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1. Synthetic retinoid-mediated preconditioning of cancer-associated fibroblasts and macrophages improves cancer response to immune checkpoint blockade

6. Pharmacologic conversion of cancer-associated fibroblasts from a protumor phenotype to an antitumor phenotype improves the sensitivity of pancreatic cancer to chemotherapeutics

8. The Origin and Contribution of Cancer-Associated Fibroblasts in Colorectal Carcinogenesis

11. The Balance of Stromal BMP Signaling Mediated by GREM1 and ISLR Drives Colorectal Carcinogenesis

17. Meflin is a marker of pancreatic stellate cells involved in fibrosis and epithelial regeneration in the pancreas

20. Correction: Pharmacologic conversion of cancer-associated fibroblasts from a protumor phenotype to an antitumor phenotype improves the sensitivity of pancreatic cancer to chemotherapeutics

25. Meflin is a marker of pancreatic stellate cells involved in fibrosis and epithelial regeneration in the pancreas.

31. Supplementary Data from Meflin-Positive Cancer-Associated Fibroblasts Inhibit Pancreatic Carcinogenesis

32. Supplemental Table S1 from Akt–Girdin Signaling in Cancer-Associated Fibroblasts Contributes to Tumor Progression

34. Supplemental Figure S1 from Akt–Girdin Signaling in Cancer-Associated Fibroblasts Contributes to Tumor Progression

42. Data from A Novel Mouse Model for De novo Melanoma

45. Data from Akt–Girdin Signaling in Cancer-Associated Fibroblasts Contributes to Tumor Progression

50. Roles of the Mesenchymal Stromal/Stem Cell Marker Meflin in Cardiac Tissue Repair and the Development of Diastolic Dysfunction

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