39 results on '"Veca, M"'
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2. Numerical test of the replica-symmetric Hamiltonian for correlations of the critical state of spin glasses in a field
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Fernández Pérez, Luis Antonio, González-Adalid Pemartín, Isidoro, Martín Mayor, Víctor, Parisi, G., Ricci-Tersenghi, F., Rizzo, T., Ruiz Lorenzo, J. J., Veca, M., Fernández Pérez, Luis Antonio, González-Adalid Pemartín, Isidoro, Martín Mayor, Víctor, Parisi, G., Ricci-Tersenghi, F., Rizzo, T., Ruiz Lorenzo, J. J., and Veca, M.
- Abstract
©2022 American Physical Society The authors wish to thank the Janus Collaboration for allowing us to analyze their data. We would like also to thank E. Marinari for interesting discussions. The analysis of the Janus configurations was performed at the ICCAEx Supercomputer Center in Badajoz, and we thank its staff for their assistance. This work was supported by the European Research Council under the European Unions Horizon 2020 Research and Innovation Programme (Grant No. 694925, G.P.), by Ministerio de Economía y Competitividad, Agencia Estatal de Investigación, and Fondo Europeo de Desarrollo Regional(FEDER)(Spain and European Union) through Grants No. PID2020-112936GB-I00 and No. PGC2018-094684-B-C21, and by Junta de Extremadura (Spain) through Grants No. GRU18079 and No. IB20079 (partially funded by FEDER). I.G.A.P. was supported by MCIU (Spain) through FPU Grant No. FPU18/02665., A growing body of evidence indicates that the sluggish low-temperature dynamics of glass formers (e.g., supercooled liquids, colloids, or spin glasses) is due to a growing correlation length. Which is the effective field theory that describes these correlations? The natural field theory was drastically simplified by Bray and Roberts in 1980. More than 40 years later, we confirm the tenets of Bray and Roberts’s theory by studying the Ising spin glass in an externally applied magnetic field, both in four spatial dimensions (data obtained from the Janus collaboration) and on the Bethe lattice., Unión Europea. Horizonte 2020, Ministerio de Economia, Industria y Competitividad (MINECO)/FEDER, Ministerio de Ciencia e Innovación (MICINN), Junta de Extremadura /FEDER, Depto. de Física Teórica, Fac. de Ciencias Físicas, TRUE, pub
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- 2022
3. Numerical test of the replica-symmetric Hamiltonian for correlations of the critical state of spin glasses in a field
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Fernandez, L. A., primary, Gonzalez-Adalid Pemartin, I., additional, Martin-Mayor, V., additional, Parisi, G., additional, Ricci-Tersenghi, F., additional, Rizzo, T., additional, Ruiz-Lorenzo, J. J., additional, and Veca, M., additional
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- 2022
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4. Numerical test of the replica-symmetric Hamiltonian for correlations of the critical state of spin glasses in a field
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Fernandez, L. A., Gonzalez-Adalid Pemartin, I., Martin-Mayor, V., Parisi, G., Ricci-Tersenghi, F., Rizzo, T., Ruiz-Lorenzo, J. J., and Veca, M.
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Condensed Matter::Soft Condensed Matter ,numerical simulations ,spin glasses ,critical phenomena ,FOS: Physical sciences ,Física ,Disordered Systems and Neural Networks (cond-mat.dis-nn) ,Condensed Matter - Disordered Systems and Neural Networks ,Condensed Matter::Disordered Systems and Neural Networks - Abstract
A growing body of evidence indicates that the sluggish low-temperature dynamics of glass formers (e.g. supercooled liquids, colloids or spin glasses) is due to a growing correlation length. Which is the effective field theory that describes these correlations? The natural field theory was drastically simplified by Bray and Roberts in 1980. More than forty years later, we confirm the tenets of Bray and Roberts theory by studying the Ising spin glass in an externally applied magnetic field, both in four spatial dimensions (data obtained from the Janus collaboration) and on the Bethe lattice., 8 pages, 4 figures, accepted in PRE
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- 2022
5. Poster presentation
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Duparc, F., Noyon, M., Ozeel, J., Gerometta, A., Michot, C., Tadjalli, M., Moslemy, H., Safaei, S., Heiman, A., Wish-Baratz, S., Melnikov, T., Smoliar, E., Hakan, A. Y., Yucel, F., Kachlík, D. K., Pešl, M. P., Báča, V. B., Stingl, J. S., Kachlík, K. D., Čech, Č. P., Báča, B. V., Mompeó, B., Marrero-Rodriguez, A., Zeybek, A., Sağlam, B., Çikler, E., Çetinel, Ş., Ercan, F., Şener, G., Kawawa, Y., Kohda, E., Tatsuya, T., Moroi, M., Kunimasa, T., Nagamoto, M., Terada, H., Labuschagne, B. C. J., van der Krieke, T. J., Hoogland, P. V., Muller, C. J. F., Lyners, R., Vorster, W., Matusz, P., Zaboi, D. E., Xu, S. C., Tu, L. L., Wang, Q., Zhang, M., Han, H., Tao, W., Jiao, Y., Pang, G., Aydin, M. E., Kopuz, C., Demir, M. T., Yildirim, M., Kale, A., Ince, Y., Khamanarong, K., Jeeravipoolvarn, P., Chaijaroonkhanarak, W., Gawgleun, W., Fujino, T., Uz, A., Apaydin, N., Bozkurt, M., Elhan, A., Sheibani, M. T., Adibmoradi, M., Jahovic, N., Alican, I., Erkanli, G., Arbak, S., Karakaş, S., Taşer, F., Güneş, H., Yildiz, Y., Yazici, Y., Aland, R. C., Kippers, V., Song, W. C., Park, S. H., Shin, C., Koh, K. S., Russo, G., Pomara, F., Veca, M., Cacciola, F., Martorana, U., Gravante, G., Tobenas-Dujardin, A. C., Laquerrière, A., Muller, J. M., Fréger, P., López-Serna, N., Álvarez-González, E., Torres-Gonzàlez, V., Laredo-López, G., Esparza-González, G. V., Álvarez-Cantú, R., Garza-González, C. E., Guzmán-López, S., Aldur, M. M., Çelik, H. H., Sürücü, S., Denk, C., Yang, H. J., Gil, Y. C., Kim, T. J., Lee, H. Y., Lee, W. J., Lee, H., Hu, K. S., Akita, K., Kim, H. J., Jung, H. S., Gurbuz, H., Balik, S., Wavreille, G., Chantelot, C., Demondion, X., Fontaine, C., Çavdar, S., Yalin, A., Saka, E., Özdoǧmuş, Ö., Çakmak, Ö., Elevli, L., Saǧlam, B., Coquerel-Beghin, D., Milliez, P. Y., Lemierre, G., Oktem, G., Vatansever, S., Ayla, S., Uysal, A., Aktas, S., Karabulut, B., Bilir, A., Uslu, S., Aktug, H., Yurtseven, M. E., Celik, H. H., Tatar, I., Surucu, S., Karaduman, A., Tunali, S., Neuhüttler, S., Kröll, A., Moriggl, B., Brenner, E., Loukas, M., Arora, S., Louis, Jr, R. G., Fogg, Q. A., Wagner, T., Tedman, R. A., Ching, H. Y., Eze, N., Bottrill, I. D., Blyth, P., Faull, R. L. M., Vuletic, J., Elizondo-Omaña, R. E., Rodríguez, M. A. García, López, S. Guzmán, de la Garza, O. Tijerina, Liu, Y. H., Zhang, K. L., Lu, D. H., Kwak, H. H., Park, H. D., Youn, K. H., Kang, H. J., Kang, H. C., Han, S. H., Ikiz, Z. A. Aktan, Ucerler, H., Uygur, M., Kutoglu, T., Dina, C., Iliescu, D., Şapte, E., Bordei, P., Lekšan, I., Marcikić, M., Radić, R., Nikolić, V., Kurbel, S., Selthofer, R., Báča, V., Doubková, A., Kachlík, D., Stingl, J., Džupa, V., Grill, R., Nam, Y. S., Paik, D. J., Shin, C. S., Kim, S. J., Kim, D. G., Jin, C. S., Kim, D. I., Lee, U. Y., Kwak, D. S., Lee, J. H., Han, C. H., Carpino, A., Rago, V., Romeo, F., Carani, C., Andò, S., Arican, R. Y., Coskun, N., Sarikcioglu, L., Sindel, M., Arican, Y. R., Altun, U., Ozsoy, U., Oguz, N., Yildirim, F. B., Nakajima, K., Duygulu, E., Aydin, H., Gurer, E. Inanc, Ozkan, O., Tuzuner, S., Özsoy, U., Çubukçu, S., Demirel, B. M., Akkin, S. M., Marur, T., Weiglein, A. H., Maghiar, T. T., Borza, C., Bumbu, A., Bumbu, G., Polle, G., Auquit-Auckbur, I., Dujardin, F., Biga, N., Olivier, E., Defives, T., Ghazali, S., Anastasi, G., Rizzo, G., Favaloro, A., Miliardi, D., Giacobbe, O., Santoro, G., Trimarchi, F., Cutroneo, G., Govsa, F., Bilge, O., Ozer, M. A., Erdogmus, S., Grizzi, F., Pelillo, F., Mori, M., Franceschini, B., Portinaro, N., Godlewski, G., Viala, M., Rouanet, J. P., Prat, D., Rahmé, Z. S., Prudhomme, M., Eken, E., Kwiatkowska, M., Liegmann, J., Chmielewski, R., Grimmond, J., Kwiatkowski, M., Schintler, M. V., Windisch, G., Wittgruber, G., Prandl, E. C., Prodinger, P., Anderhuber, F., Scharnagl, E., Gerbino, A., Buscemi, M., Leone, A., Mandracchia, R., Peri, G., Lipari, D., Farina-Lipari, E., Valentino, B., D’Arpa, S., Cordova, A., Bucchieri, F., Ribbene, A., David, S., Palma, A., Davies, D. E., Haitchi, H. M., Holgate, S. T., La Rocca, G., Anzalone, R., Campanella, C., Rappa, F., Bartolotta, T., Cappello, F., Bellafiore, M., Sivverini, G., Palumbo, D., Macaluso, F., Farina, F., Di Felice, V., Montalbano, A., Ardizzone, N., Marcianò, V., Zummo, G., Tanyeli, E., Üzel, M., Carini, F., Scardina, G. A., Varia, P., Valenza, V., Messina, P., Meiring, J. H., Schumann, C., Whitmore, I., Greyling, L. M., Hamel, O., Hamel, A., Robert, R., Garçon, M., Lagier, S., Blin, Y., Armstrong, O., Rogez, J. M., Le Borgne, J., Ifrim, C. Feng, Maghiar, A., Botea, M., Ifrim, M., Pop, O., Sandor, M., Behdadipour, Z., Saberi, M., Esfandiary, E., Gentile, C., Marconi, A., Livrea, M. A., Uzan, G., D’Alessio, P., Ridola, C. G., Grassi, N., Pantuso, G., Bottino, A., Cacace, E., Li Petri, S., Di Gaudio, F., Guercio, G., Latteri, M. A., Nobile, D., Cipolla, C., Caruso, G., Salvaggio, G., Lo Cascio, A., Fatta, G., Lagalla, R., Campisi, A., Verderame, F., Martegani, A., Cardinale, A. E., and Luedinghausen, M. V.
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- 2005
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6. Hemopoietic and angiogenetic progenitors in healthy athletes: different responses to endurance and maximal exercise
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Veca M, Ugo Testa, Eleonora Petrucci, Giuseppe Morici, Laura Chimenti, Paolo Palange, Gioia M, Maria R. Bonsignore, Alice Huertas, Anna Bonanno, Roberta Riccioni, Gualtiero Mariani, Bonsignore, MR, Morici, G, Riccioni, R, Huertas, A, Petrucci, E, Veca, M, Mariani, G, Bonanno, A, Chimenti, L, Gioia, M, Palange, P, and Testa, U
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Adult ,Male ,medicine.medical_specialty ,Physiology ,Neovascularization, Physiologic ,Antigens, CD34 ,Physical exercise ,Hematopoietic Cell Growth Factors ,Settore BIO/09 - Fisiologia ,Running ,angiopoietin ,marathon ,circulating progenitors ,growth factors ,Antigens, CD ,Endurance training ,Physiology (medical) ,Internal medicine ,medicine ,Humans ,AC133 Antigen ,Progenitor cell ,Glycoproteins ,Erythroid Precursor Cells ,biology ,Athletes ,business.industry ,Healthy subjects ,Endothelial Cells ,circulating progenitor ,Middle Aged ,Cadherins ,Hematopoietic Stem Cells ,biology.organism_classification ,Haematopoiesis ,Endocrinology ,Physical Endurance ,Cytokines ,Angiogenesis Inducing Agents ,adult ,angiogenesis inducing agents ,antigens ,athletes ,blood ,cadherins ,cd ,cd34 ,cytokines ,endothelial cells ,erythroid precursor cells ,glycoproteins ,granulocytes ,hematopoietic cell growth factors ,hematopoietic stem cells ,humans ,male ,middle aged ,neovascularization ,peptides ,physical endurance ,physiologic ,physiology ,running ,AC133 antigen ,Maximal exercise ,Peptides ,business ,Granulocytes - Abstract
J Appl Physiol. 2010 Jul;109(1):60-7. Epub 2010 May 6. Hemopoietic and angiogenetic progenitors in healthy athletes: different responses to endurance and maximal exercise. Bonsignore MR, Morici G, Riccioni R, Huertas A, Petrucci E, Veca M, Mariani G, Bonanno A, Chimenti L, Gioia M, Palange P, Testa U. SourceBiomedical Department, Internal and Specialistic Medicine (DIBIMIS), Section of Pneumology, University of Palermo, Via Trabucco, 180, 90146 Palermo, Italy. marisa@ibim.cnr.it Abstract The effects of endurance or maximal exercise on mobilization of bone marrow-derived hemopoietic and angiogenetic progenitors in healthy subjects are poorly defined. In 10 healthy amateur runners, we collected venous blood before, at the end of, and the day after a marathon race (n = 9), and before and at the end of a 1.5-km field test (n = 8), and measured hemopoietic and angiogenetic progenitors by flow cytometry and culture assays, as well as plasma or serum concentrations of several cytokines/growth factors. After the marathon, CD34(+) cells were unchanged, whereas clonogenetic assays showed decreased number of colonies for both erythropoietic (BFU-E) and granulocyte-monocyte (CFU-GM) series, returning to baseline the morning post-race. Conversely, CD34(+) cells, BFU-E, and CFU-GM increased after the field test. Angiogenetic progenitors, assessed as CD34(+)KDR(+) and CD133(+)VE-cadherin(+) cells or as adherent cells in culture expressing endothelial markers, increased after both endurance and maximal exercise but showed a different pattern between protocols. Interleukin-6 increased more after the marathon than after the field test, whereas hepatocyte growth factor and stem cell factor increased similarly in both protocols. Plasma levels of angiopoietin (Ang) 1 and 2 increased after both types of exercise, whereas the Ang-1-to-Ang-2 ratio or vascular endothelial growth factor-A were little affected. These data suggest that circulating hemopoietic progenitors may be utilized in peripheral tissues during prolonged endurance exercise. Endothelial progenitor mobilization after exercise in healthy trained subjects appears modulated by the type of exercise. Exercise-induced increase in growth factors suggests a physiological trophic effect of exercise on the bone marrow. PMID:20448032[PubMed - indexed for MEDLINE]
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- 2010
7. Processing and mechanical characterization of ultrananocrisytalline diamond films for MEMS applications
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Pachiu, C., primary, Tibeica, C., additional, Avram, A., additional, Veca, M., additional, Sandu, T., additional, Popescu, M., additional, and Popa, R., additional
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- 2017
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8. Adaptations to long-term apnoea training
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Veca, M, Muratore, F, MORICI, Giuseppe, LICCIARDI, Attilio, SCICHILONE, Nicola Alessandro, GRAVANTE, Gennaro, Veca, M, Morici, G, Licciardi, A, Scichilone, NA, Muratore, F, and Gravante, G
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hematocrit ,intermittent hypoxia ,lung volume ,fat free ma ,haemoglobin ,Settore BIO/09 - Fisiologia - Published
- 2010
9. Family history of type 2 diabetes and altered maximal aerobic capacity in elite young athletes: a cross-sectional study
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POMARA, Francesco, RUSSO G, VECA M, GRAVANTE, Gennaro, POMARA F, RUSSO G, VECA M, and GRAVANTE G
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family history ,exercise ,diabetes mellitus, type 2, genetic ,maximum aerobic power ,Settore BIO/09 - Fisiologia - Abstract
Aim. Family history of type 2 diabetes is a predictive risk factor of illness in sedentary subjects and induces early phenotypic and metabolic alterations. The aim of this study was to determine whether a family history of diabetes in young male athletes had an effect on metabolic functional parameters. Methods. The study population was 42 young male athletes with or without a family history of type 2 diabetes. Anthropometric parameters, body composition, and maximal aerobic capacity (V̇O2max) were assessed. Results. V̇O2max was lower in the 13 athletes with a family history of diabetes (FH+) than in the 29 athletes without a family history of the disorder (FH-) matched for kilogram of body weight and fat-free mass (P
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- 2008
10. Effects of exercise training and montelukast in children with mild asthma
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Maria R. Bonsignore, Anna Bonanno, Marco Virzi, Amelia Interrante, Mirella Profita, Veca M, Margherita Marchese, Giuseppe Morici, Stefania La Grutta, Giuseppina Cuttitta, Fabio Cibella, Nicola Scichilone, BONSIGNORE, MR, LA GRUTTA, S, CIBELLA, F, SCICHILONE, N, CUTTITTA, G, INTERRANTE, A, MARCHESE M, VECA, M, VIRZI, M, BONANNO, A, PROFITA, M, and MORICI, G
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Cyclopropanes ,Male ,Quinoline ,Acetates ,Settore BIO/09 - Fisiologia ,immune system diseases ,Medicine ,Orthopedics and Sports Medicine ,Anti-Asthmatic Agents ,Child ,Methacholine Chloride ,Leukotriene ,education.field_of_study ,respiratory system ,Exercise Therapy ,Asthma, Exercise-Induced ,Breath Tests ,Italy ,Exhalation ,Anesthesia ,Quinolines ,Female ,medicine.drug ,Human ,medicine.medical_specialty ,Breath Test ,Bronchoconstriction ,Population ,Physical Therapy, Sports Therapy and Rehabilitation ,Physical exercise ,Sulfides ,Settore MED/10 - Malattie Dell'Apparato Respiratorio ,Internal medicine ,Aerobic exercise ,Humans ,Anti-Asthmatic Agent ,education ,Montelukast ,Asthma ,business.industry ,Leukotriene receptor ,Acetate ,Bronchospirometry ,medicine.disease ,respiratory tract diseases ,Physical Fitness ,Physical Fitne ,Exercise Test ,Methacholine ,business - Abstract
Data from the general population suggest that habitual exercise decreases bronchial responsiveness, but the possible role of exercise in asthmatics is undefined. The leukotriene receptor antagonist montelukast decreases bronchial responsiveness and exercise-induced symptoms in asthmatic children. This randomized study in children with mild asthma evaluated the combined effects of aerobic training for 12 wk and montelukast or placebo on bronchial responsiveness (BHR) to methacholine, exercise-induced bronchoconstriction (EIB), inflammatory markers in exhaled breath condensate (EBC), and asthma exacerbations.Fifty children (mean age +/- SD: 10.2 +/- 2.4 yr) with mild stable asthma were randomly assigned to placebo (N = 25) or montelukast (N = 25). Before and after training, we assessed BHR and EIB and markers of airway inflammation-that is, exhaled nitric oxide (eNO), pH, and cysteinyl-leukotriene concentration-in EBC.Training increased maximal workload and peak minute ventilation. After training, the methacholine dose causing a 20% fall in FEV1 (PD20) increased in both groups. A decreased slope of FEV1 decline at increasing methacholine dose was found only in montelukast-treated children. EIB prevalence halved after training in both groups (EIB + children, placebo group: 10 pretraining, 4 posttraining; EIB + children, montelukast group: 8 pretraining, 5 posttraining; P0.05 by chi on all children). Resting eNO was unaffected, whereas the pH of EBC decreased after training in both groups. Cysteinyl-leukotriene concentrations were low in most children at both times. During training, montelukast-treated children showed fewer asthma exacerbations compared with the same period of the previous year.In children with mild stable asthma, exercise training decreased bronchial responsiveness to methacholine. Montelukast also decreased bronchial reactivity (FEV1 slope) and protected against exacerbations, suggesting a beneficial synergistic action of these two interventions in mild asthma.
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- 2008
11. VALUTAZIONE DELLA DENSITÀ OSSEA DI UN CAMPIONE DI SOGGETTI PARAPLEGICI. RUOLO DEL DOTTORE IN SCIENZE MOTORIE NEL RECUPERO DELL’AUTONOMIA MOTORIA
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Russo, Giuseppe, Veca, M., Gravante, G., Muratore, F., Viviano, V., Angelo CATALDO, Marcello Traina, RUSSO, G, VECA, M, GRAVANTE, G, MURATORE, F, VIVIANO, V, CATALDO, A, and TRAINA, M
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Settore BIO/09 - Fisiologia ,Settore M-EDF/01 - Metodi E Didattiche Delle Attivita' Motorie ,osso, paraplegia,densità ossea - Abstract
Scopo del lavoro è stato quello di valutare in pazienti maschi paraplegici gli adattamenti del tessuto osseo del rachide in toto e dei vari segmenti scheletrici in conseguenza della prolungata immobilità e di proporre dei nuovi protocolli d’attività motoria che possano sia limitare il decadimento progressivo delle strutture osteo-articolari, sia di preparare i pazienti all’ utilizzo di nuove tecnologie che consentano una pur parziale autonomia motoria. Allo studio hanno partecipato volontariamente venticinque soggetti paraplegici di sesso maschile con lesioni complete T3- L3, suddivisi in due gruppi in relazione agli anni di lesione: il primo gruppo è composto da 13 soggetti con lesione midollare verificatasi da meno di dieci anni; il secondo gruppo comprende 12 soggetti con lesione midollare presente da oltre dieci anni. Per la valutazione degli adattamenti del tessuto osseo del rachide e dei vari segmenti scheletrici è stata misurata la densità ossea dei soggetti con la tecnica DEXA. I valori del BMD e del T-score del total body e del tratto lombare L1-L4 dei due gruppi sono nella norma, a differenza di quelli degli arti inferiori che evidenziano uno stato di osteoporosi; questo dato dimostra che l’effetto demineralizzante dell’immobilità coinvolge in particolare le strutture non più sottoposte a stress meccanici. I nostri dati indicano che non vi è relazione tra il numero di anni dalla lesione e la perdita di massa ossea; infatti, anche nel gruppo di soggetti che presentano lesioni midollari da meno di dieci anni si ha una importante perdita di massa ossea. Il dato di normalità dei valori di BMD e T-score, riscontrati a livello del tratto lombare L1-L4 è giustificato a parer nostro da un elemento biomeccanico locale; infatti, la posizione seduta in carrozzina determina una sollecitazione meccanica continua sul rachide lombare ed in particolare sulle prime due vertebre favorendo il mantenimento della massa ossea in questo tratto. Il trattamento motorio dei soggetti mielolesi si articola in una serie di interventi, esercizi e attività finalizzate al superamento delle limitazioni imposte dalla patologia, al recupero funzionale e all'inserimento sociale del paziente.
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- 2008
12. Valutazione funzionale del fitness cardio-polmonare e metabolica di pazienti paraplegici attivi e non attivi:confronto con giovani sedentari sani
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VECA M, FEDELI L, FALDETTA E, MURATORE F, RUBINO C, CHINNICI M., RUSSO, Giuseppe, POMARA, Francesco, GRAVANTE, Gennaro, VIVIANO, Vittorio, VECA M, RUSSO G, FEDELI L, POMARA F, GRAVANTE G, VIVIANO V, FALDETTA E, MURATORE F, RUBINO C, and CHINNICI M
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- 2007
13. Circulating Progenitors in Healthy Runners after Endurance and Maximal Exercise
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BONSIGNORE, Maria Rosaria, MORICI, Giuseppe, BELLIA, Vincenzo, HUERTAS A, RICCIONI R, MARIANI G, BONANNO A, DI BELLA M, VECA M, PALANGE P, TESTA U., BONSIGNORE MR, MORICI G, HUERTAS A, RICCIONI R, MARIANI G, BONANNO A, DI BELLA M, VECA M, PALANGE P, BELLIA V, and TESTA U
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- 2007
14. Endurance training damages small airway epithelium in mice
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Alessandra Paternò, Attilio Licciardi, Anna Bonanno, Giovanni Bonsignore, Walter Guccione, Filippo Macaluso, Liboria Siena, Maria R. Bonsignore, Veca M, Laura Chimenti, Giuseppe Morici, CHIMENTI, L, MORICI, G, PATERNO', A, BONANNO, A, SIENA, L, LICCIARDI, A, VECA, M, GUCCIONE, W, MACALUSO, F, BONSIGNORE, G, and BONSIGNORE, MR
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Pulmonary and Respiratory Medicine ,Male ,Pathology ,medicine.medical_specialty ,Inflammation ,Apoptosis ,Critical Care and Intensive Care Medicine ,Settore BIO/09 - Fisiologia ,Epithelium ,Epithelial Damage ,Leukocyte Count ,Mice ,Endurance training ,Intensive care ,Physical Conditioning, Animal ,Proliferating Cell Nuclear Antigen ,medicine ,Leukocytes ,Animals ,Bronchitis ,Cell Proliferation ,Basement membrane ,Lung ,Aerobic exercise, bronchial responsivenes, methacholine, deep inspiration, leukotrienes ,business.industry ,NF-kappa B ,respiratory system ,Immunohistochemistry ,respiratory tract diseases ,Disease Models, Animal ,medicine.anatomical_structure ,Respiratory epithelium ,medicine.symptom ,business - Abstract
RATIONALE: In athletes, airway inflammatory cells were found to be increased in induced sputum or bronchial biopsies. Most data were obtained after exposure to cold and dry air at rest or during exercise. Whether training affects epithelial and inflammatory cells in small airways is unknown. OBJECTIVES: To test whether endurance training under standard environmental conditions causes epithelial damage and inflammation in the small airways of mice. METHODS AND MEASUREMENTS: Formalin-fixed, paraffin-embedded lung sections were obtained in sedentary (n = 14) and endurance-trained (n = 16) Swiss mice at baseline and after 15, 30, and 45 days of training. The following variables were assessed (morphometry and immunohistochemistry) in small airways (basement membrane length < 1 mm): (1) integrity, proliferation, and apoptosis of bronchiolar epithelium; and (2) infiltration, activation, and apoptosis of inflammatory cells. MAIN RESULTS: Compared with sedentary mice, bronchiolar epithelium of trained mice showed progressive loss of ciliated cells, slightly increased thickness, unchanged goblet cell number and appearance, and increased apoptosis and proliferation (proliferating cell nuclear antigen) (p < 0.001 for all variables). Leukocytes (CD45(+) cells) infiltrated airway walls (p < 0.0001) and accumulated within the lumen (p < 0.001); however, apoptosis of CD45(+) cells did not differ between trained and sedentary mice. Nuclear factor-kappaB translocation and inhibitor-alpha of NF-kappaB (IkappaBalpha) phosphorylation were not increased in trained compared with sedentary mice. CONCLUSIONS: Bronchiolar epithelium showed damage and repair associated with endurance training. Training increased inflammatory cells in small airways, but inflammatory activation was not increased. These changes may represent an adaptive response to increased ventilation during exercise.
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- 2006
15. Supramaximal exercise mobilizes hematopoietic progenitors
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Santoro, A., Morici, G., Zangla, D., Pelosi, E., Gioia, M., Marfia, A., Bonanno, A., Veca, M., Huertas, A., Bellia, V., Ugo Testa, Bonsignore, MR, SANTORO A, MORICI G, ZANGLA D, PELOSI E, GIOIA M, MARFIA A, BONANNO A, VECA M, HUERTAS A, BELLIA V, TESTA U, and BONSIGNORE MR
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- 2006
16. Increased numbers of inflammatory cells but decreased activation of NFkappaB pathway in small airways of endurance-trained mice
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CHIMENTI, Laura, SIENA, Liboria, LICCIARDI, Attilio, BELLIA, Vincenzo, BONSIGNORE, Maria Rosaria, MORICI, Giuseppe, PATERN A, BONANNO A, VECA M, DI BELLA M, GAGLIARDO R, CHIMENTI L, MORICI G, PATERN A, SIENA L, BONANNO A, LICCIARDI A, VECA M, DI BELLA M, GAGLIARDO R, BELLIA V, and BONSIGNORE MR
- Published
- 2006
17. Stile di vita attivo e densità ossea in donne in eta’ postmenopausale
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RUSSO, Giuseppe, BENIGNO, Arcangelo, GRAVANTE, Gennaro, FEDELI L, MURATORE F, CACCIOLA F, VECA M, RUSSO G, FEDELI L, MURATORE F, CACCIOLA F, VECA M, BENIGNO A, and GRAVANTE G
- Published
- 2006
18. Association between gene polymorphims of actn3 and srd5a and athletic performance. (2006)
- Author
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MUSSO M, BARRESI V, VECA M, CLEMENTI G, VASTA D, CONDORELLI DF, RUSSO, Giuseppe, POMARA, Francesco, GRAVANTE, Gennaro, MUSSO M, BARRESI V, VECA M, RUSSO G, POMARA F, CLEMENTI G, VASTA D, CONDORELLI DF, and GRAVANTE G
- Published
- 2006
19. Retrospective survey between active life style and bone density
- Author
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MURATORE F, FEDELI L, CACCIOLA F, VECA M, FALDETTA E, CHINNICI M., RUSSO, Giuseppe, GRAVANTE, Gennaro, VIVIANO, Vittorio, MURATORE F, RUSSO G, FEDELI L, CACCIOLA F, VECA M, GRAVANTE G, FALDETTA E, VIVIANO V, and CHINNICI M
- Published
- 2006
20. Effects of endurance training on small airways of mice
- Author
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CHIMENTI, Laura, BONSIGNORE, Maria Rosaria, PATERNO', Alessandra, MACALUSO, Filippo, LICCIARDI, Attilio, DI FELICE, Valentina, FARINA, Felicia, MORICI, Giuseppe, DE FELICE V, BONANNO A, VECA M, GUCCIONE W, SIENA A, CHIMENTI L, BONSIGNORE MR, PATERNO' A, MACALUSO F, DE FELICE V, BONANNO A, LICCIARDI A, VECA M, GUCCIONE W, DI BELLA M, SIENA A, FARINA F, and MORICI G
- Subjects
training, small airways, inflammatory cells ,Settore BIO/16 - Anatomia Umana ,Settore MED/10 - Malattie Dell'Apparato Respiratorio ,Settore BIO/09 - Fisiologia - Published
- 2006
21. Effetti dell'allenamento aerobico di 12 settimane in bambini con asma lieve intermittente
- Author
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BONSIGNORE, Maria Rosaria, MESSINEO, Barbara, MORICI, Giuseppe, BELLIA, Vincenzo, CIBELLA F, CUTTITTA G, VECA M, PROFITA M, BONANNO A, BONSIGNORE G., BONSIGNORE MR, CIBELLA F, CUTTITTA G, VECA M, MESSINEO B, PROFITA M, BONANNO A, MORICI G, BELLIA V, and BONSIGNORE G
- Published
- 2006
22. Effects of aerobic training in children with mild intermittent asthma
- Author
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VECA M, CIBELLA F, CUTTITTA G, VIRZI' M, MARCHESE M, FRANCAVILLA C, BONANNO A, PROFITA M, PARDO F, MORICI, Giuseppe, INTERRANTE, Amelia, GUCCIONE, Walter, BELLIA, Vincenzo, BONSIGNORE, Maria Rosaria, VECA M, CIBELLA F, MORICI G, CUTTITTA G, VIRZI' M, MARCHESE M, INTERRANTE A, FRANCAVILLA C, GUCCIONE W, BONANNO A, PROFITA M, PARDO F, BELLIA V, and BONSIGNORE MR
- Published
- 2006
23. Analisi dell'appoggio podalico in donne obese e normopeso durante il ciclo del passo
- Author
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RUSSO, Giuseppe, BENIGNO, Arcangelo, GRAVANTE, Gennaro, CACCIOLA F, FEDELI L, VECA M, RUSSO G, CACCIOLA F, FEDELI L, VECA M, BENIGNO A, and GRAVANTE G
- Published
- 2006
24. Beneficial effects of aerobic training+montelukast in children with mild intermittent asthma
- Author
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CIBELLA F, CUTTITTA G, VIRZI' M, MARCHESE M, VECA M, FRANCAVILLA C, BONANNO A, PROFITA M, PARDO F, BELLIA V, MORICI, Giuseppe, INTERRANTE, Amelia, GUCCIONE, Walter, BONSIGNORE, Maria Rosaria, CIBELLA F, MORICI G, CUTTITTA G, VIRZI' M, MARCHESE M, INTERRANTE A, VECA M, FRANCAVILLA C, GUCCIONE W, BONANNO A, PROFITA M, PARDO F, BELLIA V, and BONSIGNORE MR
- Published
- 2005
25. L’osteoporosi nelle lesioni del midollo spinale: analisi trasversale su paraplegici
- Author
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RUSSO, Giuseppe, MURATORE F, VECA M, POMARA, Francesco, CACCIOLA F, CHINNICI M, GRAVANTE, Gennaro, RUSSO G, MURATORE F, VECA M, POMARA F, CACCIOLA F, CHINNICI M, and GRAVANTE G
- Published
- 2005
26. Endurance raining damages small airway epithelium in mice
- Author
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CHIMENTI, Laura, LICCIARDI, Attilio, GUCCIONE, Walter, FARINA, Felicia, BONSIGNORE, Maria Rosaria, PATERNO' A, DI FELICE, Valentina, BONANNO A, VECA M, DI BELLA M, SIENA A, MORICI, Giuseppe, CHIMENTI L, PATERNO' A, DI FELICE V, BONANNO A, LICCIARDI A, VECA M, GUCCIONE W, DI BELLA M, SIENA A, FARINA F, MORICI G, and BONSIGNORE MR
- Published
- 2005
27. Endurance training damages small airway epithelium in mice
- Author
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CHIMENTI, Laura, DI FELICE, Valentina, BONANNO A, LICCIARDI, Attilio, VECA M, GUCCIONE, Walter, DI BELLA M, SIENA A, FARINA, Felicia, MORICI, Giuseppe, BONSIGNORE, Maria Rosaria, PATERNO', Alessandra, CHIMENTI L, PATERNO' A, DI FELICE V, BONANNO A, LICCIARDI A, VECA M, GUCCIONE W, DI BELLA M, SIENA A, FARINA F, MORICI G, and BONSIGNORE MR
- Subjects
Settore BIO/16 - Anatomia Umana ,Settore MED/10 - Malattie Dell'Apparato Respiratorio ,Training, bronchial epithelium, inflammatory cells ,Settore BIO/09 - Fisiologia - Published
- 2005
28. ASSOCIATION BETWEEN GENE POLYMORPHISMS OF ACTN3 AND SRD5A AND ATHLETIC PERFORMANCE
- Author
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Musso, N., Barresi, V., Veca, M, Russo, G, Pomara, F, Clementi, G, Vasta, D, Condorelli, Daniele Filippo, and Gravante, G.
- Published
- 2006
29. Modelling and analysis of electric and magnetic coupled problems under non-linear conditions
- Author
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Geri, Alberto and Veca, M. LA ROSA AND G. M.
- Published
- 1994
30. Endurance training damages small airway epithelium in mice.
- Author
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Chimenti L, Morici G, Paternò A, Bonanno A, Siena L, Licciardi A, Veca M, Guccione W, Macaluso F, Bonsignore G, and Bonsignore MR
- Abstract
Rationale: In athletes, airway inflammatory cells were found to be increased in induced sputum or bronchial biopsies. Most data were obtained after exposure to cold and dry air at rest or during exercise. Whether training affects epithelial and inflammatory cells in small airways is unknown. Objectives: To test whether endurance training under standard environmental conditions causes epithelial damage and inflammation in the small airways of mice. Methods and Measurements: Formalin-fixed, paraffin-embedded lung sections were obtained in sedentary (n = 14) and endurance-trained (n = 16) Swiss mice at baseline and after 15, 30, and 45 days of training. The following variables were assessed (morphometry and immunohistochemistry) in small airways (basement membrane length < 1 mm): (1) integrity, proliferation, and apoptosis of bronchiolar epithelium; and (2) infiltration, activation, and apoptosis of inflammatory cells. Main Results: Compared with sedentary mice, bronchiolar epithelium of trained mice showed progressive loss of ciliated cells, slightly increased thickness, unchanged goblet cell number and appearance, and increased apoptosis and proliferation (proliferating cell nuclear antigen) (p < 0.001 for all variables). Leukocytes (CD45(+) cells) infiltrated airway walls (p < 0.0001) and accumulated within the lumen (p < 0.001); however, apoptosis of CD45(+) cells did not differ between trained and sedentary mice. Nuclear factor-kappaB translocation and inhibitor-alpha of NF-kappaB (IkappaBalpha) phosphorylation were not increased in trained compared with sedentary mice. Conclusions: Bronchiolar epithelium showed damage and repair associated with endurance training. Training increased inflammatory cells in small airways, but inflammatory activation was not increased. These changes may represent an adaptive response to increased ventilation during exercise. [ABSTRACT FROM AUTHOR]
- Published
- 2007
- Full Text
- View/download PDF
31. Dielectric properties of composites containing silicone rubber and multiwall carbon nanotubes decorated with gold
- Author
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Pantazi, A., Palade, S., Berbecaru, C., Veca, M., Adrian Dinescu, Schiopu, V., Oprea, O., and Dragoman, D.
32. Scaling the graphene-silicon heterojunctions: fabrication and characterization
- Author
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Istrate, A. -I, Veca, M., Nastase, F., Baracu, A., Gavrila, R., Comanescu, F., Tucureanu, V., Dinescu, A., and Titus Sandu
33. Giano bifronte. Teoria e forme dell'aforisma italiano contemporaneo
- Author
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RUOZZI, GINO, U. ECO G. RUOZZI R. TOSI G. CALBOLI E. PASQUINI. M. T. BIASON G. CANTARUTTI. K. ELAM S. VECA. M. A. RIGONI C. VIVIANI V. RODA, G. RUOZZI, and G. Ruozzi
- Subjects
FORME BREVI ,AFORISMA ,LETTERATURA ITALIANA ,D'ANNUNZIO ,LONGANESI - Abstract
L'aforisma italiano ha conosciuto importanti modificazioni a partire dal Decadentismo, quando all'idea di pensiero scaturito da una lunga riflessione subentra quella di improvvisa illuminazione.
- Published
- 2004
34. Introduzione
- Author
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RUOZZI, GINO, U. ECO G. RUOZZI R. TOSI G. CALBOLI E. PASQUINI. M. T. BIASON G. CANTARUTTI. K. ELAM S. VECA. M. A. RIGONI C. VIVIANI V. RODA, G. RUOZZI, and G. Ruozzi
- Subjects
TEORIA DELLA LETTERATURA ,FORME BREVI ,STORIA DELLA LETTERATURA ,AFORISMA ,LETETRATURE EUROPEE - Abstract
Presentazione di elementi storici e teorici legati alla tradizione letteraria e filosofica dell'aforisma
- Published
- 2004
35. From noise on the sites to noise on the links: Discretizing the conserved Kardar-Parisi-Zhang equation in real space.
- Author
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Cavagna A, Cristín J, Giardina I, and Veca M
- Abstract
Numerical analysis of conserved field dynamics has been generally performed with pseudospectral methods. Finite differences integration, the common procedure for nonconserved field dynamics, indeed struggles to implement a conservative noise in the discrete spatial domain. In this work we present a method to generate a conservative noise in the finite differences framework, which works for any discrete topology and boundary conditions. We apply it to numerically solve the conserved Kardar-Parisi-Zhang (cKPZ) equation, widely used to describe surface roughening when the number of particles is conserved. Our numerical simulations recover the correct scaling exponents α, β, and z in d=1 and in d=2. To illustrate the potentiality of the method, we further consider the cKPZ equation on different kinds of nonstandard lattices and on the random Euclidean graph. This is a unique numerical study of conserved field dynamics on an irregular topology, paving the way for a broad spectrum of possible applications.
- Published
- 2024
- Full Text
- View/download PDF
36. Erratum: Numerical test of the replica-symmetric Hamiltonian for correlations of the critical state of spin glasses in a field [Phys. Rev. E 105, 054106 (2022)].
- Author
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Fernandez LA, Pemartin IG, Martin-Mayor V, Parisi G, Ricci-Tersenghi F, Rizzo T, Ruiz-Lorenzo JJ, and Veca M
- Abstract
This corrects the article DOI: 10.1103/PhysRevE.105.054106.
- Published
- 2023
- Full Text
- View/download PDF
37. Hemopoietic and angiogenetic progenitors in healthy athletes: different responses to endurance and maximal exercise.
- Author
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Bonsignore MR, Morici G, Riccioni R, Huertas A, Petrucci E, Veca M, Mariani G, Bonanno A, Chimenti L, Gioia M, Palange P, and Testa U
- Subjects
- AC133 Antigen, Adult, Angiogenesis Inducing Agents blood, Antigens, CD blood, Antigens, CD34 blood, Cadherins blood, Cytokines blood, Glycoproteins blood, Granulocytes physiology, Hematopoietic Cell Growth Factors blood, Humans, Male, Middle Aged, Peptides blood, Running physiology, Athletes, Endothelial Cells physiology, Erythroid Precursor Cells physiology, Hematopoietic Stem Cells physiology, Neovascularization, Physiologic, Physical Endurance physiology
- Abstract
The effects of endurance or maximal exercise on mobilization of bone marrow-derived hemopoietic and angiogenetic progenitors in healthy subjects are poorly defined. In 10 healthy amateur runners, we collected venous blood before, at the end of, and the day after a marathon race (n = 9), and before and at the end of a 1.5-km field test (n = 8), and measured hemopoietic and angiogenetic progenitors by flow cytometry and culture assays, as well as plasma or serum concentrations of several cytokines/growth factors. After the marathon, CD34(+) cells were unchanged, whereas clonogenetic assays showed decreased number of colonies for both erythropoietic (BFU-E) and granulocyte-monocyte (CFU-GM) series, returning to baseline the morning post-race. Conversely, CD34(+) cells, BFU-E, and CFU-GM increased after the field test. Angiogenetic progenitors, assessed as CD34(+)KDR(+) and CD133(+)VE-cadherin(+) cells or as adherent cells in culture expressing endothelial markers, increased after both endurance and maximal exercise but showed a different pattern between protocols. Interleukin-6 increased more after the marathon than after the field test, whereas hepatocyte growth factor and stem cell factor increased similarly in both protocols. Plasma levels of angiopoietin (Ang) 1 and 2 increased after both types of exercise, whereas the Ang-1-to-Ang-2 ratio or vascular endothelial growth factor-A were little affected. These data suggest that circulating hemopoietic progenitors may be utilized in peripheral tissues during prolonged endurance exercise. Endothelial progenitor mobilization after exercise in healthy trained subjects appears modulated by the type of exercise. Exercise-induced increase in growth factors suggests a physiological trophic effect of exercise on the bone marrow.
- Published
- 2010
- Full Text
- View/download PDF
38. Effects of exercise training and montelukast in children with mild asthma.
- Author
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Bonsignore MR, La Grutta S, Cibella F, Scichilone N, Cuttitta G, Interrante A, Marchese M, Veca M, Virzi' M, Bonanno A, Profita M, and Morici G
- Subjects
- Acetates administration & dosage, Acetates pharmacology, Anti-Asthmatic Agents administration & dosage, Anti-Asthmatic Agents pharmacology, Asthma, Exercise-Induced physiopathology, Breath Tests, Bronchoconstriction drug effects, Bronchospirometry, Child, Cyclopropanes, Exercise Test, Exhalation drug effects, Exhalation physiology, Female, Humans, Italy, Male, Methacholine Chloride administration & dosage, Physical Fitness physiology, Quinolines administration & dosage, Quinolines pharmacology, Sulfides, Acetates therapeutic use, Anti-Asthmatic Agents therapeutic use, Asthma drug therapy, Asthma physiopathology, Asthma, Exercise-Induced drug therapy, Exercise Therapy, Quinolines therapeutic use
- Abstract
Purpose: Data from the general population suggest that habitual exercise decreases bronchial responsiveness, but the possible role of exercise in asthmatics is undefined. The leukotriene receptor antagonist montelukast decreases bronchial responsiveness and exercise-induced symptoms in asthmatic children. This randomized study in children with mild asthma evaluated the combined effects of aerobic training for 12 wk and montelukast or placebo on bronchial responsiveness (BHR) to methacholine, exercise-induced bronchoconstriction (EIB), inflammatory markers in exhaled breath condensate (EBC), and asthma exacerbations., Methods: Fifty children (mean age +/- SD: 10.2 +/- 2.4 yr) with mild stable asthma were randomly assigned to placebo (N = 25) or montelukast (N = 25). Before and after training, we assessed BHR and EIB and markers of airway inflammation-that is, exhaled nitric oxide (eNO), pH, and cysteinyl-leukotriene concentration-in EBC., Results: Training increased maximal workload and peak minute ventilation. After training, the methacholine dose causing a 20% fall in FEV1 (PD20) increased in both groups. A decreased slope of FEV1 decline at increasing methacholine dose was found only in montelukast-treated children. EIB prevalence halved after training in both groups (EIB + children, placebo group: 10 pretraining, 4 posttraining; EIB + children, montelukast group: 8 pretraining, 5 posttraining; P < 0.05 by chi on all children). Resting eNO was unaffected, whereas the pH of EBC decreased after training in both groups. Cysteinyl-leukotriene concentrations were low in most children at both times. During training, montelukast-treated children showed fewer asthma exacerbations compared with the same period of the previous year., Conclusions: In children with mild stable asthma, exercise training decreased bronchial responsiveness to methacholine. Montelukast also decreased bronchial reactivity (FEV1 slope) and protected against exacerbations, suggesting a beneficial synergistic action of these two interventions in mild asthma.
- Published
- 2008
- Full Text
- View/download PDF
39. Effect of pH and ATP on the equilibrium density of lysosomes.
- Author
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Mayorga LS, De Veca MG, Colombo MI, and Bertini F
- Subjects
- Adenosine Triphosphate physiology, Animals, Deoxyglucose pharmacology, Endocytosis physiology, Ethylmaleimide pharmacology, Hydrogen-Ion Concentration, Hypoxia pathology, Hypoxia physiopathology, Iodoacetates pharmacology, Iodoacetic Acid, Liver cytology, Liver ultrastructure, Lysosomes ultrastructure, Membrane Proteins physiology, Mice, Microscopy, Electron, Subcellular Fractions, Time Factors, Trypsin pharmacology, Adenosine Triphosphate pharmacology, Lysosomes physiology
- Abstract
Lysosomes are membrane bound structures that accumulate and hydrolyze material internalized by the endocytic pathway. A very conspicuous property of this subcellular compartment is its relatively high equilibrium density. The actual mechanism that regulates lysosomal density is poorly understood. In an attempt to gain knowledge on the factors that regulate lysosomal density we have assessed the equilibrium density of lysosomal markers after in vitro incubation of a lysosome-enriched subcellular fraction. Incubation at pH 6 for 10 min at 37 degrees C causes a density shift of several lysosomal markers to light density regions of Percoll gradients. Addition of ATP was able to prevent the acid-induced density shift. Pretreatment of the vesicles with N-ethylmaleimide (NEM) or trypsin inhibited the effect of ATP. Working in intact cells, ATP depletion, a condition that causes cytoplasmic acidification, also decreases lysosomal density. The results indicate that at low pH lysosomal density is preserved by an active process that requires ATP and membrane associated proteins.
- Published
- 1993
- Full Text
- View/download PDF
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