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16 results on '"Sun, Si-Min"'

6. NLRP14 Safeguards Calcium Homeostasis via Regulating the K27 Ubiquitination of Nclx in Oocyte‐to‐Embryo Transition

Author

Meng, Tie‐Gang, primary, Guo, Jia‐Ni, additional, Zhu, Liu, additional, Yin, Yike, additional, Wang, Feng, additional, Han, Zhi‐Ming, additional, Lei, Lei, additional, Ma, Xue‐Shan, additional, Xue, Yue, additional, Yue, Wei, additional, Nie, Xiao‐Qing, additional, Zhao, Zheng‐Hui, additional, Zhang, Hong‐Yong, additional, Sun, Si‐Min, additional, Ouyang, Ying‐Chun, additional, Hou, Yi, additional, Schatten, Heide, additional, Ju, Zhenyu, additional, Ou, Xiang‐Hong, additional, Wang, Zhen‐Bo, additional, Wong, Catherine C. L., additional, Li, Zhonghan, additional, and Sun, Qing‐Yuan, additional

Published
2023
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8. SRSF10 is essential for progenitor spermatogonia expansion by regulating alternative splicing

Author

Liu, Wenbo, primary, Lu, Xukun, primary, Zhao, Zheng-Hui, primary, SU, Ruibao, primary, Li, Qian-Nan Li, additional, Xue, Yue, additional, Gao, Zheng, additional, Sun, Si-Min Sun, additional, Lei, Wen-Long, additional, Li, Lei, additional, An, Geng, additional, Liu, Hanyan, additional, Han, Zhiming, additional, Ouyang, Ying-Chun, additional, Hou, Yi, additional, Wang, Zhen-Bo, additional, Sun, Qing-Yuan, additional, and Liu, Jianqiao, additional

Published
2022
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9. Author response: SRSF10 is essential for progenitor spermatogonia expansion by regulating alternative splicing

Author

Liu, Wenbo, primary, Lu, Xukun, primary, Zhao, Zheng-Hui, primary, SU, Ruibao, primary, Li, Qian-Nan Li, additional, Xue, Yue, additional, Gao, Zheng, additional, Sun, Si-Min Sun, additional, Lei, Wen-Long, additional, Li, Lei, additional, An, Geng, additional, Liu, Hanyan, additional, Han, Zhiming, additional, Ouyang, Ying-Chun, additional, Hou, Yi, additional, Wang, Zhen-Bo, additional, Sun, Qing-Yuan, additional, and Liu, Jianqiao, additional

Published
2022
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10. SRSF2 is required for mRNA splicing and spermatogenesis

Author

Lei, Wen-Long, primary, Du, Zongchang, additional, Meng, Tie-Gang, additional, Su, Ruibao, additional, Li, Yuan-Yuan, additional, Liu, Wenbo, additional, Sun, Si-Min, additional, Liu, Meng-Yu, additional, Hou, Yi, additional, Zhang, Chun-Hui, additional, Gui, Yaoting, additional, Schatten, Heide, additional, Han, Zhiming, additional, Liu, Chenli, additional, Wang, Zhen-Bo, additional, Qian, Wei-Ping, additional, and Sun, Qing-Yuan, additional

Published
2022
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11. MAPRE2 regulates the first meiotic progression in mouse oocytes

Author

Li, Yuan-Yuan, primary, Lei, Wen-Long, additional, Zhang, Chang-Fa, additional, Sun, Si-Min, additional, Zhao, Bing-Wang, additional, Xu, Ke, additional, Hou, Yi, additional, Ouyang, Ying-Chun, additional, Wang, Zhen-Bo, additional, Guo, Lei, additional, Sun, Qing-Yuan, additional, and Han, Zhiming, additional

Published
2022
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16. Protein phosphatase 4 maintains the survival of primordial follicles by regulating autophagy in oocytes.

Author

Dong MZ, Ouyang YC, Gao SC, Gu LJ, Guo JN, Sun SM, Wang ZB, and Sun QY

Subjects
Animals, Female, Mice, Infertility, Female pathology, Infertility, Female metabolism, Infertility, Female genetics, Mice, Knockout, Signal Transduction, TOR Serine-Threonine Kinases metabolism, Autophagy, Oocytes metabolism, Ovarian Follicle metabolism, Phosphoprotein Phosphatases metabolism, Phosphoprotein Phosphatases genetics
Abstract

In mammalian ovary, the primordial follicle pool serves as the source of developing follicles and fertilizable ova. To maintain the normal length of female reproductive life, the primordial follicles must have adequate number and be kept in a quiescent state before menopause. However, the molecular mechanisms underlying primordial follicle survival are poorly understood. Here, we provide genetic evidence showing that lacking protein phosphatase 4 (PPP4) in oocytes, a member of PP2A-like subfamily, results in infertility in female mice. A large quantity of primordial follicles has been depleted around the primordial follicle pool formation phase and the ovarian reserve is exhausted at about 7 months old. Further investigation demonstrates that depletion of PPP4 causes the abnormal activation of mTOR, which suppresses autophagy in primordial follicle oocytes. The abnormal primordial follicle oocytes are eventually erased by pregranulosa cells in the manner of lysosome invading. These results show that autophagy prevents primordial follicles over loss and PPP4-mTOR pathway governs autophagy during the primordial follicle formation and dormant period., (© 2024. The Author(s).)

Published
2024
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