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12 results on '"Wei, Yuehan"'

4. Incremental diagnostic value of CMR-derived LA strain and strain rate in dialysis patients with HFpEF

Author

Zhou, Hang, primary, An, Dong-Aolei, additional, Ni, Zhaohui, additional, Xu, Jianrong, additional, Zhou, Yan, additional, Fang, Wei, additional, Lu, Renhua, additional, Ying, Liang, additional, Huang, Jiaying, additional, Yao, Qiuying, additional, Li, Dawei, additional, Hu, Jiani, additional, Chen, Binghua, additional, Shen, Jianxiao, additional, Jin, Haijiao, additional, Wei, Yuehan, additional, Ouchi, Erika, additional, Xu, Lei, additional, Wu, Lian-Ming, additional, and Mou, Shan, additional

Published
2022
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5. STING controls energy stress-induced autophagy and energy metabolism via STX17

Author

Rong, Yueguang, primary, Zhang, Shen, additional, Nandi, Nilay, additional, Wu, Zhe, additional, Li, Linsen, additional, Liu, Yang, additional, Wei, Yuehan, additional, Zhao, Yuan, additional, Yuan, Weigang, additional, Zhou, Chuchu, additional, Xiao, Guanghua, additional, Levine, Beth, additional, Yan, Nan, additional, Mou, Shan, additional, Deng, Liufu, additional, Tang, Zaiming, additional, Liu, Xiaoxia, additional, Kramer, Helmut, additional, and Zhong, Qing, additional

Published
2022
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6. Magnetic Resonance Imaging Quantification of Accumulation of Epicardial Adipose Tissue Adds Independent Risks for Diastolic Dysfunction among Dialysis Patients

Author

Zhou, Hang, primary, An, Dong‐Aolei, additional, Ni, Zhaohui, additional, Xu, Jianrong, additional, Zhou, Yan, additional, Fang, Wei, additional, Lu, Renhua, additional, Ying, Liang, additional, Huang, Jiaying, additional, Yao, Qiuying, additional, Li, Dawei, additional, Hu, Jiani, additional, Chen, Binghua, additional, Shen, Jianxiao, additional, Jin, Haijiao, additional, Wei, Yuehan, additional, Fahmy, Lara M., additional, Jing, Du, additional, Ye, Jing, additional, Xu, Lei, additional, Wu, Lian‐Ming, additional, and Mou, Shan, additional

Published
2022
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7. Myocardial Iron Deficiency Quantification and Effective Cardiac Iron Management Strategy Exploration evaluated by Cardiac T2* Mapping in End-Stage Renal Disease Patients

Author

Zhou, Hang, primary, Wei, Yuehan, additional, An, Dong-Aolei, additional, Ni, Zhaohui, additional, Xu, Jianrong, additional, Fang, Wei, additional, Lu, Renhua, additional, Ying, Liang, additional, Chen, Binghua, additional, Jin, Haijiao, additional, Huang, Jiaying, additional, Yao, Qiuying, additional, Wesemann, Luke, additional, Wu, Lian-Ming, additional, and Mou, Shan, additional

Published
2021
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8. Texture Analysis of Native T1 Images as a Novel Method for Noninvasive Assessment of Uremic Cardiomyopathy

Author

Zhou, Hang, primary, An, Dong‐Aolei, additional, Ni, Zhaohui, additional, Xu, Jianrong, additional, Fang, Wei, additional, Lu, Renhua, additional, Ying, Liang, additional, Huang, Jiaying, additional, Yao, Qiuying, additional, Li, Dawei, additional, Chen, Binghua, additional, Shen, Jianxiao, additional, Jin, Haijiao, additional, Wei, Yuehan, additional, Hu, Jiani, additional, Fahmy, Lara M., additional, Wesemann, Luke, additional, Qi, Shouliang, additional, Wu, Lian‐Ming, additional, and Mou, Shan, additional

Published
2021
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12. To target cellular senescence in diabetic kidney disease: the known and the unknown.

Author

Wei Y, Mou S, Yang Q, Liu F, Cooper ME, and Chai Z

Subjects
Humans, Animals, Autophagy, Kidney pathology, Kidney metabolism, Senescence-Associated Secretory Phenotype, Endoplasmic Reticulum Stress, Cellular Senescence, Diabetic Nephropathies metabolism, Diabetic Nephropathies pathology
Abstract

Cellular senescence represents a condition of irreversible cell cycle arrest, characterized by heightened senescence-associated beta-galactosidase (SA-β-Gal) activity, senescence-associated secretory phenotype (SASP), and activation of the DNA damage response (DDR). Diabetic kidney disease (DKD) is a significant contributor to end-stage renal disease (ESRD) globally, with ongoing unmet needs in terms of current treatments. The role of senescence in the pathogenesis of DKD has attracted substantial attention with evidence of premature senescence in this condition. The process of cellular senescence in DKD appears to be associated with mitochondrial redox pathways, autophagy, and endoplasmic reticulum (ER) stress. Increasing accumulation of senescent cells in the diabetic kidney not only leads to an impaired capacity for repair of renal injury, but also the secretion of pro-inflammatory and profibrotic cytokines and growth factors causing inflammation and fibrosis. Current treatments for diabetes exhibit varying degrees of renoprotection, potentially via mitigation of senescence in the diabetic kidney. Targeting senescent cell clearance through pharmaceutical interventions could emerge as a promising strategy for preventing and treating DKD. In this paper, we review the current understanding of senescence in DKD and summarize the possible therapeutic interventions relevant to senescence in this field., (© 2024 The Author(s).)

Published
2024
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