetry, and the Pra1-C4BP interaction is ionic in nature. The Pra1 binding domains within C4BP were localized to the complement control protein domain 4 (CCP4), CCP7, and CCP8. C4BP bound to Pra1 maintains complement-inhibitory activity. C4BP and Factor H bind simultaneously to Candida Pra1 and do not compete for binding at physiological levels. A Pra1-overexpressing C. albicans strain, which had about 2-fold Pra1 levels at the surface acquired also about 2-fold C4BP to the surface, compared with the wild type strain CAI4.A Pra1 knock-out strain showed 22% reduced C4BP binding. C4BP captured by C. albicans from human serum inhibits C4b and C3b surface depositi on and also maintains cofactor activity. In summary, Candida Pra1 represents the first fungal C4BP-binding surface protein. Pra1, via binding to C4BP, mediates human complement control, thereby favoring the immune and complement evasion of C. albicans.

Published
2011

41. Standardisation of the factor H autoantibody assay

Author

Watson, R., primary, Lindner, S., additional, Bordereau, P., additional, Hunze, E.-M., additional, Tak, F., additional, Ngo, S., additional, Zipfel, P., additional, Skerka, C., additional, Dragon-Durey, M.-A., additional, and Marchbank, K.J., additional

Published
2013
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46. Liver-kidney transplantation to cure atypical hemolytic uremic syndrome

Author

Saland, J, Ruggenenti, P, Remuzzi, G, Bekassy, Z, Bensman, A, Bresin, E, Colledan, M, Camilla, R, Coppo, R, Cruzado-Garrit, J, Daina, E, Fremeaux-Bacchi, V, Goodship, T, Gridelli, B, Hugo, C, Karpman, D, Jalanko, H, Loirat, C, Hijosa, M, Mc Kiernan, P, Noris, M, De Cordoba, S, Rota, G, Sanchez-Corral, P, Skerka, C, Tartufari, A, Zipfel, P, Saland, JM, Cruzado-Garrit, JM, Goodship, TJ, Hijosa, MM, Mc Kiernan, PJ, De Cordoba, SR, Zipfel, PF, Saland, J, Ruggenenti, P, Remuzzi, G, Bekassy, Z, Bensman, A, Bresin, E, Colledan, M, Camilla, R, Coppo, R, Cruzado-Garrit, J, Daina, E, Fremeaux-Bacchi, V, Goodship, T, Gridelli, B, Hugo, C, Karpman, D, Jalanko, H, Loirat, C, Hijosa, M, Mc Kiernan, P, Noris, M, De Cordoba, S, Rota, G, Sanchez-Corral, P, Skerka, C, Tartufari, A, Zipfel, P, Saland, JM, Cruzado-Garrit, JM, Goodship, TJ, Hijosa, MM, Mc Kiernan, PJ, De Cordoba, SR, and Zipfel, PF

Abstract

Atypical hemolytic uremic syndrome is often associated with mutations in genes encoding complement regulatory proteins and secondary disorders of complement regulation. Progression to kidney failure and recurrence with graft loss after kidney transplantation are frequent. The most common mutation is in the gene encoding complement factor H. Combined liver-kidney transplantation may correct this complement abnormality and prevent recurrence when the defect involves genes encoding circulating proteins that are synthesized in the liver, such as factor H or I. Good outcomes have been reported when surgery is associated with intensified plasma therapy. A consensus conference to establish treatment guidelines for atypical hemolytic uremic syndrome was held in Bergamo in December 2007. The recommendations in this article are the result of combined clinical experience, shared research expertise, and a review of the literature and registry information. This statement defines groups in which isolated kidney transplantation is extremely unlikely to be successful and a combined liver-kidney transplant is recommended and also defines those for whom kidney transplant remains a viable option. Although combined liver-kidney or isolated liver transplantation is the preferred therapeutic option in many cases, the gravity of risk associated with the procedure has not been eliminated completely, and assessment of risk and benefit requires careful and individual attention. Copyright © 2009 by the American Society of Nephrology.

Published
2009

47. Intravitreale Spiegel des Komplementfaktors H (CFH)

Author

Weinberger,, AWA, Skerka, C, Zipfel, PF, Weber, BHF, Becker, J, Carstesen, D, Jung, C, Keilhauer, C, Walter, P, Weinberger,, AWA, Skerka, C, Zipfel, PF, Weber, BHF, Becker, J, Carstesen, D, Jung, C, Keilhauer, C, and Walter, P

Published
2006

49. Renal histopathology

Author

Marie-Lucile, F., primary, Laure-Helene, N., additional, Yosr, C., additional, Anne, M., additional, Fadi, F., additional, Levi, C., additional, Meas-Yedid, V., additional, Daniliuc, C., additional, Karras, A., additional, Olivo-Marin, J. C., additional, Mouthon, L., additional, Guiard, E., additional, Roland, M., additional, Guillevin, L., additional, Jacquot, C., additional, Nochy, D., additional, Thervet, E., additional, Chen, Q., additional, Skerka, C., additional, Uzonyi, B., additional, Lindner, S., additional, Licht, C., additional, Hoppe, B., additional, Riedl, M., additional, Kirschfink, M., additional, Habbich, S., additional, Wolf, G., additional, Strain, L., additional, Goodship, T. H., additional, Zipfel, P. F., additional, Kfoury, H., additional, Alsuwaida, A., additional, Alsaad, K., additional, Alhejaili, F., additional, Alghonaim, M., additional, Alwakeel, J., additional, Husain, S., additional, Aloudah, N., additional, Besso, L., additional, Tamagnone, M., additional, Daidola, G., additional, Burdese, M., additional, Repetto, L., additional, Pasquale, G., additional, Colla, L., additional, Biancone, L., additional, Stratta, P., additional, Segoloni, G. P., additional, Bacalja, J., additional, Bauer Segvic, A. M., additional, Bulimbasic, S., additional, Pacic, A., additional, Knotek, M., additional, Sabljar Matovinovic, M., additional, Galesic, K., additional, Galesic Ljubanovic, D., additional, Zakharova, E., additional, Stolyarevich, E., additional, Vorobjova, O., additional, Tamouza, H., additional, Chemouny, J. M., additional, Flamant, M., additional, Raskova Kafkova, L., additional, Demion, M., additional, Laurent, M., additional, Walker, F., additional, Julian, B. A., additional, Tissandie, E., additional, Tiwari, M. K., additional, Novak, J., additional, Camara, N. O., additional, Benhamou, M., additional, Vrtovsnik, F., additional, Monteiro, R. C., additional, Moura, I. C., additional, Samavat, S., additional, Ahmadpoor, P., additional, Torbati, P., additional, Ghaderi, R., additional, Poorrezagholi, F., additional, Samadian, F., additional, Nafar, M., additional, MII, A., additional, Shimizu, A., additional, Kaneko, T., additional, Yasuda, F., additional, Fukui, M., additional, Masuda, Y., additional, Iino, Y., additional, Katayama, Y., additional, Muller, C., additional, Markovic-Lipkovski, J., additional, Simic-Ogrizovic, S., additional, Naumovic, R., additional, Cirovic, S., additional, Mitrovic, D., additional, Muller, G., additional, Wozniak, A., additional, Janicka-Jedynska, M., additional, Zurawski, J., additional, Kaczmarek, E., additional, Zachwieja, J., additional, Khilji, S., additional, Dorman, T., additional, O'kelly, P., additional, Lampty, L., additional, Leung, K., additional, Shadivan, A., additional, Varghese, C., additional, Walshe, J., additional, Saito, T., additional, Kawano, M., additional, Saeki, T., additional, Mizushima, I., additional, Yamaguchi, Y., additional, Imai, N., additional, Nakashima, H., additional, Umehara, H., additional, Shvetsov, M., additional, Popova, O., additional, Chebotareva, N., additional, Ivanov, A., additional, Bobkova, I., additional, Cremasco, D., additional, Ceol, M., additional, Peruzzi, L., additional, Mazzucco, G., additional, Giuseppina, M., additional, Vezzoli, G., additional, Cristofaro, R., additional, D'angelo, A., additional, Anglani, F., additional, Del Prete, D., additional, Coppolino, G., additional, Comi, N., additional, Bolignano, D., additional, Piraina, V., additional, Talarico, R., additional, Colombo, A., additional, Lucisano, G., additional, Fuiano, G., additional, Bernich, P., additional, Lupo, A., additional, Of Renal Biopsies, T. R., additional, Rastaldi, M. P., additional, Jercan, O. C., additional, Messa, P., additional, Alexandru, D., additional, Mogoanta, L., additional, and Uribe Villegas, V., additional

Published
2012
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50. Experimental pathology

Author

Yi Chun, D. X., primary, Alexandre, H., additional, Edith, B., additional, Nacera, O., additional, Julie, P., additional, Chantal, J., additional, Eric, R., additional, Zhang, X., additional, Jin, Y., additional, Miravete, M., additional, Dissard, R., additional, Klein, J., additional, Gonzalez, J., additional, Caubet, C., additional, Pecher, C., additional, Pipy, B., additional, Bascands, J.-L., additional, Mercier-Bonin, M., additional, Schanstra, J., additional, Buffin-Meyer, B., additional, Claire, R., additional, Rigothier, C., additional, Richard, D., additional, Sebastien, L., additional, Moin, S., additional, Chantal, B., additional, Christian, C., additional, Jean, R., additional, Migliori, M., additional, Cantaluppi, V., additional, Mannari, C., additional, Medica, D., additional, Giovannini, L., additional, Panichi, V., additional, Goldwich, A., additional, Alexander, S., additional, Andre, G., additional, Amann, K., additional, Migliorini, A., additional, Sagrinati, C., additional, Angelotti, M. L., additional, Mulay, S. R., additional, Ronconi, E., additional, Peired, A., additional, Romagnani, P., additional, Anders, H.-J., additional, Chiang, W. C., additional, Lai, C. F., additional, Peng, W.-H., additional, Wu, C. F., additional, Chang, F.-C., additional, Chen, Y.-T., additional, Lin, S.-L., additional, Chen, Y. M., additional, Wu, K. D., additional, Lu, K.-S., additional, Tsai, T. J., additional, Virgine, O., additional, Qing Feng, F., additional, Zhang, S.-Y., additional, Dominique, D., additional, Vincent, A., additional, Marina, C., additional, Philippe, L., additional, Georges, G., additional, Pawlak, A., additional, Sahali, D., additional, Matsumoto, S., additional, Kiyomoto, H., additional, Ichimura, A., additional, Dan, T., additional, Nakamichi, T., additional, Tsujita, T., additional, Akahori, K., additional, Ito, S., additional, Miyata, T., additional, Xie, S., additional, Zhang, B., additional, Shi, W., additional, Yang, Y., additional, Nagasu, H., additional, Satoh, M., additional, Kidokoro, K., additional, Nishi, Y., additional, Ihoriya, C., additional, Kadoya, H., additional, Sasaki, T., additional, Kashihara, N., additional, Wu, C.-F., additional, Chou, Y.-H., additional, Duffield, J., additional, Rocca, C., additional, Gregorini, M., additional, Corradetti, V., additional, Valsania, T., additional, Bedino, G., additional, Bosio, F., additional, Pattonieri, E. F., additional, Esposito, P., additional, Sepe, V., additional, Libetta, C., additional, Rampino, T., additional, Dal Canton, A., additional, Omori, H., additional, Kawada, N., additional, Inoue, K., additional, Ueda, Y., additional, Yamamoto, R., additional, Matsui, I., additional, Kaimori, J., additional, Takabatake, Y., additional, Moriyama, T., additional, Isaka, Y., additional, Rakugi, H., additional, Wasilewska, A., additional, Taranta-Janusz, K., additional, Deebek, W., additional, Kuroczycka-Saniutycz, E., additional, Lee, A. S., additional, Lee, J. E., additional, Jung, Y. J., additional, Kang, K. P., additional, Lee, S., additional, Kim, W., additional, Arfian, N., additional, Emoto, N., additional, Yagi, K., additional, Nakayama, K., additional, Hartopo, A. B., additional, Nugrahaningsih, D. A., additional, Yanagisawa, M., additional, Hirata, K.-I., additional, Munoz-Felix, J. M., additional, Lopez-Novoa, J. M., additional, Martinez-Salgado, C., additional, Oujo, B., additional, Arevalo, M., additional, Bernabeu, C., additional, Perez-Barriocanal, F., additional, Jesper, K., additional, Nathalie, V., additional, Pierre, G., additional, Yi Chun, D. X., additional, Iyoda, M., additional, Shibata, T., additional, Matsumoto, K., additional, Shindo-Hirai, Y., additional, Kuno, Y., additional, Wada, Y., additional, Akizawa, T., additional, Schwartz, I., additional, Schwartz, D., additional, Prot Bertoye, C., additional, Terryn, S., additional, Claver, J., additional, Beghdadi, W. B., additional, Monteiro, R., additional, Blank, U., additional, Devuyst, O., additional, Daugas, E., additional, Van Beneden, K., additional, Geers, C., additional, Pauwels, M., additional, Mannaerts, I., additional, Van den Branden, C., additional, Van Grunsven, L. A., additional, Seckin, I., additional, Pekpak, M., additional, Uzunalan, M., additional, Uruluer, B., additional, Kokturk, S., additional, Ozturk, Z., additional, Sonmez, H., additional, Yaprak, E., additional, Furuno, Y., additional, Tsutsui, M., additional, Morishita, T., additional, Shimokawa, H., additional, Otsuji, Y., additional, Yanagihara, N., additional, Kabashima, N., additional, Ryota, S., additional, Kanegae, K., additional, Miyamoto, T., additional, Nakamata, J., additional, Ishimatsu, N., additional, Tamura, M., additional, Nakagawa, T., additional, Ichikawa, K., additional, Miyamoto, M., additional, Takabayashi, D., additional, Yamazaki, H., additional, Kakeshita, K., additional, Koike, T., additional, Kagitani, S., additional, Tomoda, F., additional, Hamashima, T., additional, Ishii, Y., additional, Inoue, H., additional, Sasahara, M., additional, El Machhour, F., additional, Kerroch, M., additional, Mesnard, L., additional, Chatziantoniou, C., additional, Dussaule, J.-C., additional, Inui, K., additional, Sasai, F., additional, Maruta, Y., additional, Nishiwaki, H., additional, Kawashima, E., additional, Inoue, Y., additional, Yoshimura, A., additional, Musacchio, E., additional, Priante, G., additional, Valvason, C., additional, Sartori, L., additional, Baggio, B., additional, Kim, J. H., additional, Gross, O., additional, Diana, R., additional, Gry, D. H., additional, Asimal, B., additional, Johanna, T., additional, Imke, S.-E., additional, Lydia, W., additional, Gerhard-Anton, M., additional, Hassan, D., additional, Cano, J. L., additional, Griera, M., additional, Olmos, G., additional, Martin, P., additional, Cortes, M. A., additional, Lopez-Ongil, S., additional, Rodriguez-Puyol, D., additional, DE Frutos, S., additional, Gonzalez, M., additional, Luengo, A., additional, Rodriguez-Puyol, M., additional, Calleros, L., additional, Lupica, R., additional, Lacquaniti, A., additional, Donato, V., additional, Maggio, R., additional, Mastroeni, C., additional, Lucisano, S., additional, Cernaro, V., additional, Fazio, M. R., additional, Quartarone, A., additional, Buemi, M., additional, Kacik, M., additional, Goedicke, S., additional, Eggert, H., additional, Hoyer, J. D., additional, Wurm, S., additional, Steege, A., additional, Banas, M., additional, Kurtz, A., additional, Banas, B., additional, Lasagni, L., additional, Lazzeri, E., additional, Romoli, S., additional, Schaefer, I., additional, Teng, B., additional, Worthmann, K., additional, Haller, H., additional, Schiffer, M., additional, Prattichizzo, C., additional, Netti, G. S., additional, Rocchetti, M. T., additional, Cormio, L., additional, Carrieri, G., additional, Stallone, G., additional, Grandaliano, G., additional, Ranieri, E., additional, Gesualdo, L., additional, Kucher, A., additional, Smirnov, A., additional, Parastayeva, M., additional, Beresneva, O., additional, Kayukov, I., additional, Zubina, I., additional, Ivanova, G., additional, Abed, A., additional, Schlekenbach, L., additional, Foglia, B., additional, Kwak, B., additional, Chadjichristos, C., additional, Queisser, N., additional, Schupp, N., additional, Brand, S., additional, Himer, L., additional, Szebeni, B., additional, Sziksz, E., additional, Saijo, S., additional, Kis, E., additional, Prokai, A., additional, Banki, N. F., additional, Fekete, A., additional, Tulassay, T., additional, Vannay, A., additional, Hegner, B., additional, Schaub, T., additional, Lange, C., additional, Dragun, D., additional, Klinkhammer, B. M., additional, Rafael, K., additional, Monika, M., additional, Anna, M., additional, Van Roeyen, C., additional, Boor, P., additional, Eva Bettina, B., additional, Simon, O., additional, Esther, S., additional, Floege, J., additional, Kunter, U., additional, Janke, D., additional, Jankowski, J., additional, Hayashi, M., additional, Takamatsu, I., additional, Horimai, C., additional, Yoshida, T., additional, Seno DI Marco, G., additional, Koenig, M., additional, Stock, C., additional, Reiermann, S., additional, Amler, S., additional, Koehler, G., additional, Fobker, M., additional, Buck, F., additional, Pavenstaedt, H., additional, Lang, D., additional, Brand, M., additional, Plotnikov, E., additional, Morosanova, M., additional, Pevzner, I., additional, Zorova, L., additional, Pulkova, N., additional, Zorov, D., additional, Wornle, M., additional, Ribeiro, A., additional, Belling, F., additional, Merkle, M., additional, Nakazawa, D., additional, Nishio, S., additional, Shibasaki, S., additional, Tomaru, U., additional, Akihiro, I., additional, Kobayashi, I., additional, Imanishi, Y., additional, Kurajoh, M., additional, Nagata, Y., additional, Yamagata, M., additional, Emoto, M., additional, Michigami, T., additional, Ishimura, E., additional, Inaba, M., additional, Wu, C.-C., additional, Lu, K.-C., additional, Chen, J.-S., additional, Chu, P., additional, Lin, Y.-F., additional, Eller, K., additional, Schroll, A., additional, Kirsch, A., additional, Huber, J., additional, Weiss, G., additional, Theurl, I., additional, Rosenkranz, A. R., additional, Zawada, A., additional, Rogacev, K., additional, Achenbach, M., additional, Fliser, D., additional, Held, G., additional, Heine, G. H., additional, Miyamoto, Y., additional, Iwao, Y., additional, Watanabe, H., additional, Kadowaki, D., additional, Ishima, Y., additional, Chuang, V. T. G., additional, Sato, K., additional, Otagiri, M., additional, Maruyama, T., additional, Iwatani, H., additional, Honda, D., additional, Noguchi, T., additional, Tanaka, M., additional, Tanaka, H., additional, Fukagawa, M., additional, Pircher, J., additional, Koppel, S., additional, Mannell, H., additional, Krotz, F., additional, Virzi, G. M., additional, Bolin, C., additional, Cruz, D., additional, Scalzotto, E., additional, De Cal, M., additional, Vescovo, G., additional, Ronco, C., additional, Grobmayr, R., additional, Lech, M., additional, Ryu, M., additional, Aoshima, Y., additional, Mizobuchi, M., additional, Ogata, H., additional, Kumata, C., additional, Nakazawa, A., additional, Kondo, F., additional, Ono, N., additional, Koiwa, F., additional, Kinugasa, E., additional, Freisinger, W., additional, Lale, N., additional, Lampert, A., additional, Ditting, T., additional, Heinlein, S., additional, Schmieder, R. E., additional, Veelken, R., additional, Nave, H., additional, Perthel, R., additional, Suntharalingam, M., additional, Bode-Boger, S., additional, Beutel, G., additional, Kielstein, J., additional, Rodrigues-Diez, R., additional, Rayego-Mateos, S., additional, Lavoz, C., additional, Stark Aroeira, L. G., additional, Orejudo, M., additional, Alique, M., additional, Ortiz, A., additional, Egido, J., additional, Ruiz-Ortega, M., additional, Oskar, W., additional, Rusan, C., additional, Padberg, J.-S., additional, Wiesinger, A., additional, Reuter, S., additional, Grabner, A., additional, Kentrup, D., additional, Lukasz, A., additional, Oberleithner, H., additional, Pavenstadt, H., additional, Kumpers, P., additional, Eberhardt, H. U., additional, Skerka, C., additional, Chen, Q., additional, Hallstroem, T., additional, Hartmann, A., additional, Kemper, M. J., additional, Zipfel, P. F., additional, N'gome-Sendeyo, K., additional, Fan, Q.-F., additional, Toblli, J., additional, Cao, G., additional, Giani, J. F., additional, Dominici, F. P., additional, Kim, J. S., additional, Yang, J. W., additional, Kim, M. K., additional, Han, B. G., additional, and Choi, S. O., additional

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2012
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