35 results on '"Voeltzel, Thibault"'
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2. Human myeloid differentiation by BMP4 signaling through the VDR pathway in acute myeloid leukemia
3. The BMP pathway: A unique tool to decode the origin and progression of leukemia
4. Immature CML cells implement a BMP autocrine loop to escape TKI treatment
5. Disequilibrium of BMP2 Levels in the Breast Stem Cell Niche Launches Epithelial Transformation by Overamplifying BMPR1B Cell Response
6. A Human Bone Marrow 3D Model to Investigate the Dynamics and Interactions Between Resident Cells in Physiological or Tumoral Contexts
7. A new signaling cascade linking BMP4, BMPR1A, ΔNp73 and NANOG impacts on stem-like human cell properties and patient outcome
8. Primitive CML cell expansion relies on abnormal levels of BMPs provided by the niche and on BMPRIb overexpression
9. A minimal standardized human bone marrow microphysiological system to assess resident cell behavior during normal and pathological processes
10. Erratum for Brahimi-Horn et al., “Local Mitochondrial-Endolysosomal Microfusion Cleaves the Voltage-Dependent Anion Channel 1 To Promote Survival in Hypoxia”
11. Altered BMP2/4 Signaling in Stem Cells and Their Niche: Different Cancers but Similar Mechanisms, the Example of Myeloid Leukemia and Breast Cancer
12. A minimal standardized human bone marrow microphysiological system to assess resident cell behavior during normal and pathological processes
13. Induction of EMT by Twist Proteins as a Collateral Effect of Tumor-Promoting Inactivation of Premature Senescence
14. Gadd45a Activation Protects Melanoma Cells from Ultraviolet B-Induced Apoptosis
15. Caspase-dependent BRCA1 cleavage facilitates chemotherapy-induced apoptosis
16. The quiescent fraction of chronic myeloid leukemic stem cells depends on BMPR1B, Stat3 and BMP4-niche signals to persist in patients in remission
17. Downregulation of the histone methyltransferase SETD2 promotes imatinib resistance in chronic myeloid leukaemia cells
18. Relationships of the Antiproliferative Proteins BTG1 and BTG2 with CAF1, the Human Homolog of a Component of the Yeast CCR4 Transcriptional Complex: INVOLVEMENT IN ESTROGEN RECEPTOR α SIGNALING PATHWAY
19. The Leukemia-associated Protein Btg1 and the p53-regulated Protein Btg2 Interact with the Homeoprotein Hoxb9 and Enhance Its Transcriptional Activation
20. Local Mitochondrial-Endolysosomal Microfusion Cleaves Voltage-Dependent Anion Channel 1 To Promote Survival in Hypoxia
21. Deregulation of TWIST-1 in the CD34+ compartment represents a novel prognostic factor in chronic myeloid leukemia
22. BMP Pathway Alterations in Chronic Myelogenous Leukemia Lead to the Amplification of Primitive Progenitors Output in Chronic Phase
23. The CD10 Enzyme Is a Key Player to Identify and Regulate Human Mammary Stem Cells
24. 76: Control of human mammary cell fate by CD10-enzyme and integrin beta1
25. Twist-1 Expression Represents a Novel Prognostic Factor in Chronic Myelogenous Leukemia.
26. Caspase-dependent BRCA1 cleavage facilitates chemotherapy-induced apoptosis
27. UVB-Induced G2 Arrest of Human Melanocytes Involves Cdc2 Sequestration by Gadd45a in Nuclear Speckles
28. Distribution of VRA09, an ovarian tumor‐associated antigen, distinguishing borderline serous tumors
29. Detection of a 45-Kilodalton Antigen Overexpressed in a Cisplatin-Resistant Human Ovarian Carcinoma Cell Line
30. Antigenic properties associated with vinca alkaloid resistance in ovarian cancer cells: identification of a 92,000 DA protein
31. UVB-Induced G2 Arrest of Human Melanocytes Involves Cdc2 Sequestration by Gadd45a in Nuclear Speckles.
32. Structural probing of human lutropin using antibodies raised against synthetic peptides constructed by classical and multiple antigen peptide system approaches
33. Deregulation of TWIST-1 in the CD34+compartment represents a novel prognostic factor in chronic myeloid leukemia
34. Induction of EMT by Twist Proteins as a Collateral Effect of Tumor-Promoting Inactivation of Premature Senescence
35. The quiescent fraction of chronic myeloid leukemic stem cells depends on BMPR1B, Stat3 and BMP4-niche signals to persist in patients in remission.
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