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2. Twelve-hour normothermic liver perfusion in a rat model: characterization of the changes in the ex-situ bio-molecular phenotype and metabolism

4. Obesogenic High-Fat Diet and MYC Cooperate to Promote Lactate Accumulation and Tumor Microenvironment Remodeling in Prostate Cancer

5. Supplementary Figure S1 from Obesogenic High-Fat Diet and MYC Cooperate to Promote Lactate Accumulation and Tumor Microenvironment Remodeling in Prostate Cancer

6. Data from Obesogenic High-Fat Diet and MYC Cooperate to Promote Lactate Accumulation and Tumor Microenvironment Remodeling in Prostate Cancer

7. Supplementary Tables S1-S19 from Obesogenic High-Fat Diet and MYC Cooperate to Promote Lactate Accumulation and Tumor Microenvironment Remodeling in Prostate Cancer

8. Supplementary Information from Obesogenic High-Fat Diet and MYC Cooperate to Promote Lactate Accumulation and Tumor Microenvironment Remodeling in Prostate Cancer

9. Biochemical properties of chromatin domains define genome compartmentalization

10. Rare ATG7 genetic variants predispose patients to severe fatty liver disease

14. New entity of adult ultra-short coeliac disease: the first international cohort and case–control study

15. Identification of a Panel of miRNAs Associated with Resistance to Palbociclib and Endocrine Therapy

17. Parathyroid Tumor Microenvironment

22. 184 Complement score: A novel prognostic tool in malignant pleural mesothelioma?

32. Data from MFF Regulation of Mitochondrial Cell Death Is a Therapeutic Target in Cancer

33. Supplementary Data from MFF Regulation of Mitochondrial Cell Death Is a Therapeutic Target in Cancer

34. Figure S5 from MFF Regulation of Mitochondrial Cell Death Is a Therapeutic Target in Cancer

35. Supplementary Movie S2 from MFF Regulation of Mitochondrial Cell Death Is a Therapeutic Target in Cancer

36. Table S2 from MFF Regulation of Mitochondrial Cell Death Is a Therapeutic Target in Cancer

37. Supplementary Figure S1 from MFF Regulation of Mitochondrial Cell Death Is a Therapeutic Target in Cancer

40. Supplementary Figure 2 from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

41. Supplementary Figure 1 from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

42. Supplementary Figure 4 from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

44. Data from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

45. Supplementary Figure 3 from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

46. Supplementary Table 1 from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

47. Supplementary Figure 5 from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

48. Supplementary Figure 6 from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

49. Supplementary Table 2 from Chk2 Phosphorylation of Survivin-ΔEx3 Contributes to a DNA Damage–Sensing Checkpoint in Cancer

50. Clinical features of type 1 and 2 refractory celiac disease: Results from a large cohort over a decade

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