258 results on '"Westenfelder, Christof"'
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2. Significant expansion of the donor pool achieved by utilizing islets of variable quality in the production of allogeneic “Neo-Islets”, 3-D organoids of Mesenchymal Stromal and islet cells, a novel immune-isolating biotherapy for Type I Diabetes
3. Heme Protein–Induced Acute Kidney Injury Is Caused by Disruption of Mitochondrial Homeostasis in Proximal Tubular Cells
4. MSCs for Renal Repair
5. Treatment of Ischemia/Reperfusion Injury of the Kidney with Mesenchymal Stromal Cells
6. 841-P: Scientific Basis for the First-in-Human Trial of “Neo-Islet”–Mediated Functional Cure of T1DM: The Intraperitoneal Administration of “Neo-Islets,” 3-D Organoids of Allogeneic Islet and Mesenchymal Stromal Cells from Rodents, Dogs, and Humans Durably Cures
7. 840-P: Proof of Concept of I.P. Administered Neo-Islet (NI) T1DM Biotherapy: Omentum Becomes the New Endocrine Pancreas by Spontaneous Redifferentiation of In Vitro Dedifferentiated Islet Cells
8. Intraperitoneal administration of human “Neo-Islets”, 3-D organoids of mesenchymal stromal and pancreatic islet cells, normalizes blood glucose levels in streptozotocin-diabetic NOD/SCID mice: Significance for clinical trials
9. Cytoprotective Doses of Erythropoietin or Carbamylated Erythropoietin Have Markedly Different Procoagulant and Vasoactive Activities
10. 760-P: Allogeneic "Neo-Islets," Organoids Composed of Cultured Pancreatic Islet and Mesenchymal Stromal Cells from Diverse Donors, Afford a Marked Increase in the Global Availability of a Curative, Novel, Anti-Rejection-Drug-Independent Biotherapy of T1DM
11. Bioluminescence imaging to monitor the in vivo distribution of administered mesenchymal stem cells in acute kidney injury
12. Vasculotropic, paracrine actions of infused mesenchymal stem cells are important to the recovery from acute kidney injury
13. Administration of poly-D-glutamic acid induces proliferation of erythropoietin-producing peritubular cells in rat kidney
14. MSCs for Renal Repair
15. Administered mesenchymal stem cells protect against ischemic acute renal failure through differentiation-independent mechanisms
16. Treatment of Ischemia/Reperfusion Injury of the Kidney with Mesenchymal Stromal Cells
17. Treatment of Acute Kidney Injury with Allogeneic Mesenchymal Stem Cells: Preclinical and Initial Clinical Data
18. Contributors
19. Systemic AAV10.COMP-Ang1 rescues renal glomeruli and pancreatic islets in type 2 diabetic mice
20. 203-LB: Redosing of Suboptimally Controlled Diabetic Mice with Human Neo-islets, 3D Aggregates of Mesenchymal Stromal and Pancreatic Islet Cells, Durably Restores Euglycemia: Relevance to the Treatment of Diabetic Humans
21. 204-LB: The In Vitro Characteristics of Human Islet Cells from Diverse Donors, Coaggregated with Human Mesenchymal Stromal Cells to form “Neo-islets”, Are Consistently Identical: Relevance to Clinical Trials in Diabetic Subjects
22. Mesenchymal stem cells: a new therapeutic tool for AKI
23. In Vivo Magnetic Resonance Imaging of Iron Oxide–Labeled, Arterially-Injected Mesenchymal Stem Cells in Kidneys of Rats With Acute Ischemic Kidney Injury: Detection and Monitoring at 3T
24. Administered mesenchymal stem cells enhance recovery from ischemia∼reperfusion–induced acute renal failure in rats
25. ADMINISTERED MESENCHYMAL STEM CELLS PROTECT AGAINST ISCHEMIC ACUTE RENAL FAILURE THROUGH PARACRINE AND ANTI-INFLAMMATORY MECHANISMS: FC-10064
26. Renal SDF-1 signals mobilization and homing of CXCR4-positive cells to the kidney after ischemic injury
27. Hematopoietic Stem Cell Mobilization–Associated Granulocytosis Severely Worsens Acute Renal Failure
28. MOBILIZATION OF HEMATOPOIETIC STEM CELLS (HSC) PRIOR TO INDUCTION OF ISCHEMIC ACUTE RENAL FAILURE (ARF) IN MICE DOES NOT IMPROVE BUTSIGNIFICANTLY WORSENS OUTCOME
29. Estimation of renal interstitial adenosine and purine metabolites by microdialysis
30. Erythropoietin stimulates proliferation of human renal carcinoma cells
31. Human, rat, and mouse kidney cells express functional erythropoietin receptors
32. Interim report on the effective intraperitoneal therapy of insulin-dependent diabetes mellitus in pet dogs using “Neo-Islets,” aggregates of adipose stem and pancreatic islet cells (INAD 012-776)
33. First report on the effective intraperitoneal therapy of insulin-dependent Diabetes mellitus in pet dogs using “Neo-Islets,” aggregates of adipose stem and pancreatic islet cells
34. 262-OR: INAD 012-776 Pilot Study: Allogeneic “Neo-Islets,” Composed of Mesenchymal Stem and Islet Cells, Are Immune Protected and Dose-Dependently and Durably Control Autoimmune T1DM in Pet Dogs
35. Renal handling of furosemide in glycerol- and mercuric chloride-induced acute renal failure in rats.: 50.
36. Exposure of Mesenchymal Stromal Cells (MSCs) to INF-γ Boosts the Therapeutic Potency of Their Exosomes (Exos) in the Rescue Therapy of Severe AKI in Rats
37. Intraperitoneally Administered Neo-islets, Aggregates of Mesenchymal Stem Cells, and Cultured Islet Cells Improve Glycemic Control of Spontaneously Occurring Insulin-Dependent Diabetes Mellitus in Pet Dogs—A Pilot Study, INAD 012-776
38. Durable Control of Autoimmune Diabetes in Mice Achieved by Intraperitoneal Transplantation of “Neo-Islets,” Three-Dimensional Aggregates of Allogeneic Islet and “Mesenchymal Stem Cells”
39. A Second Dose of Allogeneic Neo-Islets (NIs) Further Reduces Insulin Need and HbA1c and Preserves Renal Function Long Term in Autoimmune T1DM Pet Dogs (INAD 012-776)
40. β-MSCs: successful fusion of MSCs with β-cells results in a β-cell like phenotype
41. Carbamylated Erythropoietin Outperforms Erythropoietin in the Treatment of AKI-on-CKD and Other AKI Models
42. Modified Hydrogels to Enhance Cellular Therapy for AKI: A Translational Challenge
43. Effect of Na−K-ATPase inhibition on hydrogen ion and potassium secretion
44. Bicarbonate reabsorption in chronic renal failure studies in man and the rat
45. Serosal Na/Ca exchange and H+ and Na+ transport by the turtle and toad bladders
46. F344/CD26mut Rats Have No Detectable CD26/DPP4 Activity F344/CD26mut Rats Have Lower Levels of Plasma SDF-1α Compared to F344/CD26wt Rats F344/CD26mut Rats Have Increased Levels of Kidney SDF-1α Compared to F344/CD26wt Rats SUMMARY RESULTS F344/CD26mut Rats (Non-Functional CD26/Dipeptidyl Peptidase-4) have Altered SDF-1α/CXCL12 Levels and Increased Mortality Following Acute Kidney Injury Compared to F344/CD26wt Rats (Wild Type CD26/DPP4 Function)
47. Chapter 21 - Treatment of Acute Kidney Injury with Allogeneic Mesenchymal Stem Cells: Preclinical and Initial Clinical Data
48. Gastric Alkalosis
49. Renal Regulation of Acid-Base Homeostasis
50. Recent advances in the understanding of acute kidney injury
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