173 results on '"Tojima, Ichiro"'
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2. Practical guide for the diagnosis and management of primary ciliary dyskinesia
3. SARS-CoV-2 induces inflammation and intracranial infection through the olfactory epithelium-olfactory bulb pathway in non-human primates
4. Immunological effects of sublingual immunotherapy with Japanese cedar pollen extract in patients with combined Japanese cedar and Japanese cypress pollinosis
5. Endoscopic Modified Lothrop (Draf Type III) Procedure for a Case of Refractory Frontal Sinusitis with a Cutaneous Fistula: A Case Report
6. Sublingual immunotherapy decreases the avidity of antigen‐specific IgE in patients with Japanese cedar pollinosis
7. Soluble ST2 suppresses IL-5 production by human basophilic KU812 cells, induced by epithelial cell-derived IL-33
8. A mechanism of interleukin-25 production from airway epithelial cells induced by Japanese cedar pollen
9. The epidermal growth factor receptor inhibitor AG1478 inhibits eosinophilic inflammation in upper airways
10. Increase in the prevalence of follicular regulatory T cells correlates with clinical efficacy of sublingual immunotherapy with house dust mites
11. Increase in the prevalence of follicular regulatory T cells correlates with clinical efficacy of sublingual immunotherapy with house dust mites.
12. Group 2 innate lymphoid cells are increased in nasal polyps in patients with eosinophilic chronic rhinosinusitis
13. Group 2 innate lymphoid cells and eosinophilic chronic rhinosinusitis
14. A decreased prevalence of group 2 innate lymphoid cells in blood is associated with good postoperative outcomes in patients with chronic rhinosinusitis.
15. A decreased prevalence of group 2 innate lymphoid cells in blood is associated with good postoperative outcomes in patients with chronic rhinosinusitis
16. Increased blood group 2 innate lymphoid cells are associated with the clinical severity of Kimura disease
17. Increased blood group 2 innate lymphoid cells are associated with the clinical severity of Kimura disease
18. Therapeutic Potential of Valproic Acid for Postviral Olfactory Dysfunction: A Single-Arm Pilot Study.
19. Therapeutic Potential of Valproic Acid for Postviral Olfactory Dysfunction: A Single-Arm Pilot Study
20. A Histochemical Analysis of Neurofibrillary Tangles in Olfactory Epithelium, a Study Based on an Autopsy Case of Juvenile Alzheimer’s Disease
21. Nasal polyp fibroblasts (NPFs)-derived exosomes are important for the release of vascular endothelial growth factor from cocultured eosinophils and NPFs
22. Endogenous Protease Inhibitors in Airway Epithelial Cells Contribute to Eosinophilic Chronic Rhinosinusitis
23. Increased blood group 2 innate lymphoid cells are involved in blood eosinophilia and itching in Kimura disease
24. Eighth Report of the Hands-on Seminar on Basic Research for Clinicians at the 60th Annual Meeting of the Japanese Rhinologic Society and the 20th Asian Research Symposium in Rhinology
25. A Case of Congenital Nasal Pyriform Aperture Stenosis Successfully Treated with Intranasal Tube Stenting
26. 17,18-Epoxyeicosatetraenoic Acid Inhibits TNF-α-Induced Inflammation in Cultured Human Airway Epithelium and LPS-Induced Murine Airway Inflammation
27. Anti-inflammatory roles of interleukin-35 in the pathogenesis of Japanese cedar pollinosis
28. Transcription of Interleukin-25 and Extracellular Release of the Protein Is Regulated by Allergen Proteases in Airway Epithelial Cells
29. Increased blood group 2 innate lymphoid cells contribute to blood eosinophilia, elevated IgE, and itching in Kimura disease
30. Recovery Over Time and Prognostic Factors in Treated Patients with Post-Infectious Olfactory Dysfunction: A Retrospective Study
31. Sinonasal undifferentiated carcinoma with skull-base invasion treated with induction chemotherapy and concurrent chemoradiotherapy consisting of cisplatin plus etoposide
32. 17,18-Epoxyeicosatetraenoic Acid Inhibits TNF-α-Induced Inflammation in Cultured Human Airway Epithelium and LPS-Induced Murine Airway Inflammation.
33. In vitro and in vivo inhibitory effects of TLR4 agonist, glucopyranosyl lipid A (GLA), on allergic rhinitis caused by Japanese cedar pollen
34. Evidence for the induction of Th2 inflammation by group 2 innate lymphoid cells in response to prostaglandin D 2 and cysteinyl leukotrienes in allergic rhinitis
35. Arachidonic acid metabolites-induced activation of group 2 innate lymphoid cell in eosinophilic upper airway inflammation
36. Anti-inflammatory effects of a novel non-antibiotic macrolide, EM900, on mucus secretion of airway epithelium
37. In vitro and in vivo inhibitory effects of TLR4 agonist, glucopyranosyl lipid A (GLA), on allergic rhinitis caused by Japanese cedar pollen.
38. Evidence for the induction of Th2 inflammation by group 2 innate lymphoid cells in response to prostaglandin D2 and cysteinyl leukotrienes in allergic rhinitis.
39. Severely infected pneumoceles of the frontal sinus in patients with mental retardation and brain atrophy treated by endoscopic sinus surgery
40. A Case of Primary Ciliary Dyskinesia with Refractory Chronic Rhinosinusitis
41. The inhibitory effects of heparin on the upper airway inflammation
42. Group 2 Innate Lymphoid Cells Are Increased in Nasal Polyps in Patients with Eosinophilic Chronic Rhinosinusitis
43. Role of Endogenous Protease Inhibitor in Airway Epithelial Cells in the Pathogenesis of Eoshinophilic Chronic Rhinosinusitis
44. Thrombin and Activated Coagulation Factor X Stimulate the Release of Cytokines and Fibronectin from Nasal Polyp Fibroblasts via Protease-Activated Receptors
45. A Case of Hodgkin Lymphoma with Dyspnea Caused by Subglottic Lesion
46. A Case of Primary Ciliary Dyskinesia with Refractory Chronic Rhinosinusitis
47. Recovery Over Time and Prognostic Factors in Treated Patients with Post-Infectious Olfactory Dysfunction: A Retrospective Study
48. Airway uric acid is a sensor of inhaled protease allergens and initiates type 2 immune responses in respiratory mucosa1
49. HMGB1-TLR4 Signaling Contributes to the Secretion of Interleukin 6 and Interleukin 8 by Nasal Epithelial Cells
50. Epithelial Cell-Derived Cytokines Contribute to the Pathophysiology of Eosinophilic Chronic Rhinosinusitis
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