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The temperature profile is an important process parameter for the autoclave curing of composite, which has an important influence on the curing time and the quality of the cured composite part. The curing period and curing quality of autoclave processed composite can be simultaneously controlled by the optimal design of the temperature profile. The numerical simulation of the curing process of a C-shaped composite part was carried out. The prediction results are in good agreement with the experimental test results, which verify the capability of the simulation method. Then, the temperature profile was optimized by using the design of experiment (DOE) method and particle swarm algorithm to achieve the simultaneous control of curing period and curing quality. Experiments were also carried out to verify the optimization results. The results show that the optimal design of temperature profile can effectively reduce the curing period and satisfy the constraints of curing uniformity, curing degree and curing deformation. The curing periods of the optimization cases are reduced by 64% and 45%, respectively.

The article presents chemical equations for the article "An Efficient Synthesis of Imidodicarbonic Diamides from 1,3-Thiazetidin-2-ones with NH2OH·HCl via Ring-Opening Reaction" by W.-Y. Tang and others, published in the periodical "Chinese Chemical Letters" in 2015.

Objective: To study the pattern of the alterations of blood glucose, insulin and insulin sensitivity after traumatic brain injury in rats, and verify the occurrence of insulin resistance after the injury., Methods: Based on Feeney's model of brain injury, the blood glucose and insulin concentration of the dogs measured 30 min before and at 6, 12, 24, 48, 72 and 120 h after injury. BG60-120, GIR60-120, and insulin sensitivity index (ISI) reflecting the insulin sensitivity were measured at 6, 24, 48, and 72 hours following severe traumatic brain injury using euglycemic-hyperinsulinemic clamp., Results: Both the blood glucose and insulin concentration increased markedly in rats following moderate and severe brain injury. BG60-120 increased markedly, and GIR60-120 and ISI decreased significantly 6, 24, 48, and 72 h after severe brain trauma as compared with those of the sham operation group. Blood glucose concentration of rats following severe injury was positively correlated with insulin concentration and BG60-120 at the corresponding time points, but negatively with GIR60-120 and ISI., Conclusion: Both the blood glucose and insulin concentration increase markedly in rats following severe brain injury. Increased blood glucose even in the presence of high-level insulin is due to acute insulin resistance occurring after traumatic brain injury.

Objective: To observe the changes of brain oxygen metabolism and neuroelectrophysiology after severe brain injury, and the effects of hypothermia on severe brain injury., Methods: 148 patients with severe brain injury (GCS 3 - 8, admitted within 10 hours from injury) were selected for this study. Patients were divided into 3 groups, Group GCS 7 - 8, Group GCS 5 - 6 and Group GCS 3 - 4. Every group were also randomly assigned to normothermia and hypothermia subgroup. Patients in the hypothermia group were cooled to 32 approximately 34 degrees C. SLSEP, BAEP, P(br)O(2) and rSaO(2) were recorded in each group at the same time., Results: In the Group GCS 7 - 8, N20 in SLSEP, I/V in BAEP and rSaO(2) were improved significantly after mild hypothermia treatment, and P(br)O(2) was decreased by hypothermia; In the Group GCS 5 - 6, N20 in SLSEP, I/V in BAEP and rSaO(2) were improved by hypothermia, and P(br)O(2) was decreased in hypothermia subgroup; In the Group GCS 3 - 4, no significant difference was found., Conclusion: Mild hypothermia has a significant effect on patients of GCS 7 - 8 and a doubt effect on patients of GCS 5 - 6. It seem no effect on patients of GCS 3 - 4. Brain oxygen metabolism and neuroelectrophysiology are important to value the therapeutic effect on severe brain injury.