32 results on '"Taylor, Shalina"'
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2. Dysregulated Smooth Muscle Cell BMPR2–ARRB2 Axis Causes Pulmonary Hypertension
3. The Right Heart Network and Risk Stratification in Pulmonary Arterial Hypertension
4. KLF4 recruits SWI/SNF to increase chromatin accessibility and reprogram the endothelial enhancer landscape under laminar shear stress
5. High Shear Stress Reduces ERG Causing Endothelial-Mesenchymal Transition and Pulmonary Arterial Hypertension
6. Multi-omic profiling of primary mouse neutrophils predicts a pattern of sex- and age-related functional regulation
7. Abstract 10270: High Shear Stress Decreases ERG Causing Endothelial to Mesenchymal Transition and Pulmonary Arterial Hypertension
8. Smooth Muscle Contact Drives Endothelial Regeneration by BMPR2-Notch1–Mediated Metabolic and Epigenetic Changes
9. Endogenous Retroviral Elements Generate Pathologic Neutrophils in Pulmonary Arterial Hypertension
10. Abstract 16861: Neutrophils in Pulmonary Arterial Hypertension Show Heightened Elastase Along With Propensity to Form Extracellular Traps and an Antiviral Signature
11. Severe Pulmonary Arterial Hypertension Is Characterized by Increased Neutrophil Elastase and Relative Elafin Deficiency
12. Monocyte released HERV-K dUTPase engages TLR4 and MCAM causing endothelial mesenchymal transition
13. Endogenous Retroviral Elements Generate Pathologic Neutrophils and Elastase Rich Exosomes in Pulmonary Arterial Hypertension
14. KLF4 Recruits SWI/SNF to Increase Chromatin Accessibility and Reprogram the Endothelial Enhancer Landscape under Laminar Shear Stress
15. Multi-omic profiling of primary mouse neutrophils reveals a pattern of sex and age-related functional regulation
16. Molecular Choreography of Acute Exercise
17. Sodium tanshinone IIA sulfonate prevents the adverse left ventricular remodelling: Focus on polymorphonuclear neutrophil‐derived granule components
18. Abstract 17192: ALDH1A3 Induces NFY to Coordinate Transcription of Cell Cycle and Metabolic Genes Necessary for Smooth Muscle Proliferation in Pulmonary Hypertension
19. The Role of Neutrophils and Neutrophil Elastase in Pulmonary Arterial Hypertension
20. Induced Pluripotent Stem Cell Model of Pulmonary Arterial Hypertension Reveals Novel Gene Expression and Patient Specificity
21. Patient-Specific iPSC-Derived Endothelial Cells Uncover Pathways that Protect against Pulmonary Hypertension in BMPR2 Mutation Carriers
22. Oxidant Sensing by TRPM2 Inhibits Neutrophil Migration and Mitigates Inflammation
23. Abstract 596: Increased Migration of Neutrophils Across Endothelial Cells from Patients with Pulmonary Arterial Hypertension is Related to Reduced Platelet Endothelial Cell Adhesion Molecule -1 and is Prevented by the Neutrophil Elastase Inhibitor Elafin
24. Abstract 17231: Contact-Mediated Interaction Between Pulmonary Artery Endothelial and Smooth Muscle Cells Promotes a BMPR2-β-catenin-Notch1 Signal Causing Hyperpolarization of Endothelial Mitochondria and a Stalk Cell-like Phenotype
25. Moesin and myosin phosphatase confine neutrophil orientation in a chemotactic gradient
26. Regulation of polyisoprenylated methylated protein methyl esterase by polyunsaturated fatty acids and prostaglandins
27. Fatty Acid Suppression of Cell Proliferation is Associated with the Inhibition of Polyisoprenylated Methylated Protein Methyl Esterase
28. Polyunsaturated Fatty Acids (PUFAs) Inhibit Polyisoprenylated Methylated Protein Methyl Esterase (PMPMEase)
29. Moesin and myosin phosphatase confine neutrophil orientation in a chemotactic gradient
30. Multi-omic profiling reveals widespread dysregulation of innate immunity and hematopoiesis in COVID-19
31. Abstract 16925: KLF and AP1 Having Opposing Roles in Regulating Chromatin Accessibility and the Endothelial Enhancer Landscape Under Laminar vs. Disturbed Shear Stress.
32. High Shear Stress Reduces ERG Causing Endothelial-Mesenchymal Transition and Pulmonary Arterial Hypertension.
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