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1. TIME-seq reduces time and cost of DNA methylation measurement for epigenetic clock construction

2. The regulatory landscape of early maize inflorescence development

3. Loss of epigenetic information as a cause of mammalian aging

4. Loss of epigenetic information as a cause of mammalian aging

5. Evolutionary Dynamics of Chromatin Structure and Duplicate Gene Expression in Diploid and Allopolyploid Cotton.

7. Reprogramming to recover youthful epigenetic information and restore vision

9. The maize W22 genome provides a foundation for functional genomics and transposon biology

10. TIME-Seq Enables Highly-Efficient Epigenetic Age Predictions in Large-Scale Human and Mouse Longevity Studies

15. Topologically associating domains are stable units of replication-timing regulation

16. TIME-Seq Enables Scalable and Inexpensive Epigenetic Age Predictions

17. The native cistrome and sequence motif families of the maize ear

19. Loss of Epigenetic Information as a Cause of Mammalian Aging

22. Arabidopsis DNA Replication Initiates in Intergenic, AT-Rich Open Chromatin

24. DNA Break-Induced Epigenetic Drift as a Cause of Mammalian Aging

25. Erosion of the Epigenetic Landscape and Loss of Cellular Identity as a Cause of Aging in Mammals

26. Reversal of ageing- and injury-induced vision loss by Tet-dependent epigenetic reprogramming

27. Chromatin structure profile data from DNS-seq: Differential nuclease sensitivity mapping of four reference tissues of B73 maize (Zea mays L)

33. The spring-loaded genome: Nucleosome redistributions are widespread, transient, and DNA-directed

36. Stimulation of the Drosophilaimmune system alters genome-wide nucleosome occupancy

37. The spring-loaded genome: nucleosome redistributions are widespread, transient, and DNA-directed.

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