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47 results on '"Snord116"'

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1. RNAi Knockdown of EHMT2 in Maternal Expression of Prader–Willi Syndrome Genes.

2. Hormonal Imbalances in Prader–Willi and Schaaf–Yang Syndromes Imply the Evolution of Specific Regulation of Hypothalamic Neuroendocrine Function in Mammals.

3. Cognitive deficits in the Snord116 deletion mouse model for Prader-Willi syndrome

4. Atypical 15q11.2-q13 Deletions and the Prader-Willi Phenotype.

5. Reference Genes across Nine Brain Areas of Wild Type and Prader-Willi Syndrome Mice: Assessing Differences in Igfbp7 , Pcsk1 , Nhlh2 and Nlgn3 Expression.

6. Phylogenetic and Molecular Analyses Identify SNORD116 Targets Involved in the Prader–Willi Syndrome.

7. Newly developed oral bioavailable EHMT2 inhibitor as a potential epigenetic therapy for Prader-Willi syndrome.

8. Patients with PWS and related syndromes display differentially methylated regions involved in neurodevelopmental and nutritional trajectory.

9. Reference Genes across Nine Brain Areas of Wild Type and Prader-Willi Syndrome Mice:Assessing Differences in Igfbp7, Pcsk1, Nhlh2 and Nlgn3 Expression

10. Atypical 15q11.2-q13 Deletions and the Prader-Willi Phenotype

11. Hypothalamus Specific Re-Introduction of SNORD116 into Otherwise Snord116 Deficient Mice Increased Energy Expenditure.

12. Ambient temperature modulates the effects of the Prader-Willi syndrome candidate gene Snord116 on energy homeostasis.

13. Phylogenetic and molecular analyses identify SNORD116 targets involved in the Prader Willi syndrome

14. Prader-Willi syndrome and atypical submicroscopic 15q11-q13 deletions with or without imprinting defects.

15. Non-coding RNA genes lost in Prader-Willi Syndrome stabilize target RNAs

16. Patients with PWS and related syndromes display differentially methylated regions involved in neurodevelopmental and nutritional trajectory

17. Reference Genes across Nine Brain Areas of Wild Type and Prader-Willi Syndrome Mice: Assessing Differences in Igfbp7, Pcsk1, Nhlh2 and Nlgn3 Expression

18. Linking oxytocin and arginine vasopressin signaling abnormalities to social behavior impairments in Prader-Willi syndrome.

19. Dysregulated adipose tissue expansion and impaired adipogenesis in Prader-Willi syndrome children before obesity-onset.

20. Behavioural characteristics of the Prader–Willi syndrome related biallelic Snord116 mouse model.

21. Novel insight into box C/D snoRNP molecular partners and related biological functions

22. Prader–Willi-Like Phenotype Caused by an Atypical 15q11.2 Microdeletion

23. Dietary Conjugated Linoleic Acid Reduces Body Weight and Fat in Snord116m+/p− and Snord116m−/p− Mouse Models of Prader–Willi Syndrome

24. A Transcriptomic Signature of the Hypothalamic Response to Fasting and BDNF Deficiency in Prader-Willi Syndrome

25. Dietary Conjugated Linoleic Acid Reduces Body Weight and Fat in Snord116 m+/p− and Snord116 m−/p− Mouse Models of Prader–Willi Syndrome.

26. Mouse models of Prader–Willi Syndrome: A systematic review.

27. High expression of miR-125b-2 and SNORD116 noncoding RNA clusters characterize ERG-related B cell precursor acute lymphoblastic leukemia

29. Increased brain age in adults with Prader-Willi syndrome

30. A Comprehensive Review of Genetically Engineered Mouse Models for Prader-Willi Syndrome Research

31. Dietary Conjugated Linoleic Acid Reduces Body Weight and Fat in Snord116 m+/p- and Snord116 m-/p- Mouse Models of Prader-Willi Syndrome.

32. A Transcriptomic Signature of the Hypothalamic Response to Fasting and BDNF Deficiency in Prader-Willi Syndrome

33. A Comprehensive Review of Genetically Engineered Mouse Models for Prader-Willi Syndrome Research.

34. High expression of miR-125b-2 and SNORD116 noncoding RNA clusters characterize ERG-related B cell precursor acute lymphoblastic leukemia

35. Hypothalamus specific re-introduction of Snord116 into otherwise Snord116 deficient mice increased energy expenditure

36. Ambient temperature modulates the effects of the Prader-Willi syndrome candidate gene Snord116 on energy homeostasis

37. Cognitive deficits in the Snord116 deletion mouse model for Prader-Willi syndrome

38. Prader–Willi-Like Phenotype Caused by an Atypical 15q11.2 Microdeletion.

39. Deletion of the Snord116/SNORD116 Alters Sleep in Mice and Patients with Prader-Willi Syndrome

40. Cognitive deficits in the Snord116 deletion mouse model for Prader-Willi syndrome.

41. Increased brain age in adults with Prader-Willi syndrome.

42. SNORD116 deletions cause Prader-Willi syndrome with a mild phenotype and macrocephaly.

43. A Transcriptomic Signature of the Hypothalamic Response to Fasting and BDNF Deficiency in Prader-Willi Syndrome.

44. High expression of miR-125b-2 and SNORD116 noncoding RNA clusters characterize ERG-related B cell precursor acute lymphoblastic leukemia.

45. Adult-onset deletion of the Prader-Willi syndrome susceptibility gene Snord116 in mice results in reduced feeding and increased fat mass.

46. Deletion of the Snord116/SNORD116 Alters Sleep in Mice and Patients with Prader-Willi Syndrome.

47. Increased brain age in adults with Prader-Willi syndrome

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