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1. A chemical probe to modulate human GID4 Pro/N-degron interactions

3. Structure-Based Discovery of Inhibitors of the SARS-CoV‑2 Nsp14 N7-Methyltransferase

6. CACHE (Critical Assessment of Computational Hit-finding Experiments): A public–private partnership benchmarking initiative to enable the development of computational methods for hit-finding

11. A chemical probe targeting the PWWP domain alters NSD2 nucleolar localization

13. Discovery of a Potent, Selective, and Cell-Active SPIN1 Inhibitor

14. Probing the SAM Binding Site of SARS-CoV-2 Nsp14 In Vitro Using SAM Competitive Inhibitors Guides Developing Selective Bisubstrate Inhibitors

15. Discovery of the SMYD3 Inhibitor BAY-6035 Using Thermal Shift Assay (TSA)-Based High-Throughput Screening

18. Chemical tools for the Gid4 subunit of the human E3 ligase C-terminal to LisH (CTLH) degradation complex

20. Chemical Tools for the Gid4 Subunit of the Human E3 Ligase C-terminal to LisH (CTLH) Degradation Complex

21. Drug Discovery in Low Data Regimes: Leveraging a Computational Pipeline for the Discovery of Novel SARS-CoV-2 Nsp14-MTase Inhibitors

22. Discovery of a Druggable, Cryptic Pocket in SARS-CoV-2 nsp16 Using Allosteric Inhibitors

23. Development of LM-41 and AF-2112, two flufenamic acid-derived TEAD inhibitors obtained through the replacement of the trifluoromethyl group by aryl rings

25. Fragment-based discovery of a chemical probe for the PWWP1 domain of NSD3

26. The Cryptosporidium parvum Kinome

27. SETD7 functions as a transcription repressor in prostate cancer via methylating FOXA1

28. Rational Design of Highly Potent SARS-CoV-2 nsp14 Methyltransferase Inhibitors

31. Author Correction: Pharmacological inhibition of PRMT7 links arginine monomethylation to the cellular stress response

32. Pharmacological inhibition of PRMT7 links arginine monomethylation to the cellular stress response

34. Discovery of a chemical probe for PRDM9

35. A chemical biology toolbox to study protein methyltransferases and epigenetic signaling

36. Development of HC-258, a Covalent Acrylamide TEAD Inhibitor That Reduces Gene Expression and Cell Migration.

37. Probing the mechanism of Cbl-b inhibition by a small-molecule inhibitor

38. SS148 and WZ16 inhibit the activities of nsp10-nsp16 complexes from all seven human pathogenic coronaviruses

39. Supplementary Dataset 2 from WDR5 Supports an N-Myc Transcriptional Complex That Drives a Protumorigenic Gene Expression Signature in Neuroblastoma

40. Supplementary Dataset 1 from WDR5 Supports an N-Myc Transcriptional Complex That Drives a Protumorigenic Gene Expression Signature in Neuroblastoma

41. Supplementary Dataset 5 from WDR5 Supports an N-Myc Transcriptional Complex That Drives a Protumorigenic Gene Expression Signature in Neuroblastoma

42. Supplementary Dataset 4 from WDR5 Supports an N-Myc Transcriptional Complex That Drives a Protumorigenic Gene Expression Signature in Neuroblastoma

43. Data from WDR5 Supports an N-Myc Transcriptional Complex That Drives a Protumorigenic Gene Expression Signature in Neuroblastoma

44. Supplementary Dataset 3 from WDR5 Supports an N-Myc Transcriptional Complex That Drives a Protumorigenic Gene Expression Signature in Neuroblastoma

45. Supplementary Methods-Figures-Tables from WDR5 Supports an N-Myc Transcriptional Complex That Drives a Protumorigenic Gene Expression Signature in Neuroblastoma

46. Discovery of Nanomolar DCAF1 Small Molecule Ligands

49. Chemical tools for the Gid4 subunit of the human E3 ligase C-terminal to LisH (CTLH) degradation complexElectronic supplementary information (ESI) available. See DOI: https://doi.org/10.1039/d3md00633f

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