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1. Correlation of allele-specific HLA loss of heterozygosity in patients with cancer with driver mutations.

2. 1223 Non-viral targeted knock-in of a KRAS G12D specific TCR, CD8αβ, and chimeric cytokine receptor in theTRAClocus outperforms lentiviral-based engineering of T cells

3. 355 Directing a high avidity KRAS G12D-specific TCR engineered with a CD8αβ co-receptor and chimeric cytokine receptor using non-viral knock-in enhances anti-tumor responses

4. Abstract A009: AFNT-111: A novel TCR-engineered T cell therapy targeting the oncogenic driver KRAS G12V

5. Supplementary Table S1 and Supplementary Figures S1-S4 from Concomitant Inhibition of PI3Kβ and BRAF or MEK in PTEN-Deficient/BRAF-Mutant Melanoma Treatment: Preclinical Assessment of SAR260301 Oral PI3Kβ-Selective Inhibitor

6. Data from Concomitant Inhibition of PI3Kβ and BRAF or MEK in PTEN-Deficient/BRAF-Mutant Melanoma Treatment: Preclinical Assessment of SAR260301 Oral PI3Kβ-Selective Inhibitor

7. A Compact Low-Power 28-nm FD-SOI CMOS 76–81 GHz Dual-Path Receiver With Phase Control for Automotive Applications

8. Nonintrusive Machine Learning-Based Yield Recovery and Performance Recentering for mm-Wave Power Amplifiers: A Two-Stage Class-A Power Amplifier Case Study

9. 244 AFNT-111, a safe and effective TCR-engineered T cell therapy targeting the oncogenic driver KRAS G12V mutation

11. 342 KRAS G12V T cell receptor-engineered T cells expressing the durability FAS-41BB switch receptor exhibit a potent, persistent, coordinated CD4/CD8 anti-tumor responsein vitroandin vivo

14. A Millimeter-Wave Substrate Integrated Waveguide Filter in Si-BCB Technology

16. mm-Wave Through-Load Element for On-Wafer Measurement Applications

17. 120-GHz 2-bit reflection-type phase shifter based on PIN diodes switched-lines.

20. Design of mm-Wave Slow-Wave-Coupled Coplanar Waveguides

23. MethylQuant: A Tool for Sensitive Validation of Enzyme-Mediated Protein Methylation Sites from Heavy-Methyl SILAC Data

24. VEGFR1-positive haematopoietic bone marrow progenitors initiate the pre-metastatic niche

27. Highly Tunable High-Q Inversion-Mode MOS Varactor in the 1–325-GHz Band

30. Essential roles for deubiquitination in Leishmania life cycle progression

31. The role of protein arginine methylation in T-lymphocyte activation

33. Preparation and optimization of new 4-(2-(indolin-1-yl)-2-oxoethyl)-2-morpholinothiazole-5-carboxylic acid and amide derivatives as potent and selective PI3Kβ inhibitors

35. Concomitant Inhibition of PI3Kβ and BRAF or MEK in PTEN-Deficient/BRAF-Mutant Melanoma Treatment: Preclinical Assessment of SAR260301 Oral PI3Kβ-Selective Inhibitor

36. Preparation and optimization of new 4-(morpholin-4-yl)-(6-oxo-1,6-dihydropyrimidin-2-yl)amide derivatives as PI3Kβ inhibitors

38. Abstract B2: Switching to aflibercept treatment resulted in greater tumor responses than continuous bevacizumab treatment in patient-derived xenograft models of colorectal cancer.

40. Abstract 3258: Discovery and characterization of SAR260301, a novel PI3Kβ-selective inhibitor in clinical development for the treatment of PTEN-deficient tumors .

41. Abstract 4638: Anti-tumor activity of pimasertib in combination with SAR245409 or SAR245408 in human primary colorectal cancer xenograft models bearing PI3K/KRas and KRas mutations.

42. Discovery and Optimization of New Benzimidazole- and Benzoxazole-Pyrimidone Selective PI3Kβ Inhibitors for the Treatment of Phosphatase and TENsin homologue (PTEN)-Deficient Cancers

44. Stromal Incorporation of VEGFR-1+, CD68+ and α-SMA+ Hemangiogenic Cells Correlates with Histologic Subtype in Non-Hodgkin’s Lymphoma.

45. Multimodular Analysis of Angiogenic Biomarkers in the Primary Treatment of Multiple Myeloma: Correlative Studies of the BiRD Trial.

47. Newly Discovered Polymorphism in the CD34+ Stem Cell Specific AC133-P1 Promoter Linked to Leukemias.

48. Discovery and Optimizationof New Benzimidazole- andBenzoxazole-Pyrimidone Selective PI3Kβ Inhibitors for the Treatmentof Phosphatase and TENsin homologue (PTEN)-Deficient Cancers.

50. Stromal Incorporation of VEGFR-1+, CD68+and α-SMA+Hemangiogenic Cells Correlates with Histologic Subtype in Non-Hodgkin's Lymphoma.

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