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8. CD4+ T cells cause renal and placental mitochondrial oxidative stress as mechanisms of hypertension in response to placental ischemia

11. Progesterone induced blocking factor improves blood pressure, mitochondrial dysfunction and reactive oxygen species in response to sFlt‐1 induced hypertension during pregnancy

13. Blockade of endogenous angiotensin II type I receptor agonistic autoantibody activity improves mitochondrial reactive oxygen species and hypertension in a rat model of preeclampsia

16. CONTINUED INVESTIGATION INTO 17-OHPC AS AN ADDITION TO THE MANAGEMENT OF PE; RESULTS FROM THE PRECLINICAL RUPP RAT MODEL OF PE

17. Interleukin-4 supplementation improves the pathophysiology of hypertension in response to placental ischemia in RUPP rats

19. Renal natural killer cell activation and mitochondrial oxidative stress; new mechanisms in AT1-AA mediated hypertensive pregnancy

20. CD4+ T cells cause renal and placental mitochondrial oxidative stress as mechanisms of hypertension in response to placental ischemia.

27. Inhibition of Angiotensin II Type 1 Receptor Agonistic Autoantibody Binding Improves Maternal Outcomes in Rats with Placental Ischemia

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