34 results on '"Stamnaes, Jorunn"'
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2. Selective activation of naïve B cells with unique epitope specificity shapes autoantibody formation in celiac disease
3. Autoantibody binding and unique enzyme-substrate intermediate conformation of human transglutaminase 3
4. Biopsy Proteome Score Performs Well as an Effect Measure in a Gluten Challenge Trial of Celiac Disease
5. Efficient T cell–B cell collaboration guides autoantibody epitope bias and onset of celiac disease
6. Characterization of the Small Intestinal Lesion in Celiac Disease by Label-Free Quantitative Mass Spectrometry
7. Separate Gut Plasma Cell Populations Produce Auto‐Antibodies against Transglutaminase 2 and Transglutaminase 3 in Dermatitis Herpetiformis
8. Expression of transglutaminase 2 in human gut epithelial cells: Implications for coeliac disease
9. Transglutaminase 2 affinity and enzyme‐substrate intermediate stability as determining factors for T‐cell responses to gluten peptides in celiac disease
10. Dissecting the interaction between transglutaminase 2 and fibronectin
11. Injection of prototypic celiac anti-transglutaminase 2 antibodies in mice does not cause enteropathy
12. TG2-gluten complexes as antigens for gluten-specific and transglutaminase-2 specific B cells in celiac disease
13. Insights from tissue “omics” analysis on intestinal remodeling in celiac disease
14. Transglutaminase 2 interactions with extracellular matrix proteins as probed with celiac disease autoantibodies
15. The propensity for deamidation and transamidation of peptides by transglutaminase 2 is dependent on substrate affinity and reaction conditions
16. Gluten T cell epitope targeting by TG3 and TG6; implications for dermatitis herpetiformis and gluten ataxia
17. In Well‐Treated Celiac Patients Low‐Level Mucosal Inflammation Predicts Response to 14‐day Gluten Challenge
18. Evidence That Pathogenic Transglutaminase 2 in Celiac Disease Derives From Enterocytes
19. Quantitative proteomics of coeliac gut during 14-day gluten challenge: low-level baseline inflammation despite clinical and histological normality predicts subsequent response
20. B cell tolerance and antibody production to the celiac disease autoantigen transglutaminase 2
21. Epitope-dependent Functional Effects of Celiac Disease Autoantibodies on Transglutaminase 2
22. Transglutaminase 2 strongly binds to an extracellular matrix component other than fibronectin via its second C‐terminal beta‐barrel domain
23. Dissecting the interaction between transglutaminase 2 and fibronectin
24. Enhanced B-Cell Receptor Recognition of the Autoantigen Transglutaminase 2 by Efficient Catalytic Self-Multimerization
25. Small Bowel, Celiac Disease and Adaptive Immunity
26. Transglutaminases in Gluten Sensitive Diseases
27. HLA-DQ Molecules as Affinity Matrix for Identification of Gluten T Cell Epitopes
28. Transglutaminase 2–Specific Autoantibodies in Celiac Disease Target Clustered, N-Terminal Epitopes Not Displayed on the Surface of Cells
29. High abundance of plasma cells secreting transglutaminase 2–specific IgA autoantibodies with limited somatic hypermutation in celiac disease intestinal lesions
30. Activation and Inhibition of Transglutaminase 2 in Mice
31. Activation of Extracellular Transglutaminase 2 by Thioredoxin
32. Redox Regulation of Transglutaminase 2 Activity
33. Activation of Extracellular Transglutaminase 2 by Thioredoxin.
34. B cell tolerance and antibody production to the celiac disease autoantigen transglutaminase 2
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