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Your search keyword '"Tang, Jing-Shi"' showing total 180 results
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180 results on '"Tang, Jing-Shi"'

1. Pain

Author

Chen, Jun, Han, Ji-Sheng, Zhao, Zhi-Qi, Wei, Feng, Hsieh, Jen-Chuen, Bao, Lan, Chen, Andrew C. N., Dai, Yi, Fan, Bi-Fa, Gu, Jian-Guo, Hao, Shuang-Lin, Hu, San-Jue, Ji, Yong-Hua, Li, Yong-Jie, Li, Yun-Qing, Lin, Qing, Liu, Xian-Guo, Liu, Yan-Qing, Lu, Yan, Luo, Fei, Ma, Chao, Qiu, Yun-Hai, Rao, Zhi-Ren, Shi, Lin, Shyu, Bai-Chuang, Song, Xue-Jun, Tang, Jing-Shi, Tao, Yuan-Xiang, Wan, You, Wang, Jia-Shuang, Wang, Ke-Wei, Wang, Yun, Xu, Guang-Yin, Xu, Tian-Le, You, Hao-Jun, Yu, Long-Chuan, Yu, Sheng-Yuan, Zhang, Da-Ying, Zhang, De-Ren, Zhang, Jun-Ming, Zhang, Xu, Zhang, Yu-Qiu, Zhuo, Min, Pfaff, Donald W., editor, Volkow, Nora D., editor, and Rubenstein, John L., editor

Published
2022
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6. Pain

Author

Chen, Jun, Han, Ji-Sheng, Zhao, Zhi-Qi, Wei, Feng, Hsieh, Jen-Chuen, Bao, Lan, Chen, Andrew C. N., Dai, Yi, Fan, Bi-Fa, Gu, Jian-Guo, Hao, Shuang-Lin, Hu, San-Jue, Ji, Yong-Hua, Li, Yong-Jie, Li, Yun-Qing, Lin, Qing, Liu, Xian-Guo, Liu, Yan-Qing, Lu, Yan, Luo, Fei, Ma, Chao, Qiu, Yun-Hai, Rao, Zhi-Ren, Shi, Lin, Shyu, Bai-Chuang, Song, Xue-Jun, Tang, Jing-Shi, Tao, Yuan-Xiang, Wan, You, Wang, Jia-Shuang, Wang, Ke-Wei, Wang, Yun, Xu, Guang-Yin, Xu, Tian-Le, You, Hao-Jun, Yu, Long-Chuan, Yu, Sheng-Yuan, Zhang, Da-Ying, Zhang, De-Ren, Zhang, Jun-Ming, Zhang, Xu, Zhang, Yu-Qiu, Zhuo, Min, Pfaff, Donald W., editor, and Volkow, Nora D., editor

Published
2016
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10. Pain

Author

Chen, Jun, Han, Ji-Sheng, Zhao, Zhi-Qi, Wei, Feng, Hsieh, Jen-Chuen, Bao, Lan, Chen, Andrew CN, Dai, Yi, Fan, Bi-Fa, Gu, Jian-Guo, Hao, Shuang-Lin, Hu, San-Jue, Ji, Yong-Hua, Li, Yong-Jie, Li, Yun-Qing, Lin, Qing, Liu, Xian-Guo, Liu, Yan-Qing, Lu, Yan, Luo, Fei, Ma, Chao, Qiu, Yun-Hai, Rao, Zhi-Ren, Shi, Lin, Shyu, Bai-Chuang, Song, Xue-Jun, Tang, Jing-Shi, Tao, Yuan-Xiang, Wan, You, Wang, Jia-Shuang, Wang, Ke-Wei, Wang, Yun, Xu, Guang-Yin, Xu, Tian-Le, You, Hao-Jun, Yu, Long-Chuan, Yu, Sheng-Yuan, Zhang, Da-Ying, Zhang, De-Ren, Zhang, Jun-Ming, Zhang, Xu, Zhang, Yu-Qiu, Zhuo, Min, and Pfaff, Donald W., editor

Published
2013
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16. The Characteristics and Related Problems of the Orbits Around the Earth-Moon Libration Points

Author

Tang Jing-shi, Liu Lin, and Hou Xi-yun

Subjects
Physics, 010308 nuclear & particles physics, Synodic day, Astronomy, Astronomy and Astrophysics, Ellipse, 01 natural sciences, Celestial mechanics, Lissajous curve, Orbit, Space and Planetary Science, Physics::Space Physics, 0103 physical sciences, Libration, Astrophysics::Earth and Planetary Astrophysics, Halo, Orbit determination, 010303 astronomy & astrophysics
Abstract

Due to the specific dynamics, the probes located at the halo orbits or Lissajous orbits around the Earth-Moon collinear libration point L1 or L2 are always studied in the synodic system to understand their trajectories. In fact, they are also orbiting the Earth in a distant Keplerian ellipse. Because of their intrinsic orbital instability, in the orbit prediction the initial errors propagate more prominently than those of the normal orbiting satellites, this requires special attention in the orbit design, maneuver, and control. Despite of all this, they are similar to the normal orbiting satellites in orbit determination and hardly require other special attentions. In this paper, the quantitative results of error propagation under the unstable dynamics, together with the theoretical analysis are presented. The results of precise orbit determination and short-arc orbit predictions are also shown, and compared with the results from the Beijing Aerospace Control Center.

Published
2019
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31. Transfer to the Collinear Libration Point L3 in the Sun-Earth+Moon System

Author

Hou, Xi-yun, Tang, Jing-shi, and Liu, Lin

Subjects
Astrodynamics
Abstract

The collinear libration point L3 of the sun-earth+moon system is an ideal place for some space missions. Although there has been a great amount of work concerning the applications of the other two collinear libration points L1 and L2, little work has been done about the point L3. In this paper, the dynamics of the libration points was briefly introduced first. Then a way to transfer the spacecraft to the collinear libration point L3 via the invariant manifolds of the other two collinear libration points was proposed. Theoretical works under the model of circular restricted three-body problem were done. For the sun-earth+moon system, this model is a good approximation. The results obtained are useful when a transfer trajectory under the real solar system is designed.

Published
2007

38. Involvement of 5-HT2A, 5-HT2Band 5-HT2Creceptors in mediating the ventrolateral orbital cortex-induced antiallodynia in a rat model of neuropathic pain

Author

Xu, Wen-Jin, Wang, Yu-Ying, Zhao, Yan, Jia, Hong, Tang, Jing-Shi, Huo, Fu-Quan, and Liu, Hua

Abstract

The present study examined the roles of 5-HT2A, 5-HT2Band 5-HT2Creceptor subtypes in mediating the ventrolateral orbital cortex (VLO)-induced antiallodynia in a rat model of neuropathic pain induced by spared nerve injury (SNI). Change of mechanical paw withdrawal threshold (PWT) was measured using von-Frey filaments. Microinjection of preferential or selective 5-HT2A/C, 5-HT2Band 5-HT2Creceptor agonists, (±)-1-(2,5-Dimethoxy-4-iodophenyl)-2-aminopropane hydrochloride (DOI), α-methyl-5-(2-thienylmethoxy)-1H-Indole-3-ethanamine hydrochloride (BW723C86) and 1-(3-Chlorophenyl)-piperazine hydrochloride (m-CPP) into the VLO significantly depressed allodynia induced by SNI, and the inhibitory effect of DOI was blocked or attenuated by selective 5-HT2A/Creceptor antagonists ketanserin (+)-tartrate salt (ketanserin) and 5-HT2Areceptor antagonist R-(+)-alpha-(2,3-dimethoxyphenyl)-1-[2-(4-fluorophenylethyl)]-4-piperidinemethanol (M100907); the effects of BW723C86 and m-CPP were antagonized by 5-HT2Breceptor antagonists N-(1-Methyl-1H-5-indolyl)-N′-(3-methyl-5-isothiazolyl)urea (SB204741) and 5-HT2Creceptor antagonist RS102221 hydrochloride hydrate (RS-102221), respectively. These results suggest that 5-HT2A, 5-HT2B, 5-HT2Creceptor subtypes are involved in mediating the VLO-induced antiallodynia in the neuropathic pain state.

Published
2020
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