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1. The arrhythmogenic cardiomyopathy phenotype associated with PKP2 c.1211dup variant

3. Multicenter clinical and functional evidence reclassifies a recurrent noncanonical filamin C splice-altering variant

5. Blood biomarkers in patients with bicuspid aortic valve disease

6. Identification of novel microcephaly-linked protein ABBA that mediates cortical progenitor cell division and corticogenesis through NEDD9-RhoA

7. Mutations in XPR1 cause primary familial brain calcification associated with altered phosphate export.

8. Expanding the genetic and phenotypic spectrum of ACTA2-related vasculopathies in a Dutch cohort

9. Expert consensus recommendations on the cardiogenetic care for patients with thoracic aortic disease and their first-degree relatives

10. Lack of evidence for a causal role of CALR3 in monogenic cardiomyopathy

11. Expanding the genetic and phenotypic spectrum of ACTA2-related vasculopathies in a Dutch cohort

12. The MAP3K7 gene: Further delineation of clinical characteristics and genotype/phenotype correlations

13. Multicenter Clinical and Functional Evidence Reclassifies a Recurrent Non-canonical Filamin C Splice-altering Variant

14. The recurrent de novo c.2011C>T missense variant in MTSS2 causes syndromic intellectual disability

15. Expanding the genetic and phenotypic spectrum of ACTA2-related vasculopathies in a Dutch cohort

18. Multicenter clinical and functional evidence reclassifies a recurrent noncanonical filamin C splice-altering variant.

19. Human KCNQ5 de novo mutations underlie epilepsy and intellectual disability

20. De novo truncating NOVA2 variants affect alternative splicing and lead to heterogeneous neurodevelopmental phenotypes

21. Multi-Omics Profiling in Marfan Syndrome: Further Insights into the Molecular Mechanisms Involved in Aortic Disease

22. The MAP3K7 gene: further delineation of clinical characteristics and genotype/phenotype correlations

23. De novo truncating NOVA2 variants affect alternative splicing and lead to heterogeneous neurodevelopmental phenotypes

24. Expanding the genetic and phenotypic spectrum of ACTA2-related vasculopathies in a Dutch cohort

25. Multi-Omics Profiling in Marfan Syndrome: Further Insights into the Molecular Mechanisms Involved in Aortic Disease

26. Mutation in the AP4M1 Gene Provides a Model for Neuroaxonal Injury in Cerebral Palsy

27. Terminal osseous dysplasia with pigmentary defects and cardiomyopathy caused by a novel FLNA variant

28. A new syndrome with noncompaction cardiomyopathy, bradycardia, pulmonary stenosis, atrial septal defect and heterotaxy with suggestive linkage to chromosome 6p

29. Terminal osseous dysplasia with pigmentary defects and cardiomyopathy caused by a novel FLNA variant

30. Terminal osseous dysplasia with pigmentary defects and cardiomyopathy caused by a novel FLNA variant

31. A de novo GLI3 mutation in a patient with acrocallosal syndrome

32. Phenotypic Variability of Atypical 22q11.2 Deletions Not Including TBX1

33. Phenotypic spectrum of the SMAD3-related aneurysms–osteoarthritis syndrome

34. MLL2 mutation spectrum in 45 patients with Kabuki syndrome

35. ADAMTS19‐associated heart valve defects: Novel genetic variants consolidating a recognizable cardiac phenotype

37. Mucopolysaccharidosis Type IIID: 12 New Patients and 15 Novel Mutations

40. Homozygous damaging SOD2 variant causes lethal neonatal dilated cardiomyopathy

41. Homozygous damaging SOD2 variant causes lethal neonatal dilated cardiomyopathy

43. Expanding Clinical Presentations Due to Variations in THOC2 mRNA Nuclear Export Factor

44. Variants in nuclear factor I genes influence growth and development

45. Homozygous damaging SOD2 variant causes lethal neonatal dilated cardiomyopathy

46. Biallelic Variants in ASNA1 , Encoding a Cytosolic Targeting Factor of Tail-Anchored Proteins, Cause Rapidly Progressive Pediatric Cardiomyopathy

47. Mortality Risk Associated With Truncating Founder Mutations in Titin

48. Cardiac Phenotypes, Genetics, and Risks in Familial Noncompaction Cardiomyopathy

50. De Novo Truncating Mutations in the Last and Penultimate Exons of PPM1D Cause an Intellectual Disability Syndrome

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