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2. Rare coding variants as risk modifiers of the 22q11.2 deletion implicate postnatal cortical development in syndromic schizophrenia

3. Chromatin regulators in the TBX1 network confer risk for conotruncal heart defects in 22q11.2DS

4. Genetic contributors to risk of schizophrenia in the presence of a 22q11.2 deletion.

5. Using common genetic variation to examine phenotypic expression and risk prediction in 22q11.2 deletion syndrome

6. Complete Sequence of the 22q11.2 Allele in 1,053 Subjects with 22q11.2 Deletion Syndrome Reveals Modifiers of Conotruncal Heart Defects

7. Contributors

9. Author Correction: Improved reference genome for the domestic horse increases assembly contiguity and composition

10. The 22q11 low copy repeats are characterized by unprecedented size and structural variability

11. Erratum: Author Correction: Improved reference genome for the domestic horse increases assembly contiguity and composition.

12. Deletion size analysis of 1680 22q11.2DS subjects identifies a new recombination hotspot on chromosome 22q11.2.

13. Improved reference genome for the domestic horse increases assembly contiguity and composition.

14. Prenatal vs postnatal diagnosis of 22q11.2 deletion syndrome: cardiac and noncardiac outcomes through 1 year of age

15. Rare Genome-Wide Copy Number Variation and Expression of Schizophrenia in 22q11.2 Deletion Syndrome

16. Comprehensive Recommendations for the Clinical Management of Pregnant Women With Noninvasive Prenatal Test Results Suspicious of a Maternal Malignancy.

18. Multiple paralogues and recombination mechanisms drive the high incidence of 22q11.2 Deletion Syndrome

21. Prenatal versus Postnatal Diagnosis of 22q11.2 Deletion Syndrome: Cardiac and Non-Cardiac Outcomes through 1 Year of Age

25. Copy number variation analysis in adults with catatonia confirms haploinsufficiency of SHANK3 as a predisposing factor

26. A multi-omics genome-and-transcriptome single-cell atlas of human preimplantation embryogenesis reveals the cellular and molecular impact of chromosome instability

27. Coffin–Siris and Nicolaides–Baraitser syndromes are a common well recognizable cause of intellectual disability

28. Non-invasive detection of genomic imbalances in Hodgkin/Reed-Sternberg cells in early and advanced stage Hodgkin's lymphoma by sequencing of circulating cell-free DNA: a technical proof-of-principle study

31. Implementation of genomic arrays in prenatal diagnosis: The Belgian approach to meet the challenges

36. Mosaic Copy Number Variation in Human Neurons

37. Balanced chromosomal rearrangements offer insights into coding and noncoding genomic features associated with developmental disorders

40. Array CGH

42. Single-cell genome-wide concurrent haplotyping and copy-number profiling through genotyping-by-sequencing

46. Chromosomal phenotypes and submicroscopic abnormalities

49. Additional file 1 of A benchmark of structural variation detection by long reads through a realistic simulated model

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