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1. An unbiased ranking of murine dietary models based on their proximity to human metabolic dysfunction-associated steatotic liver disease (MASLD)

2. Exploring the impact of the PNPLA3 I148M variant on primary human hepatic stellate cells using 3D extracellular matrix models

3. β2-spectrin (SPTBN1) as a therapeutic target for diet-induced liver disease and preventing cancer development.

4. Performance of non-invasive tests and histology for the prediction of clinical outcomes in patients with non-alcoholic fatty liver disease: an individual participant data meta-analysis

5. Transcriptional, epigenetic and metabolic signatures in cardiometabolic syndrome defined by extreme phenotypes

8. Increased serum miR-193a-5p during non-alcoholic fatty liver disease progression: Diagnostic and mechanistic relevance

10. Stem Cells Collection and Mobilization in Adult Autologous/Allogeneic Transplantation: Critical Points and Future Challenges

12. NASH limits anti-tumour surveillance in immunotherapy-treated HCC

13. Economic performance of an accredited laboratory for cell manipulation in a public health setting.

15. Bone morphogenetic protein 8B promotes the progression of non-alcoholic steatohepatitis

16. SMIM1 absence is associated with reduced energy expenditure and excess weight

21. Non-alcoholic fatty liver disease is characterised by a reduced polyunsaturated fatty acid transport via free fatty acids and high-density lipoproteins (HDL)

22. Corrigendum to: “Genome-wide association study of non-alcoholic fatty liver and steatohepatitis in a histologically characterised cohort☆” [J Hepatol (2020) 505–515]

23. Circulating endothelial cell (CEC) kinetic in patients with Multiple Myeloma (MM) who receive Au-HSCT (Autologous Hematopoietic Stem Cell Transplantation)

24. Correction: Genome-wide association study of non- alcoholic fatty liver and steatohepatitis in a histologically characterised cohort ( vol 73, pp 505-515 , 2020 )

25. Machine learning algorithm improves the detection of NASH (NAS-based) and at-risk NASH: A development and validation study

30. Hepatic steatosis risk is partly driven by increased de novo lipogenesis following carbohydrate consumption

31. The 'Hemolysis Model' for the Study of Cyto-Toxicity and Cyto-Protection by Bile Salts and Phospholipids

35. Non-alcoholic fatty liver disease features a reduced reverse polyunsaturated fatty acid transport (free fatty acids/high-density lipoprotein) from the periphery to the liver

36. Is there a murine model that fully recapitulates human NASH? An unbiased bioinformatics approach to rank pre-clinical models based on proximity to human disease

37. Transcriptomic analysis confirms that PNPLA3 I148M variant is associated with impaired mitochondrial function and antioxidant response in 3D cultured human hepatic stellate cells and liver tissue of patients with NAFLD

38. Lipidomic Approaches to Study HDL Metabolism in Patients with Central Obesity Diagnosed with Metabolic Syndrome

39. Macrophage Scavenger Receptor 1 mediates lipid-induced inflammation in non-alcoholic fatty liver disease

40. Circulating Endothelial Cell Kinetic in Patients with Multiple Myeloma Who Receive Autologous Hematopoietic Stem Cell Transplantation.

42. Global multi-stakeholder endorsement of the MAFLD definition

45. Global multi-stakeholder endorsement of the MAFLD definition

46. Increased serum miR-193a-5p during non-alcoholic fatty liver disease progression: Diagnostic and mechanistic relevance

47. Additional file 1 of Transcriptional, epigenetic and metabolic signatures in cardiometabolic syndrome defined by extreme phenotypes

48. Additional file 6 of Transcriptional, epigenetic and metabolic signatures in cardiometabolic syndrome defined by extreme phenotypes

49. Additional file 4 of Transcriptional, epigenetic and metabolic signatures in cardiometabolic syndrome defined by extreme phenotypes

50. Additional file 5 of Transcriptional, epigenetic and metabolic signatures in cardiometabolic syndrome defined by extreme phenotypes

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